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1 ese animals showing tau-positive tangles and neuropil threads.
2 ical inclusions, neurofibrillary tangles and neuropil threads.
3 , neuritic plaques, and, to a lesser extent, neuropil threads.
4 longer striations are arbitrarily considered neuropil threads.
5 brillary tangles, senile plaque neurites and neuropil threads.
6 au protein at both early and later stages of neuropil thread and tangle formation.
7 beta plaques contained densities of tangles, neuropil threads and cerebral amyloid angiopathy (CAA) s
8 ongTAR DNA-binding protein (TDP)-43-positive neuropil threads and dystrophic neurites (type C), and i
9       However, the staining pattern excluded neuropil threads and dystrophic neurites indicating that
10 neurofibrillary tangles and less reactive in neuropil threads and extracellular tangles in TPSD and F
11  the development of neurofibrillary tangles, neuropil threads and ghost tangles was rare and likewise
12 tionship between neurofibrillary tangles and neuropil threads and how they relate to cognitive functi
13 rphosphorylated tau pathology in the form of neuropil threads and neurofibrillary tangles and pre-tan
14 a direct relationship between the numbers of neuropil threads and of neurofibrillary tangles: neurofi
15                             Gallyas-positive neuropil threads and oligodendroglial coiled bodies were
16 ing in early pathological inclusions such as neuropil threads and pretangle neurons as well as coloca
17 ere examined for neurofibrillary tangles and neuropil threads and the nigra for neuronal numbers.
18 essels, astrocytes, neurofibrillary tangles, neuropil threads, and dystrophic neurites in the AD brai
19 ophic neurites surrounding neuritic plaques, neuropil threads, and granulovacuolar degeneration bodie
20  Pathologic forms of tau in neuronal somata, neuropil threads, and plaque-like clusters of neurites w
21                    Amyloid-beta plaques, tau neuropil thread burden, synapse density, tau oligomers i
22 ical inclusions, neurofibrillary tangles and neuropil threads but only in the patient who came to aut
23 ars, drug users showed a similar increase in neuropil threads compared with controls, but this reache
24   Neurofibrillary tangle-bearing neurons and neuropil threads contained NOS2 in brains from each of 1
25 llary tangles were sparse in brains that had neuropil thread counts below 200 cm(2).
26          Stereologically unbiased tangle and neuropil thread counts were obtained from the same regio
27 es were unchanged, whereas only AT8-positive neuropil thread density was statistically higher in AD s
28  characterized by neurofibrillary tangle and neuropil thread deposition, which ultimately results in
29 rillary tangles (NFT), neuritic plaques, and neuropil threads in brains from individuals with AD.
30 u accumulates as neurofibrillary tangles and neuropil threads in older persons with and without Alzhe
31 formers in pretangles, neuritic plaques, and neuropil threads in the frontal cortex tissue from AD br
32 FTs and mature NFTs, dystrophic neurites and neuropil threads in the hippocampus.
33 tau immunoreactive structures (predominantly neuropil threads) in the entorhinal and perirhinal corti
34             Hyperphosphorylated tau positive neuropil threads increased at a faster rate in drug user
35 of AD is classically described as a triad of neuropil threads, neurofibrillary tangles, and dystrophi
36 ese lesions, we sought to determine if NFTs, neuropil threads (NTs), and SPs contain RNA species.
37 , AD-like neurofibrillary tangles (NFTs) and neuropil threads (NTs).
38                 In this study, we found that neuropil threads of Alzheimer disease, rather than being
39 bset of neurofibrillary tangles and not with neuropil threads or dystrophic neurites.
40 igodendrocytic tau inclusions as well as the neuropil thread pathology in cellular processes, but the
41        In contrast, neurofibrillary tangles, neuropil threads, Pick bodies, ballooned neurons, and gl
42    The number of neurofibrillary tangles and neuropil threads was increased in AD (with or without ex
43 ther tau pathology, including pretangles and neuropil threads, was abundant and widespread.
44 umbers of nigral neurofibrillary tangles and neuropil threads were positively related to extrapyramid
45 yperphosphorylated tau positive (AT8, AT100) neuropil threads were significantly increased in the fro
46  parahippocampal neurofibrillary tangles and neuropil threads were significantly lower in persons wit