ライフサイエンスコーパス: neurotransmitter receptor

1 to contain a binding domain for a different neurotransmitter receptor.

2 sm of regulation for this important class of neurotransmitter receptor.

3 ding proteins and the rat GluR2 AMPA subtype neurotransmitter receptor.

4 on the activation dynamics of this important neurotransmitter receptor.

5 increased signaling via forebrain Gq-coupled neurotransmitter receptors.

6 cally released transmitters and postsynaptic neurotransmitter receptors.

7 and postsynaptic specialization and recruit neurotransmitter receptors.

8 loop ligand-gated ion channel superfamily of neurotransmitter receptors.

9 ve zone apposed to a postsynaptic cluster of neurotransmitter receptors.

10 cytosis model for the dendritic targeting of neurotransmitter receptors.

11 ease sites opposite clusters of postsynaptic neurotransmitter receptors.

12 ls are important determinants of function in neurotransmitter receptors.

13 roteins essential for synaptic clustering of neurotransmitter receptors.

14 of which are known to inhibit endocytosis of neurotransmitter receptors.

15 macological conditions that block inhibitory neurotransmitter receptors.

16 closer to the photoreceptors than the other neurotransmitter receptors.

17 terconnections and dominant actions for fast neurotransmitter receptors.

18 mate through several families of l-glutamate neurotransmitter receptors.

19 lling pathways, focusing on ion channels and neurotransmitter receptors.

20 n be further activated via G-protein-coupled neurotransmitter receptors.

21 ling of synaptic vesicles and trafficking of neurotransmitter receptors.

22 investigations of the mechanism of action of neurotransmitter receptors.

23 synaptic vesicular release with postsynaptic neurotransmitter receptors.

24 ich thus acquired functional Torpedo and rat neurotransmitter receptors.

25 nal growth and differentiation, and modulate neurotransmitter receptors.

26 of synaptic strength involve the turnover of neurotransmitter receptors.

27 aling complexes around specific postsynaptic neurotransmitter receptors.

28 c terminal and the activation of presynaptic neurotransmitter receptors.

29 important proteins, such as ion channels and neurotransmitter receptors.

30 nd localization of specific ion channels and neurotransmitter receptors.

31 ction pathway through a variety of monoamine neurotransmitter receptors.

32 d active zones are aligned with postsynaptic neurotransmitter receptors.

33 release and the number or responsiveness of neurotransmitter receptors.

34 interact with ion channels and cytokine and neurotransmitter receptors.

35 ase associated with these widely distributed neurotransmitter receptors.

36 tes the activity of various ion channels and neurotransmitter receptors.

37 is precisely juxtaposed to the postsynaptic neurotransmitter receptors.

38 ude compensatory changes in (1) postsynaptic neurotransmitter receptor abundance, (2) presynaptic mor

39 ze neural function through the modulation of neurotransmitter receptor abundance, ion channel density

40 by the regulation of ion channel expression, neurotransmitter receptor abundance, or modulation of pr

41 junction led to an influential model of how neurotransmitter receptors accumulate in the postsynapti

42 entiation and direct the accumulation of the neurotransmitter receptors, acetylcholine receptors (ACh

43 ar Ca2+ stores and Ca2+-permeable ionotropic neurotransmitter receptors, activate SK channels.

44 kinetic investigations of the membrane-bound neurotransmitter receptor activated by glycine.

45 o address this challenge and monitor in situ neurotransmitter receptor activation.

46 Positive and negative regulation of neurotransmitter receptor aggregation on the postsynapti

47 tracellular Ca(2+) elevations in response to neurotransmitter receptor agonists and RNA sequencing of

48 ntraventricular injection of many individual neurotransmitter receptor agonists have been well docume

49 tter biosynthetic enzymes, transporters, and neurotransmitter receptors, allows functional characteri

50 a(2+) entry takes place through postsynaptic neurotransmitter receptors, although postsynaptic calciu

51 reases the functional plasticity of this key neurotransmitter receptor and is thought to contribute t

52 Several representative examples of neurotransmitter receptors and ABC transporters with the

53 Palmitoylation of neurotransmitter receptors and associated scaffold prote

54 ized multiprotein structures associated with neurotransmitter receptors and cell-adhesion proteins fu

55 n shown to regulate the localization of both neurotransmitter receptors and downstream signaling mach

56 rsely, Foxo1 increases expression of several neurotransmitter receptors and fails to regulate target

57 n deletions decreased the synaptic levels of neurotransmitter receptors and had no effect on presynap

58 nd interrogate the relative contributions of neurotransmitter receptors and intracellular signaling c

59 The actin cytoskeleton can regulate neurotransmitter receptors and ion channels by controlli

60 Membrane proteins identified included neurotransmitter receptors and ion channels implicated i

61 Neurotransmitter receptors and ion channels shape the bi

62 Q3/Q5 and was selective over a wide range of neurotransmitter receptors and ion channels such as volt

63 ) receptor, good selectivities against other neurotransmitter receptors and ion channels, acceptable

64 Numerous neurotransmitter receptors and ion channels, including g

65 or the localization and activity of synaptic neurotransmitter receptors and ion channels.

66 LN(v)s, we blocked or activated a variety of neurotransmitter receptors and measured effects on netwo

67 uding neurotransmitter synthesizing enzymes, neurotransmitter receptors and neuropeptides, we show th

68 also regulating the expression of ionotropic neurotransmitter receptors and putative stretch receptor

69 brain depends on specialized organization of neurotransmitter receptors and scaffolding proteins with

70 te to spine-specific compartmentalization of neurotransmitter receptors and signaling molecules and t

71 N-methyl-D-aspartate (NMDA) neurotransmitter receptors and the postsynaptic density-

72 IRKs) provide a common link between numerous neurotransmitter receptors and the regulation of synapti

73 ends on the maintenance of a high density of neurotransmitter receptors and their associated scaffold

74 ng technique that can be used to investigate neurotransmitter receptors and transporters directly by

75 vities displayed by methaqualone at numerous neurotransmitter receptors and transporters in an elabor

76 ighly selective for nAChRs over the other 45 neurotransmitter receptors and transporters tested.

77 characterizing alterations in the levels of neurotransmitter receptors and transporters that are ass

78 w also that these postmortem membranes carry neurotransmitter receptors and voltage-operated channels

79 ion levels of several transcription factors, neurotransmitter receptors, and intracellular signaling

80 tiple exons in transcripts for ion channels, neurotransmitter receptors, and other synaptic proteins.

81 nse to local activation of growth factor and neurotransmitter receptors, and preferentially localize

82 ct receptors, epithelial cells often express neurotransmitter receptors, and receptors are often posi

83 derived neurons expressed neurotransmitters, neurotransmitter receptors, and synaptic proteins in vit

84 nd inverse agonists, dimerization with other neurotransmitter receptors, and the main presynaptic and

85 ion, activity is induced and modulated using neurotransmitter receptor antagonists and is measured us

86 The application of neurotransmitter receptor antagonists and putative gap j

87 d on the proper assembly of the postsynaptic neurotransmitter receptor apparatus.

88 Neurotransmitter receptors are central to communication

89 Calcium-permeable neurotransmitter receptors are concentrated into structu

90 These neurotransmitter receptors are expressed on both presyna

91 Assembly and trafficking of neurotransmitter receptors are processes contingent upon

92 either intracellular signaling molecules or neurotransmitter receptors are required for barrel forma

93 ptors (GABAAR), the brain's major inhibitory neurotransmitter receptors, are the targets for many gen

94 from mature neuronal membranes that contain neurotransmitter receptors as a sensitive detector, we f

95 Using CHO cells expressing high-affinity neurotransmitter receptors as biosensors, we show that g

96 lts identify roles for IRE1alpha and BARP in neurotransmitter receptor assembly and unlock drug disco

97 euron that helps to concentrate and organize neurotransmitter receptors at a chemical synapse.

98 n applying these analytical tools to glycine neurotransmitter receptors at inhibitory synapses, we fi

99 Aggregation of neurotransmitter receptors at pre- and postsynaptic stru

100 The surface density of neurotransmitter receptors at synapses is a key determin

101 The regulated trafficking of neurotransmitter receptors at synapses is critical for s

102 ission is ensured by a high concentration of neurotransmitter receptors at the postsynaptic membrane.

103 strength can occur by altering the amount of neurotransmitter receptors at the synapse or by altering

104 oride channels, synapse-associated proteins, neurotransmitter receptors, axon and dendrite pathfinder

105 ystem in which to analyse the segregation of neurotransmitter receptors, because muscle cells receive

106 proteins, vesicular trafficking proteins and neurotransmitter receptors becomes apparent.

107 id(A) (GABA(A)) receptor, a major inhibitory neurotransmitter receptor, belongs to a family of membra

108 multielectrode array, both with and without neurotransmitter receptor blockers or allosteric modulat

109 ithin transmembrane 4-alpha-helix bundles of neurotransmitter receptors, but confirmation of binding

110 Postsynaptic plasticity targets neurotransmitter receptors, but presynaptic mechanisms r

111 ferential sorting, delivery and retention of neurotransmitter receptors, but the mechanisms underlyin

112 ndrocyte precursor cells (OPCs) express most neurotransmitter receptors, but their function remains u

113 ilepsy-linked gamma-aminobutyric acid (GABA) neurotransmitter receptor by phenobarbital is presented.

114 Acid-sensing ion channels (ASICs) act as neurotransmitter receptors by responding to synaptic cle

115 Activation of certain neurotransmitter receptors can regulate Abeta metabolism

116 s, suggests that NMDA channels, unlike other neurotransmitter receptors, cannot open unless all bindi

117 ns acted in the synapse (34 of 40, including neurotransmitter receptors, cation channels, adhesion an

118 s mitochondria, synaptic vesicle precursors, neurotransmitter receptors, cell signaling and adhesion

119 s a mosaic of specialized microdomains where neurotransmitter receptors cluster in register with the

120 ynaptic organization by their involvement in neurotransmitter receptor clustering and signaling compl

121 However, the mechanisms directing neurotransmitter-receptor clustering and maintenance are

122 sites, in precise apposition to postsynaptic neurotransmitter receptor clusters; however, the molecul

123 Variations in synapse morphology, neurotransmitter receptor composition, and receptor dist

124 Diffuse extrasynaptic neurotransmitter receptors constitute an abundant pool o

125 acological dissection approach to identify a neurotransmitter receptor defect in Egr3(-/-) mice that

126 was used to determine whether alterations in neurotransmitter receptor densities occurred before over

127 us and a shift of the balance of hippocampal neurotransmitter receptor densities toward inhibition (p

128 on the mechanisms that maintain and regulate neurotransmitter receptor density at postsynaptic sites.

129 Neurotransmitter receptor density is a major variable in

130 tent with the hypothesis that alterations in neurotransmitter receptor density precede cell loss and

131 we find that none of the previously reported neurotransmitter receptors detected by antibodies alone

132 esult may reflect greater PTK recruitment by neurotransmitter receptors, distinct from the NMDA-R, th

133 Therefore, neurotransmitter receptor dynamism associated with rapid

134 agents act on voltage-gated ion channels and neurotransmitter receptors, enabling control of neuronal

135 ia, including defects in neuronal migration, neurotransmitter receptor expression and myelination.

136 ural fate determination and specification of neurotransmitter receptor expression.

137 The nanoscale co-organization of neurotransmitter receptors facing presynaptic release si

138 itiation and maintenance of the postsynaptic neurotransmitter-receptor field are important steps duri

139 he membrane-spanning domains of the paradigm neurotransmitter receptor for acetylcholine (AChR) displ

140 ression of a gain-of-function variant of the neurotransmitter receptor for glycine (GlyR) that is fou

141 he mechanisms underlying the activity of the neurotransmitter receptors for glutamate.

142 late to investigate the gating of eukaryotic neurotransmitter receptors, for which intermediate state

143 tromer in supporting fast, local delivery of neurotransmitter receptors from endosomes to the dendrit

144 own previously that cell membranes, carrying neurotransmitter receptors from the human postmortem bra

145 Neurotransmitter receptor function can be influenced by

146 sophila to human, inhibition of postsynaptic neurotransmitter receptor function causes a homeostatic

147 An unresolved problem in understanding neurotransmitter receptor function concerns the mechanis

148 rom fly to human, a decrease in postsynaptic neurotransmitter receptor function elicits a homeostatic

149 the complex biological processes defined by neurotransmitter receptor function.

150 studies of the Cys loop family of ionotropic neurotransmitter receptors (GABA, nACh, 5-HT(3), and gly

151 Honokiol (HNK), a phenolic neurotransmitter receptor (gamma-aminobutyric acid type

152 Interestingly, KLF11 regulates neurotransmitter receptor gene expression in differentia

153 l(1)sc and controls expression of a peptide neurotransmitter receptor gene.

154 data demonstrate comprehensive screening of neurotransmitter receptor genes in a controlled neuronal

155 we detected differential methylation in two neurotransmitter receptor genes, the gamma-Aminobutyric

156 eptide receptor genes, and 31 small-molecule neurotransmitter receptor genes.

157 arch investigating individual differences in neurotransmitter receptor genotypes has highlighted the

158 ing the cell adhesion molecule L1/NgCAM, the neurotransmitter receptor GluA2, and beta-APP.

159 ones signal by stimulating G protein-coupled neurotransmitter receptors (GPCRs), which activate G pro

160 This neurotransmitter receptor has been shown previously to b

161 mission in the central nervous system, these neurotransmitter receptors have been shown to influence

162 on the trafficking of potassium channels and neurotransmitter receptors have revealed unexpected comp

163 ivation reaction predict that, after binding neurotransmitter, receptors hesitate for approximately 4

164 ermore, recent studies demonstrate that some neurotransmitter receptor heteromers can exert an effect

165 Neurotransmitter receptor heteromers can function as pro

166 The existence of neurotransmitter receptor heteromers is becoming broadly

167 ndamental properties of these key inhibitory neurotransmitter receptors; however, the ratio of alpha1

168 , including (i) acetylcholine receptors, the neurotransmitter receptor, (ii) muscle-specific kinase (

169 annels (ASICs), a small family of excitatory neurotransmitter receptors implicated in pain and neuroi

170 c, titratable Arg analog, canavanine, into a neurotransmitter receptor in a living cell, utilizing a

171 et al. document pro-survival functions of a neurotransmitter receptor in glioma progenitor cells, wi

172 AMPA receptor (AMPA-R) is a major excitatory neurotransmitter receptor in the brain.

173 GABA(B)R2 subunits suggests a novel role for neurotransmitter receptors in controlling gene expressio

174 e GABA(A) receptors are the major inhibitory neurotransmitter receptors in mammalian brain.

175 on, and regulate the selective expression of neurotransmitter receptors in neurons and at the neuromu

176 The spatiotemporal organization of neurotransmitter receptors in postsynaptic membranes is

177 quired for the physiological organization of neurotransmitter receptors in postsynaptic specializatio

178 gating and channel properties of ionotropic neurotransmitter receptors in some hereditary epilepsies

179 , and it is highly desirable to know whether neurotransmitter receptors in such tissues are still fun

180 receptor is one of the principal inhibitory neurotransmitter receptors in the brain, and it signals

181 tors (AMPARs) are the predominant excitatory neurotransmitter receptors in the brain, where they medi

182 Psychotropic agents act on a variety of neurotransmitter receptors in the brain-gut regulatory p

183 ate receptors (AMPARs), the major excitatory neurotransmitter receptors in the brain.

184 nted research on GABA(A)Rs, a major class of neurotransmitter receptors in the central nervous system

185 AMPA receptors are the major excitatory neurotransmitter receptors in the central nervous system

186 ation regulates key functional properties of neurotransmitter receptors in the central nervous system

187 tors (NMDARs) are the predominant excitatory neurotransmitter receptors in the mammalian brain.

188 cid (AMPA) receptors are the main excitatory neurotransmitter receptors in the mammalian central nerv

189 ep in synapse formation is the clustering of neurotransmitter receptors in the postsynaptic membrane,

190 of synapse development is the clustering of neurotransmitter receptors in the postsynaptic membrane.

191 cause they are the most prevalent excitatory neurotransmitter receptors in the vertebrate central ner

192 receptors are the most prevalent excitatory neurotransmitter receptors in the vertebrate central ner

193 stems where they can be modulated by several neurotransmitter receptors including histamine H(1) rece

194 ies, and broad screening toward other common neurotransmitter receptors indicated that compound 43 is

195 ity of CTZ to interact with various types of neurotransmitter receptors indicates that the drug has m

196 Deregulated neuroendocrine and neurotransmitter receptor interactions were observed in

197 ral nervous system by coupling extracellular neurotransmitter-receptor interactions to ion channel co

198 The delivery of neurotransmitter receptors into synapses is essential fo

199 The delivery of neurotransmitter receptors into the synaptic membrane is

200 ting neuronal response through regulation of neurotransmitter receptor intraneuronal fate, we hypothe

201 ity features, such as expression of specific neurotransmitter receptors, ion channels and neuropeptid

202 tion of complex sugars to adhesion proteins, neurotransmitter receptors, ion channels and secreted tr

203 10 microM) over other P2 receptors and other neurotransmitter receptors, ion channels, and enzymes.

204 rs), rhodopsin, G-protein-coupled receptors, neurotransmitter receptors, ion channels, and so on from

205 ociated with the induction of genes encoding neurotransmitter receptors, ion channels, growth factors

206 The subunit composition of postsynaptic neurotransmitter receptors is a key determinant of synap

207 Endocytic trafficking of neurotransmitter receptors is critical to neuronal signa

208 Regulation of cell surface expression of neurotransmitter receptors is crucial for determining sy

209 Regulation of synaptic neurotransmitter receptors is currently thought to be a

210 Elucidating subunit stoichiometry of neurotransmitter receptors is preferably carried out in

211 Heterogeneity in the composition of neurotransmitter receptors is thought to provide functio

212 ticity in which perturbation to postsynaptic neurotransmitter receptors leads to a retrograde enhance

213 receptor (alpha7nAChR/CHRNA7) that is also a neurotransmitter receptor linked to cognitive function,

214 Anatomical visualization of neurotransmitter receptor localization is facilitated by

215 Candidate genes include several neurotransmitter receptor loci, particularly monoamine r

216 ogic phenotypes that arise from mutations in neurotransmitter receptors (lurcher mice) and ion channe

217 , the basis of specification of postsynaptic neurotransmitter receptors matching the newly expressed

218 Altered levels of neurotransmitter receptors may disrupt neuronal function

219 ed in memory storage or retrieval, and which neurotransmitter receptor mechanisms serve its function.

220 Ionotropic neurotransmitter receptors mediate fast synaptic transmi

221 Ionotropic neurotransmitter receptors mediate fast synaptic transmi

222 Ionotropic neurotransmitter receptors mediate rapid synaptic transm

223 fic proteins, generate action potentials and neurotransmitter receptor-mediated currents.

224 esynaptic patterns to postsynaptic cells via neurotransmitter receptor-mediated intracellular signals

225 ate receptors are the predominant excitatory neurotransmitter receptors mediating fast synaptic trans

226 cones and transsynaptically recruits the key neurotransmitter receptor mGluR6 in ON-BCs to enable syn

227 Tonic activation of metabotropic neurotransmitter receptors (mGluRs, alpha1 adrenergic re

228 s on precise apposition of release sites and neurotransmitter receptors, molecular mechanisms enablin

229 organize a multitude of receptors including neurotransmitter receptors (NMDA and AMPA receptors), si

230 now possible to determine the properties of neurotransmitter receptors of normal and diseased human

231 very little is known about the properties of neurotransmitter receptors of the AD human brain.

232 Glutamate recognition by neurotransmitter receptors often relies on Arg residues

233 rovide a new explanation for the reliance of neurotransmitter receptors on Arg side chains and highli

234 we review the evidence for the expression of neurotransmitter receptors on microglia and the conseque

235 Neurotransmitter receptors on postsynaptic cells change

236 f synaptic connections requires alignment of neurotransmitter receptors on postsynaptic dendrites opp

237 (RGCs) is unaffected by blockade of specific neurotransmitter receptors or global activity.

238 xual behavior is by increasing production of neurotransmitter receptors or of enzymes that regulate n

239 They contain neurotransmitter receptors, organelles, and signaling sy

240 y also indicate that protons and ASICs are a neurotransmitter/receptor pair critical for amygdala-dep

241 PHP remains robust to acute versus long-term neurotransmitter receptor perturbation.

242 Neurotransmitter receptors previously implicated in C. e

243 Heteromerization of neurotransmitter receptors produces functional entities

244 and temporal stages of AD, thereby altering neurotransmitter receptor properties, disrupting membran

245 receptor stimulation, an alphav integrin/PLP/neurotransmitter receptor protein complex forms that red

246 Neurotransmitter receptor recruitment at postsynaptic sp

247 hesis that all iGluRs, and potentially other neurotransmitter receptors, rely on the cooperative bind

248 the selected binding of antipsychotics to 14 neurotransmitter receptors revealed only dopamine type 3

249 ion of a presynaptic nerve terminal with the neurotransmitter receptor-rich postsynaptic apparatus.

250 rge-scale gene expression changes, including neurotransmitter receptors, signal transduction cascades

251 We hypothesized that the mER and neurotransmitter receptor signaling pathways converge to

252 ith the postsynaptic membrane that organizes neurotransmitter receptors, signaling pathways, and regu

253 nce for three possible triggers: activity of neurotransmitter receptors, signaling through adhesion p

254 alpha, encoded by Gnas, mediates hormone and neurotransmitter receptor-stimulated cAMP generation.

255 Clustering of a neurotransmitter receptor subunit in the muscle at the n

256 plicing of pre-mRNAs encoding the inhibitory neurotransmitter receptor subunits GABA(A)Rgamma2 and Gl

257 ost human autoantibodies targeting other CNS neurotransmitter receptors, such as N-methyl-d-aspartate

258 ebalancing ion channel expression, modifying neurotransmitter receptor surface expression and traffic

259 ed assembly of protein complexes composed of neurotransmitter receptors, synaptic adhesion molecules

260 derstanding this pharmacologically important neurotransmitter receptor system.

261 nervous system (CNS) functions by modulating neurotransmitter receptor systems in the brain.

262 eceptors to modulate "cross-talk" with other neurotransmitter receptor systems, it is notable that ab

263 t from the TGFbeta receptor family and known neurotransmitter receptor targets of antipsychotics.

264 Neurotransmitter receptors that are activated by various

265 of mGluR1 and mGluR5) are G-protein-coupled neurotransmitter receptors that are found in the perisyn

266 ype Ca(2+) channels is mediated primarily by neurotransmitter receptors that couple to pertussis toxi

267 iation is the array of neurotransmitters and neurotransmitter receptors that each neuron possesses.

268 Neurotransmitter receptors that inhibit the release of o

269 be gained by bringing control to endogenous neurotransmitter receptors that mediate synaptic transmi

270 lation of the B cell receptor (BCR) and/or a neurotransmitter receptor, the beta(2)-adrenergic recept

271 2 subunit of a human gamma-aminobutyric acid neurotransmitter receptor, the GABA(A) receptor, is link

272 was the chemical identification of the first neurotransmitter receptor, the nicotinic acetylcholine r

273 transcription and postsynaptic targeting of neurotransmitter receptors through distinct molecular fu

274 mediated through the G proteins that couple neurotransmitter receptors to adenylyl cyclase showed a

275 ing molecules, other adhesion molecules, and neurotransmitter receptors to bring together the key com

276 Synaptogenesis requires recruitment of neurotransmitter receptors to developing postsynaptic sp

277 Precise targeting of neurotransmitter receptors to different neuronal compart

278 smission under basal conditions by targeting neurotransmitter receptors to synapses.

279 s, from the sign of the response mediated by neurotransmitter receptors to the dynamics shaped by vol

280 nteractions, second messenger signaling, and neurotransmitter receptor trafficking and function are a

281 at regulates synapse function by controlling neurotransmitter receptor trafficking and homeostatic sy

282 Neurotransmitter receptor trafficking during synaptic pl

283 Neurotransmitter receptor trafficking is fundamentally i

284 achinery play critical roles in postsynaptic neurotransmitter receptor trafficking, which is essentia

285 b (GT1b:GM1 > 4; p < 10(-4)), three regulate neurotransmitter receptor trafficking: Thorase (ATPase f

286 synaptic transmission span the modulation of neurotransmitter receptors, transporters, activation of

287 communicate with postsynaptic partners, the neurotransmitter receptor type used to receive input fro

288 th ligands for a variety of G-protein-linked neurotransmitter receptor types that have been associate

289 t, to a decrease in synaptic distribution of neurotransmitter receptors upon deletion of neuroligins.

290 ange of potential cargoes - including mRNAs, neurotransmitter receptors, vesicles and mitochondria -

291 reciate the function and regulation of these neurotransmitter receptors, we must understand their int

292 dies and broad screening toward other common neurotransmitter receptors were also carried out to furt

293 various neural precursors express functional neurotransmitter receptors, which include G protein-coup

294 Oligodendrocytes are endowed with neurotransmitter receptors whose expression levels and p

295 eceptor ion channels (iGluRs) are excitatory neurotransmitter receptors with a unique molecular archi

296 g in the nervous system requires matching of neurotransmitter receptors with cognate neurotransmitter

297 The spatial coordination of neurotransmitter receptors with other postsynaptic signa

298 Our results show how neurotransmitter receptors with similar structures and g

299 as shallow energy traps (~3 kBT) for glycine neurotransmitter receptors, with a depth modulated by th

300 complexity and cross-talk between different neurotransmitter receptors within the same synapse or ac