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1 NgR1) and a signal transducing subunit (the neurotrophin receptor p75).
2 rats also differ in their expression of the neurotrophin receptor p75.
3 gested to be a death-inducing ligand for the neurotrophin receptor p75.
4 n and induce their death via cleavage of the neurotrophin receptor p75.
5 pressed mRNA or protein for the low-affinity neurotrophin receptor p75.
6 sine kinases, Trks, and the low affinity pan-neurotrophin receptor p75.
10 of ER-alpha with the low- and high-affinity neurotrophin receptors, p75 and trkA, and with the choli
11 tyrosine kinase domain, and the low-affinity neurotrophin receptor p75 are expressed at low levels in
12 omyosin kinase receptor B (TrkB) and the p75 neurotrophin receptor (p75) are the primary regulators o
15 osin-related kinase receptors (Trks) and p75 neurotrophin receptors (p75) compete to regulate surviva
19 etyl transferase (ChAT) and the low-affinity neurotrophin receptor (p75) in both mass and clonal cult
24 that blocks the function of the low-affinity neurotrophin receptor (p75(LNTR)) prevented LIF-induced
25 lines of in vitro evidence suggest that the neurotrophin receptor p75 mediates or exacerbates Abeta-
26 ll number and the number of cells expressing neurotrophin receptors p75(NGFR) and trkA were assessed.
29 TAp73 is a transcriptional activator of p75 neurotrophin receptor (p75(NTR)) and that p75(NTR) mRNA
32 trophic factor (BDNF)-activated TrkB and p75 neurotrophin receptor (p75(NTR)) by disrupting the endos
34 ole in Alzheimer's disease (AD), and the p75 neurotrophin receptor (p75(NTR)) has been implicated in
43 rve growth factor (NGF) signaling by the p75 neurotrophin receptor (p75(NTR)) is critical for neurona
44 distinct structural determinants in the p75 neurotrophin receptor (p75(NTR)) is crucial for the iden
50 be initiated by activation of either the p75 neurotrophin receptor (p75(NTR)) or the more selective t
51 xes with LINGO-1 and either the low-affinity neurotrophin receptor (p75(NTR)) or TROY to initiate gro
54 ular signaling networks regulated by the p75 neurotrophin receptor (p75(NTR)) substantially overlap w
56 ron loss, distal axonal degeneration and p75 neurotrophin receptor (p75(NTR)) upregulation in the per
57 Strikingly, mutant male mice lacking the p75 neurotrophin receptor (p75(NTR)) were resistant to the d
58 tingly, mutant male mice that lacked the p75 neurotrophin receptor (p75(NTR)) were seen to be resista
59 uctures of complexes formed by the DD of p75 neurotrophin receptor (p75(NTR)) with RhoGDI, for activa
60 e de novo expression of the low-affinity p75 neurotrophin receptor (p75(NTR)), a gene that plays crit
63 mined the expression of the low affinity p75 neurotrophin receptor (p75(NTR)), an excellent marker of
64 smic deletion mutant of the p75 low affinity neurotrophin receptor (p75(NTR)), indicating that this t
65 ferents, identified by the low-affinity, p75 neurotrophin receptor (p75(NTR)), with interneurons cont
66 This produced a substantial loss of both p75 neurotrophin receptor (p75(NTR))-positive and choline ac
73 induce apoptosis in neuronal cells, via the neurotrophin receptor p75(NTR) and the sortilin receptor
75 ed a marked upregulation of the proapoptotic neurotrophin receptor p75(NTR) in CA1, evident at 48 hr.
76 in PC-3 cells and siRNA directed against the neurotrophin receptor p75(NTR) in post-mitotic cultures
78 rol process by the discovery that the common neurotrophin receptor p75(NTR) interacts in a ligand-dep
81 ases, may interact with the proapoptotic pan-neurotrophin receptor p75(NTR) to induce neuronal cytosk
82 kinase TrkA (also called NTRK1), the common neurotrophin receptor p75(NTR), and the proneurotrophin
83 how that the transmembrane domain of the pan-neurotrophin receptor p75(NTR), best known for regulatin
84 ability of SorCS2 to form complexes with the neurotrophin receptor p75(NTR), required for pro-brain-d
85 tion of saporin linked to an antibody to the neurotrophin receptor p75(NTR), which eliminates cells e
90 hat neurotrophin binding to the low affinity neurotrophin receptor, p75(NTR), alone does not affect t
91 In recent studies, we have identified the neurotrophin receptor, p75(NTR), as a mediator of apopto
94 ormation for apical sorting of transmembrane neurotrophin receptors (p75(NTR)) is localized to a juxt
98 tivity for pro-nerve growth factor (proNGF), neurotrophin receptor p75 (p75(NTR)), and sortilin in th
99 hese effects are mediated by presynaptic pan-neurotrophin receptor p75 (p75(NTR)), the pro-BDNF recep
102 receptor tyrosine kinases (Trk) and the pan-neurotrophin receptor (p75) to regulate complex developm
103 d immunoprecipitation with antibodies to the neurotrophin receptors p75, trkA, trkB, and trkC showed