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1 lation of neurotoxic intermediates result in neurovisceral attacks and disease manifestations in pati
2 sionally causes frequent and crippling acute neurovisceral attacks associated with increased hepatic
3 esis characterized by life-threatening acute neurovisceral attacks precipitated by factors that upreg
4 terized clinically by skin lesions and acute neurovisceral attacks that occur separately or together.
5  Human AIP heterozygotes have episodic acute neurovisceral attacks that typically start after puberty
6                Patients can experience acute neurovisceral attacks, debilitating chronic symptoms, an
7 A) and porphobilinogen (PBG) promotes sudden neurovisceral attacks, which can be life-threatening.
8 esis characterized by life-threatening acute neurovisceral attacks.
9 iemann-Pick Type C1 (NPC1) disease is a rare neurovisceral, cholesterol-sphingolipid lysosomal storag
10                A richer understanding of the neurovisceral concomitants of this functional interplay
11                  Niemann-Pick C disease is a neurovisceral disorder caused by mutations in the NPC ge
12 use of Niemann-Pick type C2 disease, a fatal neurovisceral disorder characterized by accumulation of
13 iemann-Pick type C1 (NPC1) disease is a rare neurovisceral disorder characterized by intracellular ac
14  Niemann-Pick type C (NP-C) disease, a fatal neurovisceral disorder, is characterized by lysosomal ac
15 ize these psychophysiological processes, the neurovisceral integration model of fear (NVI-f).
16                NPC is an autosomal recessive neurovisceral lipid storage disease associated with an a
17 n-Pick type C disease (NP-C) is an inherited neurovisceral lipid storage disorder characterized by pr
18 iemann-Pick disease type C (NPC) is a severe neurovisceral lysosomal storage disorder caused by defec
19           Niemann-Pick C disease (NP-C) is a neurovisceral lysosomal storage disorder.
20 s between the presence of out-group members, neurovisceral reactions, religiosity, and ethnocentrism,
21 or Batten disease, the most common inherited neurovisceral storage disease of childhood, was identifi
22 e C (NP-C) disease is an autosomal recessive neurovisceral storage disorder in which cholesterol and
23 synthesis, cause life-threatening attacks of neurovisceral symptoms that mimic many other acute medic
24 hese disorders present clinically with acute neurovisceral symptoms which may be sporadic or recurren
25 th attacks of acute abdominal pain and other neurovisceral symptoms, with or without cutaneous manife
26 l transport causes loss of neurons and fatal neurovisceral symptoms.
27 ipids leading to neurodegeneration and fatal neurovisceral symptoms.
28 sified as follows: (1) acute porphyrias with neurovisceral symptoms: acute intermittent porphyria; de