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1 romonas gingivalis and Prevotella intermedia/nigrescens.
2 phyromonas gingivalis, Prevotella intermedia/nigrescens, Aggregatibacter actinomycetemcomitans, and F
3 ignificant decreases in levels of Prevotella nigrescens and Prevotella intermedia, serum IL-6sr, and
4 forsythus, Prevotella intermedia, Prevotella nigrescens, and Campylobacter rectus were detected in a
5  nigrescens (OR 3.23); and P. gingivalis, P. nigrescens, and T. denticola (OR 2.59); with severe peri
6  nigrescens (OR 2.91); and P. gingivalis, P. nigrescens, and T. denticola (OR 2.70) with the clinical
7 . denticola (OR 4.06); and P. gingivalis, P. nigrescens, and T. denticola (OR 3.29).
8 sythensis, Prevotella intermedia, Prevotella nigrescens, and Treponema denticola before and following
9 forsythia, Prevotella intermedia, Prevotella nigrescens, and Treponema denticola) and the potential s
10 forsythus, Prevotella intermedia, Prevotella nigrescens, and Treponema denticola.
11 phyromonas gingivalis, Prevotella intermedia/nigrescens, Bacteroides forsythus, Eubacterium species,
12 teric rods, Prevotella intermedia/Prevotella nigrescens, Capnocytophaga, Propionibacterium, yeasts, A
13        For example, the bacterium Prevotella nigrescens contains a distinctive bifunctional protein c
14 inomycetemcomitans, C. rectus, P. intermedia/nigrescens, E. corrodens, P. micros, Capnocytophaga and
15      In this study, we characterized this P. nigrescens enzyme and found that its two catalytic activ
16 ects of the two bacteria diverged in that P. nigrescens, in contrast to P. gingivalis, suppressed the
17 gens Porphyromonas gingivalis and Prevotella nigrescens induced periodontitis in mice, as evidenced b
18  1) the cell-free extracellular toxins of P. nigrescens MH1 and 2) the cell-free cell lysates of P. i
19 e: Prevotella intermedia (n = 4), Prevotella nigrescens (n = 4), and Porphyromonas gingivalis (n = 1)
20 R 2.91); P. gingivalis, B. forsythus, and P. nigrescens (OR 2.91); and P. gingivalis, P. nigrescens,
21 B. forsythus (OR 2.06); P. gingivalis and P. nigrescens (OR 2.91); P. gingivalis, B. forsythus, and P
22 R 3.23); P. gingivalis, B. forsythus, and P. nigrescens (OR 3.23); and P. gingivalis, P. nigrescens,
23 ed with coinfections of P. gingivalis and P. nigrescens (OR 3.23); P. gingivalis, B. forsythus, and P
24 h CHD among ever smokers, whereas Prevotella nigrescens (OR=1.7; 95% CI, 1.1 to 2.6), Actinobacillus
25 nema denticola, A. actinomycetemcomitans, P. nigrescens, P. intermedia, B. forsythus, and P. gingival
26 cid biosynthesis in the bacterium Prevotella nigrescens (PniDAH7PS), respectively.
27 ampylobacter Rectus, Factor 2 (Pi/Prevotella nigrescens/Prevotella melaninogenica), and the Orange-Re
28 r degree than High-M alginates from Lessonia nigrescens, showing that inhibition was related to algin
29                    Both P. gingivalis and P. nigrescens skewed the CII-specific T cell response in ly
30 st species, most often Prevotella intermedia/nigrescens, Streptococcus constellatus, or Aggregatibact
31 valis, E. corrodens, C. concisus, Prevotella nigrescens, T. forsythia, and Dialister pneumosintes.
32 s of known periodontal pathogens, such as P. nigrescens, T. forsythia, and E. corrodens, as well as C
33 sintes, Tannerella forsythia, and Prevotella nigrescens than SUP sites from patients with AgP (P <0.0
34                                   Prevotella nigrescens was the only species (p < 0.05) significantly
35  Prevotella intermedia (including Prevotella nigrescens) was investigated.
36 ivalis and especially Prevotella intermedius/nigrescens were often identified at peri-implantitis sit
37 ythia, Prevotella intermedia, and Prevotella nigrescens were statistically significantly higher in pr
38 bes (Porphyromonas gingivalis and Prevotella nigrescens) were significantly reduced (P </= 0.001) in
39 ding increased Neisseria spp. and Prevotella nigrescens, while 19 taxa had a decreased abundance.