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1 by noxious stimuli toward the outcome of non-nocifensive actions.
2 m circuit can control facial expressions for nocifensive and potentially pain-inducing stimuli.
3 lly reduces isocyanate- and tear gas-induced nocifensive behavior after both ocular and cutaneous exp
4                  We show that TRPA1-mediated nocifensive behavior can be sensitized in vivo via PKA/P
5 , we address these problems by investigating nocifensive behavior in Drosophila larvae.
6 t significantly potentiated capsaicin-evoked nocifensive behavior in female rats at proestrous and in
7 nically available compounds strongly reduced nocifensive behavior in mouse models of inflammatory and
8 ction potential (AP) generation and reducing nocifensive behavior in naive and inflamed rats.
9 urse of the MO-induced behavioral allodynia (nocifensive behavior in response to a previously innocuo
10    TRPA1-deficient mice show greatly reduced nocifensive behavior in response to isovelleral, indicat
11 Similarly, RGS4KO eliminates the duration of nocifensive behavior in the second phase of the formalin
12 herally selective dose of ACEA on MO-induced nocifensive behavior in vivo; and 3) the effects of five
13 ly inhibited ( approximately 40%) MO-induced nocifensive behavior in wild-type mice but not in TRPV1
14 urons, reduced meningeal blood flow, reduced nocifensive behavior induced by formalin injection or co
15 tween substance P levels and formalin-evoked nocifensive behavior may exist in diabetic rats, as exag
16 he shear stress responses of nociceptors and nocifensive behavior require transient receptor potentia
17 elease is unlikely to underlie the increased nocifensive behavior seen in diabetic rats.
18 of mechanical sensitivity of the abdomen and nocifensive behavior to electrical stimulation of the pa
19 ancreatic hyperalgesia was assessed by rat's nocifensive behavior to electrical stimulation of the pa
20 M1241-induced inhibition of capsaicin-evoked nocifensive behavior via peripheral actions is reversed
21                              Formalin-evoked nocifensive behavior was increased in diabetic rats but
22                                              Nocifensive behavior was only reduced in phase 1 in the
23 -Pm1a (delta/kappa-TRTX-Pm1a), that elicited nocifensive behavior when injected into mice.
24  partially recovers form3 defects in MTs and nocifensive behavior, suggesting conserved functions, th
25 ithdrawal thresholds or in capsaicin-induced nocifensive behavior.
26        Noxious pH triggers pungent taste and nocifensive behavior.
27 on of the compound induced a TRPM3-dependent nocifensive behavior.
28 , the peptides produced diverging effects on nocifensive behavior.
29  manner (EC(50) = 0.72 microg), and produced nocifensive behavior.
30      Pancreatic hyperalgesia was assessed by nocifensive behavioral response to electrical stimulatio
31 athepsin K in male mice on the expression of nocifensive behaviors after formalin injection or mechan
32 mined the role of the descending 5-HT in rat nocifensive behaviors after persistent pain by selective
33  receptor agonists induced similar levels of nocifensive behaviors and activity at spinal NMDA recept
34 cline reversed and prevented the increase of nocifensive behaviors and levels of P-p38 in rats with C
35 s a key regulator of the operating range for nocifensive behaviors and suggest strategies for produci
36  but instead served to dramatically heighten nocifensive behaviors and suppress itch.
37                                        These nocifensive behaviors can be blocked by the NK1 antagoni
38 DL1 and found that PD1 significantly reduced nocifensive behaviors evoked by algogenic capsaicin.
39 ged allodynia to hindpaw PGE(2) and enhanced nocifensive behaviors evoked by intrathecal mGluR1/5 ago
40 trations, menthol can induce lacrimation and nocifensive behaviors in a TRPM8 independent mechanism.
41 s to contribute to central sensitization and nocifensive behaviors in certain animal models of chroni
42 ensitization of knee-innervating neurons and nocifensive behaviors in mice.
43 ke allyl isothiocyanate, it failed to induce nocifensive behaviors in mice.
44 , to contribute substantially to paradoxical nocifensive behaviors in neonates and various pain state
45 nockout mice (Ctsk(-/-)) have a reduction in nocifensive behaviors in the formalin test.
46                                              Nocifensive behaviors induced by the intradermal injecti
47  these findings are associated with enhanced nocifensive behaviors specifically in TRPA1-dependent pa
48  of KET to reduce significantly a variety of nocifensive behaviors suggests that this clinically safe
49  injection of 100 microg anandamide produced nocifensive behaviors that were attenuated by pre-treatm
50 excited cutaneous C nociceptors and produced nocifensive behaviors via activation of TRPV1 receptors.
51 stration that exogenous peripheral SP causes nocifensive behaviors would seem to indicate that SP can
52 s the stimulus intensities that trigger such nocifensive behaviors, but the molecular networks that d
53 ght illumination of the skin produced robust nocifensive behaviors, evoked by the remote stimulation
54 ese cells in vivo increases anxiety-like and nocifensive behaviors.
55 crimation via TRPM8 channels without evoking nocifensive behaviors.
56 ociceptors by anandamide was associated with nocifensive behaviors.
57  vivo and if this excitation correlated with nocifensive behaviors.
58 enuation of both ongoing and movement-evoked nocifensive behaviors.
59 romedial medulla (RVM) neuronal activity and nocifensive behaviors.
60 cal hypersensitivity and show a reduction in nocifensive behaviors.
61 nput, the neural signals trigger protective (nocifensive) behaviors, and the sensory stimuli that rea
62 in the head nociceptor neuron, ASH, we study nocifensive behaviour and Ca(2+) influx.
63 rons significantly shortened the duration of nocifensive behaviour induced by hindpaw injection of br
64 tionship between ion permeation and animals' nocifensive behaviour is unknown.
65 hold mechanical nociceptors where it adjusts nocifensive behaviour under physiological and pathologic
66 second phase of formalin-induced spontaneous nocifensive behaviour, suggesting a potential role of SO
67 b8+ mice displayed a significant increase in nocifensive behaviours in the second phase of the formal
68 on neurons and/or participating in segmental nocifensive circuits.
69 onic saline that evoked comparable levels of nocifensive eye wipe behaviors when applied to the ocula
70 lating the elaborate dendrite morphology and nocifensive functions of Drosophila larval class IV dend
71 analgesia and shifts the operating range for nocifensive heat avoidance to higher temperatures.
72 rgic receptors suppressed BG flares, reduced nocifensive licking and had analgesic effects in nerve i
73                                 In contrast, nocifensive-like behavior to extreme cold-plate stimulat
74 gnitude, suggesting that ON cells facilitate nocifensive motor reactions.
75 tion of either H(2)O(2) or 15d-PGJ2 evoked a nocifensive/pain response in wild-type mice, but not in
76                                              Nocifensive paw-shaking behavior following masseteric in
77  soup of inflammatory mediators enhanced the nocifensive paw-withdrawal reflex elicited by hypotonic
78 eurons (1) are not essential for determining nocifensive reflex responsiveness to noxious thermal sti
79 l of this study was to identify neurons with nocifensive reflex-related activity in the mesopontine t
80      The mesopontine tegmentum thus contains nocifensive reflex-related neurons with neurophysiologic
81 RVM produce changes in the latency of spinal nocifensive reflexes and, when iontophoretically applied
82 nd provide the afferent limb for hyperactive nocifensive reflexes.
83 in the formalin test: (1) increased baseline nocifensive responding and (2) reduced antinociceptive a
84 ns of EP2 or BK2 agonists inhibited both the nocifensive response evoked by TRPM3 agonists, and the h
85 5 to - 6 degrees C) that induced decrease in nocifensive response starting from about a week after tr
86 d to innocuous warming with regurgitation, a nocifensive response.
87 on caused scratching, biting, and licking, a nocifensive response.
88                       In additional studies, nocifensive responses (eye swiping and lid closure) were
89 3(Lbx1) neurons): the mice lost brush-evoked nocifensive responses and conditional place aversion.
90                      In contrast, PMA-evoked nocifensive responses and sensitization of capsaicin res
91 ment attenuated formalin-induced spontaneous nocifensive responses and tissue or nerve injury-induced
92 ic activation of Bar(Crh) neurons attenuated nocifensive responses as well as tactile neuropathic pai
93 ly inhibited formalin- and capsaicin-induced nocifensive responses but did not alter baseline thermal
94 rents induced by CIM0216 in DRG neurons, and nocifensive responses elicited by this TRPM3 agonist in
95 l role of distinct lPBN efferents in diverse nocifensive responses have remained largely uncharacteri
96 ate well with the time course of behavioural nocifensive responses in rats to the same agonist descri
97 with other P2X channel agonists, can produce nocifensive responses in rodents.
98 ation, menthol (50 mM) increased tearing and nocifensive responses in TRPM8 wild type and knockout an
99 ments demonstrate that DC lesions reduce the nocifensive responses produced by noxious stimulation of
100  had no effect in TRPM8 knockout mice, while nocifensive responses remained unaffected.
101 ng pathway, were injured and then tested for nocifensive responses to a normally subnoxious stimulus.
102  injury-evoked hypersensitivity to cold, and nocifensive responses to cooling compounds.
103 tion of CeA(CAM)-LPBN projections attenuated nocifensive responses to mechanical pressure and radiant
104 rostral ventromedial medulla (RVM), inhibits nocifensive responses to noxious stimulation through a p
105  these neurons in (1) normal baseline reflex nocifensive responses to noxious thermal stimulation (ho
106  role in the endogenous modulation of reflex nocifensive responses to persistent pain in the formalin
107 onses to punctate mechanical stimulation and nocifensive responses to thermal noxious stimuli are not
108 s demonstrated by increased capsaicin-evoked nocifensive responses, increased calcitonin gene-related
109 injection in mice reduces heat and capsaicin nocifensive responses, whereas the intra-articular injec
110 e emotional-affective behaviors and modulate nocifensive responses.
111 yphenylglycol) precipitated allodynia and/or nocifensive responses.
112 e in baseline tail-flick and hotplate reflex nocifensive responses.
113  descending pathway that powerfully controls nocifensive sensory input to the brain.

 
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