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1 hearing threshold, or the susceptibility to noise-induced hearing loss).
2 istening to loud music too often, results in noise-induced hearing loss.
3 tion of drug targets and novel therapies for noise-induced hearing loss.
4 support to a role of GPx1 activity in acute noise-induced hearing loss.
5 c coding of TFS and ENV following permanent, noise-induced hearing loss.
6 air cell and synaptic ribbon loss as well as noise-induced hearing loss.
7 dBA over 24 hr) in 2013 and were at risk of noise-induced hearing loss.
8 d identify defective pexophagy as a cause of noise-induced hearing loss.
9 these Kv2.2KO mice were more susceptible to noise-induced hearing loss.
10 ated between the acute and chronic phases of noise-induced hearing loss.
11 promised hearing sensitivity and potentiated noise-induced hearing loss.
12 d enhance the resistance of the inner ear to noise-induced hearing loss.
13 nd elucidated their protective roles against noise-induced hearing loss.
14 be considered a target in the prevention of noise-induced hearing loss.
15 e did, however, show increased resistance to noise-induced hearing loss.
16 oxygen species (ROS) and auditory damage in noise-induced hearing loss.
17 Cs as potential targets for the treatment of noise-induced hearing loss.
18 profound protection from both cisplatin- and noise-induced hearing loss.
19 KSR1 wild-type mice from both cisplatin- and noise-induced hearing loss.
20 tions are implicated in both age-related and noise-induced hearing loss.
21 ing onset and with acquired (age-related and noise-induced) hearing loss.
22 ight the need for public policies to prevent noise-induced hearing loss, an entirely avoidable hearin
23 shown to be involved in the pathogenesis of noise-induced hearing loss and cochlear synaptopathy in
26 udy provides new insights into mechanisms of noise-induced hearing loss and suggests novel interventi
27 rs may be at risk of permanent, irreversible noise-induced hearing loss and that, for many individual
28 nnel-specific mechanisms of plasticity after noise-induced hearing loss and will aid in understanding
30 r data, neither group was at ongoing risk of noise-induced hearing loss, as average daily sound level
31 eficiency causes increased susceptibility to noise-induced hearing loss associated with plasma hyperh
32 57BL/6J mouse is a model for presbycusis and noise-induced hearing loss because of its age-related he
33 noise intensity dependent and contributes to noise-induced hearing loss by mediating the loss of inne
34 ence of the antioxidant glutathione (GSH) on noise-induced hearing loss by using l-buthionine-[S,R]-s
35 ment in gap detection.SIGNIFICANCE STATEMENT Noise-induced hearing loss contributes to a range of cen
36 rations of its functional properties lead to noise-induced hearing loss, highlighting the importance
37 the MOC system's protective role to prevent noise-induced hearing loss in adulthood is well establis
41 nd efficacy of ebselen for the prevention of noise-induced hearing loss in young adults in a phase 2
45 neurotransmission in this mechanism after a noise-induced hearing loss larger than 50 dB in high fre
47 formation and potential energy depletion in noise induced hearing loss (NIHL), we measured the effec
48 nflammation in the auditory cortex following noise-induced hearing loss (NIHL) and its role in tinnit
57 tively impact the resilience of the brain to noise-induced hearing loss (NIHL) later in adulthood.
58 is gap, we examined the long-term effects of noise-induced hearing loss (NIHL) on the spontaneous act
61 male and female mice and in a mouse model of noise-induced hearing loss (NIHL), also in male and fema
62 iated with audiometric notches indicative of noise-induced hearing loss (NIHL), speech frequency hear
63 of endogenous glutathione (GSH) potentiates noise-induced hearing loss (NIHL), whereas replenishment
69 e Isl1 overexpression protected the ear from noise-induced hearing loss (NIHL): both ABR threshold sh
72 ffect of the long-term inhibition of MMPs on noise-induced hearing loss was further confirmed using t
74 d on their risk for a clinically-significant noise-induced hearing loss, we found that prestin levels