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1 outh or face (6) and seizure (5, including 3 non-epileptic).
2     Furthermore, activating granule cells in non-epileptic animals evoked acute seizures of increasin
3                                   In normal, non-epileptic animals, these strongly inhibited dentate
4 were increased in epileptic mice compared to non-epileptic animals.
5 m contralateral non-seizure-generating MTL ("non-epileptic") areas.
6 ine the portrayal of epilepsy, seizures, and non-epileptic attack disorder in 62 movies produced over
7 y of life and other psychosocial issues; and non-epileptic attack disorder.
8 (iii) conversion symptoms (motor, sensory or non-epileptic attacks) (18%).
9 e diagnosis and investigation of psychogenic non-epileptic attacks, drop attacks, falls, syncope and
10               Other associated symptoms were non-epileptic attacks, migraine, fatigue and sleep paral
11 ow-up; five of whom had initial diagnoses of non-epileptic attacks.
12 urgery in 10 pediatric patients, focusing on non-epileptic background activity.
13 lly sorted human epileptic brain compared to non-epileptic brain obtained after resection from six dr
14 viation of slow-wave sleep MI of neighboring non-epileptic channels of 47 patients, whose ECoG sampli
15 y being diagnosed with epilepsy and 54% with non-epileptic conditions.
16 ilepsy rats from Strasbourg (GAERS), and its non-epileptic control (NEC) strain.
17  epileptic rats when compared to age-matched non-epileptic control rats.
18  of genetic generalized epilepsy, and inbred non-epileptic control rats.
19 mygdalas and hippocampi were conducted in 50 non-epileptic controls (age 7-79 years) and 50 patients
20 erentiated patients with focal epilepsy from non-epileptic controls (mean AUC 0.78) but failed to cla
21 ment of these mice with GABAA antagonists at non-epileptic doses causes a persistent post-drug recove
22 at least 10 years of age (7367 artefact-free non-epileptic electrodes), whereas a younger group inclu
23 ere reported in 14/25, with hyperekplexia or non-epileptic erratic myoclonus being the most prevalent
24 l autonomic manifestations were dismissed as non-epileptic events of other diseases.
25 gnificantly higher burst rates compared with non-epileptic hippocampal neurons (both p = 0.01).
26 erence in burst firing between epileptic and non-epileptic hippocampal neurons was observed.
27 roving differentiation between epileptic and non-epileptic hippocampi in humans.
28 properties recorded at rest in epileptic and non-epileptic hippocampi suggests that they cannot be us
29 sible to differentiate between epileptic and non-epileptic hippocampi using a simple odd-ball task.
30  and duration were compared in epileptic and non-epileptic hippocampi.
31        These reductions were not observed in non-epileptic hippocampi.
32  a cognitive task in epileptic compared with non-epileptic hippocampi.
33                            The occurrence of non-epileptic hyperkinetic movements in the context of d
34                             We analysed 3401 non-epileptic intracranial electrode sites from 37 child
35  deviation of modulation index (MI) from the non-epileptic mean (rated by z-score) improved the perfo
36 ment of statistical deviation of MI from the non-epileptic mean on invasive recording is technically
37  between epileptic MTL neurons compared with non-epileptic MTL neurons during SWS (p = 0.04) and REM
38 t disorder characterized by a combination of non-epileptic myoclonic jerks and dystonia.
39 al synchrony was found between epileptic and non-epileptic neurons during Aw.
40 in resulted in a 90% clinical improvement in non-epileptic paroxysmal manifestations and a normalised
41 patients with GLUT1-DS (7-47 years old) with non-epileptic paroxysmal manifestations.
42 teins expressed differently in epileptic and non-epileptic patients and their associated biological p
43 50 epileptic patients with ictal deja vu, 50 non-epileptic patients attending general neurology clini
44 ction and surgical removed brain tissue from non-epileptic patients.
45  hyperandrogenemia), this was not evident in non-epileptic primates.
46      Some evidence suggests that psychogenic non-epileptic seizures (PNES) are associated with increa
47                                  Psychogenic non-epileptic seizures (PNES) are classified with other
48 of 260 consecutive patients with psychogenic non-epileptic seizures (PNES), 5-10 years after diagnosi
49  studies of long-term outcome in psychogenic non-epileptic seizures (PNES), and none of long-term hea
50 n the treatment of patients with psychogenic non-epileptic seizures and generates ideas for future re
51 While the diagnostic features of psychogenic non-epileptic seizures have been better characterized in
52 onal seizures (FS) known also as psychogenic non-epileptic seizures or dissociative seizures, present
53 re not detected in patients with psychogenic non-epileptic seizures, and did not result from medicati
54 mples of multiple temporal lobe epilepsy and non-epileptic subjects.
55 d that, first, patients tend to transit from non-epileptic to epileptic states more often than contro