1 Previous mapping experiments between EL and
nonepileptic ABP mice identified, and a congenic strain
2 nted spike output compared with neurons from
nonepileptic animals (non-SE neurons).
3 xcitatory stimuli compared with neurons from
nonepileptic animals.
4 standing of mechanisms underlying paroxysmal
nonepileptic as well as some epileptic disorders.
5 er of cortical neurons compared with the two
nonepileptic baboons.
6 tic drugs (AEDs) are commonly prescribed for
nonepileptic conditions, including migraine headache, ch
7 tlases, using subdural EEG signals from 8251
nonepileptic electrode sites in 114 patients (ages 1.0-4
8 HFO rate were higher at seizure onset versus
nonepileptic electrode sites.
9 seizures or PDs but not during electrically
nonepileptic epochs.
10 ic seizures, single unprovoked seizures, and
nonepileptic events from those with new-onset epilepsy.
11 t that SWDs and associated immobility may be
nonepileptic in healthy outbred rats and reflect instead
12 electrocorticography (ECoG) measure from the
nonepileptic mean.
13 Noninjected control and
nonepileptic monkeys with injections into the middle and
14 In addition, the
nonepileptic paroxysmal movement disorder hyperekplexia
15 e anticonvulsants, and is among the first in
nonepileptic patients, suggesting that sudden anticonvul
16 epilepsy models but has not been observed in
nonepileptic rodents, suggesting that FRs are associated
17 ere more frequent in epileptic compared with
nonepileptic rodents; however, this feature showed limit
18 ncephalography remains the gold standard for
nonepileptic seizure diagnosis.
19 nt video electroencephalography to establish
nonepileptic seizure diagnosis.
20 The misdiagnosis of
nonepileptic seizure is costly to patients, the healthca
21 Nonepileptic seizure patients remain one of the most cha
22 es, such as frontal lobe seizures, may mimic
nonepileptic seizure semiology.
23 re that could benefit them.The first step in
nonepileptic seizure treatment is proper diagnosis.
24 The methodology in
nonepileptic seizure treatment trials is examined, descr
25 e number of publications about (psychogenic)
nonepileptic seizures (NES) over the past two decades.
26 Psychogenic
nonepileptic seizures (PNES) are diagnosed in approximat
27 burdens from diagnostic delay of psychogenic
nonepileptic seizures (PNES) requires prompt referral fo
28 er, seizures type, also known as psychogenic
nonepileptic seizures (PNES).
29 emporal lobe epilepsy (TLE), (2) psychogenic
nonepileptic seizures (PNESs) from MRI-negative epilepsi
30 wledge of longer term outcome in psychogenic
nonepileptic seizures (PNESs) patients is limited; we kn
31 between convulsive epileptic and psychogenic
nonepileptic seizures (PNESs).
32 studies assessing patients with psychogenic
nonepileptic seizures and developments in treatment.
33 Nonepileptic seizures are best conceptualized and referr
34 Finally, realizing that
nonepileptic seizures are in a spectrum of somatoform di
35 Patients with
nonepileptic seizures are prescribed antiepileptic drugs
36 Nonepileptic seizures are seizure-like symptoms that occ
37 Nonepileptic seizures occur in 10 to 20% of children who
38 cribed antiepileptic drugs that do not treat
nonepileptic seizures, have multiple laboratory tests pe
39 This review will characterize
nonepileptic seizures, identify associated factors, prop
40 Psychogenic
nonepileptic seizures, the most common conversion disord
41 r equipped to treat the underlying causes of
nonepileptic seizures.
42 that more clearly establish the diagnosis of
nonepileptic seizures.
43 ypertonia, apnea, and noise or touch-induced
nonepileptic seizures.
44 BA in epilepsy patients at seizure onset and
nonepileptic sites, cortical lesions, and from patients
45 ty from an early hyperexcitatory to a mature
nonepileptic state.
46 In the left-handed
nonepileptic subjects, there was high posttest probabili
47 r alpha1 subunit per postsynaptic density in
nonepileptic VP was 6.1 +/- 3.7, for alpha3 subunit in R