1 sympathetic neurons than in an electrically
nonexcitable cell line, and provide a framework for futu
2 Unlike the
nonexcitable cell membranes that are ubiquitous in all d
3 at ryanodine receptors are expressed in some
nonexcitable cell types and furthermore suggest that the
4 CE) is a major Ca(2+) influx pathway in most
nonexcitable cell types and is activated by any stimulus
5 ecules that play roles in both excitable and
nonexcitable cell types and with or without the pore-for
6 Only 2 (HeLa and LLC-PK1 cells) out of 11
nonexcitable cell types examined expressed ryanodine rec
7 discuss sodium channel expression in diverse
nonexcitable cell types, including astrocytes, NG2 cells
8 ryanodine receptors in several excitable and
nonexcitable cell types.
9 easing the susceptibility to apoptogens in a
nonexcitable cell.
10 lum (ER) are a major Ca(2+) entry pathway in
nonexcitable cells and are essential for T cell activati
11 is the predominant Ca(2+) entry mechanism in
nonexcitable cells and controls a variety of physiologic
12 ical, and molecular properties as I(CRAC) in
nonexcitable cells and its rate of activation during rep
13 Possible roles for these molecules in
nonexcitable cells are acute cell-volume regulation and,
14 ion potential (AP)-generating cells, whereas
nonexcitable cells are generally considered as barriers
15 ransmission; their structure and function in
nonexcitable cells are not well-defined.
16 cyclase type 8 (AC8) is activated by CCE in
nonexcitable cells but is not responsive to other forms
17 junctional resistance between excitable and
nonexcitable cells during cardiac action potential propa
18 o the role of voltage-gated Ca2+ channels in
nonexcitable cells during development.
19 t models for agonist-activated Ca2+ entry in
nonexcitable cells focus on the capacitative mechanism w
20 ium entry or store-operated calcium entry in
nonexcitable cells is a process whereby the activation o
21 e intracellular Ca(2+) concentration of many
nonexcitable cells is regulated by calcium store release
22 ole of Ca2+ in stimulus-response coupling in
nonexcitable cells is still not well understood.
23 Ca(2+) signaling in
nonexcitable cells is typically initiated by receptor-tr
24 Whether
nonexcitable cells may modulate excitable cell function
25 -7)) are widely distributed in excitable and
nonexcitable cells of vertebrates.
26 hat the consequences of their actions are in
nonexcitable cells remain critical questions.
27 pacitative (or store-operated) Ca2+ entry in
nonexcitable cells represents a switching between two di
28 identity of the calcium channels present in
nonexcitable cells such as T lymphocytes.
29 nnels are expressed in various excitable and
nonexcitable cells supporting important cellular respons
30 Astrocytes are electrically
nonexcitable cells that communicate by means of Ca(2+) s
31 activated pathway for the entry of Ca(2+) in
nonexcitable cells that is entirely separate from the wi
32 t and that the channel probably functions in
nonexcitable cells to depolarize membrane potential and/
33 sustained Ca2+ signals seen in a variety of
nonexcitable cells under conditions of maximal stimulati
34 sical mechanism to activate hHv1 channels in
nonexcitable cells upon infection or injury.
35 Chondrocytes are usually considered as
nonexcitable cells with no spontaneous [Ca(2+)](i) signa
36 The extracellular potential of excitable and
nonexcitable cells with respect to ground is approximate
37 mportant role in agonist-evoked secretion in
nonexcitable cells, although this has not been confirmed
38 coordinating local activity of electrically
nonexcitable cells, because identical patterns of ATP re
39 We have determined that, like
nonexcitable cells, both neonatal and adult cardiomyocyt
40 In
nonexcitable cells, depletion of endoplasmic reticulum C
41 In many
nonexcitable cells, depletion of the inositol 1,4, 5-tri
42 In both excitable and
nonexcitable cells, diverse physiological processes are
43 Here we show that T lymphocytes, which are
nonexcitable cells, express both regulatory beta and por
44 TGFbeta as a modulator of mechanosensing in
nonexcitable cells, identifies the TRPV4 channel as the
45 ysiological significance of this behavior in
nonexcitable cells, in which the primary mechanism of Ca
46 dentification of Orai as the SOCE channel in
nonexcitable cells, investigation of Orai function in ne
47 All cells, including
nonexcitable cells, maintain a discrete transmembrane po
48 In
nonexcitable cells, stimulation by high agonist concentr
49 Even in
nonexcitable cells, the membrane potential V(m) is funda
50 In
nonexcitable cells, the predominant mechanism for regula
51 In
nonexcitable cells, there are currently two models for i
52 In
nonexcitable cells, we had previously established that C
53 tor of store-operated Ca(2+) entry (SOCE) in
nonexcitable cells.
54 ich is a major Ca(2+) influx pathway in most
nonexcitable cells.
55 ssed at the plasma membrane of excitable and
nonexcitable cells.
56 ding of the unanticipated roles for VGCCs in
nonexcitable cells.
57 L-type voltage-gated Ca2+ channel CaV1.2 in
nonexcitable cells.
58 r molecular counterpart for Ca(2+) influx in
nonexcitable cells.
59 n electrical signaling in nerve, muscle, and
nonexcitable cells.
60 or Ca(2+) signal generation in excitable and
nonexcitable cells.
61 and membrane potential in both excitable and
nonexcitable cells.
62 g electrical signaling in both excitable and
nonexcitable cells.
63 he Ca2+-sensitive phosphatase calcineurin in
nonexcitable cells.
64 a(2+) signaling events in both excitable and
nonexcitable cells.
65 esents the principal Ca2+ entry pathway into
nonexcitable cells.
66 ype voltage-gated Ca(2+) channel Ca(V)1.2 in
nonexcitable cells.
67 ue to follow dynamic changes in potential in
nonexcitable cells.
68 iphosphate (IP3)-mediated Ca2+ liberation in
nonexcitable cells.
69 with respect to store-operated Ca2+ entry in
nonexcitable cells.
70 nnels) are widely expressed in excitable and
nonexcitable cells.
71 t Ca(2+) influx pathway in oocytes and other
nonexcitable cells.
72 d in the sustained phase of calcium entry in
nonexcitable cells.
73 a(2+) are sufficient for exocytosis in these
nonexcitable cells.
74 f whether they are expressed in excitable or
nonexcitable cells.
75 zing the membrane potential of excitable and
nonexcitable cells.
76 lux for stimulus-secretion coupling in these
nonexcitable cells.
77 apes the profile of the Ca2+ signal in these
nonexcitable cells.
78 tant in the physiology of both excitable and
nonexcitable cells.
79 y be the primary mechanism for Ca2+ entry in
nonexcitable cells.
80 ol many important processes in excitable and
nonexcitable cells.
81 calcium (SOC) influx in platelets and other
nonexcitable cells.
82 + channels in nuclear signal transduction in
nonexcitable cells.
83 and immunocytochemical analysis showed that
nonexcitable folliculostellate cells express GPR68 gene
84 ensitivity of adenylyl cyclase to opioids in
nonexcitable HEK293 cells, whereas inhibition of C-Raf o
85 e and identify ryanodine receptors (RyRs) in
nonexcitable mouse parotid acini.
86 Nonexcitable muscle membrane after direct muscle stimula
87 We investigated whether
nonexcitable muscle membrane indicates fast-twitch myofi
88 We suggest that electrophysiological
nonexcitable muscle membrane predicts preferential type
89 Patients with
nonexcitable muscle membranes (n = 15) showed smaller me
90 Proliferating cells, typically considered "
nonexcitable,"
nevertheless, exhibit regulation by bioel
91 knowledge of VGCC regulation and function in
nonexcitable tissues and cells, with the goal of providi
92 of 95 nM) and its predominant expression in
nonexcitable tissues of adult animals.
93 Our findings show how channels act in
nonexcitable tissues to regulate stem cells and may lead
94 studied, including cells from excitable and
nonexcitable tissues, such as the nervous and cardiovasc
95 hysiology in a wide variety of excitable and
nonexcitable tissues.
96 very little expression was detected in other
nonexcitable tissues.
97 in sympathetic neurons than in electrically
nonexcitable tsA201 cells.