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1 en recombination occurs without a protruding nonhomologous 3' tail, the mismatch repair protein Msh2
2 Vinculin binding sites (VBSs) from different nonhomologous actin-binding proteins use conserved helic
3 accharomyces cerevisiae is determined by two nonhomologous alleles, MATa and MATalpha.
4 equent sequence comparison showed that these nonhomologous allergens contained several similar surfac
5  of the IgE to these specific homologous and nonhomologous allergens was corroborated in an animal mo
6 ergens such as the walnut Jug r 6 or that of nonhomologous allergens, as shown for the peanut allerge
7 e demonstrate that regulatory "switching" to nonhomologous alternatives is ubiquitous, occurring acro
8 elices of N15 Cro and lambda Cro make mostly nonhomologous and nonanalogous contacts.
9 retrotransposon-rich regions that are highly nonhomologous between drug- and hemp-type alleles and ar
10                               Interestingly, nonhomologous centromere central-core sequences from S.
11  the clustering and pairwise associations of nonhomologous centromeres that precede stable pairing be
12  Data Bank and identify several sequentially nonhomologous chains that form a Hopf link and a Solomon
13 ere pairing early in meiosis I, even between nonhomologous chromosomes, and clustering of centromeres
14 to pair and instead remained associated with nonhomologous chromosomes.
15 conservation (e.g., mobile gene clusters and nonhomologous cluster alleles), our approach also implic
16  different alleles corresponded to distinct, nonhomologous clusters; and location polymorphisms, in w
17 4 in regulating homologous recombination and nonhomologous DNA end joining-mediated DSB repair in hum
18 making it relevant to the role of ARTEMIS in nonhomologous DNA end joining.
19                                   In humans, nonhomologous DNA end-joining (NHEJ) is the major pathwa
20                                              Nonhomologous DNA end-joining (NHEJ) is the predominant
21                                          The nonhomologous DNA end-joining (NHEJ) pathway is a key me
22 erase (TdT) are important components for the nonhomologous DNA end-joining (NHEJ) pathway.
23                     Moreover, Red1 exhibited nonhomologous DNA end-joining activity, thus revealing a
24 ion of the viral DNA ends by the host cell's nonhomologous DNA end-joining pathway.
25 petition between error-free HR and mutagenic nonhomologous EJ.
26            We now show that knockdown of alt-nonhomologous end joining (alt-NHEJ) components-XRCC1, L
27 PARP1-, LIG3-, and XPF-dependent alternative nonhomologous end joining (alt-NHEJ), which did not gene
28 ombination (HR) and classical or alternative nonhomologous end joining (C-NHEJ versus A-EJ).
29                                    Classical nonhomologous end joining (C-NHEJ) is a major mammalian
30                                    Classical nonhomologous end joining (C-NHEJ) is a major mammalian
31  end-joining (EJ) repair pathways [canonical nonhomologous end joining (C-NHEJ) or alternative end jo
32 double-strand break repair via the canonical nonhomologous end joining (c-NHEJ) pathway.
33  most small deletions occur during classical nonhomologous end joining (C-NHEJ).
34  homologous recombination (HR) and classical nonhomologous end joining (C-NHEJ).
35 oining of DSBs within S regions by classical nonhomologous end joining (C-NHEJ).
36 the IDPs RBM14 is required for the canonical nonhomologous end joining (cNHEJ).
37 predominantly by classical, DNA-PK-dependent nonhomologous end joining (D-NHEJ).
38 e/phosphatase-like factor (APLF) facilitates nonhomologous end joining (NHEJ) and associates with the
39 posure, which is indicative of activation of nonhomologous end joining (NHEJ) and homologous recombin
40 ables the generation of knockout alleles via nonhomologous end joining (NHEJ) and knock-in alleles vi
41            Homologous recombination (HR) and nonhomologous end joining (NHEJ) are two distinct DNA do
42 s used to repair DNA double-strand breaks by nonhomologous end joining (NHEJ) are two related family
43 tection of homologous recombination (HR) and nonhomologous end joining (NHEJ) at the same chromosomal
44                                              Nonhomologous end joining (NHEJ) can effectively resolve
45        We previously showed that the lack of nonhomologous end joining (NHEJ) DNA repair factor ligas
46 between classical (c)- and alternative (alt)-nonhomologous end joining (NHEJ) during DNA double-stran
47 ter assay, we found that COH29 could inhibit nonhomologous end joining (NHEJ) efficiency and that no
48  joining of noncomplementary DNA ends during nonhomologous end joining (NHEJ) for the repair of doubl
49 BRCA1, and deletion of Rif1 suppresses toxic nonhomologous end joining (NHEJ) induced by PARP inhibit
50                                              Nonhomologous end joining (NHEJ) is a recently described
51                A cell-based assay shows that nonhomologous end joining (NHEJ) is compromised in cells
52  by either homology-directed repair (HDR) or nonhomologous end joining (NHEJ) is tightly regulated.
53  double-strand breaks (DSBs) are repaired by nonhomologous end joining (NHEJ) or homologous recombina
54 f tools for Cas9-mediated genome editing via nonhomologous end joining (NHEJ) or homology-directed re
55       Transcriptional up-regulation of major nonhomologous end joining (NHEJ) pathway genes KU80, X-R
56 DR) mechanisms: the dominant but error-prone nonhomologous end joining (NHEJ) pathway, and the less-f
57  in mre-11(iow1) mutants are repaired by the nonhomologous end joining (NHEJ) pathway, as removing NH
58 lso suppress inappropriate activation of the nonhomologous end joining (NHEJ) pathway.
59 A breaks are ultimately resolved through the nonhomologous end joining (NHEJ) pathway.
60 ation repair and antagonizes 53BP1-dependent nonhomologous end joining (NHEJ) pathway.
61 r of double strand breaks (DSBs) through the nonhomologous end joining (NHEJ) pathway.
62 NA-dependent protein kinase (DNA-PK) and the nonhomologous end joining (NHEJ) repair pathway are intr
63 omote KSHV replication, proteins involved in nonhomologous end joining (NHEJ) repair restrict amplifi
64  of the DNA-PK enzyme, which are involved in nonhomologous end joining (NHEJ) repair, enhance amplifi
65                                              Nonhomologous end joining (NHEJ) repairs chromosome brea
66 ylation in homologous recombination (HR) and nonhomologous end joining (NHEJ) through the investigati
67                        Many bacteria rely on nonhomologous end joining (NHEJ) when only a single copy
68                                              Nonhomologous end joining (NHEJ), a major pathway of DNA
69 he efficiency of these methods is limited by nonhomologous end joining (NHEJ), an alternative DNA rep
70 pair systems: homologous recombination (HR), nonhomologous end joining (NHEJ), and single-strand anne
71 ntify DNA-PKcs complex proteins that mediate nonhomologous end joining (NHEJ), as TRIP13-binding part
72 knockout cells and organisms via error-prone nonhomologous end joining (NHEJ), but the efficiency of
73 on, which is almost exclusively dependent on nonhomologous end joining (NHEJ), CSR can occur in NHEJ-
74 ocalized sequence changes through inaccurate nonhomologous end joining (NHEJ), often leading to gene
75 subunit (DNA-PKcs) is a central component of nonhomologous end joining (NHEJ), repairing DNA double-s
76 nation (HR) but can inhibit normal repair by nonhomologous end joining (NHEJ), the main DSB repair pa
77 aired both homologous recombination (HR) and nonhomologous end joining (NHEJ), the two major DSB repa
78 early phosphorylations promote initiation of nonhomologous end joining (NHEJ), whereas ABCDE phosphor
79 rohomology-mediated end joining (MMEJ)-, and nonhomologous end joining (NHEJ)-based strategies for th
80 t that TDP-43 is a critical component of the nonhomologous end joining (NHEJ)-mediated DNA double-str
81                                              Nonhomologous end joining (NHEJ)-mediated repair of DSBs
82 tein kinase-catalytic subunit (DNA-PKcs) and nonhomologous end joining (NHEJ).
83 ensitivity is rescued by the inactivation of nonhomologous end joining (NHEJ).
84  capacity for classical, versus alternative, nonhomologous end joining (NHEJ).
85 r: the homologous recombination (HR) and the nonhomologous end joining (NHEJ).
86 B by either homologous recombination (HR) or nonhomologous end joining (NHEJ).
87 ct DNA ends from resection and thereby favor nonhomologous end joining (NHEJ).
88 epaired via homologous recombination (HR) or nonhomologous end joining (NHEJ).
89 r, increasing the involvement of error-prone nonhomologous end joining (NHEJ).
90 Bs) and their adequate recombination through nonhomologous end joining (NHEJ).
91 n revealed that OA-NO(2) inhibits HR and not nonhomologous end joining (NHEJ).
92 ) repair for the majority process of precise nonhomologous end joining (NHEJ).
93 d DNA double-stranded break (DSB) repair via nonhomologous end joining and homologous recombination.
94 volved in DNA replication and repair by both nonhomologous end joining and homologous repair is misre
95 s9 to both nuclei, combined with the lack of nonhomologous end joining and markers for positive selec
96 break produced by Ac excision: footprints by nonhomologous end joining and rearrangements by various
97 atures of the fusion fragments indicate that nonhomologous end joining and/or replication-dependent D
98 t the level of 10-20% of HR when both HR and nonhomologous end joining are available.
99               S-S junctions are joined via a nonhomologous end joining DNA repair process.
100  active cell cycle checkpoints and increased nonhomologous end joining DNA repair, suggesting that pe
101 unnos and the paralog of XRCC4 and XLF, PAXX nonhomologous end joining factor (PAXX).
102 nase catalytic subunit (DNA-PKcs), a classic nonhomologous end joining factor, antagonizes double str
103     One of the major DSBs repair pathways is nonhomologous end joining for which Ku70/80 is essential
104 es four broken DNA ends that are repaired by nonhomologous end joining forming coding and signal join
105  was an indirect effect of the repression of nonhomologous end joining in Sir(-) mutants and that the
106 ion pattern and a potential microhomology or nonhomologous end joining integration mechanism at the s
107 reaks (DSBs) by homologous recombination and nonhomologous end joining is critical for maintaining ge
108 balance between homology-directed repair and nonhomologous end joining is important for neuronal cell
109                                Specifically, nonhomologous end joining is the predominant mechanism u
110                                              Nonhomologous end joining is the primary deoxyribonuclei
111 uence analysis suggested fundamental role of nonhomologous end joining mechanism during eccDNA format
112  Cas9 lesions by homologous recombination or nonhomologous end joining mechanisms can lead to the int
113 he integrity of homology-directed repair and nonhomologous end joining of DNA breaks is impaired in K
114  Several of these genes are also involved in nonhomologous end joining of DNA double-strand break rep
115 breaks (DSBs) are generally repaired through nonhomologous end joining or homologous recombination.
116 ble-strand breaks that stimulate error-prone nonhomologous end joining or homology-directed repair at
117 ts must be removed to allow repair by either nonhomologous end joining or homology-directed repair.
118 ecombination but instead form most often via nonhomologous end joining or microhomology-mediated brea
119    Repair of DNA double-strand breaks by the nonhomologous end joining pathway is central for proper
120 genetic disruption strategies relying on the nonhomologous end joining pathway may induce compensator
121 rm of DNA damage, are mainly repaired by the nonhomologous end joining pathway, which relies on DNA-P
122 and an increase in repair by the alternative nonhomologous end joining pathway.
123 ltaAID is impaired in its ability to recruit nonhomologous end joining repair factors, resulting in a
124 , increased expression of DSB initiating and nonhomologous end joining repair machinery in newborn ne
125 n inhibitor of the DNA-PK kinase crucial for nonhomologous end joining repair of DNA DSBs, and BRCA2-
126             Double strand breaks may trigger nonhomologous end joining repair, leading to frameshift
127 s in CML progenitors was mediated by classic nonhomologous end joining repair.
128                                              Nonhomologous end joining repairs DNA double-strand brea
129 ly triggers an activation of the error-prone nonhomologous end joining response.
130  also find that MMEJ compensates for loss of nonhomologous end joining to repair rereplication DSBs i
131 lar domain, including Tel1 (ATM) activation, nonhomologous end joining, and DNA double-strand break e
132 s switch recombination, two events requiring nonhomologous end joining, at levels comparable to Atm(-
133 uggests that, in addition to its key role in nonhomologous end joining, DNA-PKcs also acts in concert
134 pair, mismatch repair, base excision repair, nonhomologous end joining, homologous recombination, and
135 gD, an ATP-dependent DNA ligase dedicated to nonhomologous end joining, in complexes with ATP that hi
136                                           In nonhomologous end joining, Ku loads onto broken DNA via
137 d that, distinct from Ku-dependent classical nonhomologous end joining, MMEJ--even with very limited
138 smatch, nucleotide excision, Fanconi anemia, nonhomologous end joining, or translesion synthesis repa
139 sis during DNA double strand break repair by nonhomologous end joining, particularly in nonreplicatin
140 major pathway for Ku-independent alternative nonhomologous end joining, which contributes to chromoso
141 mage accumulation, with a repair bias toward nonhomologous end joining.
142 n through the evolutionary loss of classical nonhomologous end joining.
143 imulate DNA double-strand break ligation via nonhomologous end joining.
144 by directly binding and activating DNA-PK in nonhomologous end joining.
145 ith disruptive mutations in genes related to nonhomologous end joining.
146 , originally identified in patients, impedes nonhomologous end joining.
147 ithin a population, which typically arise by nonhomologous end joining.
148  insertions/deletions (indels) via mutagenic nonhomologous end joining.
149 ogical functions in base excision repair and nonhomologous end joining.
150  beta (Pol beta) as mediators of alternative nonhomologous end-joining (Alt-NHEJ) events, including c
151 l components of the error-prone, alternative nonhomologous end-joining (alt-NHEJ) pathway.
152                                    Classical nonhomologous end-joining (C-NHEJ) and alternative end-j
153                                  The classic nonhomologous end-joining (c-NHEJ) pathway is largely re
154 NA polymerase II (RNAP II) and the classical nonhomologous end-joining (C-NHEJ) proteins, including P
155 translocations in wild-type versus classical nonhomologous end-joining (C-NHEJ)-deficient NSPCs revea
156 catalytic subunit (DNA-PKcs), is a classical nonhomologous end-joining (cNHEJ) factor.
157                                The classical nonhomologous end-joining (cNHEJ) pathway is a major DNA
158  of CSR is usually mediated by the classical nonhomologous end-joining (cNHEJ) pathway.
159 at are initiated on free DNA ends: classical nonhomologous end-joining (NHEJ) and ATM-dependent DNA d
160 choice between two main DSB repair pathways, nonhomologous end-joining (NHEJ) and homologous recombin
161 lved several DSB repair mechanisms, of which nonhomologous end-joining (NHEJ) and homologous recombin
162 he HPV16 E7 oncoprotein suppresses canonical nonhomologous end-joining (NHEJ) and promotes error-pron
163 esection in G1, and thereby favors repair by nonhomologous end-joining (NHEJ) as opposed to homologou
164  DNA damage repair by promoting Ku-dependent nonhomologous end-joining (NHEJ) at the expense of homol
165 ing kinases and that it is not suppressed by nonhomologous end-joining (NHEJ) components, arguing tha
166 ugh conditional deletion of TRF1 and TRF2 in nonhomologous end-joining (NHEJ) deficient cells.
167 gase 4 (LIG4), NHEJ1, and NBS1 involving the nonhomologous end-joining (NHEJ) DNA repair pathway resu
168  53BP1 gene silencing induces defects in the nonhomologous end-joining (NHEJ) DNA repair pathway.
169                 Hypomorphic mutations in the nonhomologous end-joining (NHEJ) DNA repair protein DNA
170  B cells from mice and ICF2 patients affects nonhomologous end-joining (NHEJ) during immunoglobulin c
171   Such mutant alleles result presumably from nonhomologous end-joining (NHEJ) events before the segre
172                                Ku70, a known nonhomologous end-joining (NHEJ) factor, also functions
173 t DNA ligation in vitro and assembly of core nonhomologous end-joining (NHEJ) factors on damaged chro
174                         Somatic mutations in nonhomologous end-joining (NHEJ) genes (DCLRE1C/ARTEMIS,
175 pression of key homologous recombination and nonhomologous end-joining (NHEJ) genes.
176                                              Nonhomologous end-joining (NHEJ) is a key pathway for ef
177                                              Nonhomologous end-joining (NHEJ) is a major DNA double-s
178                                              Nonhomologous end-joining (NHEJ) is a major repair pathw
179                                              Nonhomologous end-joining (NHEJ) is the major DNA double
180                Furthermore, coexpression the nonhomologous end-joining (NHEJ) machinery from the clos
181      Subsequent repair of this break via the nonhomologous end-joining (NHEJ) or homology-directed re
182 ependent large deletions are products of the nonhomologous end-joining (NHEJ) pathway and require Top
183  triggering end resection and inhibiting the nonhomologous end-joining (NHEJ) pathway in G1 phase.
184 i, suggesting that HR is compromised and the nonhomologous end-joining (NHEJ) pathway is elicited to
185                                          The nonhomologous end-joining (NHEJ) pathway is essential fo
186                                          The nonhomologous end-joining (NHEJ) pathway is the primary
187 y demonstrated that HSCs use the error-prone nonhomologous end-joining (NHEJ) pathway of DNA repair t
188 factor (XLF/Cernunnos) is a component of the nonhomologous end-joining (NHEJ) pathway of double-stran
189 u (Polmu) participates in DSB repair via the nonhomologous end-joining (NHEJ) pathway, by filling sma
190 d mutability, and abnormal regulation of the nonhomologous end-joining (NHEJ) pathway.
191                                              Nonhomologous end-joining (NHEJ) pathways repair DNA dou
192 s have proposed the participation of various nonhomologous end-joining (NHEJ) pathways.
193 ither by homologous recombination (HR) or by nonhomologous end-joining (NHEJ) pathways.
194 are suppressed by genetically eliminating Ku nonhomologous end-joining (NHEJ) protein, indicating tha
195          Here we show that FBXW7 facilitates nonhomologous end-joining (NHEJ) repair and that FBXW7 d
196 -protein kinase (DNA-PK) phosphorylation and nonhomologous end-joining (NHEJ) repair efficiency and f
197  of mice lacking Lig4, a ligase required for nonhomologous end-joining (NHEJ) repair of DNA double-st
198   Here, we show that Ku70, a core protein of nonhomologous end-joining (NHEJ) repair pathway, can dir
199 ization of homologous recombination (HR) and nonhomologous end-joining (NHEJ) repair pathways but exc
200 d impaired homologous recombination (HR) and nonhomologous end-joining (NHEJ) repair pathways, with d
201  and exonuclease activities regulate DSBR by nonhomologous end-joining (NHEJ) versus homologous recom
202  revealed 47 to 58% of reads as repaired via nonhomologous end-joining (NHEJ) with deletions and/or s
203                                     However, nonhomologous end-joining (NHEJ), an error-prone repair,
204  DSBs, namely homologous recombination (HR), nonhomologous end-joining (NHEJ), and microhomology-medi
205 cle-invasive bladder tumors are defective in nonhomologous end-joining (NHEJ), and this phenotype may
206 tein TRF2 by promoting their mobility, their nonhomologous end-joining (NHEJ), and, as we show here,
207 ence of DNA damage checkpoint components and nonhomologous end-joining (NHEJ), but not homologous rec
208 ir by both homologous recombination (HR) and nonhomologous end-joining (NHEJ), causes accumulation of
209 ment in the second major DSB repair pathway, nonhomologous end-joining (NHEJ), remains controversial.
210  occur in human cancers display hallmarks of nonhomologous end-joining (NHEJ).
211 ase (DNA-PK), a critical enzyme required for nonhomologous end-joining (NHEJ).
212 ted an increased number of DSBs processed by nonhomologous end-joining (NHEJ).
213 NA double-strand break repair pathway termed nonhomologous end-joining (NHEJ).
214  recruited to DNA damage sites to facilitate nonhomologous end-joining (NHEJ).
215 eaks, to recapitulate DSB repair via MMEJ or nonhomologous end-joining (NHEJ).
216 NA repair, which is accompanied by increased nonhomologous end-joining activity.
217        IgD CSR occurred via both alternative nonhomologous end-joining and homologous recombination p
218 ce between the two major DSB repair pathways-nonhomologous end-joining and homologous recombination r
219 se to DNA damage, which suppresses repair by nonhomologous end-joining and homologous recombination.
220 in the repair of DNA double strand breaks by nonhomologous end-joining and in the signaling of DNA da
221 fore, UbcH7-depleted cells display increased nonhomologous end-joining and reduced homologous recombi
222 y, which prevents end resection and promotes nonhomologous end-joining and therefore directly compete
223          While gene conversion and classical nonhomologous end-joining are the most physiologically p
224      PAXX was identified recently as a novel nonhomologous end-joining DNA repair factor in human cel
225        DNA-PKcs plays a critical role in the nonhomologous end-joining DNA repair pathway and provide
226 A damage sites and play a functional role in nonhomologous end-joining DNA repair.
227 and breaks in part through antagonism of the nonhomologous end-joining factor 53BP1.
228 SPR/SpCas9 system was used to knock out, via nonhomologous end-joining genome repair, the 4'OMT2 in o
229 melphalan sensitivity of the cells, with the nonhomologous end-joining inhibitor SCR7 showing the str
230 but LIG4-dependent manner, establishing that nonhomologous end-joining maintains rDNA integrity durin
231 ons by rejoining broken ends, among them the nonhomologous end-joining mechanism that utilizes a DNA
232 ologous recombination (HR) and the classical nonhomologous end-joining pathway (cNHEJ).
233 ir of DNA double-strand breaks (DSBs) by the nonhomologous end-joining pathway (NHEJ) is important no
234 d by the DCLRE1C gene, is a component of the nonhomologous end-joining pathway and participates in ha
235 lications of DNA sequence that depend on the nonhomologous end-joining pathway of DSB repair.
236 ely because they mainly used the error-prone nonhomologous end-joining pathway to repair DSBs.
237               Ku70 is a key component of the nonhomologous end-joining pathway, which is the major pa
238  these double-strand breaks by the classical nonhomologous end-joining pathway.
239 ich was necessary for DSB repair through the nonhomologous end-joining pathway.
240 pair, single strand break repair, and backup nonhomologous end-joining pathway.
241 ouble-stranded breaks (DSBs) mediated by the nonhomologous end-joining pathway.
242 DNA repair complexes that participate in the nonhomologous end-joining pathway.
243 ubunit, essential DNA repair proteins in the nonhomologous end-joining pathway.
244 NA double strand breaks that are repaired by nonhomologous end-joining pathways.
245 psis but formed in the absence of functional nonhomologous end-joining pathways.
246 thways of homologous recombination and other nonhomologous end-joining processes.
247 re partially disassembled around DSBs during nonhomologous end-joining repair in G1-arrested mammalia
248  can be efficiently ligated by the classical nonhomologous end-joining repair pathway (c-NHEJ), regen
249 ion and can ultimately become substrates for nonhomologous end-joining repair, leading to large-scale
250 HR)-mediated repair and, to a lesser extent, nonhomologous end-joining repair.
251 irectly to the DNA break and is required for nonhomologous end-joining repair.
252 s of chromothripsis in TCC-UB is mediated by nonhomologous end-joining using kilobase, rather than me
253 bination (HR) and repair efficiency, but not nonhomologous end-joining, and decreased the formation o
254 on and DNA-dependent protein kinase-mediated nonhomologous end-joining, and, when combined with olapa
255 ir through both homologous recombination and nonhomologous end-joining, implicating FUS as an upstrea
256 me of these rearrangements appear to involve nonhomologous end-joining, many must have involved mecha
257 h as KU70 and LIG4 (both involved in classic nonhomologous end-joining, NHEJ) and SMC6B (involved in
258 nt protein kinase-mediated (DNA-PK-mediated) nonhomologous end-joining, whereas DNA repair pathways m
259 nucleus" stage embryos led to high-frequency nonhomologous end-joining-mediated, mutagenic lesions in
260 d that loss of MSI1 reduces the frequency of nonhomologous end-joining.
261 akpoint of proteins that promote error-prone nonhomologous end-joining.
262 defects that could be reversed by inhibiting nonhomologous end-joining.
263 ted double-strand-break creation followed by nonhomologous end-joining.
264 subsequently ligated by DNA Ligase IV during Nonhomologous end-joining.
265 ir of DNA double-strand breaks, facilitating nonhomologous end-joining.
266 ays, homology-dependent repair and classical nonhomologous end-joining.
267                Thus, convergent evolution of nonhomologous enzymes using the 2-His-1-carboxylate faci
268 at constrain coexpression within clusters of nonhomologous eukaryotic genes and suggest that gene clu
269               In all regions, stimulation of nonhomologous fingers of the two hands elicited higher B
270 tion of residues in a homologous region from nonhomologous flanking residues).
271  coregulated, and (3) physical clustering of nonhomologous genes that encode components of secondary
272 zes the cytokine hbetaNGF was grafted into a nonhomologous human germ line V region.
273 ol, which is sufficient to protect mice from nonhomologous IAV challenge.
274 rovide substantial protection against lethal nonhomologous IAV challenge.
275  The three regions were each bordered by two nonhomologous integrase attachment (att) sites, which to
276         Likewise, Pdr6 recognizes eIF5A in a nonhomologous manner compared with the mammalian eIF5A-e
277 enes, the mechanistic logic of clustering of nonhomologous monocistronic genes in eukaryotes is not i
278 NA mixed with genomic fragments assembled by nonhomologous or microhomology-mediated joining.
279 eV F TM constructs were coexpressed with the nonhomologous parainfluenza virus 5 (PIV5) fusion protei
280  of previous studies originally based on the nonhomologous partitioning of rodent cingulate cortex.
281 ailure and synapsis with both homologous and nonhomologous partners ("chromosome tangles").
282 n viruses distinguish between homologous and nonhomologous partners during sexual RNA replication?
283 hout necessitating degenerate recognition of nonhomologous peptides.
284                               The arrays are nonhomologous, physically unlinked in mammals, and encod
285 ry modes by acquiring functionally divergent nonhomologous promoter regions.
286 onal Cu(A) center designed in a structurally nonhomologous protein, cytochrome c peroxidase (CcP), by
287 al sources with structural homology and also nonhomologous proteins arising from the same biological
288 early phases of replication was a mixture of nonhomologous recombinants.
289 suggesting that the insert was the result of nonhomologous recombination with the host genome.
290 umerous rearrangements due to homologous and nonhomologous recombination, deletions and segmental dup
291 ntrol homologous recombination-dependent and nonhomologous recombination-dependent amplification path
292  eliminated in theory by avoiding ubiquitous nonhomologous recombination.
293 odels of CGR genesis and strongly argue that nonhomologous repair of concurrently arising DNA double-
294 ignments erroneously extend through flanking nonhomologous residues.
295 ential to distinguish between homologous and nonhomologous RNA templates during sexual RNA replicatio
296 ologies (homologous subsequences embedded in nonhomologous sequence).
297  reading frame 1 of the viral genome at five nonhomologous sites, releasing six nonstructural protein
298 f ECM from fully developed native tissues at nonhomologous sites.
299     Similarly, we also observe that the more nonhomologous subunits that assemble together within a c
300 r, when the DSB end contains a 3' protruding nonhomologous tail, Msh2 promotes the rejection of misma

 
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