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1 cell-specific injury or from the pancreas of nonobese diabetic mice.
2 ferred 5 x 10(6) splenocytes from 6-week-old nonobese diabetic mice.
3 oimmune diabetes developing spontaneously in nonobese diabetic mice.
4 otocin and during development of diabetes in nonobese diabetic mice.
5 we examined the role of IL-23 in diabetes in nonobese diabetic mice.
6 tolerance and the development of diabetes in nonobese diabetic mice.
7 ng grafted and endogenous islets in diabetic nonobese diabetic mice.
8  corticosterone were investigated using male nonobese diabetic mice.
9 antigen-specific Tregs from autoimmune-prone nonobese diabetic mice.
10 the onset and severity of type 1 diabetes in nonobese diabetic mice.
11 independent in T cells from autoimmune-prone nonobese diabetic mice.
12 a small population of Ag-specific T cells in nonobese diabetic mice.
13 uction protocol is ineffective in autoimmune nonobese diabetic mice.
14 nize these peptides in type 1 diabetes-prone nonobese diabetic mice.
15 ion protein (LTbetaR-Ig) was administered to nonobese diabetic mice.
16 level of EF-1alpha mRNA was also observed in nonobese diabetic mice.
17 II molecule DQ in humans and to MHC I-Ag7 in nonobese diabetic mice.
18 e sclerosis, and prevents type 1 diabetes in nonobese diabetic mice.
19 tes mellitus by the CD4+ Th1 clone BDC2.5 to nonobese diabetic mice.
20 duced a long-term diabetes-free condition in nonobese diabetic mice.
21 markers were observed in islets derived from nonobese diabetic mice.
22  NLPs ameliorated chronic progressive EAE in nonobese diabetic mice, a model which resembles some asp
23                                      In NOD (nonobese diabetic) mice, a model of autoimmune diabetes,
24 -specific expression of TNFalpha in neonatal nonobese diabetic mice accelerated diabetes.
25 tigen-presenting cells, to treat diabetes in nonobese diabetic mice after disease onset.
26 vation and whose adoptive transfer protected nonobese diabetic mice against type 1 diabetes (T1D).
27                                   Thus, male nonobese diabetic mice allow for translational studies o
28                                              Nonobese diabetic mice also have salivary gland inflamma
29 on-associated lymphoid neogenesis, including nonobese diabetic mice, also exhibit concomitant express
30 experimental autoimmune encephalomyelitis in nonobese diabetic mice, an experimental model that resem
31             Gastric emptying was measured in nonobese diabetic mice and correlated with levels of HO-
32 cific MHC class II genes, such as I-A(g7) in nonobese diabetic mice and HLA-DQ8 in humans, suggests t
33  work from one laboratory has shown, in both nonobese diabetic mice and humans, an association betwee
34 oth muscle, and neurons were investigated in nonobese diabetic mice and organotypic cultures by immun
35 m-3 pathway blockade accelerated diabetes in nonobese diabetic mice and prevented acquisition of tran
36 completely prevented diabetes development in nonobese diabetic mice and strongly reduced its incidenc
37                                              Nonobese diabetic mice are a well-known model for human
38                              Irradiated SCID/nonobese diabetic mice are injected with a tumor of huma
39 chanisms underlying this observation, female nonobese diabetic mice at 4, 8, and 12 wk of age were gi
40 amounts of insulin (ins) to cure diabetes in nonobese diabetic mice but, unlike transplanted islets,
41  reverses established autoimmune diabetes in nonobese diabetic mice by restoring self-tolerance, and
42                             Autoimmune-prone nonobese diabetic mice deficient for B7-2 spontaneously
43                                   Studies of nonobese diabetic mice demonstrated that disruption of t
44 oantibody production, and type 1 diabetes in nonobese diabetic mice, despite normal tertiary lymphoid
45 ogenesis of diabetes, we generated NOD mice (nonobese diabetic mice developing spontaneous autoimmune
46             In adult TCR (BDC2.5) transgenic nonobese diabetic mice, DNA microarray analysis of globa
47 nce of type 1 diabetes (T1D) is decreased in nonobese diabetic mice expressing the complete cassette
48                              Macrophage from nonobese diabetic mice failed to provide signals necessa
49                                              Nonobese diabetic mice fed transformed potato tuber tiss
50  Fas (lpr) or FasL (gld) completely protects nonobese diabetic mice from autoimmune diabetes but also
51  by the beta cell have been shown to protect nonobese diabetic mice from developing diabetes.
52  isolated from the pancreatic infiltrates of nonobese diabetic mice have been shown to recognize epit
53       We show that autoimmune diabetes-prone nonobese diabetic mice have high levels of CD40(+)CD4(+)
54 by the unique class II molecule expressed by nonobese diabetic mice (I-Ag7).
55            In an alternative diabetes model, nonobese diabetic mice, IL15/IL-15Ralpha expression was
56  MM model in severe combined immunodeficient/nonobese diabetic mice in which diffuse MM lesions devel
57 n B13 autoantibodies in young diabetes-prone nonobese diabetic mice is associated with reduced inflam
58 ze that resistance to tolerance induction in nonobese diabetic mice is due to the presence of memory
59 ent population of pathogenic CD8+ T cells in nonobese diabetic mice is islet-specific glucose-6-phosp
60 n a model of passive transfer of diabetes in nonobese diabetic mice, lytic IL-2/Fc, but not nonlytic
61 ia was created by femoral artery ligation in nonobese diabetic mice (NOD mice, n = 20) and in control
62                     In an attempt to protect nonobese diabetic mice (NOD) from autoimmune diabetes, w
63 T cells at the earliest stages of disease in nonobese diabetic mice (NOD).
64 nsulin-dependent diabetes mellitus (IDDM) in nonobese diabetic mice (NOD).
65           CD4(+) T cells from JNK2-deficient nonobese diabetic mice produced less IFN-gamma but signi
66 sion of tumor necrosis factor (TNF)-alpha in nonobese diabetic mice promotes diabetes by provoking is
67 tes-inducing CD4 T cell clones isolated from nonobese diabetic mice recognize epitopes formed by cova
68 ocking the SDF-1alpha/CXCR4 axis in IFNgamma-nonobese diabetic mice resulted in diminished proliferat
69 d leukemic cell lines into immunocompromised nonobese diabetic mice resulted in significant elevation
70 enic intermediate lobe tissues into diabetic nonobese diabetic mice resulted in the restoration of ne
71                                              Nonobese diabetic mice spontaneously develop diabetes th
72            We found that CD4(+) T cells from nonobese diabetic mice spontaneously differentiate into
73                 Investigations of humans and nonobese diabetic mice suggest that proinsulin and/or a
74 e 1 or type 2 diabetes, and in the islets of nonobese diabetic mice that have developed insulitis or
75 pe recognized by pathogenic T lymphocytes in nonobese diabetic mice, the animal model for type 1 diab
76 sed Fah(-/-), Rag2(-/-), Il2r(-/-) mice with nonobese diabetic mice to create FRGN mice, whose livers
77  (Treg cells) during diabetes progression in nonobese diabetic mice was investigated to determine whe
78 ) islet transplantation into immunodeficient nonobese diabetic mice, we investigated whether A2-CAR T
79                   In spontaneous diabetes in nonobese diabetic mice, we were able to stop the ongoing
80                                   Transgenic nonobese diabetic mice were created in which insulin exp
81                                      We used nonobese diabetic mice, which develop autoimmune-mediate
82 ccelerated BDC2.5 T-cell receptor transgenic nonobese diabetic mice, which experience development of
83                  Early systemic treatment of nonobese diabetic mice with high doses of recombinant ad
84 xpression of NMI was detected in islets from nonobese diabetic mice with insulitis and in rodent or h
85                                 Treatment of nonobese diabetic mice with L11 from 1 to 4 or 8 to 12 w
86 oliferation using in vivo teratoma assays in nonobese diabetic mice with severe combined immunodefici
87                                           In nonobese diabetic mice with uncontrolled type 1 diabetes