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1 f the transgene has not resulted in complete nonresponsiveness.
2 s in the form of a postactivational phase of nonresponsiveness.
3 ring long-term remission signals immunologic nonresponsiveness.
4  appeared to be limited by induction of IL-7 nonresponsiveness.
5 of IL-17F greater than 200 pg/mL may predict nonresponsiveness.
6 en-transgenic RBCs also demonstrated induced nonresponsiveness.
7 g molecule equol was consistent with overall nonresponsiveness.
8 o determine if oral bioavailability mediates nonresponsiveness.
9  cancer is one of the major factors of tumor nonresponsiveness.
10  result in CD8 T-cell deletion or functional nonresponsiveness.
11 o define sustained periods of donor-specific nonresponsiveness.
12 n Ags typically induces Ag-specific systemic nonresponsiveness.
13 dy responses may play in the observed immune nonresponsiveness.
14               Importantly, WT TP53 predicted nonresponsiveness (7 of 8 models).
15 of BCR-ligand recognition can lead to B cell nonresponsiveness, activation, or inhibition.
16                      This activation-induced nonresponsiveness (AINR) is not simply a consequence of
17 ollowing restimulation, they rapidly develop nonresponsiveness and exhibit elevated activation-induce
18 y lead to an IL-10-induced form of anergy or nonresponsiveness and generation of the recently charact
19 donor animals could induce a state of T-cell nonresponsiveness and prevent the development of graft-v
20 PCs from RA patients displayed growth factor nonresponsiveness and sluggish cell cycle progression; m
21  might transition between responsiveness and nonresponsiveness, and between different modes of gradie
22 s associated with reversal of tumor-mediated nonresponsiveness/anergy as well as establishment of lon
23 elp at priming results in long-term antibody nonresponsiveness, antibody responses can be induced by
24 to therapy, and molecular mechanisms for the nonresponsiveness are unknown.
25  the mechanisms of immune responsiveness, or nonresponsiveness, are governed by the migration and loc
26                In addition, parasite-induced nonresponsiveness, as measured by TNF-alpha production,
27                        In conclusion, T-cell nonresponsiveness because of active suppression mediated
28  involvement of the HLA-DRB1 gene in vaccine nonresponsiveness but not altered susceptibility to vira
29 However, the ability to overcome immunologic nonresponsiveness by targeting poorly immunogenic Ags to
30 man trials suggest that tolerance (sustained nonresponsiveness) can be re-established in a subset of
31                  Anergy is a state of T-cell nonresponsiveness characterized by downregulated IL-2 pr
32             Elucidation of the mechanisms of nonresponsiveness (clonal exhaustion-deletion and immune
33  achieved a transient state of immunological nonresponsiveness correlated with higher titers of the I
34 ive potential per Tg+ cell demonstrates that nonresponsiveness due to feeding Ag results in the induc
35   We conclude that immunological pathways of nonresponsiveness follow different patterns depending bo
36 o an active induction of an antigen-specific nonresponsiveness (i.e. immunological tolerance).
37 ormally results in local and systemic immune nonresponsiveness in a process termed oral tolerance.
38  in T cells is sufficient to induce cytokine nonresponsiveness in both a T cell clone and naive prima
39 ers, indicating a prior state of immunologic nonresponsiveness in the context of AAV gene therapy.
40 hematopoietic chimeras showed donor-specific nonresponsiveness in the mixed lymphocyte reaction, lack
41 gen presentation by epithelial cells induced nonresponsiveness in the T cell clones.
42  ability of CTLA4Ig to lead to long-standing nonresponsiveness in this model depends on the nature (i
43 rus-mediated upregulation induced a state of nonresponsiveness in uninfected HIV-specific T cells.
44  asystole (after approximately 30 minutes of nonresponsiveness) in 1 case.
45 mage and repair interspersed with periods of nonresponsiveness indicative of a refractory period.
46                                       T-cell nonresponsiveness is a critical factor in immune escape
47                                In this case, nonresponsiveness is accompanied by the induction of p21
48  In this study, we demonstrate that cytokine nonresponsiveness is accompanied by the induction of the
49 rrent study, we aimed to investigate whether nonresponsiveness is an Ag/vaccine-specific phenomenon a
50                           Low responsiveness/nonresponsiveness is characterized by an insufficient im
51 ellular and molecular basis for the observed nonresponsiveness is not fully understood.
52 ve continued proliferation of AINR cells and nonresponsiveness is reversed within 1-2 days so that Ag
53                               Donor-specific nonresponsiveness is transient in most patients, and ser
54                                     However, nonresponsiveness is widely recognized and is related to
55 a suggest that anti-CD3 mAb-induced cytokine nonresponsiveness may be a consequence of hyperactivatio
56 607 T/T/APOE varepsilon3 was associated with nonresponsiveness (mean +/- SEM: +0.09 +/- 0.08 mmol/L,
57  (P2 and P3) are sufficient to induce immune nonresponsiveness (median survival time >237 days) to ca
58 flora or food Ags, it must induce a state of nonresponsiveness (mucosal tolerance).
59 ct of clopidogrel dosing on the incidence of nonresponsiveness (NR) and high post-treatment platelet
60                                         This nonresponsiveness occurs because of a lack of expression
61 sents a promising approach to overcoming the nonresponsiveness of certain cancer patients to this sel
62      To gain more insight into the purported nonresponsiveness of human liver cells to peroxisome vol
63                                          The nonresponsiveness of MV-infected human lymphocytes to mi
64 roteolytic degradation may contribute to the nonresponsiveness of patients with IBD to anti-TNF agent
65 fine the structural basis for the allosteric nonresponsiveness of sCPS and thereby provide insight in
66 rast to wild-type HSCs (WT HSCs), a complete nonresponsiveness of Tet2-/- HSCs and HPCs to changes in
67               Immune tolerance is defined as nonresponsiveness of the adaptive immune system to antig
68 f patients at high or low risk for treatment nonresponsiveness or death, and they may provide opportu
69               Identification of antiplatelet nonresponsiveness or hyporesponsiveness is highly test s
70 a and TGFbeta signaling, such that tamoxifen nonresponsiveness or resistance in breast cancer might i
71            The ability to trigger a state of nonresponsiveness represents a unique MHC-independent me
72 is the induction of T cell anergy, a form of nonresponsiveness resulting from incomplete T cell activ
73 ice congenic for slow acetylation and/or Ahr-nonresponsiveness; some groups were pretreated with beta
74 targeting to CD64 can overcome immunological nonresponsiveness to a weak immunogen.
75                                We found that nonresponsiveness to anti-integrin biologic therapies in
76 before treatment is strongly associated with nonresponsiveness to anti-TNF therapy.
77 ntervention findings associated with primary nonresponsiveness to antibiotics in adults with uncompli
78  with proinflammatory cytokine increases and nonresponsiveness to antiinfluenza vaccine in the elderl
79 CL10 may be a predictor of responsiveness or nonresponsiveness to antiviral therapy with pegylated in
80 he MAP kinases, ERKs 1 and 2, and consequent nonresponsiveness to AP-1 activation.
81 ariectomized females show no change in their nonresponsiveness to caspase-1/4 inhibition, orchiectomi
82 ti-CD40L monoclonal antibodies suggests that nonresponsiveness to cell membrane-bound antigen (e.g.,
83 hypermethylation is associated with clinical nonresponsiveness to chemotherapy in colorectal cancer.
84                 Ticagrelor therapy overcomes nonresponsiveness to clopidogrel, and its antiplatelet e
85 abolites could prevent development of T cell nonresponsiveness to cognate Ag.
86                       Importantly, GC T cell nonresponsiveness to CXCL12 correlated with high ex vivo
87 f oral tolerance in sustaining immunological nonresponsiveness to food Ags, our current understanding
88 me that high IDO activity is associated with nonresponsiveness to food allergens despite allergen sen
89 ased immune-checkpoint expression, predicted nonresponsiveness to immune-checkpoint blockade, and was
90           The factors that contribute to the nonresponsiveness to ketamine's antidepressant action re
91 -SFK pathway components may in part underlie nonresponsiveness to ketamine's antidepressant action.
92 o induce and maintain Mphi cytokine-specific nonresponsiveness to LPS.
93  positive selection, which could explain the nonresponsiveness to many nonself peptides and could imp
94 ly, CD8+ T cells from tolerant mice transfer nonresponsiveness to naive syngeneic recipients.
95 em to gain an insight into the mechanisms of nonresponsiveness to PSA, ultimately leading to strategi
96 lls lowered ped/pea-15 expression and caused nonresponsiveness to rosiglitazone, although c-jun overe
97 rgy, as CLL cells exhibit variable levels of nonresponsiveness to surface IgM stimulation that is rev
98 opment of a vascular crisis characterized by nonresponsiveness to sympathetic vasoconstrictor agents
99 g genes by miR-155 likely contributes to the nonresponsiveness to TGFbeta during SIV infection and ma
100 ositive CD8(+) T cells demonstrated profound nonresponsiveness to the specific peptide, whereas nitro
101 ne checkpoint blockade (ICB) correlated with nonresponsiveness to therapy and poor clinical outcome.
102 serum of people with RRMS is associated with nonresponsiveness to therapy with IFN-beta.
103 s have been offered to explain this apparent nonresponsiveness to warming, including the influence of
104 ings of these drugs include the induction of nonresponsiveness upon repeated use and the expansion of
105 mmed for signal- and stage-specific "nuclear nonresponsiveness" upon encounter with self-Ags.
106             Concurrent with the induction of nonresponsiveness, virus-specific cells that retained fu
107                          The rate of aspirin nonresponsiveness was 15.8%, 8.1%, and 52.8% for plain a
108                 Mucosal, but not peripheral, nonresponsiveness was abrogated by the inclusion of a ne
109                                              Nonresponsiveness was defined as <10% absolute change in
110                                      Aspirin nonresponsiveness was defined as a level of residual ser
111                                              Nonresponsiveness was defined as failure to mount an HAI
112                                              Nonresponsiveness was lower after 600 mg compared to the
113 pecific peripheral cell-mediated and humoral nonresponsiveness was maintained in both Stat 4-/- and S
114                    This donor antigen-linked nonresponsiveness was observed in four other patients wh
115                                         This nonresponsiveness was reversed by an anti-TGF-beta1-neut
116 nical isolates excluded the possibility that nonresponsiveness was the result of mutation(s) in L1.
117 vironment in the induction of orally induced nonresponsiveness, we attempted to induce tolerance to O
118  HLA-DR subtypes associated with hepatitis B nonresponsiveness were overrepresented in this group, an
119 al MHC class II molecules that induce T cell nonresponsiveness with Ag presentation.
120  plain aspirin and PL2200, this high rate of nonresponsiveness with EC aspirin was associated with lo

 
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