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1 ly symptomatic osteoarthritis managed with a nonsteroidal anti-inflammatory agent.
2 e antihistamines, mast cell stabilizers, and nonsteroidal anti-inflammatory agents.
3 ygenases are the target of aspirin and other nonsteroidal anti-inflammatory agents.
4                 Scleritis resolved with oral nonsteroidal anti-inflammatory agent alone in 1 patient
5  most common elicitors were drugs, including nonsteroidal anti-inflammatory agents and penicillin.
6                                              Nonsteroidal anti-inflammatory agents are not associated
7 odels continue to support the development of nonsteroidal anti-inflammatory agents as chemopreventive
8 were more susceptible to gastric injury from nonsteroidal anti-inflammatory agents, confirming that t
9 hol-related conditions), and medication use (nonsteroidal anti-inflammatory agents, corticosteroids,
10  is markedly attenuated, and the addition of nonsteroidal anti-inflammatory agents does not further i
11 rvations: Regional analgesia, acetaminophen, nonsteroidal anti-inflammatory agents, gabapentinoids, t
12 roke, and death) rates between celecoxib and nonsteroidal anti-inflammatory agents in CLASS.
13          Recent work has suggested that some nonsteroidal anti-inflammatory agents, including sodium
14 that NF-kappaB is a downstream target of the nonsteroidal anti-inflammatory agent indomethacin and is
15                                          The nonsteroidal anti-inflammatory agent indomethacin reduce
16 Recently, it also has been demonstrated that nonsteroidal anti-inflammatory agents inhibit tumor necr
17 anti-inflammatory activity compared with the nonsteroidal anti-inflammatory agent ketorolac.
18                                          The nonsteroidal anti-inflammatory agents ketorolac trometha
19  the synthesis of azo mutual prodrugs of the nonsteroidal anti-inflammatory agents (NSAIDs) 4-aminoph
20                                              Nonsteroidal anti-inflammatory agents (NSAIDS) inhibit g
21 k have often included patients using aspirin/nonsteroidal anti-inflammatory agents (NSAIDs) or acid-s
22                                      Topical nonsteroidal anti-inflammatory agents (NSAIDs) proved to
23                                              Nonsteroidal anti-inflammatory agents or corticosteroids
24                                              Nonsteroidal anti-inflammatory agents or NSAIDs are pote
25 PGF(2alpha) in urine cannot be suppressed by nonsteroidal anti-inflammatory agents, suggesting that i
26 h (45 mg/kg) dose of HCT 1026, a NO-donating nonsteroidal anti-inflammatory agent which has previousl
27  the same symptomatic relief as nonselective nonsteroidal anti-inflammatory agents without the same c