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1 mild pain in the thyroid) can be handled by nonsteroidal antiinflammatory drugs.
2 enases (COXs) are the therapeutic targets of nonsteroidal antiinflammatory drugs.
3 f patients with chemical gastritis caused by nonsteroidal antiinflammatory drugs.
4 d arthritis (RA) and its response to chronic nonsteroidal antiinflammatory drugs.
5 ostaglandin synthesis and are the targets of nonsteroidal antiinflammatory drugs.
6 dipyrone, and is potently inhibited by some nonsteroidal antiinflammatory drugs.
7 ions in AERD patients following ingestion of nonsteroidal antiinflammatory drugs.
8 may receive both low-dose aspirin and other nonsteroidal antiinflammatory drugs.
9 effects of COX-1 inhibition associated with nonsteroidal antiinflammatory drugs.
10 (11)C-PS13 to measure the in vivo potency of nonsteroidal antiinflammatory drugs.
11 l side effects of the indomethacin family of nonsteroidal antiinflammatory drugs.
12 de rate ratio of death was 0.68 for users of nonsteroidal antiinflammatory drugs.
13 al volunteers, even in the absence of recent nonsteroidal antiinflammatory drugs.
14 rgery, 55% of patients received prescription nonsteroidal antiinflammatory drugs, 58% received opioid
15 ling to the macrophage inhibitory cytokine-1/nonsteroidal antiinflammatory drug-activated gene-1 prom
16 ted RNA for macrophage inhibitory cytokine-1/nonsteroidal antiinflammatory drug-activated gene-1, a T
18 ng antirheumatic drugs, corticosteroids, and nonsteroidal antiinflammatory drugs, alone or in combina
20 These enzymes are pharmacological targets of nonsteroidal antiinflammatory drugs and cyclooxygenase (
21 Recommendations for the perioperative use of nonsteroidal antiinflammatory drugs and glucocorticoids
22 n of >/=6 months and inadequate responses to nonsteroidal antiinflammatory drugs and glucocorticoids)
23 nd analgesic properties yet differs from the nonsteroidal antiinflammatory drugs and inhibitors of pr
25 ic drugs were discontinued; stable dosage of nonsteroidal antiinflammatory drugs and oral corticoster
26 hite blood cell count, and use of nonaspirin nonsteroidal antiinflammatory drugs and other medication
27 e in the induction of 15-LOX-1 expression by nonsteroidal antiinflammatory drugs and related agents.
28 AF-2 activity in cells were detected: three nonsteroidal antiinflammatory drugs and the thyroid horm
30 ting for cardiovascular risk factors, use of nonsteroidal antiinflammatory drugs, and a history of ga
31 be induced by some medications, particularly nonsteroidal antiinflammatory drugs, and has been associ
32 nt angiotensin-converting enzyme inhibitors, nonsteroidal antiinflammatory drugs, and immunosuppressi
33 as weight (kg)/height (m)2), never-users of nonsteroidal antiinflammatory drugs, and those with lowe
34 esophageal adenocarcinoma but did agree that nonsteroidal antiinflammatory drugs are associated with
38 ndin biosynthesis and are the targets of the nonsteroidal antiinflammatory drugs aspirin and ibuprofe
39 on of cyclooxygenases, the central action of nonsteroidal antiinflammatory drugs (but not a prominent
40 xplain the cancer chemoprotective effects of nonsteroidal antiinflammatory drugs by defining a mechan
42 of dosing with prednisolone, methotrexate, 3 nonsteroidal antiinflammatory drugs (celecoxib, diclofen
43 rrays of bioactive lipid mediators via COX-2-nonsteroidal antiinflammatory drug-dependent oxygenation
48 Here, we show that the R-enantiomer of the nonsteroidal antiinflammatory drug etodolac inhibited tu
49 plexes of beta- and gamma-cyclodextrins with nonsteroidal antiinflammatory drugs ibuprofen, naproxen,
50 repair and create concerns about the use of nonsteroidal antiinflammatory drugs in patients with ske
51 inhibitors are as effective as nonselective nonsteroidal antiinflammatory drugs in relieving pain an
55 e reaction of three solid-state forms of the nonsteroidal antiinflammatory drug indomethacin with amm
56 f 2B5 was indistinguishable from that of the nonsteroidal antiinflammatory drug indomethacin, which b
57 tions, including inflammatory bowel disease, nonsteroidal antiinflammatory drug-induced gut injury, a
58 uch as capsule endoscopy studies demonstrate nonsteroidal antiinflammatory drug-induced small bowel e
60 tosis in human colon cancer cells by certain nonsteroidal antiinflammatory drugs involves increased e
61 3 effects to blockade by two analgesics (the nonsteroidal antiinflammatory drug ketoprofen and the mu
62 but not postoperative, administration of the nonsteroidal antiinflammatory drug ketorolac and/or reso
64 this disease, the chemopreventive effects of nonsteroidal antiinflammatory drugs may be related to th
66 research suggests that rectally administered nonsteroidal antiinflammatory drugs may reduce the incid
67 of drugs that inhibit PG production, such as nonsteroidal antiinflammatory drugs, may be deleterious
68 gs that are widely used to treat OA, such as nonsteroidal antiinflammatory drugs, may have limited ef
69 rtitioning and nature of interactions of two nonsteroidal antiinflammatory drugs, namely aspirin and
70 epidemiological studies have shown that the nonsteroidal antiinflammatory drug naproxen may be usefu
71 tioxidant ester and amide derivatives of the nonsteroidal antiinflammatory drug naproxen was designed
72 management for Helicobacter pylori-negative, nonsteroidal antiinflammatory drug-negative ulcer patien
73 -116 colon cancer cells with and without the nonsteroidal antiinflammatory drug NS-398 or the histone
74 chronic arthritis: 1) a generic nonselective nonsteroidal antiinflammatory drug (NSAID(NS)) alone; 2)
75 ee OA underwent a washout of their analgesic/nonsteroidal antiinflammatory drug (NSAID) agents (durat
76 The present study compared the effect of the nonsteroidal antiinflammatory drug (NSAID) diclofenac, w
77 estinal damage in zebrafish larvae using the nonsteroidal antiinflammatory drug (NSAID) Glafenine.
78 he small intestine is a more common site for nonsteroidal antiinflammatory drug (NSAID) toxicity than
79 abolite profiles but only modestly decreased nonsteroidal antiinflammatory drug (NSAID) turnover whil
83 Analogues of diflunisal, an FDA-approved nonsteroidal antiinflammatory drug (NSAID), were synthes
86 ces the effectiveness of both currently used nonsteroidal antiinflammatory drugs (NSAIDs) (diclofenac
88 e cancer and whether use of aspirin or other nonsteroidal antiinflammatory drugs (NSAIDs) affects ris
89 hors used PSC to assess the relation between nonsteroidal antiinflammatory drugs (NSAIDs) and 1-year
90 ardiovascular risk, studies of nonselective, nonsteroidal antiinflammatory drugs (NSAIDs) and acetami
92 rs have reported an inverse relation between nonsteroidal antiinflammatory drugs (NSAIDs) and colon c
93 tive on patient preferences for nonselective nonsteroidal antiinflammatory drugs (NSAIDs) and cycloox
94 The association between long-term use of nonsteroidal antiinflammatory drugs (NSAIDs) and non-Hod
95 pidemiologic data on the association between nonsteroidal antiinflammatory drugs (NSAIDs) and ovarian
96 investigations have identified a variety of nonsteroidal antiinflammatory drugs (NSAIDs) and structu
97 prevented by a concurrent administration of nonsteroidal antiinflammatory drugs (NSAIDS) and/or low-
98 umatic diseases and because apparently safer nonsteroidal antiinflammatory drugs (NSAIDs) are being p
103 nal CCEs, whereas treatment with 2 different nonsteroidal antiinflammatory drugs (NSAIDs) blocked neu
105 It is well established that taking multiple nonsteroidal antiinflammatory drugs (NSAIDs) can lead to
107 ntrol studies have shown that regular use of nonsteroidal antiinflammatory drugs (NSAIDs) decreases b
113 Epidemiologic studies suggest that use of nonsteroidal antiinflammatory drugs (NSAIDs) is associat
114 f cyclooxygenase-inhibiting (COX-inhibiting) nonsteroidal antiinflammatory drugs (NSAIDs) is often co
117 (LUTS), raising the possibility that use of nonsteroidal antiinflammatory drugs (NSAIDs) may inhibit
118 Several studies have suggested that use of nonsteroidal antiinflammatory drugs (NSAIDs) may reduce
120 ategies for knee monarthritis were compared: nonsteroidal antiinflammatory drugs (NSAIDs) only, NSAID
121 of nine measures of high-risk prescribing of nonsteroidal antiinflammatory drugs (NSAIDs) or selected
122 nd experimental studies suggests that use of nonsteroidal antiinflammatory drugs (NSAIDs) reduces ris
123 cal studies have shown that long-term use of nonsteroidal antiinflammatory drugs (NSAIDs) reduces the
126 c has gastrointestinal liabilities common to nonsteroidal antiinflammatory drugs (NSAIDs) that might
128 nnesota, to determine whether daily users of nonsteroidal antiinflammatory drugs (NSAIDs) were at low
130 X inhibitors, including coxibs, nonselective nonsteroidal antiinflammatory drugs (NSAIDs), and meloxi
131 ibitors of prostaglandin production, such as nonsteroidal antiinflammatory drugs (NSAIDs), and pharma
133 inuation for any reason did not differ among nonsteroidal antiinflammatory drugs (NSAIDs), but discon
134 s of other drugs, including D-penicillamine, nonsteroidal antiinflammatory drugs (NSAIDs), calcium ch
135 use of ODC inhibitors and aspirin, or other nonsteroidal antiinflammatory drugs (NSAIDs), in combina
136 of celecoxib, as compared with nonselective nonsteroidal antiinflammatory drugs (NSAIDs), remains un
137 tments for osteoarthritis (OA) pain, such as nonsteroidal antiinflammatory drugs (NSAIDs), simple ana
139 h as 5,8,11,14-eicosatetraynoic acid, and in nonsteroidal antiinflammatory drugs (NSAIDs), such as in
140 astrointestinal hemorrhage than nonselective nonsteroidal antiinflammatory drugs (NSAIDs), there has
155 coxibs and common active internal controls (nonsteroidal antiinflammatory drugs [NSAIDs], naproxen)
156 1 and COX-2 are both targets of nonselective nonsteroidal antiinflammatory drugs (nsNSAIDs) including
157 ity was apparent only among those not taking nonsteroidal antiinflammatory drugs (odds ratio = 0.49,
158 om colorectal cancer in individuals who take nonsteroidal antiinflammatory drugs on a regular basis c
159 al cancer in persons taking aspirin or other nonsteroidal antiinflammatory drugs on a regular basis.
160 nsistently indicated that sulindac and other nonsteroidal antiinflammatory drugs or cyclooxygenase in
161 wer or discontinue their background doses of nonsteroidal antiinflammatory drugs or disease-modifying
163 ct were long-term users of aspirin, ethanol, nonsteroidal antiinflammatory drugs, or a combination of
164 was found between the use of aspirin, other nonsteroidal antiinflammatory drugs, or acetaminophen an
165 ls are needed to determine whether steroids, nonsteroidal antiinflammatory drugs, or both can prevent
166 Patients were allowed to continue taking nonsteroidal antiinflammatory drugs, oral corticosteriod
167 s and adenocarcinomas, and its inhibition by nonsteroidal antiinflammatory drugs protects against col
168 n signaling was diminished by an analog of a nonsteroidal antiinflammatory drug (R-etodolac), at conc
169 ological and laboratory studies suggest that nonsteroidal antiinflammatory drugs reduce the risk of c
171 ion was stronger among participants who used nonsteroidal antiinflammatory drugs regularly (highest v
172 ere noted at the start of therapy: age, sex, nonsteroidal antiinflammatory drug-related risk factors
174 s technique to study the interactions of two nonsteroidal antiinflammatory drugs, salicylate and ibup
175 been applied to the analysis of a mixture of nonsteroidal antiinflammatory drugs separated by reverse
177 are pharmacologically important targets for nonsteroidal antiinflammatory drugs, such as aspirin and
179 umans and can measure the in vivo potency of nonsteroidal antiinflammatory drugs that competitively i
180 nt for the use or nonuse of aspirin or other nonsteroidal antiinflammatory drugs; the presence or abs
181 Indomethacin (INDO) was one of the first nonsteroidal antiinflammatory drugs to be characterized
182 mall intestine may be a more common site for nonsteroidal antiinflammatory drug toxicity than the gas
184 s who had previously received treatment with nonsteroidal antiinflammatory drugs underwent stratifica
185 d beneficiaries in New Jersey, the effect of nonsteroidal antiinflammatory drug use on 1-year all-cau
187 dex, smoking, physical activity, aspirin and nonsteroidal antiinflammatory drug use, alcohol consumpt
189 of antibiotics (primarily fluoroquinolones), nonsteroidal antiinflammatory drugs (Voltaren and ketoro
190 erence to study medications and avoidance of nonsteroidal antiinflammatory drugs were excellent.
192 PsA and a history of inadequate response to nonsteroidal antiinflammatory drugs were randomized to r