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1 pressive SOS responses before returning to a normal appearance.
2     Muller cells and rod bipolar cells had a normal appearance.
3 retrospectively judged as having a benign or normal appearance.
4 tions that produced nonfunctional Z rings of normal appearance.
5 0 days, the oral epithelium had regained its normal appearance.
6 ellum and the brainstem were of structurally normal appearance.
7  and ultimately produce an axial skeleton of normal appearance.
8 ent repeat US, the gallbladder reverted to a normal appearance.
9      In fact, after day 10, the retina has a normal appearance.
10 ng cells infected with mCMV had a relatively normal appearance 12 days after inoculation.
11 appropriate ligand (glycine or NMDA) and the normal appearance and duration of channel openings evoke
12                      Kcnn4 null mice were of normal appearance and fertility, their parotid acinar ce
13 with increased dendritic complexity, despite normal appearance and number of dendritic spines.
14 functional, layered, filtering bleb, and the normal appearance and patency of the XEN proximal segmen
15                             Knowledge of the normal appearance and variations of the post-cricoid reg
16 B::rd28 cells formed fruiting bodies of more normal appearance, and the number of viable spores incre
17  presynaptic terminals in the striatum had a normal appearance at 4 hr after ischemia but showed dege
18 ssive in Romney sheep was characterized with normal appearance at birth, but with progressive weaknes
19 yed diminished vesicular turnover, despite a normal appearance at the EM level.
20 teral ventricular morphology as having (a) a normal appearance, (b) mild, disproportionate dilatation
21                The knockout mice had grossly normal appearance, but grew approximately 20% slower tha
22 store the production of nucleocapsids with a normal appearance, but not infectious virus production.
23                                  Despite its normal appearance in conventionally examined cross-secti
24 /lacy appearance, followed by restoration of normal appearance in Plg(+) livers by day 7.
25 thyroid gland in the hyperthyroid phase to a normal appearance in the late recovery phase.
26            Four MR patterns were identified: normal appearance (n = 7), periosteal fluid only (n = 5)
27                            Every patient had normal appearance of chest organs, with the exception of
28 lycocalyx on the apical cell membrane, and a normal appearance of goblet cell mucin packets.
29 lar space is aberrantly enlarged despite the normal appearance of myofibroblasts.
30                                  Despite the normal appearance of single mutants under ideal laborato
31 f Muc1 null mice of the C57BL/6 strain had a normal appearance of surface microplicae, a well-develop
32 t 259 amino acids of SptP also disrupted the normal appearance of the cytoskeleton.
33    A ridge cortical thickness >0.75 mm and a normal appearance of the inferior mandibular cortex were
34 ic volunteers were reviewed to establish the normal appearance of the radial tunnel.
35 eversible, as microinjected cells regain the normal appearance of their actin cytoskeleton upon prolo
36 nctional K(Ca) channels at stages before the normal appearance of these channels in CG neurons develo
37                    Detailed knowledge of the normal appearance of these ligaments can serve as a base
38 valuated standard MR images to determine the normal appearances of the joint structures, especially t
39 he purpose of this review is to describe the normal appearances of the prostatic region after differe
40      Most notably, while skeletal muscle was normal, appearance of severe dystonic ataxia correlated
41 ct abnormalities in white matter that have a normal appearance on both conventional MR and DT images;
42 clerosis (TLE-MTS), and 50 TLE subjects with normal appearance on MRI (TLE-no).
43 ery preterm subjects with corpora callosa of normal appearance on structural MRI, and with seven full
44  pallor of the optic disc than in those with normal appearance or mild pallor of the optic disc.
45                       ESCs lacking Oct1 show normal appearance, self-renewal and growth but manifest
46  mutants led to embryos with morphologically normal appearance, suggesting that p53 is involved in tr
47 ation decreased, and the cells reverted to a normal appearance, the growth rate normalized, and cells
48  variable iris phenotype ranging from almost normal appearance to no iris.