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1 pressive SOS responses before returning to a normal appearance.
2 Muller cells and rod bipolar cells had a normal appearance.
3 retrospectively judged as having a benign or normal appearance.
4 tions that produced nonfunctional Z rings of normal appearance.
5 0 days, the oral epithelium had regained its normal appearance.
6 ellum and the brainstem were of structurally normal appearance.
7 and ultimately produce an axial skeleton of normal appearance.
8 ent repeat US, the gallbladder reverted to a normal appearance.
9 In fact, after day 10, the retina has a normal appearance.
11 appropriate ligand (glycine or NMDA) and the normal appearance and duration of channel openings evoke
14 functional, layered, filtering bleb, and the normal appearance and patency of the XEN proximal segmen
16 B::rd28 cells formed fruiting bodies of more normal appearance, and the number of viable spores incre
17 presynaptic terminals in the striatum had a normal appearance at 4 hr after ischemia but showed dege
18 ssive in Romney sheep was characterized with normal appearance at birth, but with progressive weaknes
20 teral ventricular morphology as having (a) a normal appearance, (b) mild, disproportionate dilatation
22 store the production of nucleocapsids with a normal appearance, but not infectious virus production.
31 f Muc1 null mice of the C57BL/6 strain had a normal appearance of surface microplicae, a well-develop
33 A ridge cortical thickness >0.75 mm and a normal appearance of the inferior mandibular cortex were
35 eversible, as microinjected cells regain the normal appearance of their actin cytoskeleton upon prolo
36 nctional K(Ca) channels at stages before the normal appearance of these channels in CG neurons develo
38 valuated standard MR images to determine the normal appearances of the joint structures, especially t
39 he purpose of this review is to describe the normal appearances of the prostatic region after differe
41 ct abnormalities in white matter that have a normal appearance on both conventional MR and DT images;
43 ery preterm subjects with corpora callosa of normal appearance on structural MRI, and with seven full
46 mutants led to embryos with morphologically normal appearance, suggesting that p53 is involved in tr
47 ation decreased, and the cells reverted to a normal appearance, the growth rate normalized, and cells