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1  in vaginal wash samples than did women with normal flora.
2  demonstrate inflammation in response to the normal flora.
3 = 0.47; P </= .01), with most concordant for normal flora.
4  and the symbiotic relation we have with our normal flora.
5 across all samples, predominantly reflecting normal flora.
6  between a natural product of the intestinal normal flora and important epithelial inflammatory and p
7 ed the plate as contaminated or growing only normal flora and not due to threshold counts.
8 , evidence is presented which shows that the normal flora are maintained on a diet of salivary factor
9 discovery of novel members of the endogenous normal flora as well as putative disease agents.
10 f growth in the human host, as a part of the normal flora, as a pathogen, or as a biofilm.
11 ovides important information for recognizing normal flora, assessing the health status of these uniqu
12 onpathogenic Gram-positive bacteria, but not normal flora bacteria.
13 l pathogen Candida albicans is a part of the normal flora but it also causes systemic candidiasis if
14 , or yeast compared with those demonstrating normal flora by 95% confidence intervals.
15 tervention on a healthy vaginal environment (normal flora confirmed by Gram stain with no candidiasis
16 ontal pathogens, opportunistic pathogens, or normal flora did not differ descriptively between groups
17 al and extraintestinal pathogenic as well as normal flora E. coli.
18                                          The normal flora furnishes the host with ecological barriers
19 ia, which include Gram-negative bacteria and normal flora Gram-positive bacteria.
20 hogenic and nonpathogenic transient, but not normal flora, Gram-positive bacteria.
21 ors that are expressed following exposure to normal flora in animals lacking IL-10.
22  in preventing excessive inflammation to the normal flora in the intestinal lumen.
23 ginal wash and serum samples from women with normal flora, intermediate flora, and BV (determined by
24                                Among the 236 normal flora isolates, 233 (98.7%) and 228 (96.6%) agree
25 pylobacter species, and 236 gastrointestinal normal flora isolates.
26  bowel disease, the loss of tolerance to the normal flora may be due in part to inappropriate toll-li
27  two groups: abnormal flora (AF; n = 41) and normal flora (NF; n = 53).
28     Gallibacterium anatis is a member of the normal flora of avian hosts and an important causative a
29 on mediated by rumen protozoa (RPz) that are normal flora of cattle.
30 of IAV and may affect both pathogens and the normal flora of different mucosal sites.IMPORTANCE Siali
31 biodiversity in aquatic environments and the normal flora of fish and shellfish.
32 us lugdunensis is often found as part of the normal flora of human skin but has the potential to caus
33       Candida albicans is a component of the normal flora of the alimentary tract and also is found o
34         Candida albicans, a component of the normal flora of the alimentary tract and mucocutaneous m
35 istic fungal pathogen and a component of the normal flora of the gastrointestinal tract, is a frequen
36 Streptococcus intermedius is a member of the normal flora of the mouth but is also an opportunistic p
37                                          The normal flora of the oropharynx may be an important sourc
38 e of this study was to compare the effect of normal flora on inflammation in mice in which the IL-10
39                                  Because the normal flora produces significant amounts of butyrate an
40 S gene is contained by invasive isolates and normal-flora strains in a region that contains genes inv
41 uced lactobacilli were less likely to regain normal flora than were those whose lactobacilli were una
42 he reduction in the number of members of the normal flora that interfere with the growth of pathogens
43 city for the pathogen without disturbing the normal flora, the low chance of bacterial resistance to
44 city for the pathogen without disturbing the normal flora, the low chance of bacterial resistance to