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1 h-affinity binding to DNA and very efficient nuclear condensation.
2 These cells appeared to be undergoing nuclear condensation.
3 GE2- and cAMP-mediated DNA fragmentation and nuclear condensation.
4 loss of membrane phospholipid asymmetry, and nuclear condensation.
5 but surprisingly NCAPH2 was dispensable for nuclear condensation.
6 n gene expression in the context of dramatic nuclear condensation.
7 u at 24 h, when 20% of infected cells showed nuclear condensation.
8 mbrane leakage preceded the manifestation of nuclear condensation.
9 glycophorin A expression, and chromatin and nuclear condensation.
10 swellings, somal chromatolytic changes, and nuclear condensation.
11 n and distention, mitochondrial swelling and nuclear condensation.
12 -3 and PARP cleavage, DNA fragmentation, and nuclear condensation.
13 motes efficient erythroblast enucleation and nuclear condensation.
14 and caused mitochondrial depolarization and nuclear condensation.
15 substrate, poly(ADP-ribose) polymerase, and nuclear condensation.
16 of mitochondrial membrane potential, before nuclear condensation.
17 procaspase-7 activation, PARP cleavage, and nuclear condensation.
18 -like activity, Annexin V incorporation, and nuclear condensation.
19 a reduction in cell size and an increase in nuclear condensation.
20 n phosphorylation, caspase-3 activation, and nuclear condensation.
21 terized by internucleosomal DNA cleavage and nuclear condensation.
22 within 30 min, before cellular shrinkage or nuclear condensation.
23 calculated as the fraction of cells showing nuclear condensation.
24 tive features such as cellular shrinkage and nuclear condensation.
25 11.8 +/- 0.50 vs. 3.3 +/- 0.26; p < 0.0001), nuclear condensation (10.9 +/- 0.58 vs. 3.4 +/- 0.20; p
26 ctivity triggered significant apoptosis, via nuclear condensation (87.5% of nuclei analyzed) and plas
28 al progenitors and other cell types leads to nuclear condensation accompanied by large-scale changes
30 sequence: formation of cytoplasmic blebs and nuclear condensation after 3 hours; nuclear fragmentatio
33 appeared to induce selective dose-dependent nuclear condensation and cell shrinkage in the treated c
34 e appears to induce selective dose-dependent nuclear condensation and cell shrinkage in treated cance
36 rapid externalization of phosphatidylserine, nuclear condensation and collapse, and single-stranded D
37 morphologic changes, histologic evidence of nuclear condensation and degeneration, and flow-cytometr
40 rous erythroid genes and is characterized by nuclear condensation and extrusion during terminal devel
43 both cell types, which was characterized by nuclear condensation and fragmentation and activation of
45 e potential (DeltaPsi), DNA degradation, and nuclear condensation and fragmentation characteristic of
48 ced by poly(ADP-ribose) polymerase cleavage, nuclear condensation and fragmentation, and hypodiploid
49 ormation of apoptotic bodies, detachment and nuclear condensation and fragmentation, in cells transfe
50 uced typical features of apoptosis including nuclear condensation and fragmentation, oligonucleosomal
51 SF-21 cell line induced apoptosis displaying nuclear condensation and fragmentation, oligonucleosomal
54 1 and caffeine, induced mitosis and abnormal nuclear condensation and increased the protein kinase ac
55 eath induced via CD95 (Fas), as evidenced by nuclear condensation and membrane blebbing, but did not
56 sequently undergo apoptosis, as indicated by nuclear condensation and poly(ADP-ribose) polymerase cle
57 ntiation associated with the onset of global nuclear condensation and reduced cell proliferation.
59 transcription, yet the relationship between nuclear condensation and transcriptional activation rema
61 from the mitochondria, caspase-3 activation, nuclear condensation, and an orderly progression of hair
63 volume changes, intracellular acidification, nuclear condensation, and chromosomal digestion ("ladder
65 cell death involving cytoplasmic shrinkage, nuclear condensation, and DNA fragmentation characterist
68 hropoiesis include global gene inactivation, nuclear condensation, and enucleation to yield circulati
70 area, persistent ckit expression, incomplete nuclear condensation, and lower rates of enucleation.
71 apoptotic signs including membrane blebbing, nuclear condensation, and reduction of mitochondrial mem
72 th vacuolization and mitochondrial swelling, nuclear condensation, and sustained plasma membrane.
74 ructs were tested on cell toxicity using our nuclear condensation assay and on mitochondrial viabilit
76 de, we observed laddering of genomic DNA and nuclear condensation, both hallmarks of apoptotic cells.
77 antly protect from cell death as measured by nuclear condensation, caspase activation, PARP degradati
78 eath by apoptosis was confirmed by increased nuclear condensation, changes in membrane morphology, an
81 s demonstrated changes resembling apoptosis: nuclear condensation, chromatin fragmentation, and forma
82 innovation of flowering plants that achieves nuclear condensation compatible with active transcriptio
83 plicing factors Sm and SC35 persisted during nuclear condensation, consistent with effective transcri
86 acrophages, which contrasted with the marked nuclear condensation displayed by control cells undergoi
87 through inducing cell apoptosis confirmed by nuclear condensation, DNA cleavage, and accumulation of
88 n was followed by apoptosis as determined by nuclear condensation, DNA fragmentation, and annexin V b
89 metry analyses were used to detect apoptotic nuclear condensation, DNA fragmentation, and changes in
91 ncer cell selective nuclear internalization, nuclear condensation, fragmentation, and eventually anti
92 , SNCEE did not induce caspase activation or nuclear condensation/fragmentation suggesting that PS ex
93 ptosis, characterized by caspase activation, nuclear condensation/fragmentation, and membrane blebbin
94 ells, including marked caspase-3 activation, nuclear condensation/fragmentation, but with swollen cyt
95 ic interrogation of the relationship between nuclear condensation, genome structure, and gene transcr
96 rial membrane potential, cell shrinkage, and nuclear condensation in a caspase-dependent fashion.
101 (1) peak in the attached cell population and nuclear condensation in the floating cell population.
102 nd propidium iodide staining showed profound nuclear condensation in the NMDA or QA-treated striatum.
105 is followed by caspase activation, apoptotic nuclear condensation, loss of membrane potential, and, f
106 jury in glutathione-depleted preOLs included nuclear condensation, margination of chromatin, and mito
107 classical hallmarks of apoptosis, including nuclear condensation, membrane blebbing, caspase activat
109 n (DCD) that is expressed as cytoplasmic and nuclear condensation, neuron shrinkage, and failure of p
110 of a plant karyopherin, which modulates the nuclear condensation of an evolutionarily conserved spli
112 Furthermore, the C127/508 cells did not show nuclear condensation or DNA fragmentation detected by in
115 such apoptotic markers as DNA fragmentation, nuclear condensation, poly(ADP-ribose) polymerase cleava
118 In apoptotic cells, DNA fragmentation and nuclear condensation result from caspase-3-mediated prot
119 sensory cells were dependent on the stage of nuclear condensation, suggesting a specific role for MMP
120 d caspase activation and activity along with nuclear condensation that occurs independent of actin cy
121 histones, HILS1 may participate in spermatid nuclear condensation through a mechanism distinct from t
122 eprivation; LY 294002-induced death included nuclear condensation, was blocked by cycloheximide, and
123 ntation and TUNEL-positive nuclei as well as nuclear condensation were abolished by the NMDA receptor
124 cells displayed irreversible cytoplasmic and nuclear condensation while maintaining intact membranes.