ライフサイエンスコーパス: nuclear division

1 to their requirement for IME1 induction and nuclear division.

2 multiple nuclei, consistent with unbalanced nuclear division.

3 rmation of delocalized septa did not require nuclear division.

4 likely to be involved in control of parasite nuclear division.

5 th retention of nuclear Clb5p at the time of nuclear division.

6 only after the nucleus enters the bud during nuclear division.

7 otype, where septation is completed prior to nuclear division.

8 inately cycle multiple times without causing nuclear division.

9 c nuclear division and the first postzygotic nuclear division.

10 lular polarity, without affecting growth and nuclear division.

11 cells that undergo closed mitosis to achieve nuclear division.

12 aughter nucleus in cells that have undergone nuclear division.

13 dc18 fail to enter S phase but still undergo nuclear division.

14 point to a central role in the regulation of nuclear division.

15 lifestyle may favor the evolution of closed nuclear division.

16 hat ensures proper genome segregation during nuclear division.

17 cannot rereplicate between bud formation and nuclear division.

18 CDK activity to allow bud formation prior to nuclear division.

19 ng cells, is normally restrained until after nuclear division.

20 ive cells, but is dispensable for growth and nuclear division.

21 t is required for chromosome segregation and nuclear division.

22 sembly of kinetochores and are essential for nuclear division.

23 cid replication is followed by two rounds of nuclear division.

24 are required for Rec8 cleavage and meiosis I nuclear division.

25 did restore JM resolution, CO formation, and nuclear division.

26 These JMs persist, preventing nuclear division.

27 ediated progression beyond the first meiotic nuclear division.

28 er checkpoint-inducing conditions that block nuclear division.

29 eres, eliminating the possibility of mitotic nuclear division.

30 Constructs that chelate InsP3 also prevent nuclear division.

31 additional round of DNA replication without nuclear division.

32 e SPB and that this hub coordinates cell and nuclear division.

33 tions in the syncytial blastoderm after 9-10 nuclear divisions.

34 efects can be detected as early as the first nuclear divisions.

35 branch formation reinitiates tip growth and nuclear divisions.

36 a maternal effect during the early embryonic nuclear divisions.

37 omatin prior to each of the three prezygotic nuclear divisions.

38 progressive epigenome decay over hundreds of nuclear divisions.

39 ty of the meiotic chromosomes during the two nuclear divisions.

40 s and circadian clock-dependent synchronized nuclear divisions.

41 delayed and reduced execution of the meiotic nuclear divisions.

42 and Cdt1 during the interval between meiotic nuclear divisions.

43 ription program and execution of the meiotic nuclear divisions.

44 activity blocks bipolar spindle assembly and nuclear divisions.

45 s that arrest development after two or three nuclear divisions.

46 ource is necessary only prior to the meiotic nuclear divisions.

47 itical role of Cep164 in G2/M checkpoint and nuclear divisions.

48 embryo and remains stable during subsequent nuclear divisions.

49 eplication (meiS) followed by two successive nuclear divisions.

50 (c.504+1G>A) of RMND1 (required for meiotic nuclear division-1) in the affected individuals.

51 Following nuclear division, a cytokinetic phragmoplast forms betwe

52 break repair and reduced DNA damage-related nuclear division abnormalities, a phenotype associated w

53 romatid cohesion (SSC) during two successive nuclear divisions allows the production of haploid gamet

54 morphology, cell cycle arrest, asynchronous nuclear division and aberrant nuclear migration.

55 ultiple nuclei indicative of a G2/M delay in nuclear division and also a defect in cytokinesis and/or

56 icated in the coordination of septation with nuclear division and cell growth.

57 Hesperadin blocked nuclear division and cytokinesis but not other aspects o

58 ur data indicate that glo1-1 plays a role in nuclear division and cytokinesis in the developing seed.

59 The link between parasite nuclear division and cytokinesis is altered during the f

60 olarized tip growth and coordination between nuclear division and cytokinesis, and it is also importa

61 dicating defects in the coordination between nuclear division and cytokinesis.

62 imply a high degree of control over parasite nuclear division and cytokinesis.

63 checkpoint is required to coordinate between nuclear division and cytokinesis.

64 rapidly resume growth, exhibiting chaining, nuclear division and FtsZ-ring formation.

65 l coordination of two key processes, meiotic nuclear division and gamete morphogenesis.

66 sulted in a 90% decrease in the enzyme, both nuclear division and growth were markedly slowed.

67 The sonA1 suppressor alleviated the nuclear division and NIMECyclin B localization defects o

68 In primary OaSC tumors, mitotic nuclear division and oxidative phosphorylation pathways

69 emperature-sensitive phoA allele caused both nuclear division and polarity defects at restrictive tem

70 This protein regulates both nuclear division and polarized growth via its ability to

71 PfCRK4 depletion led to a complete block in nuclear division and profoundly inhibited DNA replicatio

72 point machineries are both required to delay nuclear division and provide adequate time for BIR to co

73 ements for remodeling the NE during "closed" nuclear division and provide insight into the structure

74 Syncytial embryos develop through cycles of nuclear division and rearrangement within a common cytop

75 results suggest that the ability to separate nuclear division and septum formation depends upon a thr

76 to spindle pole bodies (SPBs) during meiotic nuclear division and subsequently to PSMs/SWs.

77 cnj7 and cnj8 eliminate the third prezygotic nuclear division and the first postzygotic nuclear divis

78 F-actin in one of the daughter nuclei after nuclear division and the formation of septum; and (2) di

79 function in a pathway that controls meiotic nuclear divisions and coordinates meiosis and spore form

80 of Mtrm in the early embryo caused aberrant nuclear divisions and developmental defects, and these w

81 Cytoplasmic divisions are thought to rely on nuclear divisions and mitotic signals.

82 Moreover, nuclear divisions and movements co-vary with local nucle

83 hogenesis checkpoint in budding yeast delays nuclear division (and subsequent cell cycle progression)

84 to unequal chromosome segregation, abnormal nuclear division, and aneuploidy.

85 ture linked to increased cell proliferation, nuclear division, and cell cycle activity that was local

86 synthesis, bud emergence, spindle formation, nuclear division, and cell separation.

87 cts septation and conidiation in addition to nuclear division, and cloning and biochemical analysis o

88 uires coordination between localized growth, nuclear division, and septation.

89 cnj9 eliminates the second postzygotic nuclear division, and subsequently, new macronuclei fail

90 ear fusions occur in the early cycles of the nuclear divisions, and become more prominent at later st

91 from meiotic prophase, the completion of the nuclear divisions, and spore formation.

92 cytoplasmic division cycle, coupling it with nuclear divisions, and when uncoupled, cytoplasm starts

93 While the aberrations in nuclear division are correlated with a defect in the ass

94 meiotic prophase progression and subsequent nuclear division are delayed if these haploids carry an

95 esults provide insight into how key steps in nuclear division are orchestrated.

96 We found that polar nuclear divisions are asynchronous and lag behind somati

97 Conditional alleles of MOB1 cause a late nuclear division arrest at restrictive temperature.

98 e presence of many cells undergoing aberrant nuclear division, as well as many anucleate cells, demon

99 dc1 depletion might affect bud formation and nuclear division, as well as other cellular processes, b

100 tant displays defects in cell morphology and nuclear division at 26 degrees C which are exaggerated a

101 The tsm1-512 mutation leads to delayed nuclear division at restrictive temperatures, apparently

102 We used this to predict how the nuclear division axis orientation changes upon nucleus r

103 as initially implicated in bud formation and nuclear division because cdc1(Ts) cells arrested with a

104 rminating conidia undergo multiple rounds of nuclear division before forming a septum.

105 rminating conidia undergo multiple rounds of nuclear division before the formation of the first septu

106 e fertilized egg undergoes a series of rapid nuclear divisions before the syncytial blastoderm starts

107 1 or Rad17 partially bypass the block to the nuclear divisions but do not form spores, while mutants

108 ntial specifically for spindle formation and nuclear division, but not for DNA replication, once CDKA

109 protozoans, GPIs appear to be essential for nuclear division, but not for mitochondrial duplication.

110 )/M, prp17Delta and prp22-1 strains, permits nuclear division, but suppression of the cell cycle bloc

111 he loss of linkages between chromosomes with nuclear division by restraining two other phosphatases,

112 Meiosis is a specialized nuclear division by which sexually reproducing diploid o

113 ion is followed by two consecutive rounds of nuclear divisions called meiosis I and meiosis II.

114 e cell cycle that replicates the DNA without nuclear division, causing a stepwise increase in somatic

115 During nuclear division, Cdc14 is released from the nucleolus,

116 We have characterized the septation, nuclear division, cell-cycle checkpoint defects, and DNA

117 uring megagametogenesis, including syncitial nuclear divisions, cellularization, nuclear migration an

118 as zygotic cnn expression display defects in nuclear division, chromosome alignment, and microtubule

119 ed patterns consistent with the processes of nuclear division, chromosome segregation, and transition

120 osophila, induced a reversible arrest of the nuclear divisions comparable with that induced by hypoxi

121 The microtubule-binding nuclear division cycle 80 (Ndc80) complex that is recrui

122 The nuclear division cycle 80 complex (Ndc80C) is a major mi

123 artmentalization of the cytosol for both the nuclear division cycle and branching.

124 This enzyme is required both for the nuclear division cycle and for hyphal growth, because sp

125 We found control of the nuclear division cycle and polarized morphogenesis, both

126 The aberrations in the nuclear division cycle are correlated with defects in th

127 Measurements at different stages of the nuclear division cycle showed little variation.

128 unusual features in terms of its mitotic and nuclear division cycle that presumably facilitate prolif

129 tivity and is essential for coordinating the nuclear division cycle with cytokinesis through the cyto

130 lei to form somatic lineages during the 14th nuclear division cycle, PGCs emerge from the syncytium d

131 R47 function may be modulated in the cell or nuclear division cycle.

132 f the gene prevents entry of spores into the nuclear division cycle.

133 required for proper coordination between the nuclear-division cycle and cytokinesis.

134 the SIN in coordinating cytokinesis with the nuclear-division cycle.

135 ishment and tip growth, but they have normal nuclear division cycles and can complete the asexual gro

136 olecular mechanisms that control these rapid nuclear division cycles are poorly understood.

137 processes such as cytokinesis and syncytial nuclear division cycles in Drosophila Pseudocleavage fur

138 During the rapid nuclear division cycles in early Drosophila embryos, coo

139 The nuclear division cycles of early Drosophila embryogenesi

140 The nuclear division cycles of syncytial stage Drosophila me

141 t dfmr1 embryos display defects in the rapid nuclear division cycles that precede gastrulation in nuc

142 elanogaster embryos that supports successive nuclear division cycles with native characteristics.

143 la melanogaster embryogenesis begins with 13 nuclear division cycles within a syncytium.

144 CMKC in regulating the first and subsequent nuclear division cycles.

145 We have traced these defects back to the nuclear division cycles.

146 presumptive somatic cytoplasm causes lethal nuclear division defects.

147 also show inefficient nuclear migration and nuclear division, defects in the organization of the sec

148 or proceed through mitosis in the absence of nuclear division, demonstrating an uncoupling of proper

149 e wild-type cells, clb5 clb6 mutants undergo nuclear division despite the presence of nuclear non-deg

150 Nuclear division did not occur in the mutant at 37 degre

151 cing can persist through DNA replication and nuclear divisions, disrupting uniform gene expression in

152 -sensitive snxA1 mutation leads to arrest of nuclear division during G(1) or early S. snxB1 causes hy

153 wed by a single round of DNA segregation and nuclear division during gamete budding.

154 The rapid nuclear division during this stage [4] requires that Bcd

155 ions are asynchronous and lag behind somatic nuclear divisions during syncytial cycles 9 and 10.

156 rther, the checkpoint is capable of delaying nuclear division even when SWE1 transcription is deregul

157 This results in nuclear division failure, failure to replicate DNA, and

158 ut instead triggered DNA synthesis, abnormal nuclear divisions, failure of cytokinesis, and emergence

159 During meiosis, two consecutive nuclear divisions follow a single round of deoxyribonucl

160 sac development begins with a phase of free nuclear division followed by cellularization and differe

161 leus and GFP-Cdc24p levels diminished before nuclear division followed by its reappearance in divided

162 rid undergoes multiple rounds of synchronous nuclear division, followed by cellularization, to create

163 e endosperm originates with a series of free-nuclear divisions, followed by cellularisation and subse

164 osis, a diploid cell undergoes two rounds of nuclear division following one round of DNA replication

165 owth of actin and microtubule networks after nuclear division generates reordering forces that counte

166 cytokinesis, and binucleated cells in which nuclear division had occurred without cell division.

167 lus nidulans become competent for growth and nuclear division in a process called conidial germinatio

168 In contrast, the timing of nuclear division in budded cells does not rely on Cdc28

169 The timing of nuclear division in cells that cannot make a bud is exqu

170 The pachytene checkpoint prevents meiotic nuclear division in cells that fail to complete meiotic

171 pecialized proteins are necessary for proper nuclear division in large dividing blastomeres.

172 unctions in a checkpoint pathway that delays nuclear division in response to defects in these events.

173 gous recombination repair to ensure accurate nuclear division in S. pombe.

174 f genes known to govern spindle assembly and nuclear division in spermatocytes.

175 Although the meiotic nuclear division in the ems1 mutant is normal, the micro

176 s of the SPO12 gene, which regulates meiotic nuclear divisions in budding yeast.

177 cycle components that result in synchronized nuclear divisions in Neurospora.

178 We establish that the nuclear divisions in syncytial Drosophila embryos are ac

179 cyclin E (cye-1) expression is critical for nuclear divisions in the intestine and is elevated in do

180 homolog lin-35 in negative regulation of the nuclear divisions in the intestine of C. elegans.

181 eLa cells blocked cellular cleavage, but not nuclear division, indicating a functional role for PRC1

182 Rereplication occurs prior to the meiotic nuclear divisions, indicating that this process is diffe

183 Unlike wild type, nuclear division is asynchronous in ago-2 embryos and th

184 o ensure that cytokinesis onset occurs after nuclear division is complete.

185 lins in mitosis and meiosis I, in which each nuclear division is coupled with a peak of expression of

186 lly unstable, suggesting that the process of nuclear division is error prone.

187 The lag in polar nuclear divisions is independent of grapes, which is req

188 The rigid matrix facilitated nuclear division (karyokinesis) leading to binucleation,

189 growth, induced mitotic arrest and aberrant nuclear division leading to decreased survival, and incr

190 gametophytes have a prolonged phase of free nuclear divisions leading to a variety of embryo sac abn

191 Drosophila syncytial nuclear divisions limit transcription unit size of early

192 e is not followed by cell division, although nuclear division may occur.

193 core of the meiotic process is a specialized nuclear division (meiosis I) in which homologs pair with

194 ave been shown to have roles in coordinating nuclear division, membrane trafficking and organizing th

195 to form germ cells-germ-plasm formation and nuclear division/migration.

196 ated, spo12 mutants undergo a single meiotic nuclear division most closely resembling meiosis II.

197 In Drosophila, rapid synchronous nuclear divisions must pause at the MBT to allow the for

198 us, Aspergillus nidulans, multiple rounds of nuclear division occur before cytokinesis, allowing an u

199 In Arabidopsis, two nuclear divisions occur before simultaneous cytokinesis

200 In most insects, however, rapid nuclear divisions occur in the absence of cytokinesis, a

201 s over time, and show that a large number of nuclear divisions occur in the evening.

202 vision called endomitosis, in which repeated nuclear divisions occur without cytokinesis or laying do

203 living cells and observe, track, and compare nuclear division of asexual and sexual stage parasites.

204 revented polarized growth but did not affect nuclear division of germinating spores.

205 that it may regulate oogenesis or the early nuclear divisions of embryogenesis.

206 paration gradually decreases during the four nuclear divisions of the blastoderm.

207 fragmented intestinal nuclei due to aberrant nuclear division, or defective karyokinesis.

208 recombination rates, an extended duration of nuclear division, or homolog chromosome pairing.

209 n used a histone hH1-GFP reporter to observe nuclear divisions over time, and show that a large numbe

210 pronuclear fusion, and anchoring postzygotic nuclear division products to the posterior cell cortex.

211 The homologous-pairing protein2/meiotic nuclear division protein1 (HOP2/MND1) protein complex ha

212 that the ability of this checkpoint to delay nuclear division requires the SWE1 gene, encoding a prot

213 roliferation of the parasites by controlling nuclear division, segregation, and cytokinesis.

214 ed snxA-snxD for suppressor of nimX, affects nuclear division, septation, and conidiation.

215 to germinate but had a limited capacity for nuclear division, suggesting a cell cycle defect.

216 o arrest by serum-withdrawal, EBNA3C induces nuclear division that is often divorced from cytokinesis

217 Although failing at nuclear division, these mutants septate and divide.

218 gnu (GNU embryos) under DNA synthesis but no nuclear division; this leads to the formation of a small

219 We propose that the checkpoint delays nuclear division through post-translational regulation o

220 ot chromosomes, in determining the number of nuclear divisions to execute.

221 They enlarge and undergo numerous nuclear divisions to form a spherical structure, and the

222 entally regulated, reaching a maximum during nuclear division towards the end of the intraerythrocyti

223 mstances, the morphogenesis checkpoint halts nuclear division until bud formation can resume.

224 s checkpoint, which blocks further rounds of nuclear division until cytokinesis is completed.

225 Spo13p is thought to act by delaying nuclear division until sister centromeres/chromatids und

226 stances, the morphogenesis checkpoint delays nuclear division via the mitosis-inhibitory Wee1-family

227 n typical nucleosomal structures, defects in nuclear divisions were restricted to mitotically dividin

228 Sic1 provides a failsafe mechanism promoting nuclear division when levels of mitotic cyclins are low.

229 is the exit from prophase and entry into the nuclear divisions, which in the yeast Saccharomyces cere

230 Presumably as a result of undergoing nuclear division with improperly oriented spindles, a la

231 r chromosome exchange separation and mitotic nuclear division with the upregulation of INCENP and CDC

232 on in mitosis, Nop15p-depleted cells undergo nuclear division with wild-type kinetics, activate the m

233 the mutant mother frequently show disrupted nuclear divisions with defects in chromosome condensatio

234 re, mutant cells arrest in the cell cycle at nuclear division, with a large bud, a single nucleus wit

235 opmental arrest, usually during the first 13 nuclear divisions, with a nuclear phenotype of chromatin

236 of a syncytium through successive rounds of nuclear division without cell wall synthesis.

237 with multiple rounds of DNA replication and nuclear division without cytokinesis, resulting in a mul

238 since it differs from mitosis in having two nuclear divisions without an intervening S-phase.

239 y binucleate, trimeras underwent coordinated nuclear division yielding four daughter nuclei, two of w