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1 re disassembly begins several minutes before nuclear envelope breakdown.
2 change in turn is approximately 30 s before nuclear envelope breakdown.
3 ctivate nuclear centering, DNA synthesis, or nuclear envelope breakdown.
4 cated by lack of chromosome condensation and nuclear envelope breakdown.
5 gets critical for chromatin condensation and nuclear envelope breakdown.
6 to the mitotic cytoplasm around the time of nuclear envelope breakdown.
7 ion of the mitotic spindle and the extent of nuclear envelope breakdown.
8 spindle formation, Golgi fragmentation, and nuclear envelope breakdown.
9 rapid unification of the parental genomes on nuclear envelope breakdown.
10 g it off the apical and lateral cortex after nuclear envelope breakdown.
11 nates in mitotic slippage without completing nuclear envelope breakdown.
12 ccess to the genome through DNA binding upon nuclear envelope breakdown.
13 y the release of a nuclear pool of eIF1 upon nuclear envelope breakdown.
14 eversible phase that was concurrent with the nuclear envelope breakdown.
15 destroyed in prometaphase within minutes of nuclear envelope breakdown.
16 in interfaces to promote NPC disassembly and nuclear envelope breakdown.
17 ssembly of the nuclear lamina and subsequent nuclear envelope breakdown.
18 T) capture typically occur within minutes of nuclear envelope breakdown.
19 sassembly was not due to a general defect in nuclear envelope breakdown.
20 nuclear protein import and plays roles after nuclear envelope breakdown.
21 es at the nuclear periphery and an arrest of nuclear envelope breakdown.
22 dely used in vitro to study the mechanics of nuclear envelope breakdown.
23 upon nuclear entry of cyclin B1, but before nuclear envelope breakdown.
24 o localizes to unattached kinetochores after nuclear envelope breakdown.
25 ow that centrosomes have a role in promoting nuclear envelope breakdown.
26 chromosome condensation from late G2 through nuclear envelope breakdown.
27 followed by accumulation in the nucleus then nuclear envelope breakdown.
28 microtubules and F-actin, disassembles upon nuclear-envelope breakdown.
29 maintain long centrosomal microtubules after nuclear-envelope breakdown.
31 ization with perinuclear microtubules before nuclear envelope breakdown, after which it congresses in
32 mmetric microtubule activities and polarized nuclear envelope breakdown allow for the preferential re
34 eviously, PLK-1 was shown to be required for nuclear envelope breakdown and chromosome segregation in
36 the sperm chromatin after pronuclear fusion, nuclear envelope breakdown and formation of a bipolar sp
37 vertebrates, the addition of HEI10 inhibits nuclear envelope breakdown and mitotic entry in Xenopus
38 sis inhibitor, injection of CycB accelerates nuclear envelope breakdown and mitotic remodeling of the
39 iation with kinetochores appeared soon after nuclear envelope breakdown and persisted until late anap
40 oreover, SMPD4 depletion results in abnormal nuclear envelope breakdown and reassembly during mitosis
43 Golgi, has been implicated in the process of nuclear envelope breakdown and requires interactions at
44 omosome losses in the long gap phase between nuclear envelope breakdown and the onset of spindle asse
45 uring mitosis, fluorescent tracers that mark nuclear envelope breakdown and the subsequent reformatio
46 cyte cytoskeleton during prophase I prior to nuclear envelope breakdown, and (2) extensive depolymeri
47 on of the retinoblastoma protein and lamins, nuclear envelope breakdown, and duplication of centrosom
48 clear import of the subviral complex without nuclear envelope breakdown, and integration of the viral
49 DNA damage can occur either before or after nuclear envelope breakdown, and provides an effective bl
50 leus during interphase, the cortex following nuclear envelope breakdown, and the cleavage furrow duri
51 s and orients a pair of centrosomes prior to nuclear envelope breakdown, and the spindle assembles be
53 hroughout prophase, but is disassembled upon nuclear-envelope breakdown as the mitotic spindle forms.
54 ould then be in a position to participate in nuclear envelope breakdown, as described in recent studi
55 characterized by chromosome recondensation, nuclear envelope breakdown, assembly of microtubules int
56 WAVE1 redistributes to the cytoplasm upon nuclear envelope breakdown at mitosis, and concentrates
57 before fertilization caused arrest prior to nuclear envelope breakdown at much lower concentrations
58 rom the cell nucleus in interphase but, upon nuclear envelope breakdown at the onset of mitosis, gain
59 pombe undergoes closed mitosis but 'virtual nuclear envelope breakdown' at anaphase of meiosis II, i
60 cell cycle and a tendency to arrest prior to nuclear envelope breakdown, at metaphase and at cytokine
61 ulum and the nuclear membrane contributes to nuclear envelope breakdown by coupling spindle elongatio
62 s: pronuclear meeting occurred normally, but nuclear envelope breakdown, centrosome separation, and c
63 not coupled to nuclear osmolytes released by nuclear envelope breakdown, chromatin condensation, or c
64 clusters appeared transiently at the time of nuclear envelope breakdown, disappeared at the time of f
65 r data further show that PLK-1 is needed for nuclear envelope breakdown during early embryogenesis.
66 ins, and Ce-lamin to determine the timing of nuclear envelope breakdown during mitosis in C. elegans.
67 iated lamin phosphorylation is important for nuclear envelope breakdown during mitosis, less is known
69 n transition begins with germinal vesicle or nuclear envelope breakdown (GVBD) and is critical for oo
70 port a role for the COPI coatomer complex in nuclear envelope breakdown, implicating vesiculation as
72 COPI and dominantly inhibits progression of nuclear envelope breakdown in an assay that robustly rec
75 n a closed mitosis therefore closely mirrors nuclear envelope breakdown in open mitosis(3), revealing
76 ystem that recapitulates CDK1 activation and nuclear envelope breakdown in response to mitotic cyclin
77 depletion of Myt1 in Xenopus oocytes causes nuclear envelope breakdown in vitro, we found that deple
78 We propose a new model for the mechanism of nuclear envelope breakdown in which disassembly of the n
79 membrane is compartmentalized shortly before nuclear envelope breakdown into an anterior and a poster
85 apturing microtubules, signals released upon nuclear envelope breakdown may activate proteins like Su
86 sis biosensor that monitors the time between nuclear envelope breakdown (NEB) and re-formation using
88 , these cells display mitotic collapse after nuclear envelope breakdown (NEB) characterized by defect
90 ems, the possibility of poleward flux before nuclear envelope breakdown (NEB) has not been examined.
99 in interphase and prophase but released upon nuclear envelope breakdown (NEBD) by the action of Cdk1.
100 sed the nuclear envelope and interfered with nuclear envelope breakdown (NEBD) during cell division.
101 microtubules gain access to chromatin after nuclear envelope breakdown (NEBD) during meiotic maturat
102 pendent role for Aurora A and centrosomes in nuclear envelope breakdown (NEBD) during the first mitot
103 irectly observed from prophase to just after nuclear envelope breakdown (NEBD) in early prometaphase.
109 w that Endos must be in the cytoplasm before nuclear envelope breakdown (NEBD) to be efficiently phos
110 positioning of duplicated centrosomes after nuclear envelope breakdown (NEBD), thereby preventing th
111 C first targets cyclin A2 for degradation at nuclear envelope breakdown (NEBD), we find that in zygot
119 a role for this pore protein in coordinating nuclear envelope breakdown, Nup358-specific antibodies i
124 thways, defined by centrosome maturation and nuclear envelope breakdown, plays any role in spindle as
125 reases in mice after birth, is essential for nuclear envelope breakdown prior to progression to metap
126 of Golgi spots begins to decline soon after nuclear envelope breakdown, reaches a minimum soon after
127 KIF11 further fragments the MTOCs following nuclear envelope breakdown so that they can be evenly di
128 h-18 mutant oocytes exhibit defects prior to nuclear envelope breakdown, suggesting that they are phy
129 her Nup153 or Nup358 for inhibition perturbs nuclear envelope breakdown, supporting a model in which
130 aster labeling was constant from the time of nuclear envelope breakdown, the kinetochore labeling fir
132 ted pronuclear centering, DNA synthesis, and nuclear envelope breakdown; there appeared to be a thres
133 G2 phase and activated at a set time before nuclear envelope breakdown, thereby initiating the event
134 nucleus during late prophase (<30 min before nuclear envelope breakdown) they progress normally throu
136 of metaphase cells, and traversal times from nuclear envelope breakdown to anaphase, and an override
138 t it can be recruited to kinetochores before nuclear envelope breakdown to maintain genomic stability
139 sphodynein associates with kinetochores from nuclear envelope breakdown to metaphase, but bioriented
140 rocesses ranging from vesicular transport to nuclear envelope breakdown to mitotic spindle alignment.
141 envelope and restricts the process of local nuclear envelope breakdown to the bridge midzone to prev
142 ng the first meiotic division, shortly after nuclear envelope breakdown, translational hotspots devel
145 in mitosis, KAP-GFP moved into nuclei before nuclear envelope breakdown, was again present in nuclei
146 associates with chromatin, especially after nuclear envelope breakdown when cells enter mitosis.
147 into mitosis disrupts viral assembly due to nuclear envelope breakdown, which disperses VP2 througho
148 the nuclear envelope, a process analogous to nuclear envelope breakdown, which occurs at the onset of
149 rier to mitotic establishment corresponds to nuclear envelope breakdown, which requires a decisive sh
150 iotic resumption, Plk1 cKO oocytes underwent nuclear envelope breakdown with the same timing as contr