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2 d testis-associated receptor (RTR)/germ cell nuclear factor is a nuclear orphan receptor that plays a
4 differentiation-related transcription factor nuclear factor I-A (NFI-A) controls MDSC expansion durin
6 itional deletion of the transcription factor nuclear factor I-A (NFIA) in astrocytes in the adult bra
7 tive astrocytes, we investigated the role of nuclear factor I-A (NFIA), a key transcriptional regulat
8 AA) ensembles exhibit elevated expression of nuclear factor I-A, and its selective deletion from this
10 n the flanking 5' untranslated region of the nuclear factor I/A (NFIA) gene, a strong candidate gene
11 /Notch-like EGF repeat containing (Dner) and nuclear factor I/A (Nfia), that are each heavily express
13 ir core element containing binding sites for nuclear factor I- and nuclear receptor-like factors.
14 rotein gene promoters have binding sites for nuclear factor I, as well as the glucocorticoid receptor
17 avian myeloblastosis viral oncogene homolog-nuclear factor I/B (MYB-NFIB) gene fusion that activates
18 lly cell-specific isoform usage, identifying nuclear factor I/B (NFIB) as a regulator of megakaryocyt
19 s NSC expression of the transcription factor nuclear factor I/B (NFIB) by binding to sequences in the
20 of a novel, oncogenic transcription factor, Nuclear factor I/B (Nfib), in the mouse SCLC model and i
22 The discovery of the unique role of Nfic (nuclear factor I C; a transcriptional factor) in control
24 cing (scATAC-seq) analysis reveals that NFI (nuclear factor I) factors repress Sonic hedgehog (Shh) a
25 lear Factor IA, a member of the long-studied Nuclear Factor I family of DNA-binding proteins, plays p
26 ma and show that an epigenomic shift renders Nuclear Factor I family of transcription factors oncogen
27 s of transforming cells in mice, we identify nuclear factor I family of transcription factors, known
28 sive study, the DNA recognition mechanism of Nuclear Factor I family proteins remains unresolved.
31 oRE have demonstrated the importance of both nuclear factor I (NFI) and STAT5 as well as cooperative
32 r region contains critical binding sites for nuclear factor I (NFI) and thyroid transcription factor
34 , CpG-dinucleotide content (CpGs), potential nuclear factor I (NFI) binding sites, and potential Z-DN
36 and -153 on the TTF-1 promoter contains two nuclear factor I (NFI) elements whose function is involv
46 he previously identified binding site of the nuclear factor I (NFI) family of transcription factors.
56 mal transcriptional activation potentials of nuclear factor I (NFI) proteins encoded by each of the f
61 re TFs of the basic helix-loop-helix (bHLH), nuclear factor I (NFI), SOX, and FOX families, with CREs
62 ing sites, including seven binding sites for nuclear factor I (NFI), the specific sequences within th
63 ation are up-regulated by hypophosphorylated nuclear factor I (NFI), which is dephosphorylated by the
64 onal progenitors, DPF2-S sites were bound by nuclear factor I (NFI), while DPF2-L sites were bound by
65 in the fragment -612/-580, which contains a nuclear factor I (NFI)-binding site at the region -594/-
66 dentified multiple, developmentally distinct nuclear factor I (NFI)-containing protein complexes from
69 orming regions (ZDRs) and promoter sites for nuclear factor-I (NFI) are correlated with the locations
71 microarray analyses, here we have shown that nuclear factor I, subtype A (NFI-A), a member of the NFI
72 upregulation of the genes encoding Sox9 and nuclear factor I transcription factors during developmen
74 rs, enhancer of zeste homologue 2 (EZH2) and nuclear factor I X (NFIX), coordinates with the cis-regu