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1 tivated protein kinases as well as NFkappaB (nuclear factor kappa B).
2 ak1 (p21-activated kinase -1) --> NF-kappaB (nuclear factor-kappa B).
3 -1, tribbles 3, reactive oxygen species, and nuclear factor kappa B)?
4 s both C and T alleles were found to bind to nuclear factor kappa B.
5 phosphorylation and nuclear translocation of nuclear factor kappa B.
6 opolysaccharide (LPS) -induced activation of Nuclear Factor kappa B.
7 togen-activated protein kinase and activates nuclear factor kappa B.
8 n of both interferon regulatory factor 3 and nuclear factor kappa B.
9 bition of glycogen synthase kinase-3beta and nuclear factor kappa B.
10 D91 triggers signalling cascades to activate nuclear factor-kappa B.
11 rivative of dithiocarnamate, an inhibitor of nuclear factor- kappa B.
12 hanism for induction of CDX2 is dependent on nuclear factor kappa B, a crucial transcription factor i
14 ced persistent inflammation by circumventing nuclear factor kappa B activation and its downstream cyt
15 bic acid and dehydroascorbic acid attenuated nuclear factor kappa B activation and normalized coagula
17 e course plots of Lipopolysaccharide-induced Nuclear Factor kappa B activation in mouse embryo fibrob
18 ardiac mitochondrial respiratory efficiency, nuclear factor kappa B activation, and caspase activitie
20 nhibited tumor necrosis factor-alpha induced nuclear factor-kappa B activation by 27.5% at 2 mg/ml, w
21 cyclophosphamide with mecamylamine suggested nuclear factor-kappa B activation in the chronic inflamm
22 tinamide effectively attenuated postischemic nuclear factor-kappa]B activation and exhibited robust a
23 ects that were accompanied by a reduction in nuclear factor kappa B activity and cell surface levels
24 ecrease in hepatic fibrosis (p < 0.001), and nuclear factor-kappa B activity (p < 0.05) as compared w
26 location of transcription factors, including nuclear factor kappa B, although its role in PCa was lar
27 mitogen-activated protein (MAP) kinases and nuclear factor kappa B and decreased myogenic differenti
28 ecule 1-dependent and leads to activation of nuclear factor kappa B and extracellular signal-regulate
29 nate attenuated the HS-induced activation of nuclear factor kappa B and reduced the expression of pro
30 itions, interferonopathies, and disorders of nuclear factor kappa B and/or aberrant TNF activity.
31 Immunofluorescence showed colocalization of nuclear factor kappa-B and OCN in diseased and normal va
32 crosis factor alpha]), as well as NF-kappaB (nuclear factor kappa B) and inflammasome activation.
33 reby enabling constitutive activation of the nuclear factor-kappa B, and contributing to chronic infl
34 ity of activated glucocorticoid receptor and nuclear factor kappa-b appeared to join pre-existing P30
35 es were associated with increased NF-kappaB (nuclear factor kappa B) binding activity and expression
36 tein, osteoprotegerin, receptor activator of nuclear factor kappa B, bone morphogenic protein 2, and
37 Our data demonstrate that miR-146a controls nuclear factor kappa B-dependent inflammatory responses
39 insulin receptor, which in turn activates a nuclear factor kappa B-dependent metabolic pathway, lead
40 (Hz), signals NOS induction through ERK- and nuclear factor kappa B-dependent pathways and has been d
41 human peripheral blood mononuclear cells and nuclear factor kappa B-dependent transcription in human
44 t HEK293 cells, HEK293-TLR2 cells had marked nuclear factor-kappa B-driven luciferase activity, had m
45 in inflammatory and cancer pathways, such as nuclear factor kappa B, EGF, Wnt, and B-cell lymphoma 2.
47 ee text] ([Formula: see text]) signaling via nuclear factor kappa-B ([Formula: see text]) hallmarks (
49 and III IFN responses, such as inhibitor of nuclear factor kappa B (IkappaB) kinase alpha (IKKalpha)
51 nhibitor PD98059, the Src inhibitor pp2, the nuclear factor- kappa B inhibitor caffeic acid phenethyl
53 crovascular density and higher expression of nuclear factor kappa B, interleukin-6, interleukin-8, tr
54 ylated and thereby inhibited by inhibitor of nuclear factor kappa B kinase subunit beta (IKKbeta) to
55 howed that Hsp90 interacts with inhibitor of nuclear factor kappa-B kinase (IKK) together with cochap
56 Pharmacological inhibition of inhibitor of nuclear factor kappa-B kinase subunit beta (IKKbeta), bu
58 ncreased expression of receptor activator of nuclear factor kappa B ligand (RANK-L) due to pro-inflam
61 on and inactivation of Receptor Activator of Nuclear factor Kappa B Ligand (RANKL) and osteoprotegeri
62 clast (OC) response to receptor activator of nuclear factor kappa B ligand (RANKL) and thus bone meta
63 osis factor (TNF), and receptor-activator of nuclear factor kappa B ligand (RANKL) were significantly
64 one marrow cultures to receptor activator of nuclear factor kappa B ligand (RANKL), macrophage colony
65 nonoclonal antibody to receptor activator of nuclear factor kappa B ligand, has completed its major f
66 nsing receptor and the receptor activator of nuclear factor kappa B ligand/receptor activator of nucl
67 that stimulation with receptor activator of nuclear factor kappa-B ligand (RANKL) resulted in a robu
68 oxidase (MPO), and the cytokine receptor for nuclear factor kappa-B ligand (RANKL) were significantly
69 igated if ILCs express receptor activator of nuclear factor kappa-B ligand (RANKL), a cytokine crucia
70 ze salivary levels of receptor activator for nuclear factor kappa-B ligand (RANKL), osteoprotegerin,
71 ted T cells to produce receptor activator of nuclear factor kappa-B ligand (RANKL), which, in turn, p
72 TNF-alpha), as well as receptor activator of nuclear factor kappa-B ligand (RANKL)-induced osteoclast
73 tal tissue and induces receptor activator of nuclear factor kappa-B ligand (RANKL)-RANK-osteoproteger
74 human monoclonal anti-receptor activator of nuclear factor kappa-B ligand antibody, with zoledronic
75 Change in circulating receptor activator of nuclear factor kappa-B ligand was higher in the interven
76 chemotactic protein 1, receptor activator of nuclear factor kappa-B ligand, and macrophage colony-sti
77 ctivator-related gene, receptor activator of nuclear factor kappa-B ligand, and osteoblast differenti
78 lcin, osteoprotegerin, receptor activator of nuclear factor kappa-B ligand, vascular endothelial grow
79 factor (TNF)-alpha and receptor activator of nuclear factor kappa-B ligand; these cytokines were also
80 issue homogenates, and receptor activator of nuclear factor-kappa B ligand (RANKL) activation was ana
82 , expression of activated receptor activator nuclear factor-kappa B ligand (RANKL) and bone density i
84 ) PBMCs was induced by receptor activator of nuclear factor-kappa B ligand (RANKL), either alone or i
85 osteoprotegerin (OPG)/receptor activator of nuclear factor-kappa B ligand (RANKL), increased osterix
86 histochemistry for the receptor activator of nuclear factor-kappa B ligand (RANKL), osteoprotegerin (
87 ranscription factor-2, receptor activator of nuclear factor-kappa B ligand (RANKL), sclerostin, and D
88 LR-4, osteoprotegerin, receptor activator of nuclear factor-kappa B ligand (RANKL), tumor necrosis fa
90 osorbent assay for: 1) receptor activator of nuclear factor-kappa B ligand (RANKL); 2) osteoprotegeri
91 (IL)-6, IL-8, soluble receptor activator of nuclear factor-kappa B ligand (sRANKL), osteoprotegerin
92 mmunoreactivity to the receptor activator of nuclear factor-kappa B ligand at day 7 post-treatment, s
93 nuclear factor-kappa B-receptor activator of nuclear factor-kappa B ligand interaction for osteoclast
94 interleukin-1beta and receptor activator of nuclear factor-kappa B ligand than group EP-HN019 (P <0.
96 lectrophoresis; RANKL, receptor activator of nuclear factor kappa B-ligand; NFATc1, nuclear factor of
97 eraction with cyclooxygenase-2 (COX-2), p65- nuclear factor kappa B, lipoxygenase-1 (LOX-1) and toll-
98 eracting protein 2) to coordinate NF-kappaB (nuclear factor kappa B)-mediated cytokine responses.
99 ulting in de novo lipogenesis, and increased nuclear factor kappa B-mediated inflammation act in conc
100 enomena at the organismal level, we assessed nuclear factor kappa B (NF-kappaB) activation (indicativ
101 ction in cultured hepatoma cells, leading to nuclear factor kappa B (NF-kappaB) activation and the pr
103 ogesterone exhibited an inhibitory effect on nuclear factor kappa B (NF-kappaB) activation following
104 intestinal epithelial cells and also induced nuclear factor kappa B (NF-kappaB) activation in HEK293T
107 at least three different pathways leading to nuclear factor kappa B (NF-kappaB) activation, apoptosis
112 the expression of genes reflecting enhanced nuclear factor kappa B (NF-kappaB) activity were noted i
113 r 4), interferon regulatory factor-3 (IRF3), nuclear factor kappa B (NF-kappaB) activity, and proinfl
114 ecific lipin-1 deficiency diminished hepatic nuclear factor kappa B (NF-kappaB) activity, limited liv
116 uclear export sequence (NES) of inhibitor of nuclear factor kappa B (NF-kappaB) alpha (IkappaBalpha)
117 alyses across technologies demonstrated that nuclear factor kappa B (NF-kappaB) and cholesterol biosy
122 -protein interaction (PPI) interface between nuclear factor kappa B (NF-kappaB) essential modulator (
124 TRF also showed a greater inhibition on the nuclear factor kappa B (NF-kappaB) expression than T and
125 alling pathways leading to activation of the nuclear factor kappa B (NF-kappaB) family of transcripti
130 a 12,14- Prostaglandin J2 (15dPGJ2) inhibits Nuclear factor kappa B (NF-kappaB) in human myocytes and
138 We utilized a transgenic mouse model where nuclear factor kappa B (NF-kappaB) is selectively inhibi
139 mily members such as p38 and JNK and induced nuclear factor kappa B (NF-kappaB) pathway activation.
141 (IL)12 or IL18 as well as inhibition of the nuclear factor kappa B (NF-kappaB) pathway reversed NK c
142 ) 3' untranslated region (UTR) and activates nuclear factor kappa B (NF-kappaB) pathways that contrib
143 cycle progression and I-kappa B kinase (IKK)/nuclear factor kappa B (NF-kappaB) signaling contributes
144 MRV miRNAs, we screened for their effects on nuclear factor kappa B (NF-kappaB) signaling in the pres
145 biquitinated chains from key proteins in the nuclear factor kappa B (NF-kappaB) signaling pathway.
148 We used a chick in vivo model to investigate nuclear factor kappa B (NF-kappaB) signaling, a critical
149 active and supports TGF-beta-induced EMT and nuclear factor kappa B (NF-kappaB) signaling, whereas th
151 Here we demonstrate that the dynamics of Nuclear Factor kappa B (NF-kappaB) signalling and the ce
154 CD47 expression was found to be regulated by nuclear factor kappa B (NF-kappaB) through a specific re
156 phate (NAD(P)H)-oxidase and translocation of nuclear factor kappa B (NF-kappaB) were also evaluated.
158 immune pathways that are primarily driven by nuclear factor kappa B (NF-kappaB), interferon regulator
159 acts as an upstream activator of astroglial nuclear factor kappa B (NF-kappaB), leading to the relea
161 rophages enhance myofibroblast survival in a nuclear factor kappa B (NF-kappaB)-dependent manner and
169 ated the hepatocellular damage by inhibiting nuclear factor kappa B (NF-kappaB)/cyclooxygenase 2 sign
171 decrease in PKC activity, and inhibition of nuclear factor kappa B (NF-kB) translocation, were attri
173 glucocorticoid insensitivity: respectively, nuclear factor kappa B (NF-kB), cAMP response element-bi
174 cluding TNF-alpha and IL-6 and activation of nuclear factor kappa B (NF-kB/p65), p38 mitogen-activate
176 gulators linked to TNF-alpha/TNFR signaling, nuclear factor kappa-B (NF-kappaB) activation, autophagy
177 ll receptor (BCR) signaling and constitutive nuclear factor kappa-B (NF-kappaB) activation, which is
178 response element-binding protein (CREB) and nuclear factor kappa-B (NF-kappaB) are two ubiquitous tr
179 ron regulatory factor 3 (IRF-3)-mediated and nuclear factor kappa-B (NF-kappaB) inhibitor (IkappaBalp
180 NOS1 is complex but recent data suggest that nuclear factor kappa-B (NF-kappaB) is an important trans
181 recent Immunity articles probe the roles of Nuclear Factor kappa-B (NF-kappaB) pathways in survival
182 egulated kinase (ERK), calcium (Ca(2+)), and nuclear factor kappa-B (NF-kappaB) pathways, response dy
184 transcripts and an increase in inhibitors of nuclear factor kappa-B (NF-kappaB) signaling, possibly i
186 dly induces co-occupancy of KDM7A and UTX at nuclear factor kappa-B (NF-kappaB)-associated elements i
189 ion of RelA (p65), thus repressing oncogenic nuclear factor kappa-B (NF-kB) signaling and inducing ap
190 ad of colonic tumors and increased STAT3 and nuclear factor-kappa B (NF-kappaB) activation in colon.
191 d mitogen-activated protein (MAP) kinase and nuclear factor-kappa B (NF-kappaB) activation, and the s
194 carcinoma cells (T24 cells), and TPA-induced nuclear factor-kappa B (NF-kappaB) activity with human h
195 idermis of mice lacking transcription factor nuclear factor-kappa B (NF-kappaB) activity, primary hai
196 the activation of the transcription factors nuclear factor-kappa B (NF-kappaB) and activator protein
197 ed PHB expression on TNF-alpha activation of nuclear factor-kappa B (NF-kappaB) and epithelial barrie
198 ithelia in Hashimoto's thyroiditis activates nuclear factor-kappa B (NF-kappaB) and induces pro-infla
199 GF), inducible nitric oxide synthase (iNOS), nuclear factor-kappa B (NF-kappaB) and intercellular adh
202 genotoxic and oncogenic stress activates the nuclear factor-kappa B (NF-kappaB) and p53 proteins; how
203 amic interplay of two transcription factors, nuclear factor-kappa B (NF-kappaB) and peroxisome prolif
204 UPR, there was transcriptional regulation by nuclear factor-kappa B (NF-kappaB) and RNA stabilization
205 t-7b cluster and identified several putative nuclear factor-kappa B (NF-kappaB) consensus elements.
207 tion of proinflammatory transcription factor nuclear factor-kappa B (NF-kappaB) in 7-week-old mdx mic
209 imately 40,000 genes, and tested the role of nuclear factor-kappa B (NF-kappaB) in maintaining an act
210 ic proteins and the proinflammatory molecule nuclear factor-kappa B (NF-kappaB) in the retina of Lewi
212 we have shown that the transcription factor nuclear factor-kappa B (NF-kappaB) is activated by BCR-A
213 affects expression of receptor activator of nuclear factor-kappa B (NF-kappaB) ligand (RANKL), a pot
215 constitutive activation of the antiapoptotic nuclear factor-kappa B (NF-kappaB) pathway, which can in
216 ne of these main signaling mechanisms is the nuclear factor-kappa B (NF-kappaB) pathway, which integr
219 d activators of transcription 1 (STAT1), and nuclear factor-kappa B (NF-kappaB) signaling in Lilrb3(-
220 D137 ligand and CD40 initiates activation of nuclear factor-kappa B (NF-kappaB) signaling in tumor ce
221 dy, we show that the activation of classical nuclear factor-kappa B (NF-kappaB) signaling increases t
222 nvading pathogens, their ability to regulate nuclear factor-kappa B (NF-kappaB) signaling, interleuki
224 AT3, c-jun n-terminal kinases (JNK), EKR1/2, nuclear factor-kappa B (NF-kappaB)) in the gastrocnemius
226 eading to activation of transcription factor nuclear factor-kappa B (NF-kappaB), which is a central r
227 like receptor 4 (TLR4)-dependent pathway via nuclear factor-kappa B (NF-kappaB), while simultaneously
228 so stimulated IFN-beta promoter activity and nuclear factor-kappa B (NF-kappaB)-dependent transcripti
233 cell cycle and prosurvival pathways (such as nuclear factor kappa B [NF-kappaB], AKT), pathways regul
234 monstrated a signaling pathway from HIF-1 to nuclear factor kappa B (NFkappaB) activation to enhanced
236 f intrapancreatic trypsinogen activation and nuclear factor kappa B (NFkappaB) activation, the two ma
239 in terms of interleukin (IL)-8 promoter and nuclear factor kappa B (NFkappaB) activity, were observe
240 IkappaBalpha enhances the rate of release of nuclear factor kappa B (NFkappaB) from DNA target sites
242 tion by laminar shear stress is dependent on nuclear factor kappa B (NFkappaB) subunits p50 and p65 b
243 a) is a potent cytokine that signals through nuclear factor kappa B (NFkappaB) to activate a subset o
244 or (TRPV1) and Ca(2+) to microglial IL-6 and nuclear factor kappa B (NFkappaB) translocation with ele
246 nt protein deacetylase sirtuin-6 (SIRT6) and nuclear factor kappa B (NFkappaB), sequesters GR express
247 infection of hepatic cells by HCV stimulates nuclear factor kappa B (NFkappaB)-dependent production o
250 ivator of transcription factor 6 (STAT6) and nuclear factor-kappa B (NFkappaB) in mice and cells.
251 onally activated by mTOR complex 2 (mTORC2): nuclear factor-kappa B (NFkappaB) signaling cascade.
252 essing fibroblasts induced the activation of nuclear factor-kappa B (NFkappaB), a regulator fibronect
256 ese genes were involved in interferon alpha, nuclear factor-kappa B (NFkB), extracellular signal-rela
257 onase exerts an effect via downregulation of nuclear factor kappa B (NFKbeta) by specific microRNAs (
258 cally regulated by p38delta isoform, whereas nuclear factor kappa B (NFsmall ka, CyrillicB) pathway r
260 factors, such as glucocorticoid receptor and nuclear factor kappa-b, on the three-dimensional organiz
261 Cs), a phenomenon not mediated by changes to nuclear factor-kappa B or hydrogen sulfide but that occu
262 in the presence of antagonists of NFkappaB (nuclear factor kappa B) or iNOS activity, NO synthesis a
263 factor kappa B ligand/receptor activator of nuclear factor kappa B/osteoprotegerin system involved i
264 ich also correlated with increased levels of nuclear factor kappa B p52 and decreased levels of c-Jun
267 a4beta1, which resulted in signaling through nuclear factor kappa B p65, protein kinase B1, and p38 m
268 vo that depends on Ser536 phosphorylation of nuclear factor kappa B p65; this pathway may hold valuab
269 -0.59 (-0.8, -0.3) mmol/L; P < 0.001], total nuclear factor kappa-B p65 [-0.016 (-0.022, -0.011) comp
271 e pathway that, together with the Rho-kinase nuclear factor kappa B pathway (NF-kB), are required for
272 om platelets, particularly those part of the nuclear factor kappa B pathway, are associated with BMI,
273 s) induced the expression of EMT traits, the nuclear factor kappa B pathway, autophagy, and the loss
275 ied activating mutations in the noncanonical nuclear factor-kappa B pathway could give rise to ibruti
276 as 18q21 amplification or activation of the nuclear factor-kappa B pathway, as reported previously,
277 of the mitogen-activated protein kinase and nuclear factor kappa B pathways in MDSCs was MyD88 depen
278 rough regulating the activation of canonical nuclear factor kappa B, phosphatidylinositol-4,5-bisphos
279 phosphoinositide 3-kinase/phosphorylated Akt/nuclear factor kappa B (PI3K/pAkt/NFkappaB)-signaling pa
280 e immune system signaling pathways including nuclear factor-kappa B profoundly alter intestinal epith
281 Chronic alcohol consumption inhibited the nuclear factor kappa B proinflammatory cytokine pathway
284 id-state nanopore has been used to recognize Nuclear Factor-kappa B proteins (NF-kappaB), that are in
285 lonal antibody against receptor activator of nuclear factor kappa B (RANK) ligand, with zoledronic ac
286 omeostatic conditions, receptor activator of nuclear factor kappa-B (RANK) ligand on osteoblasts driv
287 uent bone loss via the receptor activator of nuclear factor-kappa B (RANK)-RANK ligand (RANKL)-osteop
288 f clinical parameters, receptor activator of nuclear factor kappa-B (RANKL) and osteoprotegerin (OPG)
289 on and location of the receptor activator of nuclear factor-kappa B (RANKL) and of osteoprotegerin (O
290 eractions, such as the receptor activator of nuclear factor-kappa B-receptor activator of nuclear fac
291 d transcription factors, such as Bcl-2, Bax, nuclear factor kappa B, regulate the decision-making pow
293 occurs through direct targeting of upstream nuclear factor kappa B signal transducers caspase recrui
294 es the proinflammatory transcription factors nuclear factor-kappa B, signal transducer and activators
295 owing C. parvum infection could activate the nuclear factor kappa B signaling pathway and trigger inf
297 K/NFkappaB (mitogen-activated protein kinase/nuclear factor-kappa B) signals participate in regulatin
298 -responsive element (HRE)) and inflammation (nuclear factor kappa B) to obtain a chimeric promoter na
299 diac tumor necrosis factor-alpha expression, nuclear factor kappa B translocation, obesity-induced pe
300 ession of TWEAK increased, the activation of nuclear factor-kappa B without affecting the activation