ライフサイエンスコーパス: nuclear layer

1 layers or cell number in the inner and outer nuclear layer.

2 emonstrated subsequent thinning of the inner nuclear layer.

3 ithelium disruption and/or loss of the outer nuclear layer.

4 y distinct and appropriately polarized outer nuclear layer.

5 man retina exhibited positivity in the inner nuclear layer.

6 bnormal vasculature extending into the outer nuclear layer.

7 r plexiform layer (OPL) and invade the outer nuclear layer.

8 ate measure of INL thickness), and the outer nuclear layer.

9 but accurately detected the undamaged outer nuclear layer.

10 SD-OCT were localized deeper than the inner nuclear layer.

11 omogeneous invisible tumors within the inner nuclear layer.

12 ence of a single layer of cones in the outer nuclear layer.

13 esses that aberrantly intrude into the outer nuclear layer.

14 histologically with a thinning of the inner nuclear layer.

15 f the RPE in the presence of an intact outer nuclear layer.

16 inimally preserved paracentral photoreceptor nuclear layer.

17 both individuals, and thinning of the outer nuclear layer.

18 ells) have their soma displaced to the inner nuclear layer.

19 layer and stopped at the level of the inner nuclear layer.

20 dead rod photoreceptors throughout the outer nuclear layer.

21 plexiform, and to a lesser extent the outer nuclear layer.

22 ation, and nearly complete loss of the outer nuclear layer.

23 of ganglion cell, inner plexiform, and inner nuclear layers.

24 receptors, occupy the ganglion cell or inner nuclear layers.

25 ory retina between outer plexiform and outer nuclear layers.

26 to be perifoveal and those within the outer nuclear layer (8/17 eyes, 47.1%) observed to be mostly e

27 To our knowledge, microcystic inner nuclear layer abnormalities have not been investigated i

28 tinal deposits that extend through the outer nuclear layer, affect photoreceptor integrity, and are a

29 neuron (RIN)--horizontal cells (HCs), inner nuclear layer amacrine cells (iACs) and displaced amacri

30 a retraction of rod spherules into the outer nuclear layer and a sprouting of rod bipolar cell dendri

31 s were associated with thinning of the inner nuclear layer and abnormalities of both layers were asso

32 ly result in permanent thinning of the inner nuclear layer and are critical to identify in order to d

33 l progenitor cells that migrate to the outer nuclear layer and become rod precursor cells that are co

34 r outer segments extending through the outer nuclear layer and even beyond the outer limiting membran

35 for about 1% of amacrine cells in the inner nuclear layer and for up to 27% of displaced amacrine ce

36 polar cell processes extended into the inner nuclear layer and ganglion cell layer by PNM3.5.

37 by the displacement of the soma to the inner nuclear layer and has morphological similarities with ei

38 bodies in the ganglion cell layer and inner nuclear layer and immunoreactive processes in the inner

39 xiform layer, inner nuclear layer, and outer nuclear layer and in pericytes of postdevelopment day 17

40 uorescein was present in somata in the inner nuclear layer and in synaptic boutons in the inner plexi

41 on of calretinin-positive cells in the inner nuclear layer and in the ganglion cell layer is glutamic

42 The outer nuclear layer and inner retinal thicknesses were normal.

43 ral abnormalities, including a thinned outer nuclear layer and intraretinal or subretinal fluid.

44 -) mice, and transgenics had a thicker outer nuclear layer and less sub-retinal pigment epithelium de

45 omato fluorescence was detected in the inner nuclear layer and localized to type 1, 3b, and 4 OFF bip

46 s were associated with widening of the outer nuclear layer and loss of the foveal depression.

47 ter disease stages were accompanied by inner nuclear layer and nerve fiber layer abnormalities.

48 Microscopically, the inner nuclear layer and outer plexiform layer were the most af

49 rence in the thickness of the combined outer nuclear layer and outer plexiform layer when we compared

50 parafoveal thickness, presence of the inner nuclear layer and outer segment, gestational age at birt

51 vity (IR) was present primarily in the inner nuclear layer and the ganglion cell layer.

52 especially the separation between the outer nuclear layer and the Henle fiber layer.

53 ar layer; the hypointense layer 2, the outer nuclear layer and the inner and outer segments; and the

54 dition, cone opsin mislocalized to the outer nuclear layer and the outer plexiform layer in the CNGB3

55 Between the nuclear layer and the yolk, the cytoplasm was homogeneou

56 cells located in the ganglion cell and inner nuclear layers and did not alter NMDA-induced PARP-1 hyp

57 multipotent progenitors that migrate to all nuclear layers and differentiate into layer-specific cel

58 (sGC), was in somata in the inner and outer nuclear layers and in both plexiform layers.

59 , inner plexiform layer, and inner and outer nuclear layers and within the spindle-shaped cell popula

60 lized rosette-like arrangements in the outer nuclear layer, and develop abnormal vascularization, bro

61 ontinue to proliferate, migrate to the outer nuclear layer, and differentiate into photoreceptors.

62 tive EYS secretory vesicles within the outer nuclear layer, and diminished EYS protein near the conne

63 mRNA is found in cells of the retinal inner nuclear layer, and immunofluorescent confocal microscopy

64 ganglia cells, outer plexiform layer, inner nuclear layer, and outer nuclear layer and in pericytes

65 ve fiber layer, ganglion cell complex, inner nuclear layer, and outer plexiform layer thickness.

66 especially in the outer nuclear layer, inner nuclear layer, and photoreceptors.

67 the outer plexiform layer (OPL) or the inner nuclear layer, and while present in the mature retina, a

68 ed to identify A amacrine cells in the inner nuclear layer as well as widefield amacrine and small bi

69 spike in apoptosis was observed in KO outer nuclear layer at P25.

70 ent wave of Smad1/5/8 signaling in the inner nuclear layer at the end of the first postnatal week, fr

71 tions trended to have retention of the outer nuclear layer at the fovea and macular thickening, espec

72 studies used hosts with a preexisting outer nuclear layer at the time of treatment.

73 the lesions had resolved into areas of inner nuclear layer atrophy with persistence of scotomas.

74 After birth the foveal outer nuclear layer became much thicker as cone packing occurr

75 he choroid, and leading to loss of the outer nuclear layer bordered by hyporeflective wedges.

76 form ectopic synapses with rods in the outer nuclear layer but lack bipolar dendrites.

77 th arrestin1 in the inner segments and outer nuclear layer, but remained in the inner segments when a

78 om amacrine cells, were present in the inner nuclear layer by PNM9.5.

79 plaquelike lesions at the level of the inner nuclear layer by spectral-domain OCT and showed correspo

80 pithelial cell integrity and prevented outer nuclear layer cell death as examined by histopathologic

81 rs primarily affects the generation of inner nuclear layer cell types, resulting in complete loss of

82 of nearly all differentiated inner and outer nuclear layer cell types.

83 SCs had fewer amacrine cells and other inner nuclear layer cells.

84 duced number of rod photoreceptors and inner nuclear layer cells.

85 ging an ellipsoid zone interruption or outer nuclear layer changes.

86 t growth factor (FGF)-2 in the photoreceptor nuclear layer coincided with the inflammatory response i

87 and increased ectopic synapses in the outer nuclear layer compared to controls.

88 layers and increased thickness in the inner nuclear layer compared with healthy subjects (P < .05).

89 number of photoreceptor nuclei in the outer nuclear layer compared with WT controls.

90 cells progressively accumulate in the outer nuclear layer concurrently with photoreceptor degenerati

91 GFA mRNA signal was located within the inner nuclear layer corresponding to CRALBP-labeled Muller cel

92 et measurements of immunofluorescence retina nuclear layers disclosed no significant differences betw

93 ions revealed hyperreflectivity of the outer nuclear layer; disruption of the external limiting membr

94 nating controls from NPDR; and (c) the inner nuclear layer distinguishes best between controls and No

95 bserved in the ganglion cell layer and inner nuclear layer during development.

96 migrate towards the apical side of the outer nuclear layer during early postnatal retinal development

97 hages migrate to the inner part of the outer nuclear layer during photoreceptor degeneration, partici

98 ute retinal changes in PPM involve the outer nuclear layer, external limiting membrane, ellipsoid lay

99 ecific sublayers of the photoreceptor: inner nuclear layer-external limiting membrane (INL-ELM); exte

100 reactivity in the plexiform layers and outer nuclear layer fell into at least three patterns dependin

101 tool for the ImageJ platform, to quantitate nuclear layers following the retina contour.

102 jury paradigms that target different retinal nuclear layers for cell ablation.

103 whose nuclei are scattered across the outer nuclear layer, had no effect on the positioning of their

104 the deeper retinal layers, such as the outer nuclear layer, has not been previously described in mult

105 th AQP4-IgG+ NMOSD, in the Henle fiber outer nuclear layer (HFONL) and the inner segment (IS) layer,

106 The cell bodies in the inner nuclear layer, however, were larger in the peripheral re

107 (19%), and loss of ellipsoid zone and outer nuclear layer in 3 (19%).

108 bserved to be predominantly within the inner nuclear layer in all 17 eyes.

109 reatment preserved ERG b-waves and the outer nuclear layer in Bbs1(M390R/M390R) mice, and prevented o

110 zontal cell processes intrude into the outer nuclear layer in Bbs8(floxed/floxed); Rho-Cre(+) mice, b

111 of microcystoid macular changes in the inner nuclear layer in eyes with concomitant epiretinal membra

112 OCT was less accurate at detecting the inner nuclear layer in ouabain-damaged retinas, but accurately

113 of microcystoid macular changes in the inner nuclear layer in patients with idiopathic epiretinal mem

114 preservation of the ellipsoid zone and outer nuclear layer in the fovea.

115 mislocalize to the inner segments and outer nuclear layer in the Nphp4(nmf192/nmf192) mutant retina.

116 reatment preserved ERG b-waves and the outer nuclear layer in the rd10 mice to P30.

117 ied out a quantitative analysis of the inner nuclear layer in the retina of the marmoset (Callithrix

118 in CCDKO mice develops in the inferior outer nuclear layer independently of light around postnatal da

119 orizontal and bipolar cells toward the outer nuclear layer indicating impaired rod transmitter releas

120 ies formed a regular mosaic within the inner nuclear layer, indicating they represent a single amacri

121 outer nuclear layer (ONL) (p = 0.998), inner nuclear layer (INL) (p = 0.807), and ONL/INL ratio (p =

122 ine cell with their cell bodies in the inner nuclear layer (INL) and a dense layer in the middle of t

123 ti fluorescent cell bodies were in the inner nuclear layer (INL) and a few cell bodies were in the ga

124 in (HPC-1)-immunoreactive cells in the inner nuclear layer (INL) and GCL, consistent with their ident

125 The MME was restricted to the inner nuclear layer (INL) and had a characteristic perifoveal

126 monostratified cells with somas in the inner nuclear layer (INL) and medium-field monostratified cell

127 unit distributed on cell bodies in the inner nuclear layer (INL) and on processes within both the inn

128 aseline age was associated with faster inner nuclear layer (INL) and outer nuclear layer (ONL) thinni

129 ce of ectopic neuronal clusters in the inner nuclear layer (INL) and regions of disrupted retinal lam

130 in Henle's fiber layer (HFL) than the inner nuclear layer (INL) and was highly associated with hyper

131 s with outer retinal changes, isolated inner nuclear layer (INL) cysts were found in 6 of 131 eyes wi

132 cell layer (GCL) and inner part of the inner nuclear layer (INL) from 3-9 dpf; after 14 dpf, it was r

133 ic macular oedema (MMO) of the retinal inner nuclear layer (INL) has been identified in patients with

134 macular splitting were present in the inner nuclear layer (INL) in all 11 eyes and in the outer nucl

135 d in the ganglion cell layer (GCL) and inner nuclear layer (INL) in both amacrine cells and RGCs.

136 hyporeflective spaces was lower in the inner nuclear layer (INL) than in the complex formed by the ou

137 A small degree of inner nuclear layer (INL) thickening occurred in MSON eyes com

138 Inner nuclear layer (INL) thickness was significantly lower in

139 of age, outer nuclear layer (ONL) and inner nuclear layer (INL) thicknesses were measured.

140 Although type 1 lesions lead to inner nuclear layer (INL) thinning, type 2 lesions resulted in

141 and inner plexiform layer (GCIPL) and inner nuclear layer (INL) volumes were tested for association

142 of the outer plexiform layer (OPL) and inner nuclear layer (INL), and development of a hyporeflective

143 isual thresholds; total nuclear layer, inner nuclear layer (INL), and outer nuclear layer (ONL) thick

144 well as the inner plexiform layer, the inner nuclear layer (INL), and the outer plexiform layer (OPL)

145 ner segment (IS) layer, but not in the inner nuclear layer (INL), outer plexiform layer (OPL), or out

146 cell layer (GCL) and the other in the inner nuclear layer (INL), that together comprise approximatel

147 r plexiform layer (IPL), ~0.025 in the inner nuclear layer (INL), ~0.087 in the outer plexiform layer

148 nner plexiform layers (RGCL+), and the inner nuclear layer (INL).

149 in the inner plexiform layer (IPL) and inner nuclear layer (INL).

150 cell layer, with 23% displaced to the inner nuclear layer (INL).

151 the middle retina, extending from the inner nuclear layer (INL)/outer plexiform layer junction to in

152 the retina produces chronic hypoxia of inner nuclear-layer (INL) neurons and Muller glia.

153 Visual acuity and visual thresholds; total nuclear layer, inner nuclear layer (INL), and outer nucl

154 essed in the retina, especially in the outer nuclear layer, inner nuclear layer, and photoreceptors.

155 c arrestin 1 (tet-ARR1), stored in the outer nuclear layer/inner segments in the dark, modulates phot

156 plexiform layer irregularity (8%), and inner nuclear layer irregularity (6%).

157 immunoreactive cells is located in the inner nuclear layer, is immunopositive for glycine transporter

158 inner plexiform layer (mIPL), macular inner nuclear layer (mINL), macular outer plexiform layer (mOP

159 outer plexiform layer (mOPL), macular outer nuclear layer (mONL), photoreceptors (PR), and retinal p

160 n = 1), outer plexiform layer (n = 4), outer nuclear layer (n = 12), or inner segment/outer segment j

161 mRNAs were present in photoreceptors, inner nuclear layer neurons, and ganglion cells in C57BL/6 mou

162 orescent, cystoid macular edema in the inner nuclear layer, no light rise in the electro-oculography,

163 eptors nuclei at the outer edge of the outer nuclear layer of mammalian retinas.

164 own if a degenerate retina lacking the outer nuclear layer of photoreceptor cells would allow the sur

165 ted the presence of Plk1s1 mRNA in the outer nuclear layer of the mouse retina.

166 tive donor cells integrated within the outer nuclear layer of the recipient and differentiated into n

167 stic oedema predominantly involved the inner nuclear layer of the retina and tended to occur in small

168 icrocystic abnormalities involving the inner nuclear layer of the retina occurs in a subset of patien

169 inal ganglion cells (RGCs) through the inner nuclear layer of the retina with glutamate, a primary ne

170 ression is dramatically induced in the inner nuclear layer of the retina, suggesting that PGC-1alpha

171 found within the sclerad region of the inner nuclear layer of the retina.

172 is predominantly expressed within the outer nuclear layer of the retina.

173 rosette-like structures located in the outer nuclear layer of the retinae of the 4 older patients wer

174 s, and thinned the outer plexiform and inner nuclear layers of both WT and hWtEPOR 8-week-old mice.

175 No pathology was seen in the inner or outer nuclear layers of eyes with optic neuritis, suggesting t

176 er show that cells in the ganglion and inner-nuclear layers of the retina constitutively express IRF-

177 helium, choroidal thinning, undifferentiated nuclear layers of the retina, and a perivascular inflamm

178 , ganglion cells, inner plexiform, and inner nuclear layers) of eyes with previous optic neuritis (P

179 PE loss on the same day, and a reduced outer nuclear layer on day 7.

180 flectance of outer plexiform layer and inner nuclear layer on spectral-domain OCT was observed to imp

181 laque-like lesions at the level of the inner nuclear layer on spectral-domain OCT, with no fluorescei

182 loss occurred on day 14 with a reduced outer nuclear layer on the same day.

183 inner plexiform layer (IPL) (nasally), outer nuclear layer (ONL) (nasally), and inner segment (centra

184 ing in other sublayers, especially the outer nuclear layer (ONL) (p < 0.0015).

185 between TT and any observer's average outer nuclear layer (ONL) (p = 0.998), inner nuclear layer (IN

186 cquisition, the boundaries between the outer nuclear layer (ONL) and Henle's fiber layer (HFL) were w

187 When the rats were 12 weeks of age, outer nuclear layer (ONL) and inner nuclear layer (INL) thickn

188 Ellipsoid zone (EZ) width (EZW) and outer nuclear layer (ONL) and inner retinal layer (IRL) thickn

189 ion in whole retinal thickness and the outer nuclear layer (ONL) at 3 and 8 weeks (P < 0.05), along w

190 ased apoptotic nuclei in their retinal outer nuclear layer (ONL) at postnatal day (P) 22.

191 ces donor cells beneath an intact host outer nuclear layer (ONL) containing host photoreceptors.

192 ment (IS/OS) band, and thinning of the outer nuclear layer (ONL) have been identified in association

193 layer (INL) in all 11 eyes and in the outer nuclear layer (ONL) in 4 eyes.

194 h disorganized POS and thinning of the outer nuclear layer (ONL) in addition to the anomaly at the PO

195 OCT there were small foveal islands of outer nuclear layer (ONL) in those with preserved acuity.

196 are located on the apical side of the outer nuclear layer (ONL) in vertebrate retinas.

197 Assessment of the outer nuclear layer (ONL) morphology showed preserved ONL thic

198 the measurement, the thickness of the outer nuclear layer (ONL) of central horizontal B-scans increa

199 The outer nuclear layer (ONL) of KO retinas became 20% thinner bet

200 hT17M Rho was localized in the outer nuclear layer (ONL) of T17M(+/-)ERAI(+/-) photoreceptors

201 ursors were able to integrate into the outer nuclear layer (ONL) of the Rd9 retina.

202 e were small foveal islands of thinned outer nuclear layer (ONL) surrounded by thick delaminated reti

203 AF, A2E bisretinoid concentration, and outer nuclear layer (ONL) thickness in mice of different genot

204 Outer nuclear layer (ONL) thickness in T(-/-) mice was -70% of

205 s were recorded after OKT testing, and outer nuclear layer (ONL) thickness measurements were then obt

206 ificant reduction of the photoreceptor outer nuclear layer (ONL) thickness overlying 92% of the druse

207 Foveal and outer nuclear layer (ONL) thickness was measured and presence

208 cone photoreceptor packing density and outer nuclear layer (ONL) thickness within the central 15 degr

209 al macular thickness (CMT), the foveal outer nuclear layer (ONL) thickness, and tomographic structura

210 layer, inner nuclear layer (INL), and outer nuclear layer (ONL) thicknesses; and horizontal extent o

211 antly blocked photoreceptor apoptosis, outer nuclear layer (ONL) thinning, and retinal gliosis.

212 h faster inner nuclear layer (INL) and outer nuclear layer (ONL) thinning.

213 included ellipsoid zone discontinuity, outer nuclear layer (ONL) thinning; presence of hyper-reflecti

214 of Muller glia nuclei migrated to the outer nuclear layer (ONL) to divide.

215 m (RPE), photoreceptor (PR) layer, and outer nuclear layer (ONL) were segmented.

216 ion, external limiting membrane (ELM), outer nuclear layer (ONL), and outer plexiform layer (OPL).

217 eceptor inner-outer segment junction), outer nuclear layer (ONL), and total retinal thickness were me

218 The internal limiting membrane, outer nuclear layer (ONL), external limiting membrane (ELM), i

219 outer segments plus RPE (OS+) and the outer nuclear layer (ONL).

220 ter plexiform layer, and ~0.026 in the outer nuclear layer (ONL).

221 after 14 dpf, it was restricted to the outer nuclear layer (ONL).

222 layer (GCL+IPL), RNFL, outer plexiform/inner nuclear layers (OPL+INL), and outer nuclear/photorecepto

223 ere observed in the number of cells in inner nuclear layer or in ganglion cells at 12 months of age.

224 t all correlated with thickness of the inner nuclear layer or total retina.

225 ma was identified by thickening of the inner nuclear layer, outer plexiform layer, or full retina.

226 ell complex (GCC), and some sectors of outer nuclear layer (P- values </=0.05) was found with no sign

227 Patients with inner and outer nuclear layer pathology have more rapid disability progr

228 Microcystic inner nuclear layer pathology occurs in a proportion of patien

229 Identification of microcystic inner nuclear layer pathology on spectral-domain optical coher

230 and some plaques were observed in the outer nuclear layer, photoreceptor outer segment, and optic ne

231 ell layer, the anterior portion of the inner nuclear layer, photoreceptors, and choroidal stroma.

232 hickness, inner retinal thickness, and outer nuclear layer plus Henle fiber layer (ONL+HFL) thickness

233 ns at which the outer segment (OS) and outer nuclear layer plus outer plexiform layer (ONL+) thicknes

234 The thicknesses of the outer nuclear layer plus outer plexiform layer (ONL+), outer s

235 s of the outer segment (OS) layer, the outer nuclear layer plus outer plexiform layer (ONL+), the ret

236 ed with GCIP (r = -0.30; p = 0.02) and inner nuclear layer (r = -0.25; p = 0.04) atrophy rates.

237 preoperative thick measurements of the outer nuclear layer (r = 0.514; P = 0.002), high reflectance o

238 aluation revealed complete loss of the outer nuclear layer, remodeling of the inner retina, loss of r

239 The lesions extended to the outer nuclear layer, resulting in focal retinal degeneration,

240 -/-) exhibit a progressive loss of the outer nuclear layer, retinal physiology deficits, and a higher

241 ncludes photoreceptor layer thickness: inner nuclear layer-retinal pigment epithelium (INL-RPE) and t

242 serially over years, the outer photoreceptor nuclear layer showed progressive thinning.

243 of choriocapillaris flow (CC-slab) or outer nuclear layer structure (ONL-slab) were generated from O

244 d ganglion cell layer, but also in the inner nuclear layer, suggesting that retinal injury is more wi

245 r plexiform layer (S6 and N6 sectors), inner nuclear layer (T6 and N6 sectors), and outer plexiform l

246 yers of the retina at the level of the inner nuclear layer that may develop in response to ischemia o

247 mitotic nuclei remaining in the basal inner nuclear layer, the region where Muller glia typically re

248 ted of nerve fiber, ganglion cell, and inner nuclear layer; the hypointense layer 2, the outer nuclea

249 in several cell layers, including the inner nuclear layer; they are present in primary mouse Muller

250 tinal damage was assessed by measuring outer nuclear layer thickness and by electroretinogram (ERG).

251 , producing significant improvement in outer nuclear layer thickness and ERG activity.

252 For all 3 types of testing, the outer nuclear layer thickness constituted the most important p

253 26 significantly increased the retinal outer nuclear layer thickness from 6.34 +/- 1.6 to 11.7 +/- 0.

254 Outer nuclear layer thickness in CNGA3-/- retina was reduced b

255 At the fovea, retinal and inner nuclear layer thickness increased with edema severity: 1

256 ns were examined by TUNEL staining and outer nuclear layer thickness measurements.

257 paralleled reported losses of photoreceptor nuclear layer thickness over the same age range.

258 on, and there was normal outer photoreceptor nuclear layer thickness retina-wide.

259 Histological studies showed that the outer nuclear layer thickness was dramatically reduced in the

260 calized visual sensitivity and photoreceptor nuclear layer thickness were measured across the central

261 ly the GCL, inner plexiform layer, and inner nuclear layer thickness with rates of -0.11 mum/year, -0

262 1040 mum allowed the quantification of outer nuclear layer thickness, a direct correlate of photorece

263 Inner nuclear layer thickness, also in eyes without a history

264 f CME, central foveal thickness (CFT), inner nuclear layer thickness, and foveal-to-parafoveal thickn

265 sessed by TUNEL and measurement of the outer nuclear layer thickness.

266 eptor cell layer with variable loss of outer nuclear layer thickness.

267 most important predictive feature was outer nuclear layer thickness.

268 educed by injury, despite no change in outer nuclear layer thickness; and peripheral rod, but not con

269 ThicknessTool can measure retina nuclear layers thickness in a fast, accurate, and precis

270 Inner and outer nuclear layer thicknesses in patients with non-macular t

271 Amelioration of outer nuclear layer thinning indicated that vitamin E treatmen

272 ions were identified as focal areas of inner nuclear layer thinning over an anteriorly displaced oute

273 or resolved PAMM lesion was defined as inner nuclear layer thinning with outer plexiform layer (OPL)

274 scence, decreased HPLC-quantified A2E, outer nuclear layer thinning, and increased methylglyoxal (MG)

275 ation with severe GCL loss, borderline inner nuclear layer thinning, and less prominent photoreceptor

276 retinal loss, generally involving the outer nuclear layer to photoreceptors, occasionally with a cha

277 cell layer, ganglion cell complex, and inner nuclear layer volumes showed an inverse relationship wit

278 Our data indicate that regardless of which nuclear layer was damaged, MG respond by generating mult

279 he loss of photoreceptor nuclei in the outer nuclear layer was further accentuated and the number of

280 Among the intraretinal layers, the inner nuclear layer was identified as the best indicator of CR

281 The inner nuclear layer was normal or had borderline thinning.

282 y 21, but no significant damage to the outer nuclear layer was observed.

283 luding en face OCT segmentation of the inner nuclear layer was performed in all patients.

284 ression model showed that SSPiM in the inner nuclear layer was related to treatment response (P = 0.0

285 outer segments were preserved, and the outer nuclear layer was significantly thicker in the treated B

286 After 2 months, the outer nuclear layer was significantly thicker, and the photore

287 Within the nuclear layer we found an increase of the elastic and vi

288 d horizontal cells of all cells in the inner nuclear layer were comparable in central and peripheral

289 Microcystoid macular changes in the inner nuclear layer were diagnosed in 52 out of 264 eyes with

290 .3%), and hyperreflective spots in the outer nuclear layer were observed in 5 eyes (16.7%).

291 s of the retinal outer (ONL) and inner (INL) nuclear layers were evaluated at 9 weeks of age.

292 ificant thinning of both the inner and outer nuclear layers, when compared with other patients with m

293 eration reveals a proliferative advantage in nuclear layers where neurons were ablated.

294 th equals peripheral; on SDOCT, foveal outer nuclear layer (which includes HFL) and IS/OS thickens.

295 roportionate thinning of the inner and outer nuclear layers, which may be occurring as a primary proc

296 ron microscopy illustrated degenerated inner nuclear layer with disintegration of cells and loss of c

297 Neuronal loss was noted in the inner nuclear layer with focal reduction in cell density.

298 Stratified outer nuclear layer with many Muller cells suggests high sensi

299 age, resulting in a thinner but intact outer nuclear layer with residual cones expressing S- and M-op

300 mislocalizes to the inner segment and outer nuclear layer, with only approximately 20% in rod outer