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1 eractions with nuclear hormone receptors and nuclear respiratory factors.
3 tochrome c promoter mutants showed that both nuclear respiratory factor 1 (NRF-1) and cAMP-response e
4 re conserved in EWG homologs including human nuclear respiratory factor 1 (NRF-1) and the sea urchin
11 The present study tested our hypothesis that nuclear respiratory factor 1 (NRF-1) serves such a role
12 othesis, binding of the transcription factor nuclear respiratory factor 1 (NRF-1) to the cytochrome c
13 expression of a dominant negative allele of nuclear respiratory factor 1 (NRF-1), and glucose depriv
14 regulated by the same transcription factor, nuclear respiratory factor 1 (NRF-1), found recently by
15 in other COX genes, namely binding sites for nuclear respiratory factor 1 (NRF-1), nuclear respirator
17 oregulated by the same transcription factor, nuclear respiratory factor 1 (NRF-1), which regulates al
19 nscriptional activation [c-fos, c-jun, JunB, nuclear respiratory factor 1 (NRF-1)], mitochondrial pro
20 ty during disease progression and identified nuclear respiratory factor 1 (NRF1) as a master regulato
22 ing of EglN2 under hypoxic conditions reveal nuclear respiratory factor 1 (NRF1) motif enrichment in
24 X gene minimal promoter-binding activity as nuclear respiratory factor 1 (NRF1), a previously known
25 rs, such as estrogen-related receptor (ERR), nuclear respiratory factor 1 (Nrf1), GA-binding protein-
26 ecific DNA-binding transcriptional activator nuclear respiratory factor 1 (NRF1), in promoting contex
27 ed receptor-gamma coactivator-1alpha [PGC1], nuclear respiratory factor 1 [NRF1], mitochondrial trans
29 encoded by nrf is a close homologue of human Nuclear Respiratory Factor 1 and avian Initiation Bindin
30 ry mitochondrial biogenesis (no induction of nuclear respiratory factor 1 and mitochondrial transcrip
31 ear localization of 2 transcription factors, nuclear respiratory factor 1 and nuclear factor-E2-relat
32 vator 1alpha, the transcriptional activators nuclear respiratory factor 1 and nuclear respiratory fac
33 eceptor gamma coactivator 1alphaand 1betaand nuclear respiratory factor 1 mRNA and mitochondrial DNA
34 al transduction and recruitment of the NRF1 (nuclear respiratory factor 1) and THRB to the promoters
36 that the gene encoding the homolog of human Nuclear respiratory factor 1, erect wing (ewg), is auton
37 activator 1alpha, PGC1 related co-activator, nuclear respiratory factor 1, transcription factor A of
38 pha (PGC-1alpha) and its downstream proteins nuclear respiratory factor 1, transcription factor A, mi
39 ors including estrogen-related receptors and nuclear respiratory factor 1, yet its physiological role
43 reduced levels of the PGC-1alpha downstream nuclear respiratory factors 1 and 2, mitochondrial trans
44 vated receptor-gamma coactivator 1 alpha and nuclear respiratory factors 1 and 2, or to enhanced expr
45 responsive to reactive oxygen species, i.e. nuclear respiratory factor-1 (NRF-1) and mitochondrial t
46 t I RE1-like silencer and functional Sp1 and nuclear respiratory factor-1 (NRF-1) elements within a G
49 he polio virus receptor that is bound by the nuclear respiratory factor-1 (NRF-1) transcription facto
50 We reported that estradiol (E(2)) induces nuclear respiratory factor-1 (NRF-1) transcription throu
51 that control the expression of these genes - nuclear respiratory factor-1 (NRF-1) was significantly u
52 ssociate with reduced expression of multiple nuclear respiratory factor-1 (NRF-1)-dependent genes enc
57 inding algorithms predicted the existence of nuclear respiratory factor-1 (NRF1) binding sites in E2F
58 own to orchestrate mitochondrial biogenesis, nuclear respiratory factor-1 and peroxisome-proliferator
61 gene expression and nuclear accumulation of nuclear respiratory factor-1, -2alpha (Gabpa), and perox
62 ptor-gamma coactivator-1alpha (PGC-1 alpha), nuclear respiratory factor-1, and mitochondrial transcri
63 activated receptor-gamma coactivator 1-beta, nuclear respiratory factor-1, and transcription factor A
64 c mitochondrial biogenesis by enhancement of nuclear respiratory factor-1, PGC-1alpha (peroxisome pro
68 o analysis revealed tandem binding sites for nuclear respiratory factor 2 (NRF-2) on the promoter of
69 e find Zbtb11 facilitates the recruitment of nuclear respiratory factor 2 (NRF-2) to its target promo
70 at a transcription factor of the Ets family, nuclear respiratory factor 2 (NRF-2), can activate in vi
72 irt6 maintains Sirt3 levels by up-regulating nuclear respiratory factor 2 (Nrf2)-dependent Sirt3 gene
73 nes PGC1alpha, nuclear respiration factor 1, nuclear respiratory factor 2 and transcription factor A,
74 rresponded with induction of the ISR and the nuclear respiratory factor 2 antioxidant program but not
77 activators nuclear respiratory factor 1 and nuclear respiratory factor 2, mitochondrial transcriptio
79 es for nuclear respiratory factor 1 (NRF-1), nuclear respiratory factor 2/GA binding protein (NRF-2/G
80 beta/delta mRNA and protein (5 sessions) and nuclear respiratory factor-2 protein (3 sessions) while
81 els of mitochondrial transcription factor A, nuclear respiratory factor-2a, and peroxisome proliferat
82 ion of the PGC-1alpha pathway, PQQ increased nuclear respiratory factor activation (NRF-1 and NRF-2)
84 92-bp basal promoter contains sites for the nuclear respiratory factors NRF-1 and NRF-2, which have
87 box, two potential GC boxes, and a potential nuclear respiratory factor (NRF)-1 binding site, which w
90 inding analysis of VSNL1 promoter identified nuclear respiratory factor (NRF)-1/alpha-PAL as a major
91 a promoters, which for IL10 included Nfe2l2, nuclear respiratory factor (NRF)-2 (Gabpa), and MEF2, an
93 e Tfam expression through phosphorylation of nuclear respiratory factor (NRF-1) and binding to the Tf
94 or binding within multiple DH sites detected nuclear respiratory factors (NRF's) and YY1 specifically
95 man TFB1M and TFB2M promoters is governed by nuclear respiratory factors (NRF-1 and NRF-2), key trans
99 ne NDEV levels of the transcriptional type 2 nuclear respiratory factor (NRF2) which controls mitocho
101 1alpha, oestrogen-related receptor alpha and nuclear respiratory factors, resulting in a decrease in