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1 e-dependent decrease in the proliferation of nucleus pulposus cells.
2 cules, and that this regulation is unique to nucleus pulposus cells.
3 fect of HIFs on GlcAT-1 promoter function in nucleus pulposus cells.
4 umber of human studies focusing primarily on nucleus pulposus cells.
5 ommon target gene for HIF-1 and HIF-2 in the nucleus pulposus cells.
6 T-1 promoter activity and expression only in nucleus pulposus cells.
7 CCN3 and suppressed its promoter activity in nucleus pulposus cells.
8 on and promoter activity was observed in rat nucleus pulposus cells after TGFbeta treatment.
9 crease in promoter activity was seen both in nucleus pulposus cells and in N1511 chondrocytes.
10 pathologies include loss of reticular-shaped nucleus pulposus cells, disorganization of annulus fibro
11 s biology to mimic the cellular behaviour of Nucleus Pulposus cells exposed to a 3D multifactorial bi
12 lyses were used to measure ANK expression in nucleus pulposus cells from rats and humans.
13 disc disease may restore NOTCH signaling and nucleus pulposus cell function.
14 ssion and the suppression of CCN2 by CCN3 in nucleus pulposus cells further the paradigm that these C
15       Results of this study indicate that in nucleus pulposus cells, HIF-2 and HIF-1 modulate their o
16                        Here, we show that in nucleus pulposus cells, hypoxia robustly induces PHD3 ex
17 transcriptional coactivator of HIF-1alpha in nucleus pulposus cells independent of the PKM2-JMJD5 axi
18         Adenovirus-mediated gene transfer to nucleus pulposus cells may be the initial stage of a new
19 entified a subtype of locally residing human nucleus pulposus cells (NPCs), generated by certain cond
20              Adaptive response to hypoxia in nucleus pulposus cells of the intervertebral disc is reg
21 ation through controlling ADAMTS activity in nucleus pulposus cells of the intervertebral disc.
22 histochemical staining, annulus fibrosus and nucleus pulposus cells of young-adult IVD expressed oste
23 nal coactivators MRTF-A and YAP/TAZ regulate nucleus pulposus cell phenotype through cell shape.
24 ss of all the PHDs is maintained in isolated nucleus pulposus cells regardless of the disease state.
25                                        Since nucleus pulposus cells reside under conditions of hypoxi
26 erexpression of HIF-1alpha and HIF-2alpha in nucleus pulposus cells resulted in a significant suppres
27 stochemically in disc tissue, and numbers of nucleus pulposus cells staining positive for ADAMTS 4, 5
28 suppressed GlcAT-1 promoter activity only in nucleus pulposus cells, suggesting a cell type-specific
29 ng CCN3 expression in Smad3-null mice and in nucleus pulposus cells transduced with lentiviral short
30                Above 330 mosmol/kg, cultured nucleus pulposus cells up-regulated target genes TauT, B
31 xygen-dependent changes in ANK expression in nucleus pulposus cells were minimal.
32  reducing cellular aging and inflammation in nucleus pulposus cells while increasing collagen type II