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1 ation requires LIS1 and its binding protein, NudE.
2 1/2 inhibitors in reversing vasoocclusion in nude and humanized SCD mouse models of acute vasoocclusi
3 r SK-MEL-05, tumor inhibition was similar in nude and Nod-Scid mice and was less efficient than seen
8 d time-dependent tumor uptake was studied in nude BALB/c mice bearing a subcutaneous HER3 overexpress
9 p to 6 d after intravenous administration to nude BALB/c mice bearing high EpCAM-expressing HT-29 col
10 umor growth was significantly decreased when nude BALB/c mice were injected with Msi1-knockdown BCCs.
12 Astonishingly, the requirement for LIS1 or NudE can be bypassed to a significant extent by mutation
16 was performed in tumour xenografts in 15 NCr nude immunodeficient mice, which were treated with eithe
17 r the cytoplasmic dynein regulators LIS1 and NudE/L (Nde1/Ndel1), but not for the dynactin p150(Glued
18 scles were subcutaneously injected into CD-1 nude mice (CD-1 nude mice, Crl:CD1-Foxn1(nu); Charles Ri
20 el) into groups of 4-week-old athymic female nude mice (induced with subcutaneous triple negative xen
22 in human T2D M2MPhis transplanted to athymic nude mice (NMRI-Foxn1(nu)/Foxn1(nu) ) or systemic inhibi
25 cells, and in both eight-patient bloods and nude mice administered with the labeled CTCs in comparis
26 K1/2 inhibitor given to TNF-alpha-pretreated nude mice after human SSRBC infusion and onset of vasooc
27 experiments were performed on tumor-bearing nude mice after subcutaneous injection of RIN-m5F cells.
30 B or C-I inhibited tumor xenograft growth in nude mice and decreased the expression of CSC-markers an
31 ctively) to grow tumor xenografts in athymic nude mice and demonstrated the molecular specificity and
32 ed engrafting of pancreatic CSC's tumours in nude mice and displayed an antigrowth effect toward init
33 (67)Cu-CuSarTATE is well tolerated in BALB/c nude mice and highly efficacious against AR42J tumors in
34 evels of estradiol in human breast cancer in nude mice and in murine breast cancer in immune-competen
35 hibited cancer cell growth and metastasis in nude mice and inhibited cancer cell migration, invasion,
36 iments were then carried out in both healthy nude mice and nude mice with subcutaneous tumor to valid
37 s were grown as orthotopic tumors in athymic nude mice and PAF1 knockdown was induced by administrati
38 tifying the biodistribution of antibodies in nude mice and provides an alternative to PET analysis in
39 was initially established subcutaneously in nude mice and the subcutaneous tumor tissue was then ort
40 ChA-1 cells were injected into the flanks of nude mice and treated with miR-24 inhibitor or inhibitor
50 dent SSM3 mouse mammary tumors, male athymic nude mice bearing androgen-dependent CWR22 prostate canc
51 et involving ultrasound images of 23 athymic nude mice bearing C26 mouse adenocarcinomas was assemble
52 purified reagents were injected into athymic nude mice bearing CD44-positive human tumors (MDA-MB-231
55 ncer xenografts, and male and female athymic nude mice bearing estrogen-independent MDA-MB-231 human
56 jected intravenously into the BALB/c athymic nude mice bearing folate receptor (FR)-overexpressing KB
58 nd biodistribution studies were performed in nude mice bearing HCC4006 and A549 xenograft tumors.
59 s of intravenously injected nanoparticles in nude mice bearing HCT116 tumors radiosensitization was e
61 rculation and evaluated the tumor binding in nude mice bearing heterotopic cervical (HT3), esophageal
62 icacy of PEG-GIRLRG peptide was evaluated in nude mice bearing heterotopic cervical (HT3), esophageal
64 ed with [(68)Ga]Ga-FAPI-04 were conducted in nude mice bearing HT1080hFAP tumors or U87MG xenografts.
69 In vivo antitumor efficacy was tested in nude mice bearing PSMA+ PC3 PIP or PSMA- PC3 flu flank x
70 tion and SPECT/CT imaging experiments, using nude mice bearing PSMA-positive PC-310 and PSMA-negative
74 cer biodistribution was determined in BALB/c nude mice bearing subcutaneous CHL-GLP-1R xenografts.
75 of tumor cure while being well tolerated by nude mice bearing subcutaneous GPA33-positive SW1222 xen
76 One-hour dynamic PET scans were performed on nude mice bearing subcutaneous human head and neck tumor
78 cs of (89)Zr-DFO-AC-10 was studied in BALB/c nude mice bearing subcutaneous human Karpas 299 tumors (
80 nd biodistribution studies were performed on nude mice bearing U87MG and MDA-MB-231 xenografted tumor
81 /kg MEK1/2 inhibitor to TNF-alpha-pretreated nude mice before human SSRBC infusion inhibited SSRBC ad
85 in tumor xenografts grown in female athymic nude mice by small-animal PET/CT imaging and tissue biod
86 117085 significantly reduces tumor growth in nude mice compared with control untreated mice or either
90 ntal pulp on poly-l-lactic acid scaffolds in nude mice gave rise to perfect heterotopic ossicles in v
91 V1-induced cSCC from back skin, into athymic nude mice gave rise to secondary cSCCs, which lacked vir
94 of these mutant-expressing cells in athymic nude mice induced rapid tumor development, showing their
95 ties, SPECT and CT scans of HT29-xenografted nude mice injected with (177)Lu-3BP-227 were acquired, a
96 tically attenuates tumor growth in xenograft nude mice injected with human K562 leukemia cells and ce
97 Subcutaneous injection of TRIM24 iHMECs in nude mice led to growth of intermediate to high-grade tu
98 id gland scaffolds into the renal capsule of nude mice led to the differentiation of transplanted hDF
99 next evaluated in a validated orthotopic GBM nude mice model, studying the tumor growth over time by
103 ast cancer were examined using female BALB/c nude mice orthotopically implanted with human breast car
104 implantation of LOX-treated neocartilage in nude mice promoted further maturation of the neotissue,
105 se model, after tumor establishment, athymic nude mice received treatment with progesterone or vehicl
107 ansplantation of hERG1-expressing cells into nude mice resulted in an increased incidence of tumors.
108 5alpha-depleted pancreatic cancer cells into nude mice resulted in markedly reduced tumorigenicity (P
109 KD SCC1 cells into the floor of the mouth in nude mice resulted in the formation of significantly sma
110 human gliomas (U87) grown orthotopically in nude mice resulting in a more than a doubling of median
111 D25(+)IL7Ralpha(+)) after grafting recipient nude mice revealed that MPP3 cells were the most effecti
112 ografts of MKN45/5FU cells in the stomach of nude mice revealed that these cells had a high potential
114 pressed DNAJB6a formed tumors more slowly in nude mice than control cells or cells that expressed a m
115 ive MDA-MB-231 tumors to a greater extent in nude mice than in NSG mice, pointing to the potential ro
117 hyme were grafted under the renal capsule of nude mice to generate prostatelike tissues, and mice wer
118 s computed from ultrasound images of athymic nude mice to predict tumor response to treatment at an e
122 ry cholangiocarcinoma (CCA) models in BALB/c nude mice using minimally invasive ultrasound-guided int
123 ia conditions, were then assessed in healthy nude mice using the left kidney and spleen as reference
124 he tracer in the pancreatic islets of BALB/c nude mice was examined using fluorescence microscopy.
125 of WT HBx-expressing cells to form tumors in nude mice was significantly higher than that of mutant H
128 ard this end, orthotopic xenografts grown in nude mice were exposed to a fractionated radiation proto
134 1-CCK2R/A431-mock xenografted athymic BALB/c nude mice were used for biodistribution studies and smal
135 OIS and enabled tumor transplants to grow in nude mice with characteristic cell morphology of anaplas
136 In vivo treatment of A375 xenograft-bearing nude mice with cryptolepine (10 mg/Kg body weight, i.p.)
137 t overexpress MFSD2A were transferred to CD1 nude mice with dextran sodium sulfate-induced colitis, w
139 of (177)Lu-octreotate was examined in BALB/c nude mice with GOT2 tumors 1-168 h after injection with
143 ribution, and fluorescence imaging on BALB/c nude mice with orthotopically transplanted PC346C tumors
144 26 nmol/mouse, 8-9 MBq/mouse) in male BALB/c nude mice with PSMA-expressing subcutaneous LS174T-PSMA
146 ing product, (18)F-FVIIai, was injected into nude mice with subcutaneous human pancreatic xenograft t
147 11B6 uptake was performed on NMRI and BALB/c nude mice with subcutaneous LNCaP xenografts up to 14 d
148 111)In-labeled ADCs were performed on BALB/c nude mice with subcutaneous PSMA-positive LS174T-PSMA xe
149 en carried out in both healthy nude mice and nude mice with subcutaneous tumor to validate the contra
151 Methods: Two groups of 6 adult female BALB/c nude mice with subcutaneously implanted tumors underwent
153 ot R-lycosin-I inhibited tumor growth in the nude mice xenograft model without generating side effect
154 ble for small-animal PET studies in multiple nude mice xenografted with the A431 carcinoma cell line.
160 ces hypoxia in FaDu and HCT116 xenografts in nude mice, and causes a significant tumour growth delay
161 lorectal cancer metastases were generated in nude mice, and epifluorescence imaging of ICG, as well a
164 matrix proteins, subcutaneous tumor size in nude mice, and invasive behavior, including bone marrow
165 nd cervical cancer cell xenograft in vivo in nude mice, and suppress cervical cancer cell migration a
166 nt growth inhibition of MOLM13 xenografts in nude mice, and the activity correlates with inhibition o
167 ulated into the left thyroid lobe of athymic nude mice, and the orthotopic tumor growth was monitored
168 2 were injected into the pancreas of athymic nude mice, and their local and distant spread was monito
169 ously and orthotopically implanted tumors in nude mice, and was accompanied by c-SRC downregulation.
172 nhibited orthotopic prostate tumor growth in nude mice, compared with monotherapy, by reversing the e
174 taneously injected into CD-1 nude mice (CD-1 nude mice, Crl:CD1-Foxn1(nu); Charles River Laboratories
176 the OVCAR-3 and OVCAR-8 xenograft models in nude mice, demonstrating synergistic antitumor activity
177 cells were subcutaneously transplanted into nude mice, DPSC/CTL cells induced mineralized tissue for
178 man breast cancer (MCF-7 xenograft) model in nude mice, EO-33 blocked 90% of tumor growth induced by
179 utside the primary tumor microenvironment in nude mice, exhibited signatures of immune evasion, incre
181 uding HUVEC-mediated trophoblast invasion in nude mice, in vitro three-dimensional capillary tube for
182 rs and xenografts established in NOD-SCID or nude mice, low MCPIP1 levels correlated strongly with in
183 uman prostate xenograft model established in nude mice, RAD001 alone or in combination with docetaxel
186 expressing versican siRNA were injected into nude mice, the resulting tumors displayed significantly
187 n PMP tissue was i.p. grafted and grown into nude mice, then constituted into reliable and reproducib
188 and AGS, which do and do not form tumors in nude mice, to identify their genomic differences relevan
189 an ovarian SKOV3 tumors cells into 14 female nude mice, treatment with vehicle or pazopanib (2.5 mg p
190 e than WT cybrids, however, when injected in nude mice, tRNAmut cybrids produced larger tumours and s
191 on in PCa cells and induces larger tumors in nude mice, whereas its silencing decreased proliferation
192 g in aggressive tumor formation in xenograft nude mice, which could be suppressed by combined treatme
194 ive in suppressing xenograft tumor growth in nude mice, which underlines the translational potential
195 ous cell carcinoma upon inoculation into the nude mice, while parental HaCaT cells remain non-tumorig
264 ggressive orthotopic tumor models in athymic nude mice: a human PC-3 M-luc-C6 prostate tumor and a hu
266 through the layers and structures of ex vivo nude mouse ear skin and extracted pharmacokinetic parame
268 patient-derived orthotopic xenograft (PDOX) nude mouse model of a highly aggressive liver metastasis
269 nsional culture and in an in vivo orthotopic nude mouse model of HNSCC through a novel transcription-
283 ) gene showed exon skipping at exon 7, while nudE neurodevelopment protein 1 (NDE1) gene showed repla
284 resent study, we generated a novel strain of nude NOD/SCID/IL2rg(-/-) (NSIN) mice by knocking out Fox
285 nd explored their biodistribution in athymic nude, NSG, and humanized NSG mice bearing human epiderma
286 was studied in normal and immune-deficient (nude, nu/nu) BALB/c mice infected with vaccinia virus (V
290 premalignancy and (b) an in vivo orthotopic nude rat lung cancer model to evaluate response to epige
297 bcutaneous cancer esophageal xenografts, and nude rats with orthotopic esophageal cancers in four stu
298 al hair and nails, distinct from the classic nude/SCID phenotype in individuals with autosomal-recess
299 cell immune system since it did not occur in nude, severe combined immunodeficiency, or T-cell deplet
300 ecia, and nail dystrophy, accounting for the nude/severe combined immunodeficiency (nu/SCID) phenotyp