戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 e, we identify 15 SH2B1 variants in severely obese children.
2 r the direction of evidence-based therapy in obese children.
3 ence of metabolic syndrome in overweight and obese children.
4 ring childhood, especially in overweight and obese children.
5 Energy expended in activity was lower in the obese children.
6        Obese parents are more likely to have obese children.
7  hemostatic variables in a biethnic group of obese children.
8 e in regulating plasma hemostatic factors in obese children.
9 arkers, and retinal microvascular changes in obese children.
10 ut microbial composition between healthy and obese children.
11  be considered in the clinical management of obese children.
12 inflammation in a cohort of African American obese children.
13 appropriate weight-based dosing strategy for obese children.
14 rnatives more reinforcing than do overweight/obese children.
15  and dicarboxylated fatty acids were seen in obese children.
16 rylcarnitine, were significantly elevated in obese children.
17 creased steroid derivatives may be unique to obese children.
18 NAFLD was identified in 24% of the recruited obese children.
19 l NO(2) levels may improve asthma health for obese children.
20 isease (NAFLD) are frequently encountered in obese children.
21 increase physical activity in overweight and obese children.
22 ficantly longer lengths of hospital stay for obese children.
23 bserved only in adults, is also occurring in obese children.
24  allele would influence the risk of NAFLD in obese children.
25 thood between MHO children and nonoverweight/obese children.
26                  In total, 597 overweight or obese children (10.4%) were identified, and 219 of them
27  TC varies between 4.8% and 12.3% (higher in obese children [12.3%] and at the ages when TC naturally
28 lerance was detected in 25 percent of the 55 obese children (4 to 10 years of age) and 21 percent of
29                                           In obese children, 4-mo periods of physical training did no
30                   Seventy-five overweight or obese children (41 girls [55%], 34 whites [45%], 20 Hisp
31 d high-sensitivity C-reactive protein in 108 obese children, 54 with (HFF >/=5%) and 54 without NAFLD
32 geal reflux scores were higher in overweight/obese children (9.6 vs 23.2; P = .003) and appear to med
33                                           In obese children a moderate to good correlation between CA
34                                           In obese children, age, vigorous activity, diet, and baseli
35 acterization of 171 AT samples from lean and obese children aged 0 to 18 years.
36 ontrolled trials conducted in overweight and obese children aged 18 years or younger, comparing dieta
37                     The participants were 71 obese children aged 7-11 y (22 boys, 49 girls; 31 whites
38 me of distribution, or drug concentration in obese children (aged </=18 years).
39 nd 87 rodent obesity genes in 2,548 severely obese children and 1,117 controls.
40  < 1 x 10(-5)) in an additional 971 severely obese children and 1,990 controls identified 4 new loci
41 icroarray platform on sorted monocytes of 35 obese children and 16 lean controls.
42 his cross-sectional study, 45 overweight and obese children and 46 age- and sex-matched healthy indiv
43                               We studied 455 obese children and adolescents (181 Caucasians, 139 Afri
44 s58542926 SNP in a multiethnic cohort of 957 obese children and adolescents (42% Caucasians, 28% Afri
45 do whites, although the data are limited for obese children and adolescents and for boys.
46                                     Severely obese children and adolescents have lower health-related
47                    Instead of overweight and obese children and adolescents having higher confectione
48 pared with healthy children and adolescents, obese children and adolescents reported significantly (P
49                                              Obese children and adolescents show a disturbance in som
50 e risk for impaired health-related QOL among obese children and adolescents to target interventions t
51                                              Obese children and adolescents were more likely to have
52                                           In obese children and adolescents with prediabetes, intramy
53 OL total score (mean [SD], 53.8 [13.3]) than obese children and adolescents without obstructive sleep
54 ence of the metabolic syndrome is high among obese children and adolescents, and it increases with wo
55                                  In severely obese children and adolescents, inpatient treatment was
56 ose tolerance in a multiethnic cohort of 167 obese children and adolescents.
57 omains (mean [SD] total score, 67 [16.3] for obese children and adolescents; 83 [14.8] for healthy ch
58                  Systematic screening of 431 obese children and adults for mutations in the coding se
59                                              Obese children and adults, particularly those with abdom
60 plain the increased leukemia relapse rate in obese children and adults.
61 sociated with up to 6% frequency in morbidly obese children and adults.
62                                              Obese children and African-American children were more i
63 would be useful in diagnosing overweight and obese children and in developing effective strategies fo
64        We examined these genetic variants in obese children and tested whether their effects on NAFLD
65 h and without physical training on leptin in obese children and to explore the determinants of leptin
66 ectional analysis of data from overweight or obese children and young adults 3 to 19 years of age who
67 al weight, 10.8% of overweight, and 26.9% of obese children) and age (8.9% of 9- to 11-year olds and
68 e screening and assessment of overweight and obese children, and those with an elevated WHtR should u
69 oxidative stress and inflammation evident in obese children are associated with distinct metabolomic
70                                              Obese children are at increased risk for developing obst
71        Prospective pharmacokinetic trials in obese children are needed to ensure therapeutic efficacy
72 besity, and altered MAIT cell frequencies in obese children are positively associated with insulin re
73  clear, reliable assessment and treatment of obese children are still wanting in many cases.
74                                              Obese children are vulnerable to vitamin D deficiency an
75   NASH is now a significant health issue for obese children as well, leading to cirrhosis in some.
76  Steroid derivatives were markedly higher in obese children as were markers of inflammation and oxida
77    TDEE and RMR were significantly higher in obese children, as a result of their greater fat-free ma
78  cardiovascular disease does not manifest in obese children, assessment of the subclinical markers of
79 rly understood interplay might be present in obese children, assessment of the vasculature directly,
80                                              Obese children (body mass index z score >1.65) and nonob
81 ences have consistently been demonstrated in obese children, but the time course and development of a
82 patic steatosis in a series of overweight or obese children by using the imperfect gold standard meth
83 on of vitamin D deficiency in overweight and obese children by vitamin D3 supplementation with 1000 o
84 escriptions of echocardiographic findings in obese children, children engaged in athletic activities,
85                        The reason overweight/obese children commonly report worse asthma control rema
86 wing 13 years with normal weight status, and obese children could expect to live 9.8 years with obese
87  and a childhood cancer has been identified: obese children diagnosed with high-risk acute lymphoblas
88                                              Obese children displayed a distinctive monocyte gene exp
89  caregivers (PACs) as "agents of change" for obese children, evaluating the strength of evidence that
90                                              Obese children exhibited signs of increased collagen tur
91                                              Obese children experience less relapse posttransplant co
92 pharmacokinetic alterations were observed in obese children for 65% (11 of 17) of the studied drugs.
93                               Overweight and obese children, for example, may be more susceptible to
94 rown-like structures, was increased in AT of obese children from 6 years on and was associated with h
95  study, we genetically screened 225 severely obese children from consanguineous Pakistani families th
96                                     However, obese children had greater brachial diameters and restin
97                               Overweight and obese children had higher heart rates (mean 72.4 +/- 11
98                                              Obese children had lower odds of receiving a living dono
99            A total of 23.3% of the recruited obese children had NAFLD.
100 h and without OSA at initial study; however, obese children had significantly higher insulin (106.1 +
101                                              Obese children had twice the risk of high DHEAS (OR: 2.1
102         Past studies of asthma in overweight/obese children have been inconsistent.
103 enditure has shown clearly that, as a group, obese children have higher energy expenditures than do t
104 esearch purporting to show that, as a group, obese children have lower energy intakes than do lean ch
105 ene variants by exome sequencing of severely obese children, including those from consanguineous fami
106 ciation cannot rule out the possibility that obese children ingest food with higher BPA content or ha
107                      NAFLD in overweight and obese children is strongly associated with multiple card
108 drug safety, pharmacokinetics, and dosing in obese children is unknown.
109                                           In obese children, leptin concentration decreased during 4
110                                              Obese children may maintain their obese state by spendin
111 etabolic flexibility in fuel use observed in obese children may occur through the activation of alter
112                                              Obese children may show favorable changes in their behav
113 ol (Fisher exact test; P = .003); overweight/obese children more often reported shortness of breath (
114         Plasma exosomes isolated from either obese children or nonobese children with OSA were primar
115 enerally comparable to that of nonoverweight/obese children (P > 0.05 in most cases).
116 n than in metabolically abnormal, overweight/obese children (P = 0.003).
117 the investigation and treatment of asthma in obese children, particularly in comparison with current
118                 Unexpectedly, in contrast to obese children, pathogenic mutations in LEP and LEPR wer
119 isceral adiposity in sedentary overweight or obese children, regardless of sex or race.
120 Global metabolomic profiling in nonobese and obese children replicates the increased BCAA and acylcar
121 V1 1.87 vs 0.45 mg/mL; P < .012), overweight/obese children reported more than thrice frequent rescue
122 ying the hypertrophy and hyperplasia seen in obese children, respectively.
123 es (RR = 1.08) complied more, but overweight/obese children (RR = 0.81), earlier maturing children (R
124                                          The obese children spent less time engaged in activity or en
125                         The median number of obese children studied per drug was 10 (range, 1-112) an
126              The lower relative REE of older obese children suggests the importance of early interven
127 is linked to increased inflammation in AT in obese children, thereby providing evidence that obesity-
128                                              Obese children under three years of age without obese pa
129 g energy expenditure, it has been shown that obese children underreport intake significantly more tha
130                                              Obese children were 3-6 times more likely to have hypert
131       These alterations in AT composition in obese children were accompanied by decreased basal lipol
132 rmine, in vitamin D-deficient overweight and obese children, whether supplementation with vitamin D3
133                We have examined two severely obese children who are members of the same highly consan
134 .7 mIU per liter) than those in 340 severely obese children who did not have GNAS mutations (3.9 2.6
135 nd improves obesity-related comorbidities in obese children, who are insulin-resistant.
136                               Three morbidly obese children, who were congenitally deficient in lepti
137  region that includes SIM1, were reported in obese children with a Prader-Willi-like syndrome; howeve
138 he most common and is predominantly found in obese children with an early onset of walking.
139 is associated with increased morbidity among obese children with asthma and may partly explain their
140 elated asthma.Measurements and Main Results: Obese children with asthma had more memory and fewer nai
141              RATIONALE: For unclear reasons, obese children with asthma have higher morbidity and red
142  compared the CD4(+) T-cell transcriptome in obese children with asthma with that in normal-weight ch
143                             Among overweight/obese children with asthma, dysanapsis was associated wi
144 s and methylation quantitative trait loci in obese children with asthma, independent of Th-cell subty
145 sociated with pulmonary function deficits in obese children with asthma.Conclusions: We found enrichm
146                            We found that non-obese children with diagnosed asthma at a study visit we
147                                   Overweight/obese children with early-onset asthma display poorer as
148 erefore, mediate the higher risk of death in obese children with ESRD.
149 HEAS (OR: 2.16; 95% CI: 1.51, 3.09); at 7 y, obese children with high DHEAS were fatter and more cent
150                                We studied 14 obese children with impaired glucose tolerance and 14 wi
151                  A significant proportion of obese children with increased WHtRs have abnormal cardio
152                                              Obese children with indirect lung injury pediatric acute
153                                 Asymptomatic obese children with NAFLD exhibit features of early LV d
154 ed for age, gender, and pubertal status, and obese children with NAFLD were matched for body mass ind
155                                              Obese children with newly diagnosed BP-ALL are at increa
156  growth from 0 to 7 y of age in nonobese and obese children with normal and high DHEAS (>/=75th perce
157 7 +/- 0.6 nmol/L; p = 0.005) and a trend for obese children with persisting OSA to have elevated insu
158 s with insulin resistance (pre-diabetes) and obese children with type 2 diabetes, years before overt
159 s the odds of having NAFLD as overweight and obese children without metabolic syndrome.
160 diastolic blood pressure than overweight and obese children without NAFLD.
161 o have elevated insulin levels compared with obese children without OSA (p = 0.07).

 
Page Top