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1 eye movements, but not in a control region (occipital cortex).
2 ps, LO1 and LO2, in object-selective lateral occipital cortex.
3 pagation from the V1 to the V2 region of the occipital cortex.
4 nd lower in sulcal, perirolandic, and medial occipital cortex.
5 stimulus size was reflected by activation in occipital cortex.
6 on compared with healthy control subjects in occipital cortex.
7 n the right fusiform gyrus and left superior occipital cortex.
8 uded areas V1, V3a, hV4, hMT(+), and lateral occipital cortex.
9 the healthy comparison group in the superior occipital cortex.
10 increased during 20S(-), most notably in the occipital cortex.
11 h as other regions in the insular and medial occipital cortex.
12 electrodes, suggesting an origin in parieto-occipital cortex.
13 al lobe and with other areas of parietal and occipital cortex.
14 s of the inhibitory neurotransmitter GABA in occipital cortex.
15 c lamina of frontal, parietal, temporal, and occipital cortex.
16 same effect in an adjacent area, the lateral occipital cortex.
17 was correlated with rCBF in the amygdala and occipital cortex.
18 tive N1 enhancement was localized to lateral occipital cortex.
19 sses that generate the hallucinations in the occipital cortex.
20 rrupting connections between the eye and the occipital cortex.
21 which the figure region is routed to lateral occipital cortex.
22 nd intact implicit encoding in earlier-stage occipital cortex.
23 parahippocampal cortex, fusiform gyrus, and occipital cortex.
24 ivity to color, objects and faces in ventral occipital cortex.
25 ed performance nor diminished BOLD signal in occipital cortex.
26 in relative brain GABA concentration in the occipital cortex.
27 r brain areas, including the pre-frontal and occipital cortex.
28 to large-scale cross-modal plasticity within occipital cortex.
29 raine attack have been shown to occur in the occipital cortex.
30 brainstem structures preceded activation of occipital cortex.
31 iver's brain via magnetic stimulation of the occipital cortex.
32 for myo-inositol or other metabolites in the occipital cortex.
33 erent times using an input function from the occipital cortex.
34 l ranged from 22 in the caudate to 90 in the occipital cortex.
35 r medial prefrontal cortex, hippocampus, and occipital cortex.
36 nged from -33% in the caudate to -20% in the occipital cortex.
37 regions and with expression of NR(1) in the occipital cortex.
38 well as relative metabolic increases in the occipital cortex.
39 ed to be very common throughout parietal and occipital cortex.
40 ive noise and of stimulus-specific biases in occipital cortex.
41 ion, and dysplastic in-folding of the mesial occipital cortex.
42 ha-band over ipsilateral centro-parietal and occipital cortex.
43 d at increasing GABA in the anterior lateral occipital cortex.
44 reases in alpha (8-13 Hz) frequency power in occipital cortex.
45 subcortically in bilateral basal ganglia and occipital cortex.
46 rper tuned profile in more posterior ventral occipital cortex.
47 and middle frontal gyri, the insula, and the occipital cortex.
48 n cooperation with perceptual systems of the occipital cortex.
49 the anterior and posterior superior-parieto-occipital cortex.
50 bust multivariate decoding being reported in occipital cortex.
51 variance in delay-period activity in lateral occipital cortex.
52 n no longer convey visual information to the occipital cortex.
53 pramarginal gyri as well as the left lateral occipital cortex.
54 MRS data were acquired from occipital cortex.
55 be, the right anterior insula, and bilateral occipital cortex.
56 a network of cognitive control, and with the occipital cortex.
57 ty, especially in the cerebellum and parieto-occipital cortex.
58 res correlated with metabolic changes in the occipital cortex.
63 t OFC, right dorsolateral prefrontal cortex, occipital cortex, ACC, ventral striatum/nucleus accumben
64 rimary visual cortex, increased extrastriate occipital cortex activation selectively during maintaine
65 aphy (MEG) that tactile stimulation produces occipital cortex activations, starting as early as 35 ms
67 tain their connectivity with the reorganized occipital cortex and as a result influence processing of
68 dose DHA groups had greater decreases in the occipital cortex and cerebellar cortex, respectively.
69 ined the effects of inhibitory rTMS over the occipital cortex and found that the visual-vestibular po
70 ices in the frontal, parietal, temporal, and occipital cortex and in the subcortical region were obta
72 a different functional role than that in the occipital cortex and may be part of the neuronal mechani
74 set, face-selective sources in right lateral occipital cortex and right fusiform gyrus and sources in
76 al information flow between the left lateral occipital cortex and right intraparietal sulcus, as indi
77 roscopy data were acquired from the parietal-occipital cortex and supplementary motor area in all sub
78 iorally irrelevant information occurs in the occipital cortex and that representations of the informa
79 rmalities involve a circuit encompassing the occipital cortex and the cortical/subcortical systems ph
81 tree approach revealed the centrality of the occipital cortex and the peculiar aggregation of cerebel
82 omponent, which include the superior parieto-occipital cortex and the rostral superior parietal lobul
84 Glucose concentrations in gray matter-rich occipital cortex and white matter-rich periventricular t
88 s between the anterior temporal lobe and the occipital cortex, and between bilateral occipital poles.
89 lts suggest that FEF and IPS modulate visual occipital cortex, and FEF modulates IPS, in relation to
90 temporal gyrus, middle temporal gyrus, right occipital cortex, and inferior frontal cortex was found
91 posterior than anterior HC, in frontal than occipital cortex, and ipsilaterally than contralaterally
95 tween the right caudate and superior lateral occipital cortex, and the right caudate and anterior sup
96 ing the pseudo reference regions cerebellum, occipital cortex, and whole brain (WB) without ventricle
98 nial magnetic stimulation (TMS) to the human occipital cortex are immune to saccadic suppression, whe
100 ared to ATs, in the object sensitive lateral occipital cortex as well as in the face-sensitive occipi
101 ntal lobe, whereas microstate 4 involved the occipital cortex, as well as thalamic and brainstem stru
102 c TMS pulses were delivered over lateralized occipital cortex at 100, 200, or 400 ms into the retenti
103 a (rOFA), a face-selective region in lateral occipital cortex, at different latencies up to 210 ms af
104 d reduced cerebral metabolism in the primary occipital cortex (BA 17) that was revealed only by 3D-SS
106 as 1 (LO1) and 2 (LO2), in the human lateral occipital cortex between the dorsal part of visual area
107 ving words, these subjects activated lateral occipital cortex bilaterally in addition to the language
108 h significant posterior flow deficits in the occipital cortex (Brodmann's areas 18 and 19), usually s
110 ast re-routing of tactile information to the occipital cortex, but this has not been shown in humans.
111 onal partitioning of visual processes in the occipital cortex by Riddoch), but there were also other
112 his animal model, unilateral ablation of the occipital cortex causes unequivocal apoptosis of cortico
113 refrontal cortex, amygdala, temporal cortex, occipital cortex, cerebellum and thalamus (P<0.05 correc
115 rom candidate pseudoreference regions (i.e., occipital cortex, cerebellum, and whole brain) to obtain
116 e that underpins size knowledge: the lateral occipital cortex codes perceptually based aspects (stati
117 uptake in the posterior temporoparietal and occipital cortex compared to clinically normal controls,
118 ophy additionally showing elevated uptake in occipital cortex compared with early-onset Alzheimer's d
119 which in humans, is more highly expressed in occipital cortex compared with the remainder of cortex t
120 guously and slowly (3.5 +/- 1.1 mm/min) over occipital cortex, congruent with the retinotopy of the v
122 cortical activity in portions of the ventral occipital cortex, corresponding to known object areas wi
123 visual regions, only signals in the midline occipital cortex could be used to distinguish targets fr
125 roups, and the subgenual anterior cingulate, occipital cortex (cuneus), and posterior cerebellum were
127 ulate; bilateral parahippocampal gyrus; left occipital cortex) demonstrated indistinguishable activit
128 ol, the normal alpha rhythm (8-13 Hz) in the occipital cortex disappears and a frontal alpha rhythm e
129 affecting distance judgments, while rTMS to occipital cortex disrupted distance but not roughness ju
130 PCr) and inorganic phosphate (Pi) within the occipital cortex during (activation) and after (recovery
132 ion of compensatory increases in rCBF of the occipital cortex during incremental learning and the lef
133 ecuneus, middle temporal gyrus, and superior occipital cortex during the anticipation of potential re
134 dorsal occipital cortex or the right ventral occipital cortex, during the brief presentation of a tra
136 ranscranial magnetic stimulation of the left occipital cortex evoked contraction of right hand muscle
137 hese independently recorded variables, i.e., occipital cortex excitability and reactivity and EEG pha
139 tion of the changes in thickness across DF's occipital cortex, finding the most substantial loss in t
141 hey showed relative hypoactivity in the left occipital cortex for the low spatial frequency faces.
142 s are distributed widely across the inferior occipital cortex, fusiform areas, and the cingulate gyru
143 ients also appeared to have persistently low occipital cortex GABA after chronic benzodiazepine treat
144 panic disorder had a 22% reduction in total occipital cortex GABA concentration (GABA plus homocarno
145 of this study was to determine whether these occipital cortex GABA concentrations are altered after a
146 report, using this technique, suggested that occipital cortex GABA concentrations are reduced in pati
147 Depressed subjects had significantly lower occipital cortex GABA concentrations compared with healt
152 s of proton magnetic resonance spectroscopy, occipital cortex GABA concentrations were measured in 11
154 GABA neuronal response (blunted reduction of occipital cortex GABA level) to acute benzodiazepine adm
155 etic resonance spectroscopic measurements of occipital cortex GABA levels across the menstrual cycle
156 cts, who exhibited a significant decrease in occipital cortex GABA levels after this intervention.
157 a parallel-group, repeated-measures design, occipital cortex GABA responses to acute oral, open-labe
159 results indicate that superior IPS, but not occipital cortex, has a central role in VSTM storage.
161 t a silent zone at the posterior pole of the occipital cortex, implying a lack of complete cortical r
162 ntral striatum, inferior temporal gyrus, and occipital cortex in both depression and schizophrenia in
164 alpha band range from posterior thalamus to occipital cortex in congenitally blind participants.
165 e EEG recordings from frontal, parietal, and occipital cortex in freely moving rats (n = 11) during a
166 y alpha-band activity (8-15 Hz) over parieto-occipital cortex in humans plays an important role in su
167 d potential responses were recorded over the occipital cortex in patients with schizophrenia and in a
168 , posterior cingulate cortex, precuneus, and occipital cortex in patients with TLE as compared with h
169 r cingulate cortices and deactivation in the occipital cortex in response to the drug-related stimulu
172 ontal and parietal control regions to visual occipital cortex in visuospatial attention, the goal mot
173 uperior parietal lobule and the left lateral occipital cortex) included the default mode network seed
175 parietal cortices without any change in the occipital cortex, indicating that Fgf2 is necessary to r
178 to those with persistent ADHD in the medial occipital cortex, insula, parahippocampus, and prefronta
179 daptation for small shape changes in lateral occipital cortex irrespective of category membership, co
181 emianopic patients and suggest that when the occipital cortex is damaged or inhibited, and the visual
182 corroborative clinical evidence suggest that occipital cortex is engaged in tactile tasks requiring f
185 at the spatial pattern of propagation in the occipital cortex is non-concentric with a variable exten
186 siform gyrus, and insula, and extending into occipital cortex (left hemisphere) and orbitofrontal cor
187 to localize object-specific ROIs in lateral occipital cortex (LO) and scene-specific ROIs in the par
188 probe whether dorsal regions of the lateral occipital cortex (LO) are activated in tactile recogniti
189 ent fMRI studies have identified the lateral occipital cortex (LO) as a potential neural origin of th
190 with scrambled line drawings in the lateral occipital cortex (LO) of the ventral stream, an area tha
191 (2) a decrease in activation of the lateral occipital cortex (LO), and (3) a decrease in the dorsal
192 region, such as the shape-selective lateral occipital cortex (LO), must still base its activation on
194 pattern in both the retinotopic and lateral occipital cortex (LOC) in humans contains category infor
195 l FFA and bilateral object-selective lateral occipital cortex (LOC) predicted the participants' abili
196 Neurostimulation was targeted at the lateral occipital cortex (LOC), a key region for object percepti
197 C) and the higher-level visual area, lateral occipital cortex (LOC), known to be critically involved
203 on of a mid-level visual region [the lateral occipital cortex (LOC)] in a population much younger tha
206 rch suggests an enhanced excitability in the occipital cortex may underlie this reaction during IPS,
209 pital face area, fusiform face area, lateral occipital cortex, mid fusiform, parahippocampal place ar
210 length (posterior cingulate cortex, inferior occipital cortex, middle temporal cortex, hippocampus, a
211 scranial magnetic stimulation (TMS) over the occipital cortex modulated the impact of memory on searc
212 ions between the left temporal lobe and left occipital cortex not showing evidence of development unt
214 relative to total creatine (GABA/Cr) in the occipital cortex (OC), anterior cingulate cortex (ACC),
216 e report motor cortical function in the left occipital cortex of a subject who suffered a left middle
220 m the dorsolateral prefrontal cortex and the occipital cortex of elderly patients with schizophrenia
226 the right supraorbital area], but not of the occipital cortex or sham stimulation, increased the prop
227 or parietal cortex, the right or left dorsal occipital cortex or the right ventral occipital cortex,
228 me trials by (1) pretrial phase synchrony of occipital cortex oscillations in the 8-9 Hz (low alpha)
230 halamus (P = .04), frontal cortex (P = .01), occipital cortex (P = .004), and cingulate cortex (P = .
231 anterior cingulate cortex (P =.003) and the occipital cortex (P =.01) in the depressed subjects.
232 cerebellum, with intermediate values in the occipital cortex, parietal cortex, and caudate putamen.
234 ge of pERK1/2 levels in the temporal cortex, occipital cortex, parietal cortex, midbrain, and medulla
235 owed significant metabolic reductions in the occipital cortex, particularly in the primary visual cor
236 Furthermore, these findings suggest the occipital cortex plays a key role in supporting mental r
237 erior cingulate cortex (ACC) and the parieto-occipital cortex (POC) in adolescents (n=30) and emergin
239 metic problem complexity in ventral temporal-occipital cortex, posterior parietal cortex, and medial
240 havior disorder (RBD) with bilateral parieto-occipital cortex, precuneus, and ventrolateral-frontal m
241 al cerebellum and in a region in the lateral occipital cortex presumably corresponding to the area KO
252 cortex was unresponsive to Braille words and occipital cortex responded to spoken words but not diffe
256 seline BOLD response in the bilateral middle occipital cortex, selectively during the stimulus-proces
258 ntrast, superior parietal lobule and parieto-occipital cortex showed greater activation to the apart
259 recruited by patients only: the left dorsal occipital cortex showed systematic activation in all con
260 Electroencephalogram recorded over the left occipital cortex showed: 1) coherence with electromyogra
261 trastriate occipital cortex, whereas primary occipital cortex shows consistent activity across all sc
262 ttsburgh Compound B retention selectively in occipital cortex, sparing regions typically labeled in A
263 sponse decrements in right frontal and right occipital cortex, strongly supporting the compensatory r
264 ntrol (medulla and pons), and vision (dorsal occipital cortex, superior colliculi and lateral genicul
265 nificantly higher mean SUVR in PMT overlying occipital cortex than both other groups, although not in
268 tion of the ipsilateral field within lateral occipital cortex that is normally associated with visual
270 , after suppressing the activity in the left occipital cortex, the congruency-dependent response faci
271 ical areas, including portions of the medial occipital cortex, the lateral parietal cortex, and the s
273 information being encoded (fusiform/lateral occipital cortex), they each exerted opposite effects on
274 simplified reference tissue method with the occipital cortex time activity curve as an input functio
275 oops that are dominated by flow from parieto-occipital cortex to integrative frontal areas in the hig
276 scranial magnetic stimulation (TMS) over the occipital cortex to interfere with attentional reorienti
277 s in excitability affect the function of the occipital cortex to other, less provocative visual stimu
279 ed negative BOLD response (NBR) in the human occipital cortex, triggered by stimulating part of the v
280 voked potentials (SSVEPs) were measured over occipital cortex using electroencephalography (EEG).
284 brain regions, including the ventrotemporal occipital cortex (VTOC), the posterior parietal cortex,
288 unctional activation (measured with fMRI) in occipital cortex was more extensive when participants vi
289 By contrast, pattern similarity in lateral occipital cortex was related to memory only when context
290 tal, left superior parietal, and left middle occipital cortex) was evaluated using fMRI datasets acqu
291 ht posterior insula, anterior cingulate, and occipital cortex were examined in subjects at rest.
292 Granger causality from FEF and IPS to visual occipital cortex were significantly greater than both bo
293 scenes approximately 1 s before extrastriate occipital cortex, whereas primary occipital cortex shows
296 istration in overlapping areas in the middle occipital cortex while performing the same task conditio
297 ctive cathodal-inhibitory tDCS over the left occipital cortex, while, in control Experiment 2, partic
299 date nucleus to 14.5 +/- 5.3 mL/cm(3) in the occipital cortex, with intermediate values in the amygda