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1 .g. the insula) compared to others (e.g. the occipital lobe).
2 ht-dorsomedial prefrontal cortex), and iTBS (occipital lobe).
3  (relative sparing of the hippocampus and/or occipital lobe).
4 ons measured (temporal, frontal, and parieto-occipital lobes).
5  of foveal and peripheral signals within the occipital lobe.
6 ith fibres of the lateral pathway within the occipital lobe.
7 llow damage to primary visual cortex, in the occipital lobe.
8 r colour in more dorsolateral regions of the occipital lobe.
9 ure and can be traced posteriorly around the occipital lobe.
10 opographically organized visual areas of the occipital lobe.
11 rface coil to measure a 14-cm3 volume in the occipital lobe.
12  (b) dorsal posterior sigmoid gyrus, and (c) occipital lobe.
13 parietal and temporal lobes, and in the left occipital lobe.
14 ) placed in the white matter at the parietal occipital lobe.
15 in FTP-signal were only found in the lateral occipital lobe.
16 bserved in the parietal lobe followed by the occipital lobe.
17  intracranial electrodes placed on the human occipital lobe.
18  gamma peak frequency and amplitude from the occipital lobe.
19 in extrastriate visual cortex of the ventral occipital lobe.
20 emporal lobe, posterior parietal cortex, and occipital lobe.
21 m away from the most posterior aspect of the occipital lobe.
22 he right inferior frontal gyrus, but not the occipital lobe.
23 its for simple targets are restricted to the occipital lobe.
24 ical white matter of the frontal and parieto-occipital lobes.
25 urons were stained in frontal, parietal, and occipital lobes.
26 rsistent white matter changes in the parieto-occipital lobes.
27 w density in the white matter of the parieto-occipital lobes.
28 elieved to require integrity of the parietal-occipital lobes.
29 dial and lateral regions of the temporal and occipital lobes.
30  absolute regional volume in the frontal and occipital lobes.
31 crohemorrhages in the temporal, parietal and occipital lobes.
32 x differences across temporal, parietal, and occipital lobes.
33 were examined across temporal, parietal, and occipital lobes.
34 ly in frontal, temporal, medial temporal and occipital lobes.
35  across the subcortex, frontal, parietal and occipital lobes.
36 ith age but it decreased in the parietal and occipital lobes.
37 ory periphery in the parietal, temporal, and occipital lobes.
38 olandic operculum, and superior and inferior occipital lobes.
39 ciative areas of the temporal, parietal, and occipital lobes.
40 d right and left superior and right inferior occipital lobes.
41 d, localized to portions of the temporal and occipital lobes.
42 emotor regions, and also in the parietal and occipital lobes.
43  prefrontal, the right parietal and the left occipital lobes.
44 ntion would be disproportionately greater in occipital lobes.
45 nd parietal lobes but not in the frontal and occipital lobes.
46 ickening was demonstrated in the frontal and occipital lobes.
47 specially the frontal, lateral temporal, and occipital lobes.
48 us and regions of the temporal, parietal and occipital lobes.
49 ost commonly affected region was the parieto-occipital lobes (100%), however, other atypical regions
50 1, volume: 11,296 mm3, P < 0.001), bilateral occipital lobe (12.1 15.0 mL . 100 g-1 . min-1, volume:
51 ons for CVAs in UC patients were frontal and occipital lobes (19% and 18%, respectively).
52 3 +/- 3.6 mL/100 g/min; P = 0.007) and right occipital lobes (-4.3 +/- 5.6 mL/100 g/min; and -3.9 +/-
53   ARIA-E was more frequently observed in the occipital lobe (90%).
54 ions, and identify additional sources in the occipital lobe, a region expected to be active in our pa
55 nimals selectively activated the left medial occipital lobe--a region involved in the earliest stages
56               We observed a higher MI in the occipital lobe across all ages, and occipital MI increas
57 r hippocampal functional connectivity to the occipital lobe and cerebellum (left hippocampus: 9176 mm
58 le there were no notable distinctions in the occipital lobe and insula cortex.
59 iated with reduced gray matter volume in the occipital lobe and left cerebellar lobule VIIb, which is
60 d associations between conception and larger occipital lobe and nucleus accumbens volume, increased s
61 spheric communication between regions of the occipital lobe and of the superior parietal lobules.
62 pathways, including the lingual gyrus in the occipital lobe and the inferior parietal lobe, which had
63 ent spatial frequencies were mapped over the occipital lobe and then over the entire brain.
64 cal spread to the limbic system, whereas the occipital lobes and cerebellar hemispheres were unaffect
65 matter from frontal, parietal, temporal, and occipital lobes and cerebellum as well as basal ganglia.
66  in anterior temporal lobes, parietal lobes, occipital lobes and cerebellum.
67  functional connectivity in the temporal and occipital lobes and deep gray matter structures and with
68 rcts localized to the frontal, temporal, and occipital lobes and thalamus were associated with greate
69 nts showed activation in the fusiform gyrus, occipital lobe, and inferior frontal cortex relative to
70 nterior-posterior stretch in the frontal and occipital lobes, and superior-inferior stretch in right
71 frontal cortices; the temporal, parietal and occipital lobes; and the hippocampus, thalamus, and amyg
72 dle frontal gyri bilaterally and in the left occipital lobe as a control region.
73 bplate, and intermediate zones of the monkey occipital lobe as early as E70.
74 gyrus, lateral and medial temporal lobe, and occipital lobe as well as caudate and putamen nuclei, af
75 cluding the frontal, parietal, temporal, and occipital lobes, as well as the insula and cingulate cor
76 mpal (B = -0.08; 95% CI, -0.13 to -0.02) and occipital lobe (B = -0.70; 95% CI, -1.19 to -0.21) volum
77 d with significantly decreased volume in the occipital lobe (B, 0.02; 95% CI, 0.002 to 0.04; P = .03)
78 adoxical ability of some human subjects with occipital lobe brain damage to discriminate unseen stimu
79 ller gray matter volumes in the temporal and occipital lobes, but no difference between groups was fo
80 g light results in similar activation in the occipital lobes by fMRI.
81 bolic reductions were most pronounced in the occipital lobe, caudate nucleus, and temporal lobe.
82 impairment (MCI) and AD in the frontal lobe, occipital lobe, cerebellum, temporal, and parietal lobe,
83  least two characteristics localizing to the occipital lobe (clinical symptoms, interictal focus, ict
84 reased in the precuneus and the parietal and occipital lobes compared with subjects with subjective c
85                                          The occipital lobe contains retinotopic representations of t
86 measured glutamate concentrations in ACC and occipital lobe cortex (OCC) in 13 subjects with T1D with
87 f five white matter tracts connecting to the occipital lobe, cortical spinal tract, limbic system, pi
88 The lateral temporal, frontal, parietal, and occipital lobe cortices appeared normal for age 66 years
89 revealed that activity in large parts of the occipital lobe covaried with each.
90                Seven human participants with occipital lobe damage enrolled in a visual training prog
91  39 patients (12 female) with hemianopia and occipital lobe damage.
92  disorder; and neuroimaging demonstration of occipital lobe damage.
93 gyri than sulci and in frontal compared with occipital lobes, decreasing consistently throughout the
94  seizure control, outcome after resection of occipital lobe developmental abnormalities is less unifo
95 anscranial magnetic stimulation of the human occipital lobe disrupts the normal perception of objects
96 od flow increases were observed in the right occipital lobe during the memory task relative to the co
97                         Medically refractory occipital lobe epilepsies are increasingly treated with
98 s had unilateral frontal lobe epilepsy, five occipital lobe epilepsy (OLE), six parietal lobe epileps
99 main pathological substrates of uncontrolled occipital lobe epilepsy are gliomas and developmental ab
100 n was present, intact cortex in the lesioned occipital lobe exhibited conventional retinotopic organi
101 fiber tracking (FT) were used to measure the occipital lobe fiber tracts connecting the two hemispher
102  dissections demonstrated connections to the occipital lobe from the fusiform gyrus along with longer
103 ammonis region 1, but not in the thalamus or occipital lobes from the same brains.
104              The results revealed regions of occipital lobe, frontal lobe, supplementary motor area,
105 lear magnetic resonance spectroscopy, serial occipital lobe GABA and homocarnosine concentrations wer
106                    Baseline concentration of occipital lobe GABA+ (GABA plus coedited macromolecules)
107 icipants with WS showed reduced thalamic and occipital lobe gray matter volumes and reduced gray matt
108 lying solely on these four electrodes of the occipital lobe had the potential to serve as an objectiv
109  + iTBS (L-DLPFC), iTBS (L-DLPFC), and iTBS (occipital lobe) had a higher response rate (k = 23); cTB
110  bodies (DLB) have both been associated with occipital lobe hypometabolism on (18)F-FDG PET, whereas
111 calized to the temporal lobe in 9 and to the occipital lobe in 1; seizure onset was poorly localized.
112 phere could be traced posteriorly around the occipital lobe in 45 (90%).
113 ct connection between the olfactory bulb and occipital lobe in any species and is a step toward furth
114 eactivity was significantly decreased in the occipital lobe in both presymptomatic (mean BOLD change
115 in meninges and calvarial bone overlying the occipital lobe in migraine with visual auras.
116 shown in the cortex of the left parietal and occipital lobe in the first patient and symmetrically in
117              Demyelination involved parietal-occipital lobes in 90%, leading to visual and auditory p
118 ost prominent in the frontal, cingulate, and occipital lobes in all high-risk individuals compared wi
119 l lobes had a lower uptake than parietal and occipital lobes in general.
120 P levels were found in frontal, parietal and occipital lobes in MCI and late AD compared to controls
121 ease in F(4)-NPs was present in parietal and occipital lobes in MCI compared to controls and a signif
122 haracteristics of the frontal, parietal, and occipital lobes in the memory encoding stage of MCI prov
123 ional thinning of the parietal, temporal and occipital lobes in the VLBW group, whereas regional thic
124 dal neurons in tissue from human frontal and occipital lobes, in 11 cases with (M(age) = 79 +/- 7 yea
125 he cuneus, lingual and fusiform gyri, middle occipital lobe, inferior parietal lobule, and also cingu
126 timulation is performed there, activation of occipital lobes is seen by fMRI.
127  (total and frontal, parietal, temporal, and occipital lobes), lateral ventricular cerebrospinal flui
128 ippocampus and right middle temporal gyrus), occipital lobe (left lingual gyrus), anterior cingulate,
129 st RT group than in the slow RT group in the occipital lobes, left sensorimotor cortices, and supplem
130 ), a common visual impairment resulting from occipital lobe lesions, affects a significant number of
131 ), right posterior cingulate gyrus, and left occipital lobe (lingual gyrus).
132 erns of hypometabolism involving the lateral occipital lobe, lingual gyrus, cuneus, precuneus, poster
133 erns of hypometabolism involving the lateral occipital lobe, lingual gyrus, cuneus, precuneus, poster
134 linical variables were also unrelated to the occipital lobe location of abnormality.
135                                  Outcome and occipital lobe location were analyzed with respect to su
136 ionship to etiology, pathological substrate, occipital lobe location, surgical approach, and electroc
137  from those where the line sloped upwards in occipital lobe (lvPPA-high), representing unusually high
138 bjects in which the line sloped downwards in occipital lobe (lvPPA-low), representing low uptake, wer
139 y decreased cortical grey matter rCBF in the occipital lobe (mean difference -11.1 mL/100 g per min,
140 lective involvement of parietal, frontal and occipital lobes might explain some of the clinical and n
141  = 137; parietal lobe n = 240 vs n = 93; and occipital lobe, n = 241 vs n = 97, respectively; P < .00
142                                       In the occipital lobe, NAA and ADP are 11.9 +/- 1.9mM and 32 +/
143 nit, and contrast them to image onset and to occipital lobe neurons.
144 ns were obtained: (1) four electrodes in the occipital lobe (OCL4); (2) three electrodes in the prefr
145 hates, and ADP levels in the hippocampus and occipital lobe of 15 healthy volunteers.
146 icant grey matter atrophy bilaterally in the occipital lobe of PDD patients compared with Parkinson's
147 ective areas in the fusiform gyrus (FFA) and occipital lobe (OFA).
148 g of the retinal nerve fibre layer following occipital lobe/optic radiation damage due to stroke.
149  showed greater hypometabolism in the medial occipital lobe, orbitofrontal cortex, anterior temporal
150 y differ with respect to location within the occipital lobe (overall medial [50%] or lateral [38%]);
151 nt activation decreases in the left anterior occipital lobe (P=.016), right posterior temporal lobe (
152 n bilateral, frontal, parietal, temporal and occipital lobes (P < 0.001), while FTD patients had a th
153  the posterior P2 component over the parieto-occipital lobe peaked significantly earlier for the uniq
154                                              Occipital lobe perfusion defects have been identified on
155 es of connectivity between both temporal and occipital lobes predicted reading errors.
156 d early involvement of the left temporal and occipital lobes, progressing to extensive left hemispher
157 a R = +0.62, p < 3 x 10(-4), whereas, in the occipital lobe, R = +0.67, p < 5 x 10(-5).
158 f actual to expected hemorrhages = 1.37) and occipital lobes (ratio = 1.45, p < 0.0001).
159  the thalamus and decreased rCBF in the left occipital lobe, right cerebellum, and temporal cortex bi
160 lateral middle frontal, temporal and lateral occipital lobes, right precentral and superior frontal c
161 ns of interest in anterior temporal lobe and occipital lobe sections corresponding closely to those u
162 inar central lateral (CL) nucleus and medial occipital lobe seen in controls (p < 0.001, paired t-tes
163          Initial signs or symptoms suggested occipital lobe seizure onset in 13 of 16 patients.
164 tients who underwent surgery for intractable occipital lobe seizures were retrospectively evaluated.
165 sterior parietal cortex or the right ventral occipital lobe selectively induced a significant shift t
166 c variances in the central sulcus region and occipital lobe suggest potential difficulties in control
167 logy of the rolandic cortex) and the ICOE-G (occipital lobe symptomatology), while the autonomic clin
168 6.47) gyri, and one right ILF end point, the occipital lobe (T = 5.36).
169                                   The caudal occipital lobe, temporal pole, genu and prefrontal white
170 ose hypometabolism (P < 0.001) involving the occipital lobes, temporal lobes, limbic system, cerebell
171 ose hypometabolism (P < 0.001) involving the occipital lobes, temporal lobes, limbic system, cerebell
172 hat have shown that following lesions of the occipital lobe, the retinal nerve fibre layer thickness
173 campus and parahippocampal gyrus, and in the occipital lobe, the right frontal lobe and the left pari
174               The activation volumes in both occipital lobes, the left sensorimotor cortex, and the s
175 ately 8% in the frontal lobes and 12% in the occipital lobes-these regions also showed significant vo
176 dema of the white matter of the parietal and occipital lobes; this syndrome is potentially reversible
177  5 days followed by (1)H MRS scanning in the occipital lobe to measure the change in intracerebral gl
178                         Whereas resection of occipital lobe tumors associated with chronic epilepsy p
179 aseline and during visual stimulation in the occipital lobe using (31)P magnetic resonance spectrosco
180 thout (95% CI = -8.71, -0.84; P = 0.02), and occipital lobe volume was 1.86 cc lower for those with l
181 the foot as a control, and activation in the occipital lobes was not observed.
182 hole brain (frontal, oriental, temporal, and occipital lobes) was active in creative processes of eng
183 g transcranial magnetic stimulation over the occipital lobe, we showed a clear impairment in the accu
184 nterior & posterior temporal lobe and in the occipital lobe were found, with amplitude increases of 3
185       In addition, some brain regions in the occipital lobe were significantly more sensitive to cont
186 pth) in the frontal, parietal, temporal, and occipital lobes were calculated.
187                                          The occipital lobes were obtained after death from six adult
188 rences in the brains were small, even in the occipital lobe where only a small region of the primary
189 cts were most prominent for the parietal and occipital lobe, which showed divergent effects as early
190 C in the prefrontal, temporal, parietal, and occipital lobes, while volume was reduced only in the te
191 tive processes are mainly in the frontal and occipital lobes; while the whole brain (frontal, orienta
192 rmal optic nerves and only subtle changes to occipital lobe white and gray matter.
193 us, right inferior temporal gyrus, and right occipital lobe white matter.
194 ed a low-frequency sinusoidal current to the occipital lobe with an offset frequency to the flash sti
195 s was found in all brain regions, except the occipital lobe, with large effect sizes in the parietote

 
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