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1 ence in rabbits (based on survival following ocular infection).
2 ne model of naturally-acquired P. aeruginosa ocular infection.
3 y to delay its clearance from the eye during ocular infection.
4  of infected mice between days 1 and 5 after ocular infection.
5 fic effector T (T(eff)) cells induced during ocular infection.
6 oss lymphoid and extralymphoid tissues after ocular infection.
7 mice than in BALB/c and CBA/J mice following ocular infection.
8 ndicating the critical role of HVEM in HSV-1 ocular infection.
9 le of CXCL10 during the acute phase of HSV-1 ocular infection.
10 d disease occurs in humans following primary ocular infection.
11 imulation in susceptibility to P. aeruginosa ocular infection.
12 nd enteric washes and were protected against ocular infection.
13  for the treatment of symptomatic adenoviral ocular infection.
14 TMCs) and an IFNAR1-deficient mouse model of ocular infection.
15 A1 (sncRNA1) encoded within LAT during HSV-1 ocular infection.
16 and disease progression in a murine model of ocular infection.
17 the conjunctiva did not result in successful ocular infection.
18 ymphotropic dissemination of HSV-1 following ocular infection.
19 ding virulence and immune responses to viral ocular infection.
20 n models: mouse footpad infection and rabbit ocular infection.
21 s into immunopathological responses to viral ocular infection.
22 tics in order to gain effective control over ocular infection.
23 SE by acyclovir treatment provided 4 d after ocular infection.
24  of either receptor attenuates disease after ocular infection.
25  against herpes simplex virus type 1 (HSV-1) ocular infection.
26 ls of testican-1 after P. aeruginosa-induced ocular infection.
27 ratitis (AK) is a rare but sight-threatening ocular infection.
28 al wild-type adenoviral serotypes that cause ocular infections.
29 unoglobulin for treatment and prophylaxis of ocular infections.
30 therapy for the treatment of adenoviral (Ad) ocular infections.
31 monas aeruginosa are the leading isolates in ocular infections.
32 ence showing excellent potency in the war on ocular infections.
33 f this antiviral drug in treating adenoviral ocular infections.
34 visual outcomes in endophthalmitis and other ocular infections.
35 teria are the major contributor of bacterial ocular infections.
36 was aimed to review the bacterial profile of ocular infections.
37 ibility pattern among patients with external ocular infections.
38 s hospital infections and community-acquired ocular infections.
39 ibacterials used for empirical management of ocular infections.
40 marily on innate immunity to protect against ocular infections.
41 n be a vector for transmission of adenovirus ocular infections.
42 live attenuated vaccines against genital and ocular infections.
43 is known about the role of magA in secondary ocular infections.
44 lens could be used as a treatment for fungal ocular infections.
45  differentiate Fusarium spp. responsible for ocular infections.
46 he role of NK cells in the modulation of CMV ocular infection, 9.0 x 10(2) plaque-forming units of th
47                                    Following ocular infection, all three viruses produced similar acu
48  simplex virus type 1 (HSV-1) during primary ocular infection and after reactivation of latent infect
49 of CD4 knockout (KO) mice after both primary ocular infection and challenge with RH tachyzoites.
50 n four districts, we incorporated testing of ocular infection and serology into routine trachoma impa
51 al trachoma but low levels of C. trachomatis ocular infection and seropositivity.
52 ns that cause sexually transmitted diseases, ocular infections and atypical pneumonias.
53 ds are widely prescribed in the treatment of ocular infections and disorders.
54  Pneumococcus is also a major cause of human ocular infections and is commonly isolated in cases of b
55                  Bacteria are major cause of ocular infections and possible loss of vision.
56 ed in the eyes of control mice after primary ocular infection, and near-normal histology with few tac
57 ease in HSV-1-infected mice.IMPORTANCE HSV-1 ocular infections are the leading cause of corneal blind
58 s conducted among 160 patients with external ocular infections at Borumeda hospital, Northeast Ethiop
59             Most of the isolates of Fusarium ocular infections belong to the F. solani or F. oxysporu
60 s latent HSV infection in the mouse model of ocular infection but has no impact on the maintenance of
61 of explant reactivation at the lower dose of ocular infection but not at the higher dose.
62 ratory tract suspected to be responsible for ocular infections but no well-described case of D. pigru
63                                              Ocular infection by Chlamydia trachomatis is the leading
64 are highly antibiotic resistant, and primary ocular infection by ESBL E coli has rarely been reported
65 ns of DCs to the protection afforded against ocular infection by immunization against HSV-1 and their
66 t sncRNA1 has a protective role during acute ocular infection by modulating the innate immune respons
67           PPK1 is essential for a successful ocular infection by P. aeruginosa.
68                                              Ocular infection by this organism is the leading cause o
69 ion of neutrophils during primary chlamydial ocular infection by using the guinea pig model of Chlamy
70 -associated MRSA is an important pathogen of ocular infections; CA-MRSA and HA-MRSA ocular infections
71 arises during Pseudomonas aeruginosa-induced ocular infection can trigger tissue damage resulting in
72                                       Fungal ocular infections cause significant ocular morbidity, pa
73                                 Trachoma, an ocular infection caused by Chlamydia trachomatis, is the
74 ulation approach to suppress the severity of ocular infections caused by herpes simplex virus infecti
75 e overlapping functions.IMPORTANCE Recurring ocular infections caused by HSV-1 can cause corneal scar
76                                        While ocular infections caused by S. maltophilia are documente
77 ility (meeting release criteria) and safety (ocular infection, corneal perforation, or graft detachme
78 y and duration of Chlamydia trachomatis (Ct) ocular infections decrease with age, suggesting developm
79         Successful outcome for patients with ocular infection depends on close collaboration between
80 en of ocular infections; CA-MRSA and HA-MRSA ocular infections differ demographically and clinically,
81 The rare descriptions, in the literature, of ocular infections due to Pasteurella multocida include:
82 ch has targeted immune mechanisms in primary ocular infections, events that could impact chronic resp
83 erature ( degrees C) was not associated with ocular infections for any pathogen type.
84 rachomatis is a leading cause of genital and ocular infections for which no vaccine exists.
85                            Following primary ocular infection, herpes simplex virus type 1 (HSV-1) es
86                                    Following ocular infection, herpes simplex virus type 1 (HSV-1) es
87                                        After ocular infection, HSV-specific CD8+ T cells migrate to a
88                                              Ocular infections, if left untreated, can damage the str
89 d treatment of Staphylococcus aureus-induced ocular infection in both rats and rabbits compared to go
90 with the peripheral nervous system following ocular infection in mice.
91  a transcriptional level during latent HSV-1 ocular infection in mice.
92  is a dearth of information on prevalence of ocular infection in our population.
93 nesis of herpes simplex virus type 1 (HSV-1) ocular infection in the mouse.
94                                    Following ocular infection in vivo, clusters formed exclusively in
95 , and topical application suppressed primary ocular infection in vivo.
96 tunistic pathogen Serratia marcescens causes ocular infections in healthy individuals.
97 4, has only been reported to cause wound and ocular infections in humans.
98 a topical antiviral treatment for adenoviral ocular infections in the target population.
99 ainst the Zika virus (ZIKV), in general, and ocular infection, in particular, has never been investig
100 he role of neutrophils in primary chlamydial ocular infection, indicates a previously unappreciated r
101                                     External ocular infection is a public health problem in Ethiopia.
102                       Mycobacterium chimaera ocular infection is a rare disease that is linked to byp
103  that the anatomic host site in pneumococcal ocular infections is important.
104 l as well as molecular investigation on HAdV ocular infections is rather absent in Greece, which has
105 ), a subgroup D virus associated with severe ocular infections, is unable to use CAR efficiently to i
106 icroflora isolates, 0.80 (CI, 0.54-0.94) for ocular infection isolates, and 1.0 (CI, 0.45-1.0) for st
107                                        Among ocular infection isolates, invasive and cytotoxic strain
108 ied a potential target for modulation during ocular infection, macrophage migration inhibitory factor
109       In immunized mice during primary HSV-1 ocular infection, macrophages play an important role in
110 nanosuspension's potential for comprehensive ocular infection management by reducing treatment freque
111                                              Ocular infections may arise spontaneously or following p
112  anti-HSV-1 mechanisms of murine IFN-beta in ocular infection, mice were transduced with an adenovira
113 hese phenotypes are also mirrored in a mouse ocular infection model.
114 st to provide evidence that in P. aeruginosa ocular infection, mouse strains favoring development of
115                                    Following ocular infection, mouse survival decreased in the DeltaC
116                    In the 7-day period after ocular infection of BALB/c mice, the replication of HSV-
117                    At 11 days postinfection, ocular infection of C57BL/6 mice with all of the strains
118 ollowing herpes simplex virus type 1 (HSV-1) ocular infection of C57BL/6 mice, activated CD8(+) T cel
119             We have reported previously that ocular infection of different strains of mice with recom
120                         Following footpad or ocular infection of mice lacking type I IFN receptors, H
121     Conversely, forced expression of CD80 by ocular infection of mice with a recombinant HSV-1 exacer
122 dministration begun at different times after ocular infection of mice with HSV could influence the se
123                           Here, we show that ocular infection of mice with HSV-1 suppressed expressio
124 nd when applied topically, inhibited primary ocular infection of mice.
125 n) failed to infect ganglionic neurons after ocular infection of mice.
126 cessfully infecting ganglionic neurons after ocular infection of mice.
127 lactic Delta41Delta29 vaccination on primary ocular infection of NIH inbred mice with HSV-1 McKrae, a
128 ut most importantly in virulence as shown by ocular infection of nonhuman primates.
129                                    Following ocular infection of rabbits, dLAT1.5 reactivated at a lo
130 an wild-type or marker-rescued viruses after ocular infection of rabbits.
131 l vaccine, gD2 alum, protected against acute ocular infection only.
132 cular necrosis in response to either primary ocular infection or challenge.
133 without evidence of either systemic or local ocular infection or inflammation.
134 d intravenously, intramuscularly, topically (ocular infections), or orally.
135 KT cells in anti-HSV-1 immunity during HSV-1 ocular infection (P.
136          To mitigate the burden of bacterial ocular infections, physicians should regard on risk redu
137                                    Bacterial ocular infections pose significant risks to vision and i
138  the many problems associated with recurrent ocular infection, reducing virus reactivation should be
139 patient with signs or symptoms suggestive of ocular infection regardless of Candida septicemia.
140              However, its regulatory role in ocular infection remains unclear.
141    There have been 5 cases of M. haemophilum ocular infections reported in the literature.
142 ing IL-18 to the cornea of mice before HSV-1 ocular infection resulted in reduced angiogenesis and di
143                        At-risk patients with ocular infections should be asked about close contact wi
144 IL-10(-/-) animals depleted of nTregs before ocular infection showed more severe SK lesions as compar
145 r System, Central Nervous System Infections, Ocular Infections, Soft Tissue Infections of the Head an
146 r System, Central Nervous System Infections, Ocular Infections, Soft Tissue Infections of the Head an
147                    In Pseudomonas aeruginosa ocular infection, T-helper cell 1-responsive mouse strai
148  DeltasncRNA1 virus were more susceptible to ocular infection than their wild-type (WT) counterparts.
149 osa keratitis (or pink eye) is a challenging ocular infection that causes serious complications due t
150 C. trachomatis infection causes trachoma, an ocular infection that leads to blindness, and sexually t
151 nduced fungal keratitis is a rare but severe ocular infection that may result in significant vision i
152 tible C57BL/6J (B6) mouse to resistant after ocular infection through modulation of the inflammatory
153                             Sixty days after ocular infection, trigeminal ganglia (TG) were removed f
154 ated population prevalence of C. trachomatis ocular infection was approximately 17.5%.
155                                       Latent ocular infection was associated with significant up-regu
156                                         When ocular infection was caused by an invasive strain in viv
157 moral immunity in herpes simplex virus (HSV) ocular infections was studied in immunoglobulin mu chain
158                                       Herpes ocular infections was suspected.
159 potheses, guinea pigs with primary C. caviae ocular infections were depleted of neutrophils by using
160         All patients with suspected external ocular infections were examined under slit lamp microsco
161                       Although isolates from ocular infections were found in all four groups, they ha
162         We identified 274 patients with MRSA ocular infections, which comprised 181 CA-MRSA and 93 HA
163            IMPORTANCE HSV-1 causes recurrent ocular infections, which is the leading cause of corneal
164 participants with symptomatic ocular or peri-ocular infections who were enrolled using a consecutive
165 oxin (Ac-CT) or with saline, before or after ocular infection with A. castellanii.
166 implex virus type 1 (HSV-1) during the acute ocular infection with and without AED treatment focusing
167 scertain the disease pattern of trachoma and ocular infection with C trachomatis in a trachoma hypere
168 ll households were examined for trachoma and ocular infection with C. trachomatis at baseline, and 6
169                 Trachoma, caused by repeated ocular infection with Chlamydia trachomatis (Ct), is tar
170 s where trachoma is mesoendemic suggest that ocular infection with Chlamydia trachomatis can be elimi
171                                              Ocular infection with Chlamydia trachomatis can lead to
172                    It is caused by recurrent ocular infection with Chlamydia trachomatis in childhood
173 tibiotic treatment could reduce trachoma and ocular infection with Chlamydia trachomatis in hyperende
174  inflammation-follicular (TF) is common, but ocular infection with Chlamydia trachomatis is not.
175 reventable blindness, is produced by chronic ocular infection with Chlamydia trachomatis, an obligate
176                 Trachoma, caused by repeated ocular infection with Chlamydia trachomatis, is an impor
177           Here, we examine the prevalence of ocular infection with CT and previous exposure to CT in
178            The host inflammatory response to ocular infection with herpes simplex virus (HSV) can be
179 erpetic stromal keratitis (HSK) that follows ocular infection with herpes simplex virus (HSV) is sugg
180                                              Ocular infection with herpes simplex virus (HSV) results
181                                              Ocular infection with herpes simplex virus (HSV) sets of
182 inding immunoinflammatory reaction caused by ocular infection with herpes simplex virus (HSV).
183 ammatory lesion stromal keratitis induced by ocular infection with herpes simplex virus (HSV).
184 tory stromal keratitis (SK) lesion caused by ocular infection with herpes simplex virus (HSV).
185 f immunization strategies to protect against ocular infection with herpes simplex virus 1 (HSV-1) mus
186                                              Ocular infection with herpes simplex virus 1 (HSV-1) res
187                                              Ocular infection with herpes simplex virus 1 (HSV-1) set
188  infiltrates in the cornea of mice following ocular infection with herpes simplex virus 1 (HSV-1).
189                                              Ocular infection with herpes simplex virus 1 can result
190             Stromal keratitis resulting from ocular infection with herpes simplex virus is a common c
191                                              Ocular infection with herpes simplex virus leads to an i
192 plicated in the modulation of the outcome of ocular infection with herpes simplex virus type 1 (HSV-1
193 Treatment was begun at different times after ocular infection with HSV and the outcome was assessed c
194                                              Ocular infection with HSV causes a chronic T cell-mediat
195                                              Ocular infection with HSV causes corneal neovascularizat
196                                              Ocular infection with HSV may result in the blinding imm
197                                              Ocular infection with HSV results in a blinding immunoin
198 sion was up-regulated (10- to 20-fold) after ocular infection with HSV, an event that involved the pr
199 e between VEGF-A and sVR-1 that occurs after ocular infection with HSV, which causes prominent neovas
200                                        Mixed ocular infection with HSV-1 strains CJ394 and OD4 yield
201  octamer (8mer) as an eye drop 1 hour before ocular infection with HSV-1 was investigated.
202 ny-stimulating factor 1 (CSF-1) DNA prior to ocular infection with HSV-1.
203 5-80%) experiencing development of HSE after ocular infection with HSV-1.
204 e are highly susceptible to HSV-1 infection, ocular infection with HSV-IL-4 resulted in 100% survival
205 A in the cornea and draining lymph node upon ocular infection with HSV.
206 ical changes and their implication following ocular infection with HSV.
207        Here we present a case of M. chimaera ocular infection with no CNS M. chimaera lesions on brai
208 s potential importance, the role of IL-12 in ocular infection with P. aeruginosa remains unexplored a
209                                     EXPOSURE Ocular infection with recombinant adeno-associated viral
210                                        After ocular infection with the HSV-1 strain McKrae, virus rep
211 study was conducted to determine whether the ocular infection with this recombinant virus induces opt
212 prevalent among staphylococcal isolates from ocular infections, with many strains demonstrating multi
213 vation.IMPORTANCE HSV-1 is a common cause of ocular infections worldwide and a significant cause of p
214        Bacteria are the major contributor of ocular infections worldwide.
215                   This impedes therapies for ocular infections, wound healing, and dry-eye disease th

 
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