戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (1語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 erentiation of odontoblasts throughout later odontogenesis.
2  the expression of several genes involved in odontogenesis.
3 nown stimulators of epithelial growth during odontogenesis.
4 erning of the first branchial arch (BA1) and odontogenesis.
5 smaller subunits in the dentin matrix during odontogenesis.
6 aining sequences encoded by exons 8/9 during odontogenesis.
7 play a crucial role during the initiation of odontogenesis.
8  in regulating CNC cell proliferation during odontogenesis.
9 /or repression of PAX9 effector genes during odontogenesis.
10 rom each other with respect to their rate of odontogenesis.
11 f tooth development during the initiation of odontogenesis.
12 ecies and is expressed at high levels during odontogenesis.
13 stricted to the dental epithelium throughout odontogenesis.
14  to the first morphological manifestation of odontogenesis.
15 actions that occur throughout the process of odontogenesis.
16 eneral, but realistic, mathematical model of odontogenesis.
17 in dental mesenchyme results in an arrest in odontogenesis.
18 d dental epithelium to establish its role in odontogenesis and craniofacial developmental.
19 se model, we have observed severe defects in odontogenesis and dentin formation with the removal of t
20 ritical role in regulation of both the early odontogenesis and later differentiation of hard tissue-p
21 eletion of the Dmp1 gene leads to defects in odontogenesis and mineralization.
22 ergistically activate gene expression during odontogenesis and miR-200a-3p attenuates their expressio
23 e kidney of periostin, a protein involved in odontogenesis and osteogenesis, has been suggested as a
24 cal functions of Runx2, Osx, and Dspp during odontogenesis and osteogenesis.
25 he non-cell-autonomous nature of Msx1 during odontogenesis, and disclose an additional late survival
26      FoxJ1(-/-) mice present with defects in odontogenesis, and we correlate these defects to hierarc
27                                 Mutations of Odontogenesis-Associated Phosphoprotein (ODAPH, OMIM *61
28  the E12.5 bud stage, is a key player during odontogenesis, being responsible for the initiation of t
29 on of BMP2 and DSPP is detected during mouse odontogenesis by in situ hybridization assay, and BMP2 u
30 tes several signaling pathways essential for odontogenesis, ciliary defects can interrupt the latter
31            Thus, Shh has dual roles in early odontogenesis, first in bud formation by stimulating epi
32 xpression of these three genes during murine odontogenesis from epithelial thickening to cytodifferen
33                                  Research in odontogenesis has shown that the oral epithelium of the
34  how DMP1 controls dentin mineralization and odontogenesis in vivo.
35                                              Odontogenesis is accomplished by reciprocal signaling be
36  the role of DPP-mediated Wnt5a signaling in odontogenesis is not well understood.
37 pproach to the investigation of this step of odontogenesis on the molecular level.
38                                              Odontogenesis or tooth development is a highly regulated
39 s NF-kappaB activity thus induces an ectopic odontogenesis program that is usually suppressed under p
40                Moreover, we showed that this odontogenesis-regulated expression of C5L2 is specifical
41 nt, the early signaling pathways involved in odontogenesis remain inducible in Aves and suggest that
42      However, the normal function of DLX3 in odontogenesis remains unknown.
43 oupled to complex shape pinpoints aspects of odontogenesis that might be re-evolved in the lab to sol
44 ral expression of the amelogenin gene during odontogenesis, the mouse amelogenin promoter was systema
45  expression of secreted signals that promote odontogenesis throughout the oral cavity.
46  both early and late odontoblasts for normal odontogenesis to proceed.
47                                       During odontogenesis, we find Islet1 to be exclusively expresse
48 tive signaling properties required to direct odontogenesis, which are not found in other branchial ar