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1 ents for the transforming actions of diverse oncogene proteins.
2 clude Ras, Dbl family, and G-protein-coupled oncogene proteins.
3 st in their more celebrated cousins, the Ras oncogene proteins.
4  observed by overexpressing c-Myc (Myc proto-oncogene protein), a downstream target of Pim kinases.
5                                          Ras oncogene proteins are plasma membrane-associated signal
6         Further, we identified the MYC proto-oncogene protein as a second CPSF1 substrate and show th
7        Contained in this sequence is the Myb oncogene protein binding site, TAACTG, which was shown p
8 l molecule and a disordered peptide from the oncogene protein c-Myc, we describe a "specific-diffuse"
9  increased levels of beta-catenin/Tcf target oncogene proteins c-myc and cyclin D1.
10                    The closely related proto-oncogene proteins CrkII and CrkL consist of one SH2 and
11 rticularly the case of proteins, such as the oncogene protein E7 of human papillomavirus type 16, whi
12 transforming proteins and include Dbl family oncogene proteins, G protein-coupled receptors and G pro
13 ammatory protein-4 (MIP-4), growth-regulated oncogene protein (GRO), monocyte chemoattractant protein
14                   This study defines IRSs as oncogene proteins in vivo and provides new models to dev
15 ression of the HGF receptor, the c-met proto-oncogene protein, is uniformly found in the human bronch
16 ng (activation of v-akt murine thymoma viral oncogene/protein kinase B [AKT], inhibition of glycogen
17 on by forming a novel complex with two other oncogenes, protein kinase C, iota and epithelial cell tr
18 Due to their ability to function as dominant oncogenes, protein kinases have become favored targets i
19           In addition, AURKA and N-myc proto-oncogene protein (MYCN) associate with each other to reg
20  the tumor suppressor protein p53, the viral oncogene protein pp60src, or a ubiquitin activating enzy
21 rs of transformation by a diverse variety of oncogene proteins that include Ras, Dbl family, and G-pr
22               Vav is a member of a family of oncogene proteins that share an approximately 250-amino-
23         CML is associated with increased abl oncogene protein tyrosine kinase (PTK) activity.
24 il activator (ENA-78) and the growth-related oncogene proteins, was markedly suppressed in PMN from s
25 aps due to an association of the HER-2 proto-oncogene protein with resistance to hormone and/or chemo