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1 utionary depths is an important predictor of oncogenic effect.
2 a deficiency alone does not exhibit a strong oncogenic effect.
3 with CDC73 in HEK293 cells abrogated its pro-oncogenic effect.
4 teins to lysosomes for degradation, with pro-oncogenic effects.
5  of Jak2 tyrosine phosphorylation in Bcr-Abl oncogenic effects.
6 n drugs reduce its expression and downstream oncogenic effects.
7 on of gene expression, and likely additional oncogenic effects.
8 n extracellular CD44 ligand with established oncogenic effects.
9  This interaction is primarily linked to pro-oncogenic effects.
10 Substance P and its truncated receptor exert oncogenic effects.
11  apoptosis, partially recapitulating SRSF1's oncogenic effects.
12 ve TpoR mutants, a function required for the oncogenic effects.
13 s while v-Src activates STAT3 to promote its oncogenic effects.
14 hat sequesters CLCF1, thereby inhibiting its oncogenic effects.
15 85% of patients, attempts to demonstrate its oncogenic effect alone have repeatedly failed, suggestin
16 ition of EZH2 abolish EPIC1's immune-related oncogenic effect and its suppression of interferon-y sig
17 UTR structure can significantly increase its oncogenic effect and worsen the clinical course of MCL p
18  stabilization in the stomach has negligible oncogenic effects and requires MYC activation to induce
19 ers are limited by existing knowledge on the oncogenic effects and therapeutic benefits of specific v
20  that GPC3 overexpression and its associated oncogenic effects are linked to the down-regulation of m
21       Studies in cell lines suggest that its oncogenic effects are mediated through the induction of
22 21(Cip1) induction, suggesting that Bcl11a's oncogenic effects are mediated, in part, through suppres
23         This study sheds light on contextual oncogenic effects associated with FGFR1 alterations and
24 ts indicate that SYT-SSX2 exerts part of its oncogenic effect by altering cytoskeletal architecture i
25 uced in the skin by UVR protects against its oncogenic effects by inhibiting Hedgehog signaling, wher
26             The corepressor Evi-1 exerts its oncogenic effects by repressing TGF-beta/Smad3-mediated
27            Mechanistically, ATDC exerted its oncogenic effects by suppressing miR-29 and subsequent u
28                                  Despite its oncogenic effect, cells with TSC deficiency were more se
29 an induce local inflammation and distant pro-oncogenic effects compared with hepatic radiofrequency a
30 of Akt is cholesterol sensitive and that the oncogenic effects conferred by myristoylation arise, in
31 with NFATc1 activation, indicating that NFAT oncogenic effects depend on cell types and tissue contex
32 regulated CRLF2 and potentiated the JAK-STAT oncogenic effects during leukemogenesis.
33 may be exploited in the future to target its oncogenic effects for patient benefit.
34  the distal gut, mutant p53 had the expected oncogenic effect; however, in the proximal gut and in tu
35 mor suppressor effect of SULF1, SULF2 has an oncogenic effect in HCC mediated in part through up-regu
36  raise the possibility that ErbB2 exerts its oncogenic effect in part by impairing TGFalpha-dependent
37 is tumor suppressor might have a paradoxical oncogenic effect in some hematopoietic cells.
38 fects of increased Bcr expression on Bcr-Abl oncogenic effects in a more physiological system, we tes
39 le gammaretroviruses have well-characterized oncogenic effects in animals, they have not been shown t
40           PGE(2) has been shown to exert pro-oncogenic effects in colorectal neoplasia through produc
41  genes dysregulated by loss of SIRT6 possess oncogenic effects in hepatocarcinogenesis.
42 al DNA viruses are highly suspicious to have oncogenic effects in humans.
43 nctions but also promotes inflammation, with oncogenic effects in males and teratogenic effects in fe
44  polycomb repressive complex 2 (PRC2) exerts oncogenic effects in many tumour types.
45 hanism through which B-Raf(V600E) exerts its oncogenic effects in melanoma.
46 ntrast, rap-2 was found to cause significant oncogenic effects in OVCA cells, while SKIIP promotes on
47 ely, our results argue that MYC mediates its oncogenic effects in part by altering mevalonate metabol
48 ggest that the AR and miR-21 axis exerts its oncogenic effects in prostate tumors by downregulating T
49          B-cell CLL/lymphoma 6 (BCL6) exerts oncogenic effects in several human hematopoietic maligna
50 (eIF-4E), a downstream effector of mTOR, has oncogenic effects in vivo and cooperates with c-Myc in B
51 t how activated Notch1 signaling exerts this oncogenic effect is not completely understood.
52 less, the downstream signaling mediating its oncogenic effects is not well defined.
53       The molecular network underlying EphB4 oncogenic effects is still unclear.
54 olves genetic and epigenetic alterations and oncogenic effects mediated by viral proteins in the acti
55 ivity to near normal levels and reversed the oncogenic effects (morphology changes and foci formation
56       Our studies reveal a novel cooperative oncogenic effect of AEG-1 and c-Myc that might explain t
57 proliferation, and that efforts to block the oncogenic effect of aerobic glycolysis must target react
58             We propose that a portion of the oncogenic effect of AIB1 could be through control of EGF
59 ciated with tumor progression, suggesting an oncogenic effect of amplified REL in B-lymphoid cells th
60         However, the underlying cause of the oncogenic effect of Arg882His in DNMT3A is not fully und
61                         Here, we address the oncogenic effect of Bcl3 in vivo and describe how this S
62 ced transformation, and MITF potentiates the oncogenic effect of BRAF(V600E) in these progenitor cell
63                                 To study the oncogenic effect of chronically elevated insulin on hepa
64                       This suggests that the oncogenic effect of distinct Ras mutants has a different
65                    Next, we investigated the oncogenic effect of EIF4A1 on cancer cell proliferation
66 CA mutation was found to have alleviated the oncogenic effect of either the TP53 mutation or MYC ampl
67                          We propose that the oncogenic effect of ETS fusion oncoproteins is in part m
68      These data demonstrate that part of the oncogenic effect of EWS/FLI1 is to transcriptionally der
69  Given IL-15 has been implicated in the anti-oncogenic effect of exercise and is being explored as an
70 which may be clinically relevant in the anti-oncogenic effect of exercise and repetitive exposure to
71  increased myokines associated with the anti-oncogenic effect of exercise and the magnitude of respon
72   These observations suggest a model for the oncogenic effect of high-risk HPV16 E7.
73 nd the loss of this immunity-rather than the oncogenic effect of HPVs-causes the markedly increased r
74 pressor activity of IRF-1 expression and the oncogenic effect of IRF-2 in human and murine tumor mode
75 hich suggests that pancreatitis enhances the oncogenic effect of KRAS through induction of IER3 expre
76    Absence of wild-type Kras potentiates the oncogenic effect of KRASG12D, while incidentally inducin
77  GTPase reaction rate that characterizes the oncogenic effect of many of the p21 mutants found in hum
78                                  The elusive oncogenic effect of mutating Gln61 is also explored.
79 ion in our simulations, we conclude that the oncogenic effect of mutation of Gln61 is indirect and is
80                             Furthermore, the oncogenic effect of Notch1 on primary melanoma cells was
81  the development of new drugs to silence the oncogenic effect of SET/TAF-Ibeta's histone chaperone ac
82               Last, we demonstrated that the oncogenic effect of SLPI may be due to protection of gro
83            We have previously shown that the oncogenic effect of SULF2 in HCC may be mediated in part
84                             This potentially oncogenic effect of tetraploidy is countered by a p53-de
85 fection is associated with CCA, owing to the oncogenic effect of the associated chronic biliary tract
86 is likely to significantly contribute to the oncogenic effect of the inactivation of BRCA1 or BRCA2.
87 on occurrences in cancer that potentiate the oncogenic effect of upstream lesions on the PI3K pathway
88 ch functions downstream of AKT, mediates the oncogenic effects of activated PI3K/AKT in ALK+ ALCL.
89 ses the oncogenic AKT, and mTOR mediates the oncogenic effects of AKT.
90                                          The oncogenic effects of Akt2 and Akt3 described here are in
91  function of the WWW element may explain the oncogenic effects of an alternative splicing variant of
92 and paracrine interactions that underlie the oncogenic effects of androgens and IGF1 and open up new
93                                        These oncogenic effects of APEX1 were mediated by the upregula
94 indings that Bcr(64-413) interferes with the oncogenic effects of Bcr-Abl and therefore has the poten
95 r's inhibitory effects but also enhanced the oncogenic effects of Bcr-Abl in a solid tumor model and
96 at BCR gene expression strongly inhibits the oncogenic effects of Bcr-Abl in NOD/scid mice, yielding
97 wn that c-Myc expression is required for the oncogenic effects of Bcr-Abl, and that over-expression o
98 d that expression of Bcr interferes with the oncogenic effects of Bcr-Abl.
99 estigated the role of the Bcr protein in the oncogenic effects of Bcr-Abl.
100 hosphoserine form of Bcr, which inhibits the oncogenic effects of BCR-ABL.
101  generated and may be targeted to reduce the oncogenic effects of beta-catenin in intestinal cells.
102 tegrin/FAK signaling profoundly inhibits the oncogenic effects of both NRP1 variants.
103 hus, CDK4 provides a direct link between the oncogenic effects of c-MYC and cell-cycle regulation.
104                        In cancer, these anti-oncogenic effects of CDK5 can provide selective pressure
105 that miRNA sequestration is critical for the oncogenic effects of CEP170, NUCKS1, and ZC3H11A mRNAs.
106 fferentiated cells are more sensitive to the oncogenic effects of certain signaling abnormalities tha
107  and tobacco smoke exposure and the relative oncogenic effects of chemically stable versus unstable D
108 vestigated the contribution of ceRNAs to the oncogenic effects of CNAs.
109               We investigated cell-cycle and oncogenic effects of cyclin E1 overexpression in tissues
110 ltaNp63 in Kras-driven LUAD and mediates the oncogenic effects of DeltaNp63 in both LUAD and LUSC.
111 ion-transplantation approach to evaluate the oncogenic effects of E2a-Hlf on murine B-cell progenitor
112  the tumor microenvironment in promoting the oncogenic effects of EBV.
113                                   These anti-oncogenic effects of exercise were associated with the e
114                                          The oncogenic effects of Gab2 in HepG2 cells were promoted b
115 a comprehensive understanding of the general oncogenic effects of HH/GLI signaling on the formation o
116 , these results strongly argue that the anti-oncogenic effects of LO on ras-mediated transformation a
117 fic factors determine the sensitivity to the oncogenic effects of loss or overexpression of Runx fact
118 rmal HAI-1 expression completely negated the oncogenic effects of matriptase.
119              Unexpectedly, these data reveal oncogenic effects of mitochondrial dysfunction that are
120 hat ETV5 is a key downstream mediator of the oncogenic effects of mutant FGFR3, as its knockdown in F
121 sting that TOR activity is essential for the oncogenic effects of mutant Rheb.
122 ream signaling node for the pleiotropic, pro-oncogenic effects of Nanog.
123                                    Given the oncogenic effects of Notch2, we analyzed its gene dosage
124                                          The oncogenic effects of nutrients were reversed by SIRT3, w
125                The results highlight the pro-oncogenic effects of pair-bond disruption, point to the
126 exogenous BMI1 overexpression mitigates anti-oncogenic effects of PLK1 inhibition and overcomes senes
127 direct pharmacological target that overcomes oncogenic effects of PML/RARalpha by triggering its degr
128 and functional genomics assays and find that oncogenic effects of PPM1D truncation converge on regula
129 nctions may be partially responsible for the oncogenic effects of PPM1D when it is amplified and over
130 lin-like growth factor 2 (IGF2) mediated the oncogenic effects of PRC2 heterogeneity in tumor growth.
131 ecific signaling circuitry sensitizes to the oncogenic effects of RB1 mutations.
132 hus, strategies to identify and suppress pro-oncogenic effects of RFA are urgently required to furthe
133 ependent signaling pathway that mediates the oncogenic effects of secondary BAs in gastrointestinal c
134 vidence for a novel mechanism underlying the oncogenic effects of secondary bile acids.
135 ts that will guide further research into the oncogenic effects of SF3B1 mutation.
136    Thus, STAT3 is a critical mediator of the oncogenic effects of somatic EGFR mutations and targetin
137    We found that ARPs were required for many oncogenic effects of Src including Mmp1 expression and i
138 hylating agent, led to reversal of the above oncogenic effects of succinate in ccRCC cells.
139  v-Jun from JNK signaling that underlies the oncogenic effects of the delta-domain deletion; however,
140            Overall, these results define the oncogenic effects of the GARP-TGFbeta axis in the tumor
141 nd as a result this SH2 binding inhibits the oncogenic effects of the oncoprotein.
142                  The primary mediator of the oncogenic effects of the Wnt signaling pathway is beta-c
143 ure a critical level of gene product for the oncogenic effects of these fusions.
144         Studying the molecular, cellular and oncogenic effects of these mutations can reveal novel me
145 ls in the cell and likely contributes to the oncogenic effects of these pathways in human cancer.
146          To understand the developmental and oncogenic effects of two closely positioned point mutati
147 ever, we found that GRIM-19 inhibits the pro-oncogenic effects of v-Src independently of STAT3.
148                                        These oncogenic effects of YAP were associated with activation
149   The tumor suppressor p53 mediates its anti-oncogenic effect on cells by functioning as a sequence-s
150 ing functional p53, RUNX1 has apparently pro-oncogenic effects on cell growth that include cytoskelet
151 ular mechanisms by which menin decreases the oncogenic effects on cell morphology and other phenotype
152       Coexpression of CD40 and CD40L confers oncogenic effects on immortalized human epithelial cells
153 regulates MDM2 protein levels and exerts its oncogenic effects on p53 in tumor cells.
154 on of the miR-3622 locus at 8p21 reduced the oncogenic effects on tumor progression and metastasis.
155             Here, we report that GARP exerts oncogenic effects, promoting immune tolerance by enrichi
156              Mechanistically, PHF19-mediated oncogenic effect relies on its PRC2-interacting and chro
157 pendent prognostic biomarker that exerts its oncogenic effects through miRNA-mediated post-transcript
158 ults suggest that COX-2/PGE(2) may exert pro-oncogenic effects through synergistic induction of recep
159 able targets in glioblastoma, relating their oncogenic effects to activation of the Wnt/beta-catenin
160 ese results demonstrate that WAP-Int3 has no oncogenic effect upon PI-MECs and that the expansion of
161 anscriptional activity of RNF6 underlies its oncogenic effect, we performed integrated chromatin immu
162 y component in this pathway may have similar oncogenic effects, we studied the relationship between a
163                                    Moreover, oncogenic effects were further potentiated by inflammato
164 r mutations that are then selected for their oncogenic effects, whereby AID promotes aggressiveness i
165  to a better understanding of underlying pro-oncogenic effects, which is needed to develop CDK8/CDK19
166 anscription factor STAT3 and ablates its pro-oncogenic effects while v-Src activates STAT3 to promote
167 pression of cytokine expression and the anti-oncogenic effect with inhibition of angiogenesis.
168 , colorectal and thyroid cancers, suggesting oncogenic effects with the activation of signaling pathw

 
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