ライフサイエンスコーパス: oncotic pressure

1 effects of (in)Abeta depended on changes in oncotic pressure.

2 RPF), blood pressure, hematocrit, and plasma oncotic pressure.

3 der basal conditions to result in changes in oncotic pressure.

4 icantly faster in groups 2 to 4, and colloid oncotic pressure after resuscitation was greater in grou

5 This suggests that plasma oncotic pressure alone may be a more important determina

6 We tested the hypothesis that plasma oncotic pressure alone, not the plasma-to-lymph oncotic

7 tic pressure (piGC) from knowledge of plasma oncotic pressure and the filtration fraction revealed th

8 arious levels throughout the body, regulates oncotic pressure and transports ligands.

9 ratio, plasma protein concentration, plasma oncotic pressure, and myocardial contractility.

10 Total solids, colloid oncotic pressure, arterial oxygen content, Hb, lactate,

11 e to hemodilution caused by its high colloid oncotic pressure, but may facilitate diffusive oxygen tr

12 vestigate this further, Lp and the effective oncotic pressure difference (f3DeltaPi) acting across th

13 Thus, the effective oncotic pressure difference (sigma black triangle down p

14 hese results support our hypothesis that the oncotic pressure difference across the tumor microvascul

15 otic pressure alone, not the plasma-to-lymph oncotic pressure difference, modulates pulmonary transva

16 ar fluid filtration than the plasma-to-lymph oncotic pressure difference.

17 c pressures were estimated as the sum of the oncotic pressure due to HA alone plus the oncotic pressu

18 ficients to HA and A, there was an effective oncotic pressure (E pi) of between 3.46 and 6.0 cm H2O o

19 Oncotic pressure, effective at inducing VDAC closure, al

20 e circulation and the resulting reduction in oncotic pressure exerted by the plasma, although the fat

21 ncentration and the decreased transcapillary oncotic pressure gradient.

22 rol animals, even though the plasma-to-lymph oncotic pressure gradients were equal.

23 Starling's equation indicates that reduced oncotic pressure gradients will favor edema formation, a

24 4.5 and 17.7 +/- 2.2 mm Hg; plasma-to-lymph oncotic pressure gradients, respectively, were 4.4 +/- 0

25 Whereas oncotic pressure in blood plasma of various species is k

26 um total protein is the chief determinant of oncotic pressure in humans.

27 ecies is known, no data are available on the oncotic pressure in the interstitial space of tumors.

28 ecause of the leaky nature of tumor vessels, oncotic pressure in tumor interstitium should be close t

29 The high oncotic pressure in tumors is consistent with the elevat

30 ability to expand plasma volume and improve oncotic pressure in various clinical settings, such as i

31 aised left atrial pressure elevation, plasma oncotic pressures in dextran and control sheep, respecti

32 We found interstitial oncotic pressures in four human tumor xenografts to be h

33 ic wick method for the direct measurement of oncotic pressures in the interstitial fluid of tumors gr

34 Neither the plasma oncotic pressure nor the mean arterial pressure differed

35 he oncotic pressure due to HA alone plus the oncotic pressure of albumin (A) in an HA matrix.

36 ross the endothelial glycocalyx and that the oncotic pressure of interstitial fluid does not directly

37 cromolecules was assessed from the effective oncotic pressure (omega delta pi) exerted by the perfusa

38 as determined, blood was sampled, and plasma oncotic pressure (pi(A)) was measured.

39 Computation of glomerular intracapillary oncotic pressure (piGC) from knowledge of plasma oncotic

40 min (HSA) serves not only as a physiological oncotic pressure regulator and a ligand carrier but also

41 ntial contribution of medullary interstitial oncotic pressure to the net balance of forces influencin

42 ents in serum albumin, immunoglobulin G, and oncotic pressure values were 31, 32, and 26%, respective

43 glomerular filtration during HPP, perfusate oncotic pressure was reduced by lowering the concentrati

44 Total interstitial oncotic pressures were estimated as the sum of the oncot

45 ack triangle down pi was near 70% of luminal oncotic pressure when the tissue concentration equalled