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1 cialized intercellular structures (fungi and oomycetes).
2  losses to the water mold Phytophthora spp. (Oomycetes).
3 in the protection against the fungus and the oomycete.
4 , informative microsatellite markers in this oomycete.
5 rolegnia spp. which are basal members of the oomycetes.
6 or future P450 annotations in newly explored oomycetes.
7  and 31 P450 subfamilies were newly found in oomycetes.
8 ee hundred and fifty-six P450s were found in oomycetes.
9 homology data revealed P450 family blooms in oomycetes.
10 ution of phytopathogenic traits in fungi and oomycetes.
11 oactivity against plant pathogenic fungi and oomycetes.
12  independently in plant pathogenic fungi and oomycetes.
13 , protects plants against diseases caused by oomycetes.
14 infection structures of pathogenic fungi and oomycetes.
15 production and bioactivity against fungi and oomycetes.
16 ted for RxLR-effectors from plant pathogenic oomycetes.
17 ttern of cross-kingdom HGT between fungi and oomycetes.
18 ribution; they occur in bacteria, fungi, and oomycetes.
19 to pathogen Phytophthora infestans and other oomycetes.
20 s is not the case for several subfamilies in oomycetes.
21 ved in the perception of bacteria, fungi and oomycetes.
22 e encoded by the genomes of plant pathogenic oomycetes.
23 e population structure within these obligate oomycetes.
24 or delivery are uncharacterized in fungi and oomycetes.
25 ies and catalogues the effector secretome of oomycetes.
26 o new methods to suppress diseases caused by oomycetes.
27 ent in a few eukaryotic lineages such as the Oomycetes.
28 sively extracellular and unique to fungi and oomycetes.
29 gen-associated molecular patterns (PAMPs) in oomycetes.
30 ced by plant pathogenic bacteria, fungi, and oomycetes.
31 t the fungal kingdom, and in the fungus-like oomycetes.
32 e are shared with plant-associated fungi and oomycetes.
33  from the same or closely related species of oomycetes.
34 ially destructive plant-associated fungi and oomycetes.
35 , SWEETs had not been identified in fungi or oomycetes.
36 his economically devastating fish-pathogenic oomycete A. invadans.
37 hogen of potato and a model organism for the oomycetes, a distinct lineage of fungus-like eukaryotes
38 diseases of plants and animals are caused by oomycetes, a group of eukaryotic pathogens important to
39                                              Oomycetes accomplish parasitic colonization of plants by
40 biotrophic, hemibiotrophic, and necrotrophic oomycetes affect disease progression.
41                   Physiological races of the oomycete Albugo candida are biotrophic pathogens of dive
42 ve secondary metabolites, including the anti-oomycete and antifungal haterumalide, oocydin A and the
43 for the biosynthesis of the antifungal, anti-oomycete and antitumor haterumalide, oocydin A (ooc).
44 ay enhanced resistance to infection with the oomycete and bacterial pathogens.
45                                              Oomycete and fungal effectors with RXLR and RXLR-like mo
46 rs specifically influencing the emergence of oomycete and fungal EPPs, including new introductions th
47                Effector proteins secreted by oomycete and fungal pathogens have been inferred to ente
48 is to unrelated (hemi)biotrophic filamentous oomycete and fungal pathogens.
49                                         Many oomycete and fungal plant pathogens are obligate biotrop
50                                   Bacterial, oomycete and fungal plant pathogens establish disease by
51 nal variants of the RXLR motif, and that the oomycete and fungal RXLR motifs enable binding to the ph
52                                              Oomycete and fungal symbionts have significant impacts o
53 l wall architecture important for pathogenic oomycete and symbiotic bacterial interactions in legumes
54 o mutualism) with their hosts include fungi, oomycetes and actinomycete bacteria.
55 earch on eukaryotes such as animals, plants, oomycetes and fungi has shown that P450s profiles in the
56 her than the so-called Crinkler effectors of oomycetes and fungi, these effectors are encoded by othe
57 abidopsidis, which represents the kingdom of oomycetes and is phylogenetically distant from fungi, em
58 onal secreted proteins from plant pathogenic oomycetes and its similarity to a host-targeting signal
59  last 150 yr between plant pathogens (fungi, oomycetes and plasmodiophorids) and vascular plants.
60 d nematodes), common host-targeting signals (oomycetes and protozoans) and specialized intercellular
61 d economical impact of the animal pathogenic oomycetes and review the recent advances in this emergin
62 he understanding of the relationship between oomycetes and their host plants.
63  The resource is focused on fungi, protists (oomycetes) and bacterial plant pathogens that have genom
64 stance by challenging EP plants with fungal, oomycete, and bacterial pathogens and an insect pest.
65 ease resistance of plants against bacterial, oomycete, and fungal pathogens and has a unique mode of
66 ntribute to plant defense against bacterial, oomycete, and fungal pathogens.
67 d by other plant resistance genes and virus, oomycete, and nematode effectors and for host susceptibi
68 etween 184 effectors from bacterial, fungal, oomycete, and nematode pathogens with 25 Arabidopsis aut
69 olite not previously shown to be produced by oomycetes, and two proteins with homology to vertebrate
70                         For bacteria, fungi, oomycetes, and viruses, we illustrate that host range is
71 tion of the kelp Macrocystis pyrifera by the oomycete Anisolpidium ectocarpii using TEM, in vivo auto
72 bium meliloti as well as with the pathogenic oomycete Aphanomyces euteiches.
73                                              Oomycetes are a phylogenetically distinct group of organ
74 sequences similar to those seen in fungi and oomycetes are also found in the animal kingdom, but rath
75 ytes are well preserved in Rhynie rocks, and oomycetes are also present.
76                                              Oomycetes are fungal-like eukaryotic microbes in the kin
77 in bacteria, equivalent systems in fungi and oomycetes are poorly understood.
78                                              Oomycetes are responsible for multi-billion dollar damag
79  able to identify known sequences present in oomycetes as well as identify novel sequences.
80 e, we report the identification of SWEETs in oomycetes as well as SWEETs and a potential SemiSWEET in
81 toplasm, consistent with the hypothesis that oomycetes, as is the case with bacteria and fungi, activ
82 keleton confers resistance against fungi and oomycetes, AtADF4 is not involved in resistance against
83 ily of 700 proteins with similarity to known oomycete avirulence genes.
84                             Similar to other oomycete avirulence proteins, AVR3aKI carries a signal p
85              Recent characterization of four oomycete Avr genes revealed that they encode effector pr
86 defense responses that are effective against oomycete, bacterial and viral pathogens, pointing to a c
87 are consistent with the hypothesis that some oomycetes became successful plant parasites by multiple
88                       Research revealed that oomycetes belonging to different orders contain distinct
89 nes resistance to downy mildew caused by the oomycete Bremia lactucae carrying the cognate avirulence
90 and Saccharomycotina, and in phytopathogenic Oomycetes, but neither other eukaryotes nor prokaryotes.
91 icals to control plant and animal pathogenic oomycetes cannot be used anymore; due to resistance in t
92          The three examined plant pathogenic oomycetes carry complex and diverse sets of RXLR effecto
93                                              Oomycetes cause devastating plant diseases of global imp
94                              Many species of oomycetes cause economic and environmental damage owing
95                              Phytopathogenic oomycetes cause some of the most devastating diseases af
96                                              Oomycete cell wall carbohydrates were recognized by fish
97    We uncover a robust molecular response to oomycete colonization in Marchantia that consists of con
98 T has played a role in shaping how fungi and oomycetes colonize plant hosts.
99 ranches of life (ascomycete, eubacteria, and oomycete) converge onto the Arabidopsis TCP14 transcript
100 gests that prostaglandins may be involved in oomycete development.
101 s, we outline some of the reasons fungal and oomycete diseases cause such significant losses to tropi
102 ing effective long-term control measures for oomycete diseases.
103                                 We study the oomycete downy mildew pathogen of Arabidopsis, Hyalopero
104 amily includes all experimentally identified oomycete effector and avirulence genes, and its rapid pa
105                               Recently, four oomycete effector genes have been isolated, and several
106 ted programmed cell death upon bacterial and oomycete effector recognition as well as decreased resis
107                  Here the latest findings on oomycete effector secretion, delivery and function are d
108                                  For several oomycete effectors (i.e., the RxLR-effectors) it has bee
109                                              Oomycete effectors identified to date contain a targetin
110 lusion that RXLR and dEER serve to transduce oomycete effectors into host cells indicates that the >3
111 ctions of the very large number of predicted oomycete effectors that contain them.
112 at protein belonging to an ancient family of oomycete effectors that rapidly evolves to escape host d
113 tionally interchangeable with RxLR motifs of oomycete effectors.
114 displays similarity to the WY domain core in oomycete effectors.
115 otifs, RXLR and EER, present in translocated oomycete effectors.
116 idly moving toward genome-wide catalogues of oomycete effectors.
117 ponse induced by non-host flagellins and the oomycete elicitor INF1.
118 kthroughs in live-cell imaging of fungal and oomycete encounter sites, including live-cell imaging of
119 ng in several distinct lineages of fungal or oomycete-feeding nematodes.
120 challenge by pathogenic bacteria, fungi, and oomycetes, for whom they provide a resource of living sp
121                                          The oomycetes form a phylogenetically distinct group of euka
122 ubset of these genes is conserved in related oomycetes from the Phytophthora genus.
123 mato, potato and barley to viral, bacterial, oomycete, fungal and nematode infections.
124 yotic proteins, which include effectors from oomycetes, fungi and other parasites.
125 ve major pathogen groups (viruses, bacteria, oomycetes, fungi, and nematodes), has contributed to our
126 din biosynthesis have been identified in the oomycete genome.
127                                              Oomycete genomes display a strongly bipartite organizati
128      The high level of synteny between these oomycete genomes extends to the ABC superfamily, where 1
129 fector genes have been isolated, and several oomycete genomes have been sequenced.
130                        Our analysis of other oomycete genomes revealed that both uridine phosphorylas
131 enigmatic, but the expansion of NLP genes in oomycete genomes suggests they are important.
132 tative effectors have now been identified in oomycete genomes, the sequences of which show evidence o
133        Although research on plant pathogenic oomycetes has flourished in recent years, the animal pat
134 ished in recent years, the animal pathogenic oomycetes have received less attention.
135 uring the compatible interaction between the oomycete Hyaloperonospora arabidopsidis (Hpa) and its ho
136     Here, we demonstrate that the biotrophic oomycete Hyaloperonospora arabidopsidis (Hpa) exhibits a
137   Here, we report the genome sequence of the oomycete Hyaloperonospora arabidopsidis (Hpa), an obliga
138 expression of BABA-IR against the biotrophic oomycete Hyaloperonospora arabidopsidis is associated wi
139  the tandem WY-domain effector ATR1 from the oomycete Hyaloperonospora arabidopsidis through direct a
140 Golovinomyces orontii and Erysiphe pisi, the oomycete Hyaloperonospora arabidopsidis, and the bacteri
141 eudomonas syringae pv tomato, the biotrophic oomycete Hyaloperonospora arabidopsidis, and the necrotr
142  basal defense against RKN as well as to the oomycete Hyaloperonospora arabidopsidis.
143 domonas syringae pv. tomato DC3000 or to the oomycete Hyaloperonospora arabidopsidis.
144  in response to infections by the pathogenic oomycete Hyaloperonospora parasitica.
145 conditions and treatment with the pathogenic oomycete, Hyaloperonospora parasitica, wild type had a h
146  in pollen tubes, root hairs, and fungal and oomycete hyphae and is the most widely distributed unidi
147 he presence or absence of the mycelia of the oomycetes in both shaking and static conditions.
148  only form of extracellular SOD in fungi and oomycetes, in stark contrast to the extracellular Cu/Zn-
149               The eukaryotic microbes called oomycetes include many important saprophytes and pathoge
150       The genus Aphanomyces (Saprolegniales, Oomycetes) includes species with a variety of ecologies
151                        When plant-pathogenic oomycetes infect their hosts, they employ a large arsena
152  demonstrate that the Marchantia response to oomycete infection displays evolutionarily conserved fea
153 tants were more susceptible and resistant to oomycete infection, respectively, showing that the inten
154 nylpropanoid (flavonoid) biosynthesis during oomycete infection.
155 S10 was specifically expressed in roots upon oomycete infection.
156 mportantly, for restriction of bacterial and oomycete infections.
157     Current research is helping us learn how oomycetes interact with host and environment, understand
158                     We suggest that bacteria/oomycete interactions should be considered not only in t
159 tion of RxLR effectors from plant pathogenic oomycetes into the cytoplasm of their host is currently
160                                         This oomycete is highly variable and rapidly overcomes resist
161 nt that the effector secretome of pathogenic oomycetes is more complex than expected, with perhaps se
162  largest group of translocated proteins from oomycetes is the RxLR effectors, defined by their conser
163 te having limited secondary metabolism, many oomycetes make chemicals for communicating within their
164  unparalleled opportunities to determine how oomycetes manipulate hosts to establish infection.
165                                Understanding oomycete metabolism is fundamental to understanding thes
166 ss effective, there is a need for studies on oomycete metabolism to help identify promising and more
167 biotic gene transfer events have diversified oomycete metabolism, resulting in biochemical pathways t
168  act as ecological amplifiers for fungal and oomycete mycelial networks in soils, extending their pot
169 ngdom as well as in phylogenetically distant oomycetes or "pseudofungi" species.
170                               The eukaryotic oomycetes, or water molds, contain several species that
171                                              Oomycete P450 patterns suggested host influence in shapi
172  that is distinct from that triggered by the oomycete PAMP INF1.
173 otein RAC1 that determines resistance to the oomycete pathogen Albugo candida.
174 se can inhibit germination of spores of this oomycete pathogen and inhibit tobacco leaf infection by
175          We used Medicago truncatula and its oomycete pathogen Aphanomyces euteiches to elucidate ear
176 he AM fungus Rhizophagus irregularis and the oomycete pathogen Aphanomyces euteiches.
177 ry of currently known bacterial, fungal, and oomycete pathogen effectors that induce biotic and abiot
178  that the effector protein HaRxL106 from the oomycete pathogen Hyaloperonospora arabidopsidis co-opts
179 nced disease resistance against the virulent oomycete pathogen Hyaloperonospora arabidopsidis Noco2,
180 e toward a virulent strain of the biotrophic oomycete pathogen Hyaloperonospora arabidopsidis Noco2.
181 terial pathogen Pseudomonas syringae and the oomycete pathogen Hyaloperonospora arabidopsidis.
182 d plants was also more resistant against the oomycete pathogen Hyaloperonospora arabidopsidis.
183 terial pathogen Pseudomonas syringae and the oomycete pathogen Hyaloperonospora arabidopsidis.
184      Hyaloperonospora parasitica is a native oomycete pathogen of Arabidopsis and is related to other
185 R1(NdWsB) in Hyaloperonospora parasitica, an oomycete pathogen of Arabidopsis.
186 ctor protein Avr1b of Phytophthora sojae, an oomycete pathogen of soybean (Glycine max), we show that
187 mination and blue mold disease caused by the oomycete pathogen Peronospora tabacina.
188  spore germination and leaf infection by the oomycete pathogen Peronospora tabacina.
189 ine phosphorylases, PcUP1 and PcUP2 from the oomycete pathogen Phytophthora capsici.
190 um) nuclear proteome during infection by the oomycete pathogen Phytophthora capsici.
191                                          The oomycete pathogen Phytophthora infestans, the agent of t
192  causes increased susceptibility only to the oomycete pathogen Phytophthora infestans.
193  Marchantia polymorpha to infection with the oomycete pathogen Phytophthora palmivora.
194 esponse to inoculation with the downy mildew oomycete pathogen Sclerospora graminicola.
195 ellular proteins in Phytophthora and Pythium oomycete pathogen species.
196  mildew of spinach is caused by the obligate oomycete pathogen, Peronospora effusa.
197                                          The oomycete pathogen, therefore, is under selection pressur
198  but also show defects in colonization by an oomycete pathogen, with the absence of appressoria forma
199 E2 contributes to innate immunity against an oomycete pathogen.
200 e microdomains in plant susceptibility to an oomycete pathogen.
201 nd presentation of information on fungal and Oomycete pathogenicity genes and their host interactions
202 g molecular genetic and genomic knowledge of oomycete pathogenicity is essential to gain the full con
203 icipates in nonhost resistance to fungal and oomycete pathogens and is required for full penetration
204                                              Oomycete pathogens cause serious damage to a wide spectr
205                                   Fungal and oomycete pathogens cause some of the most devastating di
206                                              Oomycete pathogens contain large complements of predicte
207                              Many fungal and oomycete pathogens differentiate a feeding structure nam
208                This is a novel strategy that oomycete pathogens exploit to modulate host defense.
209 cally and agronomically important fungal and Oomycete pathogens for intervention with synthetic chemi
210 factors that mediate plant susceptibility to oomycete pathogens is relatively unexplored.
211                                          The oomycete pathogens Phytophthora infestans and P. capsici
212                           Known effectors in oomycete pathogens possess an RXLR-EER motif in their am
213                                              Oomycete pathogens secrete numerous effectors to manipul
214 ine phosphorylases are not found in obligate oomycete pathogens such as Hyaloperonospora arabidopsidi
215 different queries with a focus on fungal and oomycete pathogens were performed, leading to 510 up-reg
216 s are released into plants upon infection by oomycete pathogens, suggesting they may elicit plant def
217                                       In the oomycete pathogens, two conserved N-terminal motifs, RXL
218 nd effector genes from bacterial, fungal and Oomycete pathogens, which infect human, animal, plant, i
219 ative analysis of P450s in 13 newly explored oomycete pathogens.
220 enhanced resistance to virulent bacterial or oomycete pathogens.
221 disease resistance to virulent bacterial and oomycete pathogens.
222 nificantly reduced infection capacity of the oomycete pathogens.
223 istance of Arabidopsis against bacterial and oomycete pathogens.
224 hogen of Arabidopsis and is related to other oomycete phytopathogens that include several species of
225 ecretion, because effector proteins from the oomycete Phytophthora infestans and virulence determinan
226            Filamentous pathogens such as the oomycete Phytophthora infestans infect plants by develop
227 on using a different pathogen of tomato, the oomycete Phytophthora infestans that is distantly relate
228 g a secreted protein from the hemibiotrophic oomycete Phytophthora infestans that is specifically exp
229                                          The oomycete Phytophthora infestans, causal agent of the tom
230 e to virulent strains of P. syringae and the oomycete Phytophthora sojae.
231 ased leaf susceptibility to infection by the oomycetes Phytophthora infestans and Phytophthora palmiv
232                                   Fungal and oomycete plant parasites are among the most devastating
233 and Nuk12 candidate effector proteins of the oomycete plant pathogen Phytophthora infestans.
234 aliana) ecotype Columbia-0 is nonhost to the oomycete plant pathogen Phytophthora sojae and the funga
235    Here, we show that two effectors from the oomycete plant pathogen Phytophthora sojae suppress RNA
236                   We recently found that the oomycete plant pathogen Phytophthora sojae uses nuclear
237      Phytophthora palmivora is a destructive oomycete plant pathogen with a wide host range.
238      Phytophthora palmivora is a devastating oomycete plant pathogen.
239 cesses that lead to speciation of fungal and oomycete plant pathogens and provide an outline of how s
240                     The sequenced genomes of oomycete plant pathogens contain large superfamilies of
241                                              Oomycete plant pathogens deliver effector proteins insid
242  effector proteins produced by bacterial and oomycete plant pathogens have been elucidated in recent
243            They are closely related to other oomycete plant pathogens such as Phytophthora species an
244                                   Fungal and oomycete plant pathogens translocate effector proteins i
245  hyper-susceptible to fungal, bacterial, and oomycete plant pathogens, demonstrating that Pip1 is an
246  We used the draft genome sequences of three oomycete plant pathogens, Phytophthora sojae, Phytophtho
247 cations of population genomics to fungal and oomycete plant pathogens.
248                            Since the natural oomycete population is too low to reach a quorum necessa
249                 One type of plant pathogens, oomycetes, produces effector proteins with N-terminal RX
250 cation in the DNA recognition helices of the oomycete proteins.
251 rasites, including viruses, bacteria, fungi, oomycetes, protozoa, insects and nematodes.
252 ated zoospores that are produced by the soil oomycete Pythium aphanidermatum as a biological vector,
253 fection of rice (Oryza sativa) with the root oomycete Pythium graminicola.
254 We show that the hyphae of the mycelial soil oomycete Pythium ultimum function as active translocatio
255 rt of the PAH fluorene (FLU) by the mycelial oomycete Pythium ultimum that was grown along the air-wa
256  to extreme susceptibility to a root-rotting oomycete (Pythium spp), demonstrating that these genes a
257                                          Two oomycetes, Pythium oligandrum and Pythium aphanidermatum
258                                        While oomycete R2R3 proteins contain c-Myb-like helices, R1R2R
259 ealed that PITG_04584, which lacks close non-oomycete relatives, was involved in zoosporogenesis, cys
260 rsensitive cell death response (HR) and full oomycete resistance, but not for salicylic acid inductio
261      P. ultimum sequences with similarity to oomycete RXLR and Crinkler effectors, Kazal-like and cys
262  determine the myriad virulence functions of oomycete RXLR effector proteins.
263 hts into structure/function relationships of oomycete RXLR effectors and how these proteins engage wi
264  structures of the effector domains from two oomycete RXLR proteins, Phytophthora capsici AVR3a11 and
265 e present in at least half of the identified oomycete RXLR-dEER effector candidates, and we show that
266                         A related pathogenic oomycete's HT signal export is dependent on PI(3)P bindi
267 uckeri, Flavobacterium columnare, and/or the oomycete Saprolegnia ferax.
268                                              Oomycetes secrete both extracellular and intracellular e
269                                    Fungi and oomycetes secrete many specialized effector proteins for
270 bition of Pythium ultimum, a phytopathogenic oomycete sensitive to pyoluteorin.
271  sweet2 mutants were more susceptible to the oomycete, showing impaired growth after infection.
272 rium catenoides, a free-living sister of the oomycetes, shows that these transfers largely converge w
273 tionarily conserved effectors from different oomycete species can suppress immunity in plant species
274                                              Oomycetes such as these Phytophthora species share the k
275                             Plant pathogenic oomycetes, such as the Irish potato famine pathogen Phyt
276                             Plant pathogenic oomycetes, such as the potato (Solanum tuberosum) and to
277  to an enhanced susceptibility to a virulent oomycete, suggesting a role for BAP1 in basal defense re
278 a-aminobutyric acid (BABA) revealed IMPAIRED OOMYCETE SUSCEPTIBILITY1 (IOS1) as a critical PTI player
279 a-aminobutyric acid (BABA) revealed IMPAIRED OOMYCETE SUSCEPTIBILITY1 (IOS1) as a critical PTI player
280 abidopsis thaliana) receptor kinase IMPAIRED OOMYCETE SUSCEPTIBILITY1 (IOS1) as required for successf
281 This is supported by the observation that an oomycete that does not form zoospores, Hyaloperonospora
282       Saprolegnia parasitica is a freshwater oomycete that is capable of infecting several species of
283 guish isolates within several species of the oomycetes that cause downy mildew diseases.
284                                    Fungi and oomycetes that colonize living plant tissue form extensi
285 ers largely converge within the radiation of oomycetes that colonize plant tissues.
286 ng function has been recruited by pathogenic oomycetes to facilitate their own invasion.
287 icroorganisms ranging from fungi, algae, and oomycetes to testate amoebozoans, and even cyanobacteria
288 ed by eukaryotic microbes, such as fungi and oomycetes, to host plants and contribute to the establis
289                            During infection, oomycetes translocate effector proteins into host cells,
290 ically diverse: viruses, bacteria, protozoa, oomycetes, true fungi, parasitic plants, and many types
291      Understanding the mechanisms underlying oomycete virulence and the genomic processes by which th
292         Several common mechanisms underlying oomycete virulence, including protein toxins and cell-en
293 lcNAc) epitopes were not identified when the oomycete was grown in vitro or while infecting the roots
294 lar and molecular events in response to this oomycete, which has a broad host range.
295 ning and biochemical studies have shown that oomycetes, which belong to the kingdom Stramenopila, sec
296                                       Unlike oomycetes, which employ RXLR effectors to suppress host
297 quences and other sequenced plant pathogenic oomycetes with 91% of the hybrid assembly derived sequen
298 ering effectors, have emerged from comparing oomycetes with different genome characteristics, parasit
299  plant defenses against pathogenic fungi and oomycetes with limited, indirect evidence.
300 lacking chromalveolates such as ciliates and oomycetes would be explained by plastid loss in these li

 
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