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1 ired pOKR were found to exhibit long-lasting optokinetic after nystagmus (OKAN) as opposed to those t
2 estingly, a long-lasting and robust negative optokinetic afternystagmus (OKAN) was evoked upon light
6 vement recordings showed decreased gains for optokinetic and vestibulo-ocular reflexes, confirming an
7 gth of directed eye movements (i.e. combined optokinetic and voluntary tracking) for stimuli moving a
8 ibution suggests that monocular nasotemporal optokinetic asymmetry is partly attributable to subcorti
9 le in maintaining the monocular nasotemporal optokinetic asymmetry seen in patients with infantile es
10 y mutants required nearly an hour to recover optokinetic behavior after return to bright light, where
11 ed calcium imaging in awake zebrafish during optokinetic behavior to record transgenically identified
12 is, reduced neuronal connection, and reduced optokinetic behavioral response in zebrafish larvae.
13 Its applicaton for explaining a variety of optokinetic behaviors in other insects assumes that neur
16 ent of motion responsiveness for pursuit and optokinetic eye movements (optokinetic nystagmus [OKN]).
18 e of virtual reality, vibrotactile feedback, optokinetic flow, YouTube videos, and innovative methods
19 ters (saccadic latency, smooth pursuit gain, optokinetic gain), motor proficiency (BOT-2 subtests), a
22 Moreover, these cells are capable of driving optokinetic head tracking and visually guided behaviour
30 ical deviation (DVD), monocular asymmetry of optokinetic nystagmus (MOKN), monocular asymmetry of smo
31 otions; i.e., they give better responses for optokinetic nystagmus (OKN) and visually evoked potentia
35 e of this study was to characterize vertical optokinetic nystagmus (OKN) in normal human subjects, co
38 We asked if an involuntary eye movement, optokinetic nystagmus (OKN), could serve as an objective
39 designed to elicit smooth pursuit, saccades, optokinetic nystagmus (OKN), vestibulo-ocular reflex (VO
43 13%, 28%, and 30%), and nasotemporal pursuit/optokinetic nystagmus asymmetry (23%, 38%, and 54%).
48 sed on sub-conscious, reflex responses (e.g. optokinetic nystagmus) that don't require involvement of
52 ctive saccades, vergence smooth pursuit, and optokinetic nystagmus, was measured annually with a head
53 lexibly responded to rotational stimuli with optokinetic nystagmus-like head movements, independent o
59 Visual function was assessed with a rotating optokinetic (OKN) drum at ages 13 and 18 months and neur
60 ent of dye results in significantly improved optokinetic (OKR) ( 43 fold, p < 0.001) and visualmotor
62 ore the feasibility of using Saccadic Vector Optokinetic Perimetry (SVOP) to differentiate glaucomato
65 ity of the vestibulo-ocular reflex (VOR) and optokinetic reflex (OKR) allows for optimal combined gaz
66 cued water maze (WM) behavioral test and the optokinetic reflex (OKR) measurement at different times
69 A prime example of such behaviours is the optokinetic reflex (OKR), an innate eye movement mediate
70 haracterized as blind, these mutants lack an optokinetic reflex (OKR), but in another behavioral assa
71 on gaze stabilization behaviors, such as the optokinetic reflex (OKR), to perceive and correct for gl
72 (HOKS) decreases the gain of the horizontal optokinetic reflex and evokes the second phase of optoki
73 ntaneously firing neurons, is engaged during optokinetic reflex compensation for inner ear dysfunctio
75 encoding in the superior colliculus and the optokinetic reflex follow a novel motion integration rul
77 Similar to what was observed neuronally, the optokinetic reflex occurred more reliably at low contras
79 e and after disease onset by quantifying the optokinetic reflex responses and to compare them to the
83 Similarly within the afferent arm of the optokinetic reflex we showed expression in the developin
84 on wiring of the ocular motor system and the optokinetic reflex, impairing horizontal eye movements.
85 dorsal oculomotor neurons impairs the larval optokinetic reflex, suggesting that neuronal clustering
86 eye-head angle to a default position via the optokinetic reflex, we developed an efficient and unbias
90 d those on retinal function were analyzed by optokinetic response (OKR) and electroretinography (ERG)
92 brates large, moving visual scenes induce an optokinetic response (OKR) control of eye movements to s
94 ly evoked smooth eye movements, known as the optokinetic response (OKR), have been studied in various
101 mprovement of visual function as assessed by optokinetic response and looming-induced escape behavior
102 was assessed using the electroretinogram and optokinetic response and retinal morphology investigated
105 physiology of the zebrafish visual mutant no optokinetic response c (nrc) and to identify the genetic
106 o optokinetic reflex could be elicited in no optokinetic response c (nrc) mutant animals under any te
107 Here we describe a zebrafish mutant, no optokinetic response f(w21) (nof), with a nonsense mutat
110 re reduced visual acuity, measured using the optokinetic response, and vascular leakage continued to
111 Recovery of Ribeye a levels rescues the optokinetic response, increases the number of PKCalpha-p
112 ed visual function, as evidenced by improved optokinetic response, restored b-wave amplitude in elect
113 il and assessed for visual function using an optokinetic response, with subsequent samples taken for
117 d movements in their surroundings, displayed optokinetic responses (OKR) or optomotor responses (OMR)
118 of RPE-expressed rlbp1b selectively impaired optokinetic responses (OKR) with a ~50% reduction in sac
124 etinal motion input in generating horizontal optokinetic responses in patients with infantile strabis
126 viated eye can supplement temporal monocular optokinetic responses in the fixating eye under binocula
127 All patients showed poor temporally directed optokinetic responses that instantaneously improved when
129 y was measured behaviorally, using optomotor/optokinetic responses to rotating square-wave stimuli.
133 g: decreased survival; hypokinesia; impaired optokinetic responses; neurodegeneration; neuroinflammat
135 t, gaze holding, convergence, vestibular and optokinetic slow phases, and cancellation of vestibular
139 These VEPRs are not simple responses to optokinetic stimulation, but are modulated by the config
140 sensory adaptation process during continued optokinetic stimulation, which, when the stimulus is rem
144 = 9) while pairing yaw rotation with a pitch optokinetic stimulus, resulting in cross-axis adaptation
146 nistic motion stimulus from this subcortical optokinetic system facilitates development of the unstab
147 evelop in infancy, this phylogenetically old optokinetic system, which is normally operative in the f
150 65(T/-) mice visual function was measured by optokinetic tracking (OKT) and electroretinography (ERG)
151 ression, selected visual cycle proteins, and optokinetic tracking (OKT) in streptozotocin (STZ)-induc
152 ction as measured by electroretinography and optokinetic tracking and resulted in retinal morphologic
154 ould improve visual function (evaluated with optokinetic tracking response) of diabetic mice, potenti
156 ks of hyperglycemia for visual function with optokinetic tracking weekly visual acuity and monthly co
157 logical (electroretinogram), psychophysical (optokinetic tracking), and pharmacological techniques.
158 opening, in which daily threshold testing of optokinetic tracking, amid otherwise normal visual exper
162 n folium IXcd of the flocculus, such that an optokinetic zone spans a ZII+/- pair: the HA zones span