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1 ired pOKR were found to exhibit long-lasting optokinetic after nystagmus (OKAN) as opposed to those t
2 estingly, a long-lasting and robust negative optokinetic afternystagmus (OKAN) was evoked upon light
3 inetic reflex and evokes the second phase of optokinetic afternystagmus (OKAN-II).
4 e with severe visual defects, as assessed in optokinetic and optomotor response assays.
5                     Behaviorally, discordant optokinetic and vestibular input induced appropriate hig
6 vement recordings showed decreased gains for optokinetic and vestibulo-ocular reflexes, confirming an
7 gth of directed eye movements (i.e. combined optokinetic and voluntary tracking) for stimuli moving a
8 ibution suggests that monocular nasotemporal optokinetic asymmetry is partly attributable to subcorti
9 le in maintaining the monocular nasotemporal optokinetic asymmetry seen in patients with infantile es
10 y mutants required nearly an hour to recover optokinetic behavior after return to bright light, where
11 ed calcium imaging in awake zebrafish during optokinetic behavior to record transgenically identified
12 is, reduced neuronal connection, and reduced optokinetic behavioral response in zebrafish larvae.
13   Its applicaton for explaining a variety of optokinetic behaviors in other insects assumes that neur
14                               This nonfoveal optokinetic contribution suggests that monocular nasotem
15 ction in visual function on testing with the optokinetic drum and the circadian running wheel.
16 ent of motion responsiveness for pursuit and optokinetic eye movements (optokinetic nystagmus [OKN]).
17 lly constrained vector summation also guides optokinetic eye movements.
18 e of virtual reality, vibrotactile feedback, optokinetic flow, YouTube videos, and innovative methods
19 ters (saccadic latency, smooth pursuit gain, optokinetic gain), motor proficiency (BOT-2 subtests), a
20              Animals with vision had reduced optokinetic gains by 24 h, while the OKR response for an
21  latency and increases in smooth pursuit and optokinetic gains were observed (all p < 0.05).
22 Moreover, these cells are capable of driving optokinetic head tracking and visually guided behaviour
23         The pigeon flocculus receives visual-optokinetic information and is important for generating
24                             Nasally directed optokinetic input to the esodeviated eye can supplement
25 ted rostrally were most persistent following optokinetic input.
26 to generate accurate responses to full-field optokinetic input.
27 were differentially accessed by saccadic and optokinetic inputs.
28 suppresses the perception of this full-field optokinetic motion during active pursuit.
29 s uncovered, exposing it to nasally directed optokinetic motion.
30 ical deviation (DVD), monocular asymmetry of optokinetic nystagmus (MOKN), monocular asymmetry of smo
31 otions; i.e., they give better responses for optokinetic nystagmus (OKN) and visually evoked potentia
32            To evaluate the efficacy of using optokinetic nystagmus (OKN) as an objective measurement
33                                              Optokinetic nystagmus (OKN) assists stabilization of the
34                                         Look optokinetic nystagmus (OKN) consists of voluntary tracki
35 e of this study was to characterize vertical optokinetic nystagmus (OKN) in normal human subjects, co
36 f consciousness, inferred from the reflexive optokinetic nystagmus (OKN) pattern.
37         Modifying experimental conditions of optokinetic nystagmus (OKN) result in different outcomes
38     We asked if an involuntary eye movement, optokinetic nystagmus (OKN), could serve as an objective
39 designed to elicit smooth pursuit, saccades, optokinetic nystagmus (OKN), vestibulo-ocular reflex (VO
40             The effect of aging on torsional optokinetic nystagmus (tOKN) is unknown.
41 s for pursuit and optokinetic eye movements (optokinetic nystagmus [OKN]).
42                                  We used the optokinetic nystagmus and pupil size to objectively and
43 13%, 28%, and 30%), and nasotemporal pursuit/optokinetic nystagmus asymmetry (23%, 38%, and 54%).
44                             The direction of optokinetic nystagmus correlates with visual perception
45 ed head saccades to reset gaze, analogous to optokinetic nystagmus in primates.
46                                          The optokinetic nystagmus is a gaze-stabilizing mechanism re
47                            Abnormal vertical optokinetic nystagmus was present in 19 (68%) of 28 subj
48 sed on sub-conscious, reflex responses (e.g. optokinetic nystagmus) that don't require involvement of
49         To find out whether flies perform an optokinetic nystagmus, and how it may be affected by loc
50                                   Asymmetric optokinetic nystagmus, latent nystagmus, and dissociated
51             These include fast phases of the optokinetic nystagmus, visual scanning in stationary ani
52 ctive saccades, vergence smooth pursuit, and optokinetic nystagmus, was measured annually with a head
53 lexibly responded to rotational stimuli with optokinetic nystagmus-like head movements, independent o
54 ion and the role of nasally biased monocular optokinetic nystagmus.
55 de-field motion by a mechanism similar to an optokinetic nystagmus.
56 during leftward and rightward slow phases of optokinetic nystagmus.
57 es (Macaca mulatta) during the slow phase of optokinetic nystagmus.
58                                          The optokinetic (OK) stimulus subtended 72 degrees horizonta
59 Visual function was assessed with a rotating optokinetic (OKN) drum at ages 13 and 18 months and neur
60 ent of dye results in significantly improved optokinetic (OKR) ( 43 fold, p < 0.001) and visualmotor
61                                  Subcortical optokinetic pathways seem to play an important role in m
62 ore the feasibility of using Saccadic Vector Optokinetic Perimetry (SVOP) to differentiate glaucomato
63  and eye tracking perimetry (saccadic vector optokinetic perimetry, SVOP) was performed.
64  fixation, and a reduction in vestibular and optokinetic quick phases.
65 ity of the vestibulo-ocular reflex (VOR) and optokinetic reflex (OKR) allows for optimal combined gaz
66 cued water maze (WM) behavioral test and the optokinetic reflex (OKR) measurement at different times
67                                          The optokinetic reflex (OKR) serves as a vital index for vis
68                          Across species, the optokinetic reflex (OKR) stabilizes vision during self-m
69    A prime example of such behaviours is the optokinetic reflex (OKR), an innate eye movement mediate
70 haracterized as blind, these mutants lack an optokinetic reflex (OKR), but in another behavioral assa
71 on gaze stabilization behaviors, such as the optokinetic reflex (OKR), to perceive and correct for gl
72  (HOKS) decreases the gain of the horizontal optokinetic reflex and evokes the second phase of optoki
73 ntaneously firing neurons, is engaged during optokinetic reflex compensation for inner ear dysfunctio
74          At 6 d post-fertilization (dpf), no optokinetic reflex could be elicited in no optokinetic r
75  encoding in the superior colliculus and the optokinetic reflex follow a novel motion integration rul
76                                              Optokinetic reflex measurements showed that Jimpy mice h
77 Similar to what was observed neuronally, the optokinetic reflex occurred more reliably at low contras
78                              Whilst arrested optokinetic reflex pathway development is implicated in
79 e and after disease onset by quantifying the optokinetic reflex responses and to compare them to the
80                                          The optokinetic reflex resulting in optomotor head tracking
81                                    Using the optokinetic reflex to evaluate visual function, we obser
82                                              Optokinetic reflex was not detectable horizontally.
83     Similarly within the afferent arm of the optokinetic reflex we showed expression in the developin
84 on wiring of the ocular motor system and the optokinetic reflex, impairing horizontal eye movements.
85 dorsal oculomotor neurons impairs the larval optokinetic reflex, suggesting that neuronal clustering
86 eye-head angle to a default position via the optokinetic reflex, we developed an efficient and unbias
87  and despite abolishing the gaze-stabilizing optokinetic reflex.
88 d defective eye movements as measured by the optokinetic reflex.
89 ay of retinal signals into the brainstem for optokinetic reflexes.
90 d those on retinal function were analyzed by optokinetic response (OKR) and electroretinography (ERG)
91                                          The optokinetic response (OKR) consists of smooth eye moveme
92 brates large, moving visual scenes induce an optokinetic response (OKR) control of eye movements to s
93                                          The optokinetic response (OKR) to a visual stimulus moving a
94 ly evoked smooth eye movements, known as the optokinetic response (OKR), have been studied in various
95            This behavior, which is termed an optokinetic response (OKR), is a reflex that appears in
96 ations (SOs) and, in several cases, reversed optokinetic response (OKR).
97                                          The optokinetic response (tracking eye movements) was evoked
98 wo alleles of the recessive lethal mutant no optokinetic response a (noa).
99                                Additionally, optokinetic response analysis performed at 5dpf indicate
100           Fish deficient in Ribeye a lack an optokinetic response and have shorter synaptic ribbons i
101 mprovement of visual function as assessed by optokinetic response and looming-induced escape behavior
102 was assessed using the electroretinogram and optokinetic response and retinal morphology investigated
103        A red-blind zebrafish mutant, partial optokinetic response b (pob), has been isolated by measu
104                The zebrafish mutant, partial optokinetic response b (pob), was isolated using an N-et
105 physiology of the zebrafish visual mutant no optokinetic response c (nrc) and to identify the genetic
106 o optokinetic reflex could be elicited in no optokinetic response c (nrc) mutant animals under any te
107      Here we describe a zebrafish mutant, no optokinetic response f(w21) (nof), with a nonsense mutat
108                                          The optokinetic response for wfs1b(-/-) zebrafish was signif
109                              We measured the optokinetic response gain of immobilised zebrafish larva
110 re reduced visual acuity, measured using the optokinetic response, and vascular leakage continued to
111      Recovery of Ribeye a levels rescues the optokinetic response, increases the number of PKCalpha-p
112 ed visual function, as evidenced by improved optokinetic response, restored b-wave amplitude in elect
113 il and assessed for visual function using an optokinetic response, with subsequent samples taken for
114 the whole brains of zebrafish performing the optokinetic response.
115 ysis of optic flow and the generation of the optokinetic response.
116 y optic system (AOS) where they initiate the optokinetic response.
117 d movements in their surroundings, displayed optokinetic responses (OKR) or optomotor responses (OMR)
118 of RPE-expressed rlbp1b selectively impaired optokinetic responses (OKR) with a ~50% reduction in sac
119 nse to light, of which the optomotor and the optokinetic responses are the most widely studied.
120                                 In contrast, optokinetic responses are unaffected in the opposite con
121                                              Optokinetic responses became unstable but were generally
122                           Moreover, bistable optokinetic responses cannot be entirely attributed to s
123                          This improvement in optokinetic responses did not necessitate a fixation shi
124 etinal motion input in generating horizontal optokinetic responses in patients with infantile strabis
125 y designed to identify larvae with defective optokinetic responses in red but not white light.
126 viated eye can supplement temporal monocular optokinetic responses in the fixating eye under binocula
127 All patients showed poor temporally directed optokinetic responses that instantaneously improved when
128                               Measurement of optokinetic responses to plaid stimuli revealed that mic
129 y was measured behaviorally, using optomotor/optokinetic responses to rotating square-wave stimuli.
130         Visual predators rely on fast-acting optokinetic responses to track and capture agile prey.
131                                    Subnormal optokinetic responses were found in a subgroup of obliga
132 nced by changes in the direction of elicited optokinetic responses.
133 g: decreased survival; hypokinesia; impaired optokinetic responses; neurodegeneration; neuroinflammat
134 under certain stimulus conditions to mediate optokinetic signals in the brain.
135 t, gaze holding, convergence, vestibular and optokinetic slow phases, and cancellation of vestibular
136                         Long-term horizontal optokinetic stimulation (HOKS) decreases the gain of the
137                 During a sustained period of optokinetic stimulation in 5-day-old wild-type zebrafish
138                                              Optokinetic stimulation induces nystagmus that can be us
139      These VEPRs are not simple responses to optokinetic stimulation, but are modulated by the config
140  sensory adaptation process during continued optokinetic stimulation, which, when the stimulus is rem
141  the oculomotor integrator after saccadic or optokinetic stimulation.
142 duration and the adaptation to the preceding optokinetic stimulation.
143 ike activity (CSA) in response to rotational optokinetic stimuli.
144 = 9) while pairing yaw rotation with a pitch optokinetic stimulus, resulting in cross-axis adaptation
145 ross their retinas, creating a contraversive optokinetic stimulus.
146 nistic motion stimulus from this subcortical optokinetic system facilitates development of the unstab
147 evelop in infancy, this phylogenetically old optokinetic system, which is normally operative in the f
148 OKT thresholds were measured using a virtual optokinetics system.
149                                              Optokinetic testing was performed in 7 patients with iso
150 65(T/-) mice visual function was measured by optokinetic tracking (OKT) and electroretinography (ERG)
151 ression, selected visual cycle proteins, and optokinetic tracking (OKT) in streptozotocin (STZ)-induc
152 ction as measured by electroretinography and optokinetic tracking and resulted in retinal morphologic
153       Microbead-injected eyes showed reduced optokinetic tracking as well as cell death.
154 ould improve visual function (evaluated with optokinetic tracking response) of diabetic mice, potenti
155           Visual function was measured using optokinetic tracking to determine spatial frequency and
156 ks of hyperglycemia for visual function with optokinetic tracking weekly visual acuity and monthly co
157 logical (electroretinogram), psychophysical (optokinetic tracking), and pharmacological techniques.
158 opening, in which daily threshold testing of optokinetic tracking, amid otherwise normal visual exper
159                               We devised the optokinetic uncover test to examine the role of peripher
160 ysfunction arising due to instability of the optokinetic-vestibular systems.
161 ing nystagmus arises from instability of the optokinetic-vestibular systems.
162 n folium IXcd of the flocculus, such that an optokinetic zone spans a ZII+/- pair: the HA zones span
163                                        These optokinetic zones relate to the ZII stripes in folium IX

 
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