ライフサイエンスコーパス: organ-specific autoimmune disease

1 ed immune systems, in the pathogenesis of an organ specific autoimmune disease.

2 A-deficient mice did not develop systemic or organ-specific autoimmune disease.

3 of CD40 plays a key role in the etiology of organ-specific autoimmune disease.

4 (EAO), 2 classical T cell-mediated models of organ-specific autoimmune disease.

5 ry T cells; T(regs)) can function to control organ-specific autoimmune disease.

6 thway has immunoregulatory potential in this organ-specific autoimmune disease.

7 tulates observations made in other models of organ-specific autoimmune disease.

8 on of HLA-DQ8 confers susceptibility to this organ-specific autoimmune disease.

9 reactive T cell response and T cell-mediated organ-specific autoimmune disease.

10 bgroups, one of which may be associated with organ-specific autoimmune disease.

11 epitopes that are sufficient to produce this organ-specific autoimmune disease.

12 suppressor T cells that prevent induction of organ-specific autoimmune disease.

13 multiple chemokines in vivo during a complex organ-specific autoimmune disease.

14 udy the immunopathogenic features of a human organ-specific autoimmune disease.

15 lerance have not been studied in spontaneous organ-specific autoimmune disease.

16 ell tolerance and lead to the development of organ-specific autoimmune disease.

17 uction and skin blistering in a prototypical organ-specific autoimmune disease.

18 ential role in resistance or pathogenesis of organ-specific autoimmune diseases.

19 s into the pathogenesis of both systemic and organ-specific autoimmune diseases.

20 opment and pathogenesis of both systemic and organ-specific autoimmune diseases.

21 ctive at blocking the development of certain organ-specific autoimmune diseases.

22 se molecules promote or inhibit systemic and organ-specific autoimmune diseases.

23 T cells are essential in the protection from organ-specific autoimmune diseases.

24 hypersensitivity reactions, and induction of organ-specific autoimmune diseases.

25 e immunopathogenic mechanisms of uveitis and organ-specific autoimmune diseases.

26 ism to explain the etiology of virus-induced organ-specific autoimmune diseases.

27 the immunopathology responsible for several organ-specific autoimmune diseases.

28 more heterogeneous and had both systemic and organ-specific autoimmune diseases.

29 antigenically distinct CD4+ T cell-mediated, organ-specific, autoimmune diseases.

30 Graves disease, a common organ-specific autoimmune disease affecting humans, diff

31 ells that can prevent both the initiation of organ-specific autoimmune disease after day 3 thymectomy

32 ies in the pathogenesis of most systemic and organ-specific autoimmune diseases, although there is co

33 ings represent a critical component of human organ-specific autoimmune disease and may have important

34 mune disorders comprise more than 30% of all organ-specific autoimmune diseases and are characterized

35 Many human organ-specific autoimmune diseases are characterized by

36 ommon among patients with Down syndrome, but organ-specific autoimmune diseases are common.

37 The mechanisms underlying organ-specific autoimmune diseases are incompletely unde

38 autoimmune encephalomyelitis (EAE) and other organ-specific autoimmune diseases are induced by autoan

39 nitiate and maintain MHC class II-associated organ-specific autoimmune diseases are poorly defined.

40 Organ-specific autoimmune diseases are usually character

41 s have been implicated in the development of organ-specific autoimmune diseases as well as pathogen-s

42 h can be customized for application to other organ-specific autoimmune diseases as well.

43 Thus, EAM represents a model of an organ-specific autoimmune disease associated with a Th2

44 IL-23) play crucial roles in pathogenesis of organ-specific autoimmune diseases by inducing the diffe

45 ontaneous autoimmune thyroiditis (SAT) is an organ-specific autoimmune disease characterized by chron

46 As organ-specific autoimmune diseases do not become manifes

47 te this, mice lacking DRAK2 are resistant to organ-specific autoimmune diseases due to defective auto

48 gle self-antigens can be sufficient to cause organ-specific autoimmune disease, even in otherwise sel

49 ls, and endothelia as well as participate in organ-specific autoimmune disease, graft rejection, and

50 gers of systemic autoimmunity, but a role in organ-specific autoimmune disease has not been demonstra

51 ions both in the induction and regulation of organ-specific autoimmune diseases have been defined and

52 has advanced more slowly than that of other organ-specific autoimmune diseases, however, largely bec

53 peripheral tolerance, and the development of organ-specific autoimmune disease in adult mice.

54 eg accumulation and function during an acute organ-specific autoimmune disease in vivo.

55 We now recognize that MN is an organ-specific autoimmune disease in which circulating a

56 xplained excess of type 1 diabetes and other organ-specific autoimmune diseases in children with Down

57 ffecting the skin, is one of the most common organ-specific autoimmune diseases in humans.

58 The development of organ-specific autoimmune diseases in mice thymectomized

59 IL-15 disorders play pathogenetic roles in organ-specific autoimmune diseases including celiac dise

60 The target autoantigens in several organ-specific autoimmune diseases, including type 1 dia

61 ysis bullosa acquisita (EBA) is a prototypic organ-specific autoimmune disease induced by autoantibod

62 tal autoimmune anterior uveitis (EAAU) is an organ-specific autoimmune disease induced by immunizatio

63 CD4(+)CD25(+) regulatory T cells inhibit organ-specific autoimmune diseases induced by CD4(+)CD25

64 CD4(+)CD25(+) regulatory T cells inhibit organ-specific autoimmune diseases induced by CD4(+)CD25

65 tal autoimmune encephalomyelitis (EAE) is an organ-specific autoimmune disease inducible in susceptib

66 ssue destruction and point to a new model of organ-specific autoimmune disease involving lipid Ag pre

67 The highly selective nature of organ-specific autoimmune disease is consistent with a c

68 e production of pathogenic autoantibodies in organ-specific autoimmune diseases is largely T cell dep

69 Recovery from organ-specific autoimmune diseases largely relies on the

70 e diseases, suggesting that resistance to an organ-specific autoimmune disease may be regulated at le

71 Hence, virus-induced organ-specific autoimmune diseases may be dependent on v

72 D) in mice, a slowly progressing Ab-mediated organ-specific autoimmune disease of the thyroid induced

73 To understand the pathogenesis of organ-specific autoimmune disease requires an appreciati

74 Infection may exacerbate organ-specific autoimmune disease such as glomerulonephr

75 , a member of herpes viridae, causes various organ-specific autoimmune diseases, such as autoimmune g

76 The induction of organ-specific autoimmune diseases, such as experimental

77 Type 1 diabetes is an organ-specific autoimmune disease that is mediated by au

78 biliary cholangitis (PBC) is a prototypical organ-specific autoimmune disease that is mediated by au

79 ons are also critical in the pathogenesis of organ-specific autoimmune diseases that were previously

80 In many organ-specific autoimmune diseases, the identity of the

81 in almost all of the animal models of human organ-specific autoimmune diseases, transplant rejection

82 Organ-specific autoimmune disease was present in 7 of th

83 implicated in the immunopathology of certain organ-specific autoimmune diseases whereas a role as reg