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1 r, reticulin fibrosis/renal dysfunction, and organomegaly.
2 iferative disorder including lymphadenopathy/organomegaly.
3 progressive myeloproliferative disorder with organomegaly.
4 lanine aminotransferase, petechial rash, and organomegaly.
5 ) gene results in widespread hyperplasia and organomegaly.
6 ies, including gender-specific gigantism and organomegaly.
7 compared with those of patients lacking such organomegaly.
8 h male gender, worse performance status, and organomegaly.
9 s with A-MDS were older and had little or no organomegaly.
10 ll of these patients had bulky adenopathy or organomegaly.
11  These mice develop gigantism and widespread organomegaly.
12  was requested to depict lymphadenopathy and organomegaly.
13 linical manifestations including cytopenias, organomegaly and bone disease.
14 severe lysosome pathology (hypopigmentation, organomegaly, and delayed myelination and development, "
15 tion and maturation block, thrombocytopenia, organomegaly, and extensive fibrosis.
16 variant associated with developmental delay, organomegaly, and hypopigmentation resulting from lysoso
17 ren had delayed myelination and development, organomegaly, and hypopigmentation, but neither had oste
18 tions of ALPS include autoimmune cytopenias, organomegaly, and lymphadenopathy.
19                                       Fever, organomegaly, and sepsis were the next most frequent fin
20 ohistiocytosis, lymphoproliferative disease, organomegaly, and/or malignancy.
21 anifestations of HLH, including weight loss, organomegaly, anemia, thrombocytopenia, hypercytokinemia
22 ether sclerotic bone lesions, effusions, and organomegaly are present.
23 g Cux-1 developed multiorgan hyperplasia and organomegaly, but not an overall increase in body size.
24 ion, which clinically manifests with fevers, organomegaly, cytopenias, laboratory derangements, and r
25 atment prevents disability in Polyneuropathy Organomegaly Endocrinopathy Monoclonal-protein and Skin
26 syndrome (coined to refer to polyneuropathy, organomegaly, endocrinopathy, M protein, and skin change
27                              Polyneuropathy, organomegaly, endocrinopathy, monoclonal gammopathy, and
28                       POEMS (polyneuropathy, organomegaly, endocrinopathy, monoclonal gammopathy, and
29 sociated MCD (HHV8-MCD), and polyneuropathy, organomegaly, endocrinopathy, monoclonal plasma cell dis
30  and organomegaly) or POEMS (polyneuropathy, organomegaly, endocrinopathy, monoclonal plasma cell dis
31 eflects the common features: Polyneuropathy, Organomegaly, Endocrinopathy, Monoclonal protein and Ski
32 ic disorder characterized by polyneuropathy, organomegaly, endocrinopathy, monoclonal protein, and sk
33 y (PN), a clonal plasma cell disorder (PCD), organomegaly, endocrinopathy, skin changes, edema, scler
34 rbid manifestations of the disease including organomegaly, growth and pubertal retardation, and osteo
35 evels and severe mediator symptoms, all with organomegaly had systemic disease, and none without orga
36 egaly had systemic disease, and none without organomegaly had systemic disease.
37 er features, including acanthosis nigricans, organomegaly, hyperandrogenism, and diabetes.
38 ites, reticulin fibrosis, renal dysfunction, organomegaly (iMCD-TAFRO) or iMCD-not otherwise specifie
39 (135% of normal birthweight) and generalized organomegaly, kinky tail, postaxial polydactyly, heart a
40           Instead, patients show progressive organomegaly, leukocytosis, anemia, and thrombocytosis.
41  The POEMS syndrome (polyradiculoneuropathy, organomegaly, multiple endocrinopathies, monoclonal prot
42 ndicated a uniform increase in mass, with no organomegaly or associated pathology.
43  included: a bone lesion, Castleman disease, organomegaly (or lymphadenopathy), endocrinopathy, edema
44 enia, ascites, fever, reticulin fibrosis and organomegaly) or POEMS (polyneuropathy, organomegaly, en
45 did not cause obvious neurological symptoms, organomegaly, or a reduction in lifespan.
46  symptoms including urticaria pigmentosa and organomegaly (P < .02).
47 c features of HLS, including fever, multiple organomegaly, pancytopenia, hemophagocytosis, hyperfibri
48                        We will summarize the organomegaly phenotypes associated with Pten tissue-spec
49 he disease, that is, polyradiculoneuropathy, organomegaly, potentially including coexisting Castleman
50 e reticulo-endothelial systems, with massive organomegaly resulting from the presence of engorged, li
51 p27(Kip1) develop widespread hyperplasia and organomegaly similar to those developed by p18-deficient
52 lets <100 x 10(9)/L, bulky adenopathy and/or organomegaly, symptomatic hyperviscosity, peripheral neu
53 less than 100 x 10(9)/L, bulky adenopathy or organomegaly, symptomatic hyperviscosity, peripheral neu
54 lder age, high white blood cell (WBC) count, organomegaly, T-lineage immunophenotype, ability of leuk
55  reticulin myelofibrosis, renal dysfunction, organomegaly (TAFRO) clinical subtype.
56                                              Organomegaly was a strong indicator of systemic disease.
57                                              Organomegaly was confirmed by means of ultrasound.
58                                              Organomegaly with cardiopulmonary compromise most freque
59         However, these mice developed severe organomegaly with disorganized lymphoid tissues.