ライフサイエンスコーパス: oryzae

1 rculans) to 34.9% (K. lactis), and 19.5% (A. oryzae).

2 and bacterial blight (Xanthomonas oryzae pv. oryzae).

3 a cell wall-degrading enzyme of X. oryzae pv oryzae).

4 growth, development and pathogenicity in M. oryzae.

5 f MoGSK1 produced deformed appressoria in M. oryzae.

6 ytoplasmic localization as MoGsk1 does in M. oryzae.

7 rp1 as the yeast Vrp1 homolog in Magnaporthe oryzae.

8 rmation plays a critical role in Magnaporthe oryzae.

9 ll-to-cell movement of invasive hyphae in M. oryzae.

10 involving the rice blast fungus Magnaporthe oryzae.

11 llular ROS signalling and pathogenesis in M. oryzae.

12 two thioredoxin genes, TRX1 and TRX2, in M. oryzae.

13 without subsequent elicitation with Rhizopus oryzae.

14 mechanism of the blight disease caused by M. oryzae.

15 ented with Monascus purpureus or Aspergillus oryzae.

16 (ETFDH) in the rice blast fungus Magnaporthe oryzae.

17 nfection of bacterium Xanthomonas oryzae pv. oryzae.

18 t the virulence of compatible isolates of M. oryzae.

19 athogens: Magnaporthe oryzae and Xanthomonas oryzae.

20 dent disease susceptibility to X. oryzae pv. oryzae.

21 13 induction by 42 isolates of X. oryzae pv. oryzae.

22 C is essential for viability of Magnaporthe oryzae.

23 a model plant pathogenic fungus, Magnaporthe oryzae.

24 plain enhanced susceptibility to Magnoporthe oryzae.

25 ria tritici, Ustilago maydis and Magnaporthe oryzae.

26 pment of infection structures in Magnaporthe oryzae.

27 MoHMT1 in the rice blast fungus, Magnaporthe oryzae.

28 thogens Pseudomonas syringae and Xanthomonas oryzae.

29 f laccase (LAC) originating from Aspergillus oryzae.

30 enhanced susceptibility of rice plants to M. oryzae.

31 ctors of the rice blast pathogen Magnaporthe oryzae.

32 on etiologic agent of mucormycosis, Rhizopus oryzae.

33 infection by the fungal pathogen Magnaporthe oryzae.

34 y step in the infection cycle of Magnaporthe oryzae.

35 s septin assembly and host penetration by M. oryzae.

36 by peroxidases, leading to resistance to M. oryzae.

37 mmunity against the blast fungus Magnaporthe oryzae.

38 agnaporthe oryzae and Xanthomonas oryzae pv. oryzae.

39 known to inactivate plant MAP kinases in M. oryzae.

40 as validated in the hemibiotroph Magnaporthe oryzae.

41 cally to infection by pathogenic Pyricularia oryzae.

42 formation and plant infection in Magnaporthe oryzae.

43 growth, conidiation and pathogenicity of M. oryzae.

44 culans, Kluyveromyces lactis and Aspergillus oryzae.

45 In the devastating blast fungus Magnaporthe oryzae(1), powerful glycoprotein-rich mucilage adhesives

46 The main fungal species were Rhizopus oryzae (32%) and Lichtheimia species (29%).

47 erboard assays for 4 clinical isolates of R. oryzae (48-hour fractional inhibitory concentration inde

48 treatment with either Xanthomonas oryzae pv oryzae (a bacterial pathogen) or lipaseA/esterase (LipA;

49 o examine the effects of statins on Rhizopus oryzae, a common cause of mucormycosis.

50 factor of RNAP of host bacterium Xanthomonas oryzae, a major rice pathogen.

51 For the fermentation by Aspergillus oryzae, all the function responses were influenced by X(

52 is involved in all three MAPK pathways in M. oryzae although its functions differ in each pathway.

53 ophilin (Rhi o 2) was purified from Rhizopus oryzae, an indoor mold causing allergic sensitization.

54 annotation improvements for A. nidulans, A. oryzae and A. fumigatus genomes based on recently availa

55 d to the proteolytic activity of Aspergillus oryzae and Aspergillus flavipes enzymes, as well as to a

56 aerospermum, Paecilomyces formosus, Rhizopus oryzae and Aspergillus niger.

57 ase of rice caused by the fungus Magnaporthe oryzae and can result in loss of a third of the annual g

58 aseX, SODX) led to enhanced resistance to M. oryzae and increased hydrogen peroxide (H(2) O(2) ) accu

59 Development of M. oryzae and initiation of infection are critically depend

60 including Zymoseptoria tritici, Magnaporthe oryzae and Neurospora crassa, exhibited PAMP activity, i

61 We review recent studies of M. oryzae and other relevant appressorium-forming fungi whi

62 pathogens Xanthomonas oryzae pathovar (pv.) oryzae and pv. oryzicola produce numerous transcription

63 nfection of M. grisea, Xanthomonas oryzae pv.oryzae and rice stripe virus were analyzed.

64 ciation between the cereal weevil Sitophilus oryzae and the bacterium Sodalis pierantonius as a model

65 esistance to the fungal pathogen Magnaporthe oryzae and the RING-type E3 ligase AVRPIZ-T INTERACTING

66 Gram-negative E. coli and plant pathogens X. oryzae and X. campestris, as well as against human funga

67 nced susceptibility to the rice pathogens M. oryzae and Xanthomonas oryzae pv oryzae (Xoo).

68 ent immunity: increased susceptibility to M. oryzae and Xanthomonas oryzae pv. oryzae, hemibiotrophic

69 resistance against the pathogens Magnaporthe oryzae and Xanthomonas oryzae pv. oryzae.

70 gainst two major rice pathogens: Magnaporthe oryzae and Xanthomonas oryzae.

71 t, as well as enhanced resistance to both M. oryzae and Xoo.

72 hanced resistance to rice blast (Magnaporthe oryzae) and bacterial blight (Xanthomonas oryzae pv. ory

73 izoctonia solani, and Xanthomonas oryzae pv. oryzae) and several chemical elicitors.

74 he normal physiology and pathogenicity of M. oryzae, and it sheds new light on the mechanism of the b

75 s to MAP kinase kinase kinase in Magnaporthe oryzae, and urate oxidase (designated ClUrase) were func

76 acearum, Xanthomonas campestris, Xanthomonas oryzae, and Xylella fastidiosa T2S also occurs in nonpat

77 s species: Aspergillus nidulans, Aspergillus oryzae, Aspergillus fumigatus and Aspergillus niger.

78 The reductive aminase from Aspergillus oryzae (AspRedAm) was combined with a single alcohol deh

79 dependent reductive aminase from Aspergillus oryzae (AspRedAm, Uniprot code Q2TW47) that can catalyse

80 ybP-ykoY riboswitch aptamer from Xanthomonas oryzae at 2.96 angstrom resolution, revealing two confor

81 ne susceptible and the other resistant to M. oryzae, at multiple time points during the fungal infect

82 ne chitosan nanocomposites bound Aspergillus oryzae beta-galactosidase.

83 pproximately 40-50% with B. circulans and A. oryzae beta-galactosidases, and at 95% lactose depletion

84 aria oryzae, Gibberella fujikuroi, Bipolaris oryzae, Burkholderia glumae, Xanthomonas oryzae, Erwinia

85 inhardtii, Mucor circinelloides and Rhizopus oryzae but was dissimilar to the non-oleaginous Aspergil

86 fungal development and plant infection in M. oryzae by regulation of fatty acid metabolism, turgor es

87 gens Xanthomonas vesicatoria and Xanthomonas oryzae, capable of inhibiting disease symptoms in detach

88 the first case of a successfully treated A. oryzae catheter-associated bloodstream infection in an i

89 The fungus Magnaporthe oryzae causes blast, the most devastating disease of cul

90 The fungus Magnaporthe oryzae causes devastating diseases of crops, including r

91 Here, we report that Magnaporthe oryzae CKS1 encodes a cyclin-dependent kinase subunit, w

92 s-dependent cell death, and resistance to M. oryzae containing AvrPiz-t.

93 Additionally, expression of R. oryzae CotH was increased within hours of incubation wit

94 hreshold of posaconazole activity against R. oryzae could be achieved with 2-fold lower drug concentr

95 report that a purine-requiring mutant of M. oryzae could develop functional appressoria, penetrate h

96 ional heterologous expression in Aspergillus oryzae coupled with isolation and detailed structural el

97 e rice blast disease, the fungus Magnaporthe oryzae develops a pressurized dome-shaped cell called an

98 ast disease, the fungal pathogen Magnaporthe oryzae develops a specialized infection structure called

99 resistance to PthXo2-dependent X. oryzae pv. oryzae due to promoter variations of OsSWEET13 in japoni

100 he roles of the GPI anchoring in Magnaporthe oryzae during plant infection.

101 sativa) NLR RGA5 recognizes the Magnaporthe oryzae effector AVR-Pia through direct interaction.

102 also mediate recognition of the unrelated M. oryzae effector AVR-Pia, indicating that the correspondi

103 t factors that interact with the Magnaporthe oryzae effector AVR-Pii.

104 uired for the recognition of the Magnaporthe oryzae effector AVR1-CO39.

105 Here, we show that the M. oryzae effector AvrPiz-t interacts with the bZIP-type tr

106 Development of the M. oryzae effector-secreting biotrophic interfacial complex

107 The rice blast fungus Magnaporthe oryzae elaborates a specialized cell called an appressor

108 ts corroborate a model whereby X. oryzae pv. oryzae enhances the release of sucrose from host cells i

109 ris oryzae, Burkholderia glumae, Xanthomonas oryzae, Erwinia chrysanthemi, Pseudomonas syringae, and

110 Additionally, R. oryzae exposed to lovastatin showed macroscopic loss of

111 statins had MICs of >64 microg/mL against R. oryzae Exposure of R. oryzae to statins decreased germli

112 tive extraction methods based on Aspergillus oryzae fermentation and alpha-amylase hydrolysis are als

113 in pyrazines was observed in the Aspergillus oryzae-fermented samples, while higher levels of furans

114 Slp1 is required by M. oryzae for full virulence and exerts a significant effec

115 icant reductions in cutaneous lesions and R. oryzae fungal burden, compared with animals that receive

116 is work, the effect of SSF with the Rhizopus oryzae fungus on the phenolic acid content of rice bran

117 The blast fungus Magnaporthe oryzae gains entry to its host plant by means of a speci

118 We demonstrate that Magnaporthe oryzae generates NO during germination and in early deve

119 R. oryzae germination, DNA fragmentation, susceptibility to

120 In bioassays with Pyricularia oryzae, Gibberella fujikuroi, Bipolaris oryzae, Burkhold

121 cluster was reconstructed within Aspergillus oryzae giving production of pleuromutilin in an ascomyce

122 After penetrating the leaf cuticle, M. oryzae grows as a biotroph in intimate contact with livi

123 oid triggering plant defences that impact M. oryzae growth and BIC development.

124 Consequently, R. oryzae has a 2- to 10-fold enrichment in gene families a

125 We conclude that M. oryzae has a robust anti-oxidant defence system and main

126 receptors for Candida albicans and Rhizopus oryzae has been demonstrated in experimental animal mode

127 Fermentation by using Aspergillus oryzae has been reported to increase antioxidant activit

128 lity to M. oryzae and Xanthomonas oryzae pv. oryzae, hemibiotrophic pathogens, but enhanced resistanc

129 ited antifungal activity against Magnaporthe oryzae (IC(50), 5.21 ug/mL).

130 of the devastating rice pathogen Magnaporthe oryzae impaired for de novo methionine biosynthesis.

131 t of the important rice pathogen Magnaporthe oryzae in leaf cells.

132 ld cell death, and enhanced resistance to M. oryzae in the non-Piz-t background.

133 olimus, alone and in combination, against R. oryzae in vitro, using multiple methods (ie, hyphal meta

134 and development of diverse cell types in M. oryzae, including conidia, appressoria, and asci.

135 cies (ROS) and enhances susceptibility to M. oryzae, indicating that AvrPiz-t functions to suppress p

136 tivation of Bsr-d1 expression by Magnaporthe oryzae infection and degradation of H(2) O(2) by peroxid

137 Expression of OsMYB30 was induced during M. oryzae infection or when Bsr-d1 was knocked out or downr

138 The rice blast fungus Magnaporthe oryzae infects plants with a specialized cell called an

139 S) data was applied for classification of S. oryzae infestation in rice grains.

140 to 84.75% of rice with variable degree of S. oryzae infestation.

141 ive sensors was further applied to detect S. oryzae infested rice with PCA and MLR techniques.

142 nd reduce lesion sizes caused by Magnaporthe oryzae Inhibition of EXO70 by ES2-14 in Botrytis cinerea

143 anchoring facilitates the penetration of M. oryzae into host cells by affecting the cell wall integr

144 fungus Magnaporthe oryzae (syn: Pyricularia oryzae) into three major globally distributed clades bas

145 l resistant and susceptible plants during M. oryzae invasion discovered distinct pathways triggered i

146 e cycle of the rice blast fungus Magnaporthe oryzae involves a series of morphogenetic changes, essen

147 A. oryzae IOC 3999/1998 expressed beta-glucosidase activity

148 efatted soybean flour (DSF) with Aspergillus oryzae IOC 3999/1998 or Monascus purpureus NRRL 1992 was

149 Acidovorax oryzae is a bacterium that has never before been reporte

150 The rice blast fungus Magnaporthe oryzae is a model for studying fungal-plant interactions

151 The fungus Magnaporthe oryzae is a serious pathogen of rice and other grasses.

152 e reductive aminase (RedAm) from Aspergillus oryzae is combined with either (i) a 1 degrees alcohol/a

153 disease by strains of Xanthomonas oryzae pv. oryzae is dependent on major transcription activation-li

154 ted and the distribution of the operon in X. oryzae is investigated in over 100 isolates.

155 ips and show that the closest relative of A. oryzae is not A. flavus, but A. minisclerotigenes or A.

156 ycogen synthase kinase 3 (GSK3) MoGSK1 in M. oryzae is regulated by Mps1 MAP kinase, particularly und

157 Magnaporthe oryzae is the causal agent of rice blast disease, the mo

158 The blast fungus Magnaporthe oryzae is the most devastating pathogen of cultivated ri

159 m-negative bacterium, Xanthomonas oryzae pv. oryzae, is a tyrosine sulfotransferase.

160 epeat protein, confers resistance against X. oryzae isolates by recognizing multiple TALEs.

161 -galactosidase preparations from Aspergillus oryzae, Kluyveromyces lactis and Bacillus circulans.

162 f sialyl Lewis(a), Lewis(x), and Aspergillus oryzae lectin [AOL] and binding of wheat germ agglutinin

163 splasia, abnormal DNA ploidy and Aspergillus oryzae lectin) can identify patients at high risk for de

164 r terephthalate-to-adipate ratio by Rhizopus oryzae lipase and Fusarium solani cutinase.

165 rs by two fungal esterases (FsC and Rhizopus oryzae lipase) at different temperatures.

166 redox-responsive E3 ubiquitin ligases in M. oryzae mediate Sir2 accumulation-dependent antioxidation

167 t pathogen, the rice blast fungusMagnaporthe oryzae(Mo), was expressed inPichia pastoris.Mo-MnLOX was

168 so increased fungal growth and attenuated R. oryzae neutrophil-mediated damage.

169 m-negative bacterium, Xanthomonas oryzae pv. oryzae; NH1, the rice ortholog of NPR1, a key regulator

170 We show that the M. oryzae NMO2 gene is required for mitigating damaging lip

171 Here, we characterized the M. oryzae nucleoside diphosphate kinase-encoding gene NDK1

172 tivation (SSC) time of rice bran by Rhizopus oryzae on gamma-oryzanol recovery and its antioxidant pr

173 r (DSF) by Monascus purpureus or Aspergillus oryzae on the bioactive compounds.

174 tructure of the DBD of PCG2, the Magnaporthe oryzae orthologue of MBP1, bound to MCB-DNA.

175 show that the rice blast fungus Magnaporthe oryzae overcomes this first line of plant defense by sec

176 Wheat blast caused by the fungus Magnaporthe oryzae pathotype Triticum (MoT) is an emerging threat to

177 The rice pathogens Xanthomonas oryzae pathovar (pv.) oryzae and pv. oryzicola produce n

178 oc (> 90%), but absent in the other major X. oryzae pathovar.

179 sponse to diverse TAL effectors from both X. oryzae pathovars.

180 hyma cells near the epidermis, inhibiting M. oryzae penetration at the early stage of infection.

181 reviously, we showed that during Xanthomonas oryzae phage Xp10 infection, the phage protein P7 inhibi

182 cription regulator P7 encoded by Xanthomonas oryzae phage Xp10.

183 -containing SM cluster, ACE1, in a clonal M. oryzae population (Clade 2).

184 eport that the rice blast fungus Magnaporthe oryzae possesses two distinct secretion systems to targe

185 Piz-t from the rice blast fungus Magnaporthe oryzae preferentially accumulates in the specialized str

186 The enzyme from A. oryzae produced the highest yield and specific productiv

187 es and soluble protein fractions of Azospira oryzae PS, as well as soluble protein fractions encapsul

188 ase, acetylxylan esterase, and a Xanthomonas oryzae putative a-L: -arabinofuranosidase.

189 following treatment with either Xanthomonas oryzae pv oryzae (a bacterial pathogen) or lipaseA/ester

190 the rice pathogens M. oryzae and Xanthomonas oryzae pv oryzae (Xoo).

191 se (LipA; a cell wall-degrading enzyme of X. oryzae pv oryzae).

192 a) xylem and mesophyll pathogens Xanthomonas oryzae pv. oryzae (Xoo) and pv. oryzicola (Xoc) encode n

193 ion pathway of the rice pathogen Xanthomonas oryzae pv. oryzae (Xoo) in a semi-isolated environment r

194 The biotrophic pathogen Xanthomonas oryzae pv. oryzae (Xoo) produces a sulfated peptide name

195 esistance to a broad spectrum of Xanthomonas oryzae pv. oryzae (Xoo) races that cause bacterial bligh

196 of the bacterial blight pathogen Xanthomonas oryzae pv. oryzae (Xoo) that deliver the TAL effector Av

197 and delivered by the pathogenic Xanthomonas oryzae pv. oryzae (Xoo), in revealing the new function o

198 The pathogen, Xanthomonas oryzae pv. oryzae (Xoo), secretes one or more of six kno

199 XA3 receptor confer immunity to Xanthomonas oryzae pv. oryzae (Xoo), the causal agent of bacterial l

200 nfection by pathogenic bacterium Xanthomonas oryzae pv. oryzae (Xoo), which causes a vascular disease

201 terial blight diseases caused by Xanthomonas oryzae pv. oryzae (Xoo).

202 ce to bacterial blight caused by Xanthomonas oryzae pv. oryzae (Xoo).

203 ce to bacterial blight caused by Xanthomonas oryzae pv. oryzae (Xoo).

204 of rice (Oryza sativa) caused by Xanthomonas oryzae pv. oryzae (Xoo).

205 for bacterial blight, caused by Xanthomonas oryzae pv. oryzae (Xoo).

206 ed immunity X (RaxX) produced by Xanthomonas oryzae pv. oryzae (Xoo).

207 recessive resistance to PthXo2-dependent X. oryzae pv. oryzae due to promoter variations of OsSWEET1

208 The results corroborate a model whereby X. oryzae pv. oryzae enhances the release of sucrose from h

209 preventing disease by strains of Xanthomonas oryzae pv. oryzae is dependent on major transcription ac

210 ram-negative bacterial pathogen, Xanthomonas oryzae pv. oryzae upon recognition of a small protein, A

211 oryzae, Rhizoctonia solani, and Xanthomonas oryzae pv. oryzae) and several chemical elicitors.

212 he oryzae) and bacterial blight (Xanthomonas oryzae pv. oryzae).

213 susceptibility to M. oryzae and Xanthomonas oryzae pv. oryzae, hemibiotrophic pathogens, but enhance

214 rom the Gram-negative bacterium, Xanthomonas oryzae pv. oryzae, is a tyrosine sulfotransferase.

215 sed by the gamma-proteobacterium Xanthomonas oryzae pv. oryzae, which utilizes a group of type III TA

216 ays after infection of bacterium Xanthomonas oryzae pv. oryzae.

217 thXo2-dependent disease susceptibility to X. oryzae pv. oryzae.

218 led OsSWEET13 induction by 42 isolates of X. oryzae pv. oryzae.

219 pathogens Magnaporthe oryzae and Xanthomonas oryzae pv. oryzae.

220 to the Gram-negative bacterium, Xanthomonas oryzae pv. oryzae; NH1, the rice ortholog of NPR1, a key

221 e bacterial leaf streak pathogen Xanthomonas oryzae pv. oryzicola (Xoc) contains a homologous operon.

222 The rice bacterial pathogen Xanthomonas oryzae pv. oryzicola (Xoc) has been demonstrated to cont

223 e bacterial leaf streak pathogen Xanthomonas oryzae pv. oryzicola (Xoc).

224 Xanthomonas oryzae pv. oryzicola causes bacterial leaf streak of ric

225 that a native TAL effector from Xanthomonas oryzae pv. oryzicola drives expression of a target with

226 ce to the strains in the African clade of X. oryzae pv. oryzicola, representing the first dominant re

227 e to the infection of M. grisea, Xanthomonas oryzae pv.oryzae and rice stripe virus were analyzed.

228 derivative flg22(Xo) peptides of Xanthomonas oryzae pvs. oryzae (Xoo) and oryzicola (Xoc).

229 ior to elicitation with Rhizopus oligosporus/oryzae (R) was investigated for its potential to enhance

230 wo fungal CFI protein classes in Magnaporthe oryzae: Rbp35/CfI25 complex and Hrp1.

231 Lyophilized protease from A. oryzae reached 1251.60 U/g and yield of 155010.66 U/kg o

232 i such as the rice blast fungus (Magnaporthe oryzae) remains unclear.

233 the hemibiotrophic rice pathogen Magnaporthe oryzae requires plant defence suppression to facilitate

234 Here, we show that plant infection by M. oryzae requires two independent S-phase cell-cycle check

235 fection in the rice blast fungus Magnaporthe oryzae, requires very-long-chain fatty acids (VLCFAs), w

236 ingle mutants display modest but opposite M. oryzae resistance in the non-Piz-t background.

237 soy flour fermented with M. purpureus or A. oryzae, respectively.

238 xposure to lovastatin on the virulence of R. oryzae RESULTS: All statins had MICs of >64 microg/mL ag

239 Recent genome sequencing of Rhizopus oryzae revealed evidence of a whole-genome duplication e

240 with three major rice pathogens (Magnaporthe oryzae, Rhizoctonia solani, and Xanthomonas oryzae pv. o

241 knowledge deficiency in the mechanism of M. oryzae-rice interactions and underscore how effector-med

242 succinate) (PBS) and the lipase of Rhizopus oryzae (RoL), we detected complete hydrolysis of PBS thi

243 te fermentation (Bacillus subtilis, Rhizopus oryzae, Saccharomyces cerevisiae, Lactobacillus helvetic

244 Aspergillus oryzae showed stability at all pH values studied.

245 Here, we report that a M. oryzae sirtuin, MoSir2, plays an essential role in rice

246 The rice blast fungus Magnaporthe oryzae spreads in rice biotrophically early during infec

247 were expressed in TAL effector-deficient X. oryzae strain X11-5A, and assessed in 21 rice varieties.

248 llular nucleotide pools were perturbed in M. oryzae strains lacking NDK1 through targeted gene deleti

249 lations of the rice blast fungus Magnaporthe oryzae (syn: Pyricularia oryzae) into three major global

250 es, we enriched and cultivated anti-Rhizopus oryzae T cells from healthy individuals.

251 The roles of X. oryzae TAL effectors in diverse rice backgrounds, howeve

252 led two related non-LTR retrotransposons (M. oryzae Telomeric Retrotransposons or MoTeRs) inserted in

253 he presence of retrotransposons (Magnaporthe oryzae Telomeric Retrotransposons-MoTeRs) inserted in th

254 we show how, in the blast fungus Magnaporthe oryzae, terminating rice innate immunity requires a dyna

255 Magnaporthe oryzae, the causal agent of blast disease, is one of the

256 A mutant of Rhizopus oryzae, the most common cause of mucormycosis, with redu

257 In Magnaporthe oryzae, the Mst11-Mst7-Pmk1 MAP kinase pathway is essent

258 vo activity of posaconazole against Rhizopus oryzae, the Mucorales species most commonly associated w

259 urface signals are recognized by Magnaporthe oryzae to activate the Pmk1 MAP kinase that is essential

260 t, gene silencing of CEBiP in rice allows M. oryzae to cause rice blast disease in the absence of Slp

261 cose, and iron, augmenting the ability of R. oryzae to invade and subsequently damage endothelial cel

262 th anti-CotH Abs abolished the ability of R. oryzae to invade host cells and protected DKA mice from

263 Exposure of R. oryzae to statins at concentrations below their MICs dec

264 4 microg/mL against R. oryzae Exposure of R. oryzae to statins decreased germling formation, induced

265 ong telomeric repeat sequence of Aspergillus oryzae together with reverse-transcription-PCR and ident

266 bacterial pathogen Pseudomonas syringae pv. oryzae, transgenic EFR wheat lines had reduced lesion si

267 e bacterial pathogen, Xanthomonas oryzae pv. oryzae upon recognition of a small protein, Ax21, that i

268 me isolates of the rice pathogen Xanthomonas oryzae use truncated versions of TALEs (which we term in

269 ial Aspergillus species, such as Aspergillus oryzae, used in food fermentation and enzyme production,

270 vivo infections of rice demonstrate that M. oryzae virulence is enhanced, quite paradoxically, when

271 ion antitermination mechanism of Xanthomonas oryzae virus Xp10 protein p7, which binds host RNA polym

272 A. oryzae was further established as a platform for bio-con

273 ere the causative species was identified, R. oryzae was present in 85% of rhinocerebral forms compare

274 culans, Kluyveromyces lactis and Aspergillus oryzae) was analysed in detail, at 4 and 40 degrees C.

275 activity of beta-galactosidase (Aspergillus oryzae) was evaluated.

276 genesis of the rice blast fungus Magnaporthe oryzae, we identified MoGlo3 as an ArfGAP protein that i

277 tter understanding of redox regulation in M. oryzae, we measured the amount and redox potential of gl

278 Clinical strains of R. oryzae were exposed to lovastatin, atorvastatin, and sim

279 ed by heterologous expression in Aspergillus oryzae, whereas tropB and tropC were successfully expres

280 ment of pathogenic fungi such as Magnaporthe oryzae which causes rice blast.

281 gamma-proteobacterium Xanthomonas oryzae pv. oryzae, which utilizes a group of type III TAL (transcri

282 between the domesticated fungus Aspergillus oryzae, whose saccharification abilities humans have har

283 s essential for growth and development of M. oryzae with extensive downstream targets in addition to

284 lg22(Xo) peptides of Xanthomonas oryzae pvs. oryzae (Xoo) and oryzicola (Xoc).

285 d mesophyll pathogens Xanthomonas oryzae pv. oryzae (Xoo) and pv. oryzicola (Xoc) encode numerous sec

286 of the rice pathogen Xanthomonas oryzae pv. oryzae (Xoo) in a semi-isolated environment represented

287 e biotrophic pathogen Xanthomonas oryzae pv. oryzae (Xoo) produces a sulfated peptide named RaxX, whi

288 o a broad spectrum of Xanthomonas oryzae pv. oryzae (Xoo) races that cause bacterial blight disease.

289 erial blight pathogen Xanthomonas oryzae pv. oryzae (Xoo) that deliver the TAL effector AvrXa27.

290 red by the pathogenic Xanthomonas oryzae pv. oryzae (Xoo), in revealing the new function of two previ

291 The pathogen, Xanthomonas oryzae pv. oryzae (Xoo), secretes one or more of six known transcri

292 or confer immunity to Xanthomonas oryzae pv. oryzae (Xoo), the causal agent of bacterial leaf blight.

293 pathogenic bacterium Xanthomonas oryzae pv. oryzae (Xoo), which causes a vascular disease in rice, o

294 ht diseases caused by Xanthomonas oryzae pv. oryzae (Xoo).

295 rial blight caused by Xanthomonas oryzae pv. oryzae (Xoo).

296 rial blight caused by Xanthomonas oryzae pv. oryzae (Xoo).

297 yza sativa) caused by Xanthomonas oryzae pv. oryzae (Xoo).

298 athogens M. oryzae and Xanthomonas oryzae pv oryzae (Xoo).

299 ial blight, caused by Xanthomonas oryzae pv. oryzae (Xoo).

300 X (RaxX) produced by Xanthomonas oryzae pv. oryzae (Xoo).