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1 ight into cellular processes involved in the osmotic stress response.
2 n medium with salt, urea failed to stimulate osmotic stress response.
3 the dynamic protein-DNA interactions during osmotic stress response.
4 ates the DNA synthesis, independently of the osmotic stress responses.
5 r (AtWRKY15) modulates plant growth and salt/osmotic stress responses.
6 ABA-dependent and -independent pathways for osmotic stress responses.
7 (BON) proteins are critical for all of these osmotic stress responses.
8 n in osm-8 that constitutively activates the osmotic stress response also promoted xanthine stone acc
9 with distinct temporal patterns in our yeast osmotic stress response and axolotl limb regeneration ca
10 factor AtUAP1 is a negative regulator of the osmotic stress response and enhances the multi-Ub chain
11 e as a starting point for future research on osmotic stress response and help develop better strategi
13 In summary, LRRC8A is key to bidirectional osmotic stress responses and cell survival under hyperto
14 ndings indicate that NLR signaling represses osmotic stress responses and that BON proteins suppress
15 suggest that the ability of FRAP to mediate osmotic stress response (and glucose deprivation respons
21 ther, our real time-qPCR results showed that osmotic stress-response genes that are dependent on the
28 ctivated protein kinase pathway mediates the osmotic stress response in Saccharomyces cerevisiae, act
29 protein kinase (MAPK) pathway regulates the osmotic stress response in the yeast Saccharomyces cerev
30 We applied our method to extend pathways in osmotic stress response in yeast and identified several
34 assa osmosensing MAPK pathway, essential for osmotic stress responses, is a circadian output pathway
37 essary for the constitutive activity and the osmotic stress response of rat aldose reductase transcri
38 h distinct regions affect differentially the osmotic stress response (OSA) and meiotic recombination
39 etic selection for negative regulators of an osmotic stress response pathway called HOG, for high osm
40 e models of transcriptional dynamics for the osmotic stress response pathway in Saccharomyces cerevis
44 ance mutants, which genetically activate the osmotic stress response, strongly inhibited the formatio
45 ntial blunting in transcription of important osmotic stress response target genes and reduced cell vi
46 rounded by uncellularized endosperm mount an osmotic stress response that is connected to increased l
50 We also found that mutants with impaired osmotic stress response were better adapted to alternati
51 found that the defects of the bon mutants in osmotic stress responses were suppressed by mutations in
52 es a role for PIAL1 and 2 in salt stress and osmotic stress responses, whereas under standard conditi