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1 vating TRAF6-dependent signaling pathways in osteoclast progenitors.
2 sarcoma oncogene family, protein B (MAFB) in osteoclast progenitors.
3 oting mitochondrial apoptotic death of early osteoclast progenitors.
4 tly discovered a novel function for MDSCs as osteoclast progenitors.
5 identify the quiescent PPARgamma(+) cells as osteoclast progenitors.
6 F-kappaB and JNK activation in RANKL-treated osteoclast progenitors.
7 essed by transfected cell lines and purified osteoclast progenitors.
8 ctive metabolite of vitamin A, on periosteal osteoclast progenitors.
10 ly expressed by isolated bone marrow-derived osteoclast progenitors and by mature osteoclasts in vivo
11 steoclastogenesis both directly by acting on osteoclast progenitors and indirectly by increasing expr
12 ed bone formation, but increased bone marrow osteoclast progenitors and osteoclast numbers on bone su
13 enhanced expression of genes associated with osteoclast progenitors and precursors in Notch2(tm1.1Eca
14 lity that estrogen exerts a direct effect on osteoclast progenitors, and does so through the estrogen
15 calcium concentrations, increased committed osteoclast progenitors, and mature osteoclasts as well a
18 ings not only identify the long-sought-after osteoclast progenitors but also establish unprecedented
19 owever, the role of B cells is not to act as osteoclast progenitors but may be to act as osteoclast s
22 We observed a synergistic potentiation of osteoclast progenitor cell differentiation and formation
23 protein expression of several macrophage and osteoclast progenitor cell markers, such as macrophage c
24 flammatory signals led to the recruitment of osteoclast progenitor cell potential from ex vivo-isolat
25 mediated by enhancing expression of RANK in osteoclast progenitor cells and by upregulating secretio
26 f Rac1 restored the pathological increase in osteoclast progenitor cells in Nf1+/- mice and was suffi
28 indings, in vitro experiments using RAW264.7 osteoclast progenitor cells showed that TRPV2 mRNA was i
31 examine the effects of estrogen loss at the osteoclast progenitor colony forming unit-granulocyte ma
32 unique cell surface phenotype of clonogenic osteoclast progenitors (colony-forming unit-osteoclast [
33 that ERalpha-mediated estrogen signaling in osteoclast progenitors decreases "oxidative phosphorylat
36 dies demonstrated that B cells do not act as osteoclast progenitors in estrogen-replete or estrogen-d
37 ulation ~3 nM) on the numbers of macrophages/osteoclast progenitors in the bone cell cultures, as ass
38 r primary M-MuLV infection of osteoclasts or osteoclast progenitors in the bone marrow, and they sugg
39 he bone marrow and an expansion of monocytes/osteoclast progenitors in the periphery, resulting in ag
41 The signaling pathway activated by WNT16 in osteoclast progenitors is noncanonical, whereas the path
45 of clusters expressing genes associated with osteoclast progenitors, osteoclast precursors, and matur
46 urface, but display a normal distribution of osteoclast progenitor populations in the bone marrow and
48 rescent protein) reporter mice, we show that osteoclast progenitors reside specifically in the PPARga
51 y support the contention that in addition to osteoclast progenitors such as CFU-GM, earlier hematopoi
52 mulating factor 1 (CSF1) is known to promote osteoclast progenitor survival, but its roles in osteocl
53 aired osteoblast mineralization and enhanced osteoclast-progenitor survival, leading to increased ost
54 g tumor progression, in this case as a novel osteoclast progenitor that specifically drives bone meta
55 hat PKD activity promotes differentiation of osteoclast progenitors through increased expression of D
56 dy demonstrates that irisin acts directly on osteoclast progenitors to increase differentiation and p
58 eased osteoclast formation was observed when osteoclast progenitors were cocultured with oim/oim-deri