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1 (human transferrin, human serum albumin, and ovalbumin).
2 H2 responses after allergic sensitization to ovalbumin.
3 e of Galbeta(1-->4)GlcNAc ligands on chicken ovalbumin.
4 pidly after sensitization and challenge with ovalbumin.
5 ollowed by injection of 2,4,6-trinitrophenyl-ovalbumin.
6 from mice sensitized to native or aggregated ovalbumin.
7 rcutaneous challenge by the protein allergen ovalbumin.
8 airway epithelium were tolerized to inhaled ovalbumin.
9 deled in mice sensitized and challenged with ovalbumin.
10 ngth) produced a Th2-biased response against ovalbumin.
11 nternalized by dendritic cells and delivered ovalbumin.
12 ntraperitoneally toward the primary antigen, ovalbumin.
13 ild-type (WT) mice sensitized and exposed to ovalbumin.
14 etic) system and induced allergic asthma via ovalbumin.
15 d desensitization to the model food allergen ovalbumin.
20 3beta) sequences following immunization with ovalbumin administered with complete Freund's adjuvant (
21 eally injected into ovalbumin-sensitized and ovalbumin aerosol-challenged leukotriene C4 synthase-def
22 accurately identify a CD4 T cell response to ovalbumin against a background response to the complex m
23 als were exposed repeatedly to aspiration of ovalbumin alone or together with environmental adjuvants
25 problem by co-encapsulating a model antigen ovalbumin along with TLR9 and STING ligands within lipos
26 model of allergic asthma was developed with ovalbumin-alum in female Cd39 wild type (Cd39(+/+) ) and
27 Mice were sensitized with intraperitoneal ovalbumin-alum, followed by intranasal challenge with ov
29 erformed intranasally or epicutaneously with ovalbumin and a secondary antigen, keyhole limpet hemocy
31 nd exposed to two separate allergic stimuli (ovalbumin and Aspergillus extract), genetic removal of M
33 of glycopeptides from fetuin, glycophorin A, ovalbumin and gp120 tryptic digests were used to build a
35 ferent murine models of experimental asthma (ovalbumin and house dust mite); miRNAs deregulated in bo
40 LDI-MS identified proteolytic fragments from ovalbumin and lysozyme, exhibiting varied resistance to
44 tion against the anaphylaxis associated with ovalbumin and peanut allergy and affects the epigenome o
48 We hypothesize that ACC is stabilized by ovalbumin and/or lysozyme or additional vesicle proteins
50 responses were determined in the setting of ovalbumin- and house dust mite-induced lung inflammation
51 ngly, serpins are homologs of the well-known ovalbumin antigen and are associated with autoimmunity.
52 ) anti-OVA TCR transgenic mice sensitized to ovalbumin antigen, B cell depletion also impaired allosp
53 d standards from an Abl kinase sensor and an ovalbumin antigenic peptide were then employed to identi
55 s shown by both the astringency method (with ovalbumin as a precipitant) and the tannins determinatio
58 coding a CD8(+) T cell receptor epitope from ovalbumin, as well as a viral peptide-specific major his
59 1, 7a, 17, and 23 (10 mg/kg) protected in an ovalbumin/Aspergillus mouse asthma model, and PEG conjug
61 yptophan depletion in myofibrillar proteins, ovalbumin, beta-lactoglobulin, soy protein and human ser
62 rates of lysozyme, cytochrome c, myoglobin, ovalbumin, bovine serum albumin, and etanercept were inv
64 revealed that calcium inhibited glycation of ovalbumin by a mixed non-competitive mechanism in both d
65 A knockout mice (P < .005) and in vivo after ovalbumin challenge (P < .05), higher levels of apoptosi
69 mulin plasmids were detected in the lungs of ovalbumin-challenged asthmatic mice up to 27days after a
71 Approximately 90% of initially internalized ovalbumin-conjugated micelles were retained in cells aft
74 umin ratio, yolk index, air cell size, and S-ovalbumin content of UC were significantly the most supe
75 systemically applied (64)Cu-labeled chicken ovalbumin (cOVA)-TCR transgenic T cells into the pulmona
76 s on proteins (as caseins, whey proteins and ovalbumin) declared in the label of several sport nutrit
80 otein effects with bovine serum albumin, (S-)ovalbumin, egg white, whole egg, defatted egg yolk, whea
81 sters, which were mainly composed of chicken ovalbumin-encapsulated AuNCs, can recognize ricin B beca
87 an in vivo mouse model of nasal tolerance to ovalbumin, F. prausnitzii A2-165 enhanced ovalbumin-spec
88 g of CD103(+) dendritic cells (DCs) from the ovalbumin-fed DAF(-/-) mice showed impaired expression o
92 n of non-entrapped drug present) or protein (ovalbumin >90% reduction of OVA present) and organic sol
93 sing the HOD antigen (hen egg lysozyme [HEL]-ovalbumin-human transmembrane Duffy(b)) were transfused
94 irments included selective elevation in anti-ovalbumin IgE in plasma following immunization, reduced
96 bumin-dependent colitis in mice that express ovalbumin in biliary epithelia (ASBT-OVA mice) and cross
100 Unlike oral feeding or ocular injection of ovalbumin in wild-type (WT) mice, which induced dominant
102 el and a steroid-insensitive model combining ovalbumin-induced AAD with C muridarum (Cmu) respiratory
105 ay disease, and prevented the development of ovalbumin-induced airway hyperreactivity, eosinophilia,
106 e experimental models of house dust mite- or ovalbumin-induced airway inflammation and influenza A vi
110 n-1) PSCs was studied in an in vivo model of ovalbumin-induced allergic inflammation and an in vitro
114 rway inflammation was assessed in vivo in an ovalbumin-induced asthma model by using invasive spirome
115 isothiocyanate contact hypersensitivity and ovalbumin-induced dermatitis as models for TH2-mediated
116 D(4) blocked LTC(4)-mediated potentiation of ovalbumin-induced eosinophilic inflammation; recruitment
119 Haemophilus respiratory infection-mediated, ovalbumin-induced severe, steroid-resistant allergic air
120 ytial virus respiratory tract infections and ovalbumin-induced, severe, steroid-insensitive allergic
121 These nanoparticles efficiently delivered ovalbumin into the cytosol of dendritic cells and demons
123 knockout mice with EpCAM-deficient LCs with ovalbumin led to increased induction of type 2 Ova-speci
125 of activation of CD8+ T cells in an in vitro ovalbumin model and illustrated that acid-labile polymer
128 control vs sensitized and/or challenged with ovalbumin, n=3-5/group) 6 hour, 24 hour, and 48 hour aft
129 etween heat-induced aggregates of ovalbumin (ovalbumin nanoparticles, OVAn) and linoleic acid (LA) we
131 wed by five oral dosing with purified AHC or ovalbumin on alternate days and continued on HFD for ano
134 this murine model was exposed to allergens (ovalbumin or house dust mite extract) to decipher in viv
135 RAs) and assessed their abilities to reverse ovalbumin or peanut allergies in mouse models, as well a
136 C-RAs to half-maximally sensitized mice with ovalbumin or peanut allergy reduced anaphylactic respons
137 ols for food allergy induction, using either ovalbumin or peanut extract as allergens for sensitizati
138 ction when it was in formulation with either ovalbumin or recombinant hemagglutinin B (rHagB) antigen
139 fluenza and, once recovered, subjected to an ovalbumin- or house dust mite-induced experimental asthm
140 lly block nociceptors, substantially reduced ovalbumin- or house-dust-mite-induced airway inflammatio
141 I cells and cognate peptide stimulation with ovalbumin (OVA) 323-339 plus the inflammatory adjuvant,
145 aneously or immunized intraperitoneally with ovalbumin (OVA) and challenged intranasally with antigen
147 SIGIRR(-/-) C57BL/6 mice were sensitized to ovalbumin (OVA) and challenged with OVA aerosol to induc
148 -deficient (miR-155(-/-) ) mice were used in ovalbumin (OVA) and house dust mite (HDM) models of AAI.
149 xpression and activity were evaluated in the ovalbumin (OVA) and house dust mite (HDM) murine models.
151 dine or activated using dimaprit in both the ovalbumin (OVA) and house dust mite extract (HDM) murine
153 icroencapsulated via complex coacervation of ovalbumin (OVA) and sodium alginate (AL), and the microc
154 sh) mice were epicutaneously sensitized with ovalbumin (OVA) and then challenged orally with OVA.
155 eceptor alpha(-/-) mice were sensitized with ovalbumin (OVA) and then infected with M pneumoniae or S
161 evidence in a transgenic mouse system using ovalbumin (OVA) as a model surrogate TRA that the de nov
163 ose on anaphylactic symptoms induced by oral ovalbumin (OVA) challenge in sensitized mice were invest
165 ng a heterologous CD4(+) T cell epitope from ovalbumin (OVA) consisting of OVA residues 323 to 339 (O
166 e epicutaneously sensitized female mice with ovalbumin (OVA) followed by epicutaneous sensitization a
169 6 mice by intraperitoneal sensitization with ovalbumin (OVA) in combination with intranasal (i.n) exp
170 04, on allergic lung inflammation induced by ovalbumin (OVA) in mice and by house dust mite (HDM) in
172 tes uptake and processing of soluble antigen ovalbumin (Ova) in primary human macrophages and dendrit
177 fractionally coated with a powder mixture of ovalbumin (OVA) model allergen, CpG, and 1,25-dihydroxyv
178 c T cell receptor (TCR) with specificity for ovalbumin (OVA) on CD4(+)-T cells and cMy-mOVA mice expr
180 to wild-type recipient mice, challenged with ovalbumin (OVA) or house dust mite (HDM), and accessed f
183 e that received an irrelevant immunodominant ovalbumin (OVA) peptide, OVA323-339, mice that received
184 iAD and extrinsic AD, eAD), and established ovalbumin (OVA) percutaneous sensitized AD model and pas
185 rformed by applying a skin patch soaked with ovalbumin (OVA) plus CpG, and its suppressor activity wa
187 oli BL21 and E coli BL21_HTW were gavaged to ovalbumin (OVA) sensitized and challenged mice to invest
191 y, amyloids assembled from the model protein ovalbumin (OVA) were found to release the precursor prot
194 N microparticles co-loaded with the antigen, ovalbumin (OVA), and adjuvant, CpG 1826, (PNSN(OVA + CpG
195 /-) mice were sensitized and challenged with ovalbumin (OVA), and the development of AAD was ascertai
197 uce allergic responses to a second allergen, ovalbumin (OVA), in mice sensitized dually to OVA and Fe
198 a foreign, crosslinked protein, chicken egg ovalbumin (OVA), in the absence of an external adjuvant.
200 ) was used to produce exosomes together with ovalbumin (OVA), the resulting Dexo vaccine strongly sti
201 the dose and penetration of a model vaccine, Ovalbumin (OVA), to depths of 500mum into porcine skin.
205 XCL1 (KC) expression were also determined in ovalbumin (OVA)-challenged wild-type female recipient mi
208 to T and B cell responses of unimmunized and ovalbumin (OVA)-immunized BALB/c mice, and furthermore,
210 s was assessed in whole lungs from mice with ovalbumin (OVA)-induced allergic airway inflammation (AA
212 to determine the effect of Ptx3 deletion on ovalbumin (OVA)-induced allergic inflammation in a murin
214 nt susceptibility to pneumococcal infection, ovalbumin (OVA)-induced allergic lung inflammation (ALI)
224 he draining lymph nodes of tape-stripped and ovalbumin (OVA)-sensitized skin were examined for their
235 o-mid mass proteins (Trypsin Inhibitor (TI); Ovalbumin (OVA); Bovine Serum Albumin (BSA)), we observe
236 CAF09 and a surface-adsorbed model antigen [ovalbumin (OVA)] showed that a significantly larger frac
237 ntracellular uptake of model cancer antigen (ovalbumin, OVA) by THP-1-differentiated macrophage-like
239 formation between heat-induced aggregates of ovalbumin (ovalbumin nanoparticles, OVAn) and linoleic a
240 ntioxidant peptides, which were derived from ovalbumin, ovotransferrin and cystatin were isolated fro
241 nant MPO peptide (MPO(409-428)) or a control ovalbumin peptide (OVA(323-339)) to splenocytes and indu
243 O4@Al2O3 MNPs are functionalized with pigeon ovalbumin (POA), which contains Gal-alpha(1-->4)-Gal-bet
244 ctron tomography to measure the structure of ovalbumin precipitate particles salted out with ammonium
245 fic antibodies and enhanced proliferation of ovalbumin-reactive T cells associated with increased acc
249 n and challenge of mIgE-ITT-mutant mice with ovalbumin resulted in diminished serum IgE titers and re
250 S analysis of N-linked glycans released from ovalbumin revealed that several mobility features previo
252 nanostructures comprised of crystallites of ovalbumin self-assemble into a well-defined bicontinuous
253 ype (WT) and EP2(-/-) mice were subjected to ovalbumin sensitization and acute airway challenge.
257 ex vivo, were intratracheally injected into ovalbumin-sensitized and ovalbumin aerosol-challenged le
258 ent of airway epithelial barrier function in ovalbumin-sensitized control and prenatally stressed off
259 of lungs from prenatally stressed, non-BMC, ovalbumin-sensitized females unveiled a deregulated expr
264 involved in asthmatic-like attacks using the ovalbumin-sensitized murine model of the disease, and id
266 se, cholangitis developed in mice expressing ovalbumin simultaneously in biliary epithelia and entero
268 2 but significantly less IL-10 production by ovalbumin-specific CD4(+) T cells, resulting in prolifer
270 A) showed significant enhancement in antigen ovalbumin-specific immunoglobulin responses compared to
272 months of age also had reduced frequency of ovalbumin-specific regulatory B cells compared to egg-to
275 e analyzed by flow cytometry for presence of ovalbumin-specific regulatory T cells, using activation
276 nce to egg were associated with expansion of ovalbumin-specific T and B regulatory cells, which may b
277 to ovalbumin, F. prausnitzii A2-165 enhanced ovalbumin-specific T cell proliferation and reduced the
278 rausnitzii A2-165 stimulated BMDCs increased ovalbumin-specific T cell proliferation and reduced the
279 s examined by assessment of proliferation of ovalbumin-specific T cells in rat insulin promoter-membr
281 by co-cultures of DCs from infected mice and ovalbumin-specific, MHC class II-restricted alpha/beta (
283 This work illustrates the links between ovalbumin structure after heating and allergenicity pote
285 d tolerance induced by inhalation of soluble ovalbumin, suppressing the normal generation of forkhead
287 cells in rat insulin promoter-membrane-bound ovalbumin transgenic mice after sham or IRI procedures.
289 the present work, we determined that murine ovalbumin-transgenic (OT-1) CD8(+) T cells recognize the
290 ia hepatic nuclear factor 4alpha and chicken ovalbumin upstream promoter transcription factor I/II.
291 n partially through up-regulation of chicken ovalbumin upstream promoter transcription factor II (COU
292 ale mouse embryos lacking Coup-tfII (chicken ovalbumin upstream promoter transcription factor II) in
296 lated by an orphan nuclear receptor, chicken ovalbumin upstream promoter-transcription factor II (COU
299 nulation capacities of native and aggregated ovalbumin were measured with sera from egg-allergic chil