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1 r prolonged labor was a secondary pathway to overweight.
2 ect the risks of preterm birth and childhood overweight.
3 and non-transmitted maternal GRS with child overweight.
4 mall size for gestational age, and childhood overweight.
5 d 120,000 (95% UI 34,000-240,000) fewer with overweight.
6 es were present in 28%, including asthma and overweight.
7 aternal BMI, being weaker among mothers with overweight.
8 cations and seems to influence later risk of overweight.
9 ated with parental body mass index (BMI) and overweight.
10 ive to separate programming for stunting and overweight.
11 nal age, but with a higher risk of childhood overweight.
12 is associated with increased food intake and overweight.
13 maternal smoking, with the risk of childhood overweight.
15 mong obese adolescents (270/100,000) than of overweight (179/100,000), normal weight (154/100,000), a
16 ory: underweight (31%), normal weight (24%), overweight (19%), obese class I (16%), obese class II (1
17 y the square of the height in meters], <25), overweight (25 to <30), moderate obesity (30 to <35), an
18 egorized into normal weight (20-24.9 kg/m2), overweight (25-29.9 kg/m2), and obesity (30+ kg/m2).
20 : 48%; underweight: 30%; normal weight: 23%; overweight: 33%; obesity: 14%; predominant cancer sites:
21 (57%), were at nutritional risk (57%), were overweight (53%), had visceral obesity (62%), had a norm
22 ecause the majority of Gulf War Veterans are overweight, a second objective was to determine if a hig
23 ower hazard of death among ever-smokers (for overweight, adjusted hazard ratio (aHR) = 0.56, 95% conf
24 nterval [CI] 1.22-1.83; P < .05), the OR for overweight adolescents was 1.42 (95% CI 1.08-1.92; P < .
25 take, weight loss, and metabolic outcomes in overweight adults who could choose to follow Mediterrane
26 CI: 0.48, 0.92), but never-smokers did not (overweight, aHR = 1.41, 95% CI: 0.66, 3.03; obesity, aHR
27 the major direct cause is rapid increases in overweight allows identifying selected crucial drivers a
28 esarean section and first pregnancy to child overweight and atopy share many common mediators of the
33 ptoms, post-traumatic stress disorder, being overweight and negatively loaded by pain self-efficacy a
35 significantly higher in obese, compared with overweight and normal-weight adolescents (23.05 (8.79) v
36 ants relied less on model-based control than overweight and normal-weight participants, with no diffe
40 ms were to determine, in vitamin D-deficient overweight and obese children, whether supplementation w
41 ensitivity, which are generally disturbed in overweight and obese individuals, may improve by increas
47 ssociation was stronger among those who were overweight and obese than among those of normal weight (
50 weekdays was associated with higher odds of overweight and obesity [Odds ratio (95% confidence inter
51 countries develop, and project the burden of overweight and obesity among the poor for 103 countries.
59 emain high in many regions, whereas those of overweight and obesity have increased in all age and soc
62 erence in sleep duration was associated with overweight and obesity in females, but not in males.
64 es in sleep timing, were not associated with overweight and obesity in the overall population, althou
69 reported data, possible reverse causality of overweight and obesity on wealth, and the lack of physic
71 ght to analyze associations between maternal overweight and obesity severity and rates of complex and
73 aiming to reduce the prevalence of childhood overweight and obesity should focus on maternal weight s
74 nships between sleep duration and timing and overweight and obesity status in adolescents, with a spe
79 ous manifestation of both undernutrition and overweight and obesity, affects most low-income and midd
80 iet which increases adipose tissue favouring overweight and obesity, and housed in either an enriched
81 tive interventions and treatment options for overweight and obesity, and the medical complications an
82 livery, sex, and birth weight with childhood overweight and obesity, but we observed statistically si
83 fiable risk factors for CVD include smoking, overweight and obesity, diabetes, elevated blood pressur
85 rt of successful prevention and treatment of overweight and obesity, which should be further tested i
91 affect the intestinal microbiome, including overweight and obesity; physical activity; and dietary i
92 trition and mental health, using measures of overweight and suicidal ideation and planning which some
93 Urban residents had higher odds of being overweight and/or obese (OR: 1.89, 95% CI: 1.62-2.20) an
94 ds were associated with higher odds of being overweight and/or obese (P-trend < 0.001) and lower odds
95 verweight and/or obesity as 49% of them were overweight and/or obese and nearly 39% at the lowest wea
96 hest wealth quintile were more vulnerable to overweight and/or obesity as 49% of them were overweight
97 d socioeconomic variation in underweight and overweight and/or obesity prevalence in the country, adj
99 alth risks (tobacco smoking, binge drinking, overweight, and anaemia); and social determinants of hea
100 ition, stunting, micronutrient deficiencies, overweight, and children not reaching their developmenta
104 ood glucose, raised blood pressure, smoking, overweight, and obesity) at the household, community, di
105 19.5% among individuals with normal weight, overweight, and obesity, respectively (P(interaction) <
106 e Diet Index (SDI), and the risk of obesity, overweight, and weight gain in French adults, with a pro
108 ividuals who have a normal weight or who are overweight are at high risk if they have an excess of vi
109 should be used for the in vivo situation of overweight-associated type 2 diabetes reflecting both th
110 eight from childhood to adulthood and having overweight at both ages were associated with higher risk
111 Kidney recipients, younger children, those overweight at transplant, and those with higher cumulati
112 h premature menopause were more likely to be overweight, Black, have >=20 pack years of smoking, hist
113 gories: normal weight (BMI 18.5-23.9 kg/m2), overweight (BMI 24.0-27.9 kg/m2), and obese (BMI >= 28 k
114 MI < 22.5), normal weight (BMI = 22.5-24.9), overweight (BMI = 25-29.9), and obesity (BMI >= 30)-and
117 o 30 kg/m2) is highest among the poor, while overweight (body mass index greater than or equal to 25
118 RSs, based on adult BMI, contribute to child overweight, but in overweight mothers other genetic and
120 ndency of NR oral availability and safety in overweight, but otherwise healthy men and women, an 8-we
123 categories than to lower categories, whereas overweight children had similar rates of transition to t
124 he following for underweight, normal weight, overweight, class 1 obesity, class 2 obesity, and class
126 ted GRSs (SD-units) increased odds for child overweight equally [OR: 1.30 (95% CI 1.16; 1.46) and 1.3
127 r susceptibility to infectious diseases, and overweight, especially in terms of increased risk of add
128 included maternal prepregnancy and paternal overweight, excessive gestational weight gain, raised fa
129 In a subset of 13,160 women, development of overweight from childhood to adulthood and having overwe
130 rage childhood BMI values and development of overweight from childhood to adulthood were associated w
132 e normal weight (5-84th BMI percentile), 15% overweight (>=85-94th BMI percentile), and 35% obese (>=
133 e following: having a father with T2D, being overweight, having higher blood pressure and higher leve
134 e ratio at 3 months was the dominant path to overweight; higher Enterobacteriaceae/Bacteroidaceae rat
135 MiR-26a is reduced in serum exosomes of overweight humans and is inversely correlated with clini
136 obesity susceptibility phenotypes, and that overweight humans exhibit increased striatal Rgs4 protei
137 We aimed to examine whether parents being overweight in childhood, adolescence, or adulthood is as
138 ntervention that addresses both stunting and overweight in children aged 4 years and older by providi
139 and non-transmitted genetic contributions to overweight in children from the Danish National Birth Co
142 ignificant effect of fathers' onset of being overweight in puberty on offspring's asthma without nasa
143 ct was seen with HDI, with lowest chances of overweight in the lowest tertile compared with the highe
144 obesity by 4.6% (95% UI 1.4%-9.5%) and with overweight (including obesity) by 3.6% (95% UI 1.1%-7.4%
148 re to early undernutrition followed by later overweight increases the risk of non-communicable diseas
149 95% CI 1.12-1.68) in diabetes odds among non-overweight individuals (BMI < 25 kg/m2) without a family
150 r reductions in body weight were observed in overweight individuals and those with diabetes and metab
152 d the lowest HR of all-cause mortality among overweight individuals with high mMED (HR 0.94; 95% CI 0
153 disease, CVD, and all-cause mortality while overweight individuals, despite a higher odds of CAC, sh
155 people exhibit probability distortion (they overweight low probabilities), loss aversion (losses loo
160 BMI, contribute to child overweight, but in overweight mothers other genetic and environmental facto
161 in a wide BMI range [normal-weight (n = 31), overweight (n = 29), obese (n = 30)] performed a sequent
162 ers (N = 106) and non-responders (N = 97) of overweight non-diabetic middle-aged Danes to two earlier
163 k randomized placebo-controlled trial in 126 overweight, non-insulin sensitive (HOMA-IR >=1.30), Chin
165 mal weight (NW: 18.5-24.9 kg/m2, n = 88) and overweight/obese (OW: 25-35 kg/m2, n = 86) women between
168 postpartum, 10 metabolites differed between overweight/obese and lean groups with nominal P < 0.05,
169 om Provinces 2, and 7 were less likely to be overweight/obese and more likely to be underweight (refe
170 o-controlled, single ascending-dose study in overweight/obese human participants, subcutaneous BFKB84
171 I (>90% with successful replication in 1,584 overweight/obese individuals from a community-based coho
172 bstructive pulmonary disease (COPD), whereby overweight/obese individuals have improved survival, has
174 Compared with normal-weight participants, overweight/obese participants had lower hazard of death
176 risk among certain subgroups (eg firstborns, overweight/obese pre-pregnancy BMI), but associations we
180 utritional intervention was conducted in 305 overweight/obese volunteers involving 2 energy-restricte
184 y tackle both undernutrition and problems of overweight, obesity, and diet-related non-communicable d
186 ional migrants had roughly twice the risk of overweight/obesity (adjusted mean risk = 51.7% versus 23
187 migrants had significantly higher levels of overweight/obesity (adjusted mean risk = 51.7% versus 37
190 levels (except ALT) and increased risks for overweight/obesity (odds ratio, 6.41 [95% CI, 2.95-15.56
192 and transcriptomic profiles from 11 men with overweight/obesity after TRF (8 h day(-1)) and extended
193 quartile were 1.93 (95% CI: 1.69, 2.20) for overweight/obesity and 2.07 (95% CI: 1.74, 2.46) for cen
194 Other factors, such as the prevalence of overweight/obesity and diabetes, have increased consider
195 e context of cancer, the association between overweight/obesity and mortality is complicated by conco
196 udies have investigated associations between overweight/obesity and risk of developing rheumatoid art
198 also observed an interconnectedness between overweight/obesity and suicide ideation with planning am
199 The overall prevalence of underweight, and overweight/obesity at start of CPD was 8.9% and 19.7%, r
200 t were positively associated with adolescent overweight/obesity but not with suicidal ideation with p
201 ssie-FIT, a weight-loss program for men with overweight/obesity delivered in Australian Football Leag
202 % CI 59% to 29%, p < 0.001) lower chances of overweight/obesity for girls and boys, respectively.
203 across their full ranges, with the risks of overweight/obesity in early (2.0-5.0 years), mid (5.0-10
204 total of 55,295 adolescents had a measure of overweight/obesity status, and 59,061 adolescents report
207 ot been previously explored in children with overweight/obesity who present a different white matter
208 otal of 28.8% of boys and 28.1% of girls had overweight/obesity, and 7.5% of boys and 17.5% of girls
209 t increases in central adiposity and risk of overweight/obesity, but there was no consistent pattern
210 nvironmental adversities, including maternal overweight/obesity, diabetes/hypertensive disorders, or
212 term recordings in a cohort of children with overweight/obesity, young adults and middle-age adults.
216 white matter microstructure in children with overweight/obesity; those findings indicate that the ass
217 s associated with a higher risk of childhood overweight (odds ratio [OR] 1.17 [95% CI 1.02-1.35], P v
218 usted odds of CAC >0 were increased in those overweight (odds ratio, 1.13 [95% CI, 1.1-1.2]) and obes
219 with increasing BMI, that was evident in the overweight (odds ratio, 1.39; 95% CI 1.13 to 1.71; crude
220 its) had a strong association with childhood overweight [Odds ratio (OR): 2.01 (95% confidence interv
221 disrupted probabilistic reasoning, namely to overweighting of sensory evidence, in patients with SCZ.
224 alysis, we found that 56.5% of all SIVH were overweight or had obesity, 2.4% reported hypertension, 1
226 body mass index < 25) than in those who were overweight or obese (body mass index >= 25), at 99.2% ve
227 = 0.42) and proportion of patients who were overweight or obese (OR 1.3; 95% CI 0.70, 2.51; p = 0.34
228 risk of EOCRC, whereas aspirin use and being overweight or obese (relative to normal BMI) were signif
229 the whole, the subjects in both groups were overweight or obese and insulin resistant; they displaye
231 ional design, we investigated postmenopausal overweight or obese female subjects who either underwent
232 ng physically active, were less likely to be overweight or obese in adolescence and early adulthood.
234 e to determine whether for young women being overweight or obese is associated with a higher risk of
235 lementation of 1000 mg/d for 6 wk in healthy overweight or obese men and women increased skeletal mus
237 etyl-CoA, is highly induced in the kidney of overweight or obese patients with CKD and ob/ob BTBR mic
241 th the increasing numbers of persons who are overweight or obese, higher rates of cardiomyopathy can
245 wk intervention in adults (18-60 y old) with overweight or obesity (body mass index 25-40 kg/m2).
247 specimens in the NHANES 2014 and had data on overweight or obesity [body mass index (kg/m2) >=25] and
249 2%, 12%; P for trend = 0.001) lower risk of overweight or obesity during adolescence and young adult
251 Additionally, despite the lower risk of overweight or obesity for current smokers, normal weight
252 nancy with body mass index (BMI) and risk of overweight or obesity of grandchildren during adolescenc
253 ed with reduced-fat (0.1-2%) milk, the OR of overweight or obesity was 0.61 (95% CI: 0.52, 0.72; P <
254 nship between smoking amount and the risk of overweight or obesity was U-shaped, and the trends were
259 ternal smoking was associated with childhood overweight (OR 1.21 [95% CI 1.16-1.27], P value < 0.001)
260 2.07-2.23], P value < 0.001), and childhood overweight (OR 1.42 [95% CI 1.35-1.48], P value < 0.001)
261 ransmitted GRS was not associated with child overweight [OR 0.98 (95% CI 0.88; 1.10)] suggesting no s
262 n patients with cardiovascular risk factors (overweight [OR, 2.6, P=0.023], diabetes mellitus type II
265 yzed: AAG was associated with higher risk of overweight (p = 0.008), hypertriglyceridemia (p = 0.040)
266 mass index, increasing from 3.90% in lean or overweight participants to 17.73% in obese participants.
269 adipose tissue samples were obtained from 17 overweight patients undergoing elective abdominal surger
271 data for analyses, and we excluded 119 (26%) overweight patients, leaving a final cohort of 256 (68%)
272 by nephrectomy; after exclusion of 59 (39%) overweight patients, our final cohort consisted of 93 (6
274 lity was lower in both the normal-weight and overweight populations than in the underweight populatio
277 cate that as countries develop economically, overweight prevalence increased substantially among the
279 ndex (z-score; 1.72 aHR; 95% CI: 1.39-2.14), overweight status (2.63 HR; 95% CI: 1.71-4.04), and youn
280 overweight status and offspring's childhood overweight status (odds ratio, 2.23 [95% CI, 1.45-3.42]
281 and age 30 years with offspring's childhood overweight status (potential mediator) and offspring's a
283 between both fathers' and mothers' childhood overweight status and offspring's childhood overweight s
287 identified greater body weight reductions in overweight subjects (1.88%, p < 0.0001) and in females (
288 22%; beta = 0.12; 95% CI: (-0.03, 0.27)) and overweight subjects (27%; beta = 0.19; 95% CI: (-0.004,
290 pants in Q1 had a higher risk of obesity and overweight than participants in Q5 (HR comparing Q1 with
291 and 1.27 (95% CI, 1.01 to 1.60; P = .04) for overweight versus lean groups and were 1.32 (95% CI, 1.0
292 r 16 comparisons (obese vs normal weight and overweight vs normal weight for 8 outcomes), these findi
293 ne, a higher percentage of participants with overweight was observed in the first SDI Q, reflecting t
297 e was a lower risk of all-cause mortality in overweight women (hazard ratio, 0.79 [95% CI, 0.63-0.98]
299 = 0.12) and FBG (R(2) = 0.07, P = 0.06); (4) overweight young adults had higher urinary mRNA levels o