ライフサイエンスコーパス: oxidative

1 E, protected ROS-exposed Caco2 cells against oxidative (78%) and cellular (90%) damage at a 3 ug/L co

2 Oxidative addition and reductive elimination are definin

3 ved by exploiting the lower barrier for C-CN oxidative addition and reductive elimination at benzylic

4 o assist catalysts and show that the rate of oxidative addition between palladium catalysts and alkyl

5 bone of a bisphosphine ligand on the rate of oxidative addition of bromobenzene to a ligand-coordinat

6 nd but also the ambiphilic nature allows for oxidative addition of Si-H, N-H, and even C-C bonds at t

7 This consists of oxidative addition of the aryl halide (ArX) to the Pd(0)

8 results chart a path to actively controlling oxidative addition on an Au surface using an applied bia

9 irst step in several catalytic cycles is P-H oxidative addition to yield intermediate metal hydride c

10 Pd-catalyzed sequences involving oxidative addition, cyclization, and termination through

11 nd of elementary steps: radical addition-SET-oxidative addition-reductive elimination.

12 SOD3 preserves HA homeostasis by inhibiting oxidative and enzymatic hyaluronidase-mediated HA breakd

13 hy young men is not enough to confer a major oxidative and inflammatory burden on the brain, but it d

14 glucose/lactate exchange, and biomarkers of oxidative and inflammatory stress were also measured.

15 scade C-H activation; regioselective [4 + 2] oxidative annulation; and lactonization of aromatic acid

16 ccess highly oxidized diterpenes by a hybrid oxidative approach that strategically combines chemical

17 Herein, we report a rhodium-catalyzed oxidative arene alkenylation from arenes and styrenes to

18 shot measurements of a consistent measure of oxidative balance can inform our understanding of differ

19 vidual and temporal variation in measures of oxidative balance were substantial, we found high consis

20 Yet, examination of changes in oxidative balance within wild animals across time, space

21 y, we use the cases of nitrile hydration and oxidative biotransformation reactions to show that the c

22 ontribute to the neuronal iron elevation and oxidative burden that feature in AD pathology.

23 on exist, but overaccumulation results in an oxidative burden that, if unchecked, would lead to cell

24 strated improved neutrophil function: normal oxidative burst (in 3 of 3 patients tested), corrected p

25 odestly increased phagocytosis/killing by an oxidative burst of murine neutrophils in vitro Intravita

26 e report a regioselective and chemoselective oxidative C(sp(3))-H methylation method that is compatib

27 n and neonatal upregulation of mitochondrial oxidative capacity may protect against oxidative stress

28 espiratory exchange rates and higher hepatic oxidative capacity.

29 e (gasotransmitter) produced endogenously by oxidative catabolism of heme, and the understanding of i

30 R to less energy demanding, and value-added, oxidative chemistry.

31 bisperoxides with high selectivity over the oxidative cleavage of C=C bond that usually forms the ke

32 to activate molecular oxygen for subsequent oxidative cleavage of glycosidic bonds.

33 It was established that flavonol oxidative cleavage reaction and oxidative polymerization

34 transformation involving a sigma complex and oxidative cleavage transition state.

35 We, therefore, present a simple thermal-oxidative compositional inversion (TOCI) method to intro

36 In contrast, 2(2-) degrades under oxidative conditions, which we associate to the impossib

37 dy provides a new way of stereoselective C-C oxidative coupling via a photocatalytic means using spec

38 arly 4 folds at the interface as a result of oxidative cross-linking, even though the modulus of the

39 trically, over the pH range 4-10, the OCl(-) oxidative current was found to correlate extremely well

40 The synthesis approach involves oxidative cutting of short range ordered carbon derived

41 thesized through the iodine-mediated [3 + 2] oxidative cyclization of 2-aminoheteroarenes and isothio

42 id and its ability to adapt to and cope with oxidative damage and immune clearance.

43 AT1 has potential to protect the retina from oxidative damage and to prevent or slow down diabetic re

44 ings indicate that a strain's sensitivity to oxidative damage can be elucidated from the structural p

45 Interestingly, infected chub exhibited lower oxidative damage compared to uninfected fish, irrespecti

46 can reverse isoprenaline (ISO)-induced renal oxidative damage in rats, a model that mimics SNS overst

47 ttractiveness may be costly due to increased oxidative damage in the postmenopausal period.

48 The leading cause of mutation due to oxidative damage is 8-oxo-2'-deoxyguanosine (8-oxoG) mis

49 analyzed immunofluorescence distribution of oxidative damage markers, and of SOD2 (superoxide dismut

50 omic-level physicochemical properties and of oxidative damage mechanisms for multiple strains in a sp

51 However, it did not lead to oxidative damage of the enriched batches or affect the m

52 Oxidative damage to DNA is a threat to the genomic integ

53 models, mitochondrial H(2)O(2) emission and oxidative damage were greater in Taz(KD) than in wild-ty

54 is causes an increase of intravascular heme, oxidative damage, and inflammation in which macrophages

55 fects on reactive oxygen species production, oxidative damage, and telomere shortening, at the indivi

56 iciency makes red cells highly vulnerable to oxidative damage, and therefore susceptible to hemolysis

57 on rate possibly through the accumulation of oxidative damage, in particular in the mitochondrial gen

58 m of cell death that relies on iron-mediated oxidative damage.

59 Selective oxidative deamination has long been considered to be an

60 heptane scaffold through the C-C coupling or oxidative deborylation reactions.

61 Oxidative decomposition of soil organic matter determine

62 tion and its relationship with systemic anti-oxidative defence capacity in normal individuals versus

63 Thermally accelerated oxidative degradation of wolfberry pulp was kinetically

64 UvrC, the [4Fe4S] cofactor is susceptible to oxidative degradation with aggregation of apo species.

65 tified as highly selective catalysts for the oxidative dehydrogenation (ODH) of alkanes to olefins.

66 tified as highly selective catalysts for the oxidative dehydrogenation of alkanes such as propane.

67 ified as a highly efficient catalyst for the oxidative dehydrogenation of propane (ODHP) reaction, th

68 family members, FTO and ALKBH5, both act as oxidative demethylases of N6-methyladenosine (m6A) but f

69 The oxidative difluorination of alkenes represents an import

70 ine to oxidative stress and the induction of oxidative DNA damage in spermatozoa.

71 ot a key species participating in endogenous oxidative DNA damage.

72 These guanine-derived oxidative DNA lesions interfere with both replication an

73 Fungal biomass, richness, and oxidative enzyme potential were reduced by N deposition

74 eliminate wild microorganisms and inactivate oxidative enzymes.

75 ssibility of efficiently stabilizing several oxidative equivalents via charge delocalization, resulti

76 ce adsorbed iodide enable the site-selective oxidative etching of Au(0), which leads to nonuniform gr

77 ost-translational modifications, denoted as 'oxidative eustress'.

78 neurons 3 h following learning, in a form of oxidative eustress.

79 racellular collagen assembly begins with the oxidative folding of ~30-kDa C-terminal propeptide (C-Pr

80 l-catalyzed C-C insertions and eliminations, oxidative fragmentation using hypervalent iodine reagent

81 tion, this study suggests that the extent of oxidative genome damage resulting from various cellular

82 ive substances (IC(50) = 23 +/- 2 ug/mL) and oxidative haemolysis (IC(50) at 60 min = 46.0 +/- 0.8 ug

83 different electrochemical strategies for the oxidative homocoupling of benzophenone imine, a readily

84 ul applications in red-light-induced aerobic oxidative hydroxylation of arylboronic acids and benzyli

85 d to myocardial infarction, whereas high HDL oxidative-inflammatory index values (OR(1SD), 1.53; 95%

86 n in mouse and human lung is associated with oxidative injury and pathogenic inflammation.

87 strate that NADPH oxidase 2 (NOX2) generated oxidative injury causes upregulation of a constitutively

88 hemical molecular redox probes, Formaldehyde oxidative latent probe (FOLP) and dihydroxy-formaldehyde

89 tent probe (FOLP) and dihydroxy-formaldehyde oxidative latent probe (HFOLP), for the selective profil

90 gh obesity but also through inflammatory and oxidative mechanisms and insulin resistance, even in the

91 ies, ethanolic celery extracts inhibited the oxidative-mediated DNA damage induced by tert-butylhydro

92 pharmacological inhibition of mitochondrial oxidative metabolism attenuates EMCV-mediated beta-cell

93 led that migratory cells selectively utilize oxidative metabolism during the process of migration to

94 Thereafter, a phenotypic switch to oxidative metabolism with lipid utilization to fuel inva

95 lism, including glucose disposal, lipolysis, oxidative metabolism, and energy expenditure.

96 that synapses can switch from glycolytic to oxidative metabolism, but to do so, they rely on activit

97 ant increase in 2 transcriptional drivers of oxidative metabolism, PGC1alpha and PPARD, suggesting an

98 infusion on muscle force and skeletal muscle oxidative metabolism.

99 e study of potential direct sirtuin cysteine oxidative modifications has been performed.

100 insulin-stimulated leg blood flow and a more oxidative muscle fiber type distribution.

101 High-altitude mice had a more oxidative muscle phenotype than low-altitude mice.

102 nction were overlaid upon the generally more oxidative phenotype of highlanders.

103 Targeting oxidative phosphorylation (OXPHOS) with BDQ and simultan

104 red for energy generation processes, such as oxidative phosphorylation (OXPHOS).

105 ulation in human and mouse were enriched for oxidative phosphorylation and adipogenesis.

106 reprogramming of tumor metabolism involving oxidative phosphorylation and fatty acid oxidation (FAO)

107 ate adenocarcinoma consumes citrate to power oxidative phosphorylation and fuel lipogenesis, enabling

108 ion of levels associated with each hallmark; oxidative phosphorylation and G(2)-M checkpoint were ass

109 mes and identified nuclear genes involved in oxidative phosphorylation and glycolysis (OXPHOG) as a c

110 lomic profile that reflects a combination of oxidative phosphorylation and glycolysis.

111 pid oxidation, glycolysis, and mitochondrial oxidative phosphorylation are common strategies to cope

112 udies support the notion that glycolysis and oxidative phosphorylation are rheostats in immune cells

113 vated receptor gamma coactivator 1-alpha and oxidative phosphorylation complex II and III were signif

114 ance of specific nuclear-encoded subunits in oxidative phosphorylation complexes I and V increased in

115 WT) hearts, but there were no differences in oxidative phosphorylation coupling efficiency or membran

116 r predicted mitochondrial function, of which oxidative phosphorylation emerged as the top-most enrich

117 n a normal surveillance state, microglia use oxidative phosphorylation for their energy supply, but r

118 hifting between glycolysis and mitochondrial oxidative phosphorylation has been implicated in macroph

119 Oxidative phosphorylation in the presence of substrates

120 ain driver of allogeneic T cell-driven GVHD, oxidative phosphorylation is a main driver of Treg suppr

121 This compromised oxidative phosphorylation, causing severe oxidative stre

122 n-regulated genes included those involved in oxidative phosphorylation, mitochondrial dysfunction, nu

123 pplementing NAD+ for glycolysis and NADH for oxidative phosphorylation.

124 lomics upregulated glycolysis and suppressed oxidative phosphorylation.

125 uding up-regulation of genes associated with oxidative phosphorylation.

126 been proposed as a master regulator of tumor oxidative phosphorylation.

127 enetic machinery needed for local control of oxidative phosphorylation.

128 t antigenic stimulation impaired ADP-coupled oxidative phosphorylation.

129 hat flavonol oxidative cleavage reaction and oxidative polymerization are main chemical routes which

130 uggest that sirtuin activity is regulated by oxidative post-translational modifications of cysteines

131 a kinetic study of the primary and secondary oxidative process was run and the kinetic parameters (ra

132 Oxidative processes frequently contribute to organic pol

133 ecause it provides an understanding of their oxidative properties.

134 from formaldehyde, an obligate by-product of oxidative protein demethylation during transcription reg

135 rythroid 2-like 2 (NRF2) protects cells from oxidative, proteotoxic, and metabolic stress in normal c

136 ectively, indicating that both reductive and oxidative quenching catalytic cycles can be operative, a

137 est performing system and we found that fast oxidative quenching of the exciton occurs (picoseconds)

138 s and the availability of a reductive and an oxidative quenching pathway.

139 cular atom-transfer radical addition through oxidative quenching.

140 cal synthesis using a fired brick to control oxidative radical polymerization and deposition of a nan

141 dro-beta-carbolines undergo sodium periodate oxidative ring expansion in the presence of formaldehyde

142 of life due to the protective mechanisms of oxidative shielding and revealed only after menopause wh

143 ROS and oxidative signals arising from metabolism or phytohormon

144 thus V O(2) , especially within fast-twitch oxidative skeletal muscle.

145 l plates for live cell, fluorescent reactive oxidative species (ROS) and viability observations, or o

146 driven by a, so far, not-described reactive oxidative species (ROS)-regulated calcium influx.

147 e insights for the design and development of oxidative-species-selective materials for more effective

148 P > 0.05) on dry-aged beef lipid and protein oxidative stabilities and proteolysis pattern compared t

149 eographical origin, post-harvest treatments, oxidative stability and sensory quality.

150 nt (TPC), radical scavenging activity (RSA), oxidative stability index (OSI), fatty acid composition

151 in the oil/water ratio increased the overall oxidative stability of emulsions but decreased the antio

152 The oxidative stability of hydrolysate-stabilized emulsions

153 sonication process resulted in the increased oxidative stability of lipids as confirmed by low PV, TB

154 The high lipid and protein oxidative stability of long-term frozen lean beef produc

155 Therefore, we investigated the oxidative stability of soy protein-based products subjec

156 Physical and oxidative stability of the mayonnaises were analyzed bas

157 ubility of quercetin in Coil and enhance its oxidative stability.

158 signalling pathway activation as well as the oxidative status in tumours to clarify the mechanisms in

159 n Fe(3+) and Fe(2+) as well as the different oxidative steps of the various ferroxidase centers alrea

160 8.5+/-4.1, P=0.014) accompanied by increased oxidative stress (dihydroethidium fluorescence: sham, 1.

161 n throughout a lifetime and may cause higher oxidative stress (OS).

162 emical (malonyl-aldehyde [MDA], glutathione, oxidative stress [OSI], tumor necrosis factor [TNF]-alph

163 tathione synthesis, the Gclm knockout mouse, oxidative stress activated MMP9 (matrix metalloprotease

164 fractions showed bioactive properties, with oxidative stress amelioratory effects, and could be a po

165 rast, induction of cydDC sensitizes cells to oxidative stress and aminoglycosides, which can be suppr

166 treatment reduces CoQ synthesis and promotes oxidative stress and apoptosis in tumors when administer

167 The homeostatic link between oxidative stress and autophagy plays an important role i

168 The relationship between oxidative stress and cardiac stiffness is thought to inv

169 1-specific siRNA mitigated radiation-induced oxidative stress and cellular injury.

170 cellular antioxidants under H(2)O(2) induced oxidative stress and disturbances caused to mitochondria

171 xygen species are linked to the induction of oxidative stress and inflammation in the liver.

172 sis and TCA cycle, a milieu which can hasten oxidative stress and inflammation.

173 ated by mitigation of vascular mitochondrial oxidative stress and inflammation.

174 tion; (iii) Cellular signaling changes; (iv) Oxidative stress and inflammatory responses.

175 JunD downregulation was associated with oxidative stress and left ventricular dysfunction assess

176 g physiological responses (plasma TH levels, oxidative stress and mitochondrial density).

177 ffects of the reciprocal interaction between oxidative stress and neuroinflammation, impacting on PVI

178 rmore, Sspo defects are sufficient to induce oxidative stress and neuroinflammatory responses implica

179 art mitochondria is unchanged in the face of oxidative stress and point to a critical role of obesity

180 tored visual pigment formation and decreased oxidative stress and retinal degeneration, which resulte

181 beta1 signaling is a target for iron-induced oxidative stress and suggest that baseline PI-PLCbeta1 q

182 this life extension is due to a reduction of oxidative stress and the activation of the transcription

183 ncreased ULBP2/5 expression was dependent on oxidative stress and the antioxidants N-acetylcysteine a

184 on the vulnerability of the male germline to oxidative stress and the induction of oxidative DNA dama

185 sregulation of placental Htra1 and placental oxidative stress are features of preeclamptic placentas

186 sensitivity to microbial toxin, osmotic and oxidative stress are seen in both mutants albeit to diff

187 drial oxidative capacity may protect against oxidative stress associated with birth while ensuring en

188 diac pathological remodeling, apoptosis, and oxidative stress associated with both onset and advancem

189 ere, we assess ribosomal integrity following oxidative stress both in vitro and in cells to elucidate

190 lay in the maintenance of the ribosome under oxidative stress conditions.

191 Inadequate nutrient intake leads to oxidative stress disrupting homeostasis, activating sign

192 Mutations indicative of oxidative stress do not increase with increasing latency

193 Conversely, prevention of mitochondrial oxidative stress during chronic T cell stimulation allow

194 Our results provide evidence that reducing oxidative stress following allotransplantation of PVPON/

195 ination of mitochondrial stresses, including oxidative stress from low levels of purine metabolites a

196 ng CNS innate immune cells identified a core oxidative stress gene signature coupled to coagulation a

197 Oxidative stress in adipocyte plays a central role in th

198 pment of chronic, low-grade inflammation and oxidative stress in age-related dry eye.

199 To test whether increased oxidative stress in Atg7 (Delta/Delta) mice was responsi

200 PGRPs induce oxidative stress in bacteria through a block in the resp

201 he inflammatory processes and the effects of oxidative stress in Caco-2 cells, and preserved the inte

202 increases mitochondrial activity and reduces oxidative stress in children with SCD/VOE.

203 ne oxidase for dopamine metabolism, triggers oxidative stress in dopaminergic neurons and alpha-Syn a

204 astrocyte activation, neuroinflammation, and oxidative stress in FBN-ARO-KO mice.

205 atus, providing a mechanism for the enhanced oxidative stress in pre-eclampsia.

206 ized cells provide insights into the role of oxidative stress in senescence bypass and immortalizatio

207 of A. baumannii to form biofilms and resist oxidative stress in the respiratory tract facilitates sy

208 of GSK3 enhanced Nrf2 activity and prevented oxidative stress in the retina of diabetic mice.

209 (REDD1) is necessary for the development of oxidative stress in the retina of streptozotocin-induced

210 Our results suggest that the oxidative stress induced high mutation frequency on mtDN

211 Oxidative stress induces coccoid formation and is associ

212 Oxidative stress inhibits E2F1 transcriptional activity,

213 Oxidative stress is a central part of innate immune-indu

214 We show that oxidative stress is a key factor in the mode of action o

215 elevance for human amyloid diseases in which oxidative stress is often an associated hallmark.

216 em (CNS) innate immune cells contributing to oxidative stress is unknown, and therapies to target the

217 -treated rats not only prevents elevation of oxidative stress markers but also rescues levels of depl

218 ted mitochondrial dysfunction and associated oxidative stress might induce senescence in joint tissue

219 ss glucose is present; and (c) mitochondrial oxidative stress must precede the insulin stimulus to ca

220 ng proteinopathy of AD, possibly by inducing oxidative stress or ferroptotic cell death, or may be re

221 upon transition to latency or by decreasing oxidative stress or iron content.

222 ures of kidney function, tubular injury, and oxidative stress over time in a cohort of children with

223 nction, suppression of neuroinflammatory and oxidative stress pathways, and a reduction in persistent

224 Oxidative stress plays a key role in the activation of a

225 e of DNA-binding protein associated with the oxidative stress response and that this molecular functi

226 aracterized proteins involved in prokaryotic oxidative stress response are rare, we sought to learn m

227 on of C. albicans drug-efflux, regulation of oxidative stress response, and maintenance of cell membr

228 for scavenging free radicals in wolfberries oxidative stress response.

229 nd data set here could provide insights into oxidative stress responses in the heart and avail the se

230 lbs of affected human scalp remains unclear, oxidative stress sensing appears to be a key factor invo

231 glycerides in liver of young mice, caused by oxidative stress that activated FOXO1 to promote express

232 l presence reflects the cumulative amount of oxidative stress the brain has endured.

233 xonal and myelin loss attributed to elevated oxidative stress through NADPH oxidase in lineage-traced

234 hannels, while hydrogen peroxide distributes oxidative stress to sensitize the network to mitochondri

235 Thus, oxidative stress transcriptomics identified neurotoxic C

236 es mitochondrial fission leading to elevated oxidative stress via DRP1.

237 ty for providing cellular protection against oxidative stress while serving as a reactive oxygen spec

238 f normal mitochondrial function and elevated oxidative stress within the lymphatic muscle.

239 l hypoxia, immune dysfunction, angiogenesis, oxidative stress) causes various disruptions in offsprin

240 enin are pleiotropic, resulting in decreased oxidative stress, activation of pro-proliferative and pr

241 Mechanistically, this is due to increasing oxidative stress, alterations to interactions with the m

242 ltration, tissue repair enzymes, pathways of oxidative stress, and altered intestinal barrier functio

243 hin the aerobic respiratory chain, mitigates oxidative stress, and contributes to gene expression in

244 asmic reticulum (ER) stress response, causes oxidative stress, and induces apoptosis.

245 yr) is produced in cells under conditions of oxidative stress, and m-Tyr has been shown to be toxic t

246 pathogens in circulation, kill them through oxidative stress, and present them to the antigen-presen

247 Also, they reduced weight loss, oxidative stress, and the anthracnose (Colletotrichum gl

248 To avoid iron-mediated oxidative stress, bacteria utilize iron-dependent global

249 responsive to inflammatory mediators and to oxidative stress, consistent with a key role in CF lung

250 electrofusion of rabbit SCNT embryos induced oxidative stress, disturbed the epigenetic state, and ca

251 ty through multiple mechanisms including the oxidative stress, DNA damage, lysosomal dysfunction, inf

252 e cells and proline metabolism to counteract oxidative stress, during growth at 4 degrees C compared

253 t how peripheral changes in a key marker for oxidative stress, glutathione (GSH), may associate with

254 or 15 (GDF-15), a marker of inflammation and oxidative stress, has emerged as a biomarker for arteria

255 bolites upon exposure to PCB-153, leading to oxidative stress, hemolytic anemia, and tumor developmen

256 role of lipid peroxidation, a potent form of oxidative stress, in mediating RV hypertrophy and failur

257 substrate for the heart and kidneys, reduced oxidative stress, lowered serum uric acid level, reduced

258 s, which identified consistent enrichment in oxidative stress, mitochondrial dysfunction, and transcr

259 With oxidative stress, MSH3 with this deletion (Delta27bp MSH

260 l mitochondrial hyper-function and increased oxidative stress, possibly resulting in neurodegeneratio

261 2 in a manner that decreased and scaled with oxidative stress, respectively.

262 reactive oxygen species (ROS) and consequent oxidative stress, specifically in the gut.

263 There was evidence for oxidative stress, tissue injury and microscopic intersti

264 ac dysfunction, myocardial inflammation, and oxidative stress, underlining the importance of inflamma

265 here is a disconnect between respiration and oxidative stress, whereby mitochondrial oxidant levels d

266 anslation quality control breaks down during oxidative stress, wherein ThrRS is rendered inactive.

267 r, we accomplished both light- and energetic/oxidative stress-gated control of this interaction.

268 on by protecting multiple myeloma cells from oxidative stress-induced apoptosis.

269 al studies found noise to be associated with oxidative stress-induced vascular and brain damage, medi

270 ns as a redox sensor to prevent drug-induced oxidative stress-mediated DNA damage and execution with

271 Transcriptional profiling of oxidative stress-producing CNS innate immune cells ident

272 s and regulates the transcription of various oxidative stress-response genes.

273 elevates ROS level and induces expression of oxidative stress-responsive genes.

274 ceptor type 1 (AT(1) R) axis associated with oxidative stress.

275 enetic factors involved in the regulation of oxidative stress.

276 ioxidant that may protect against As-induced oxidative stress.

277 ed oxidative phosphorylation, causing severe oxidative stress.

278 rotein synthesis during cellular response to oxidative stress.

279 requirement A1 (Htra1), a marker of cellular oxidative stress.

280 lso controls cellular defense against ER and oxidative stress.

281 ta activity, possibly useful during cellular oxidative stress.

282 ucing CD36 scavenger receptor expression and oxidative stress.

283 ypoxia, nutrient deprivation, metabolic, and oxidative stress.

284 changing environmental conditions and avoid oxidative stress.

285 protein kinase (MAPK) pathway responding to oxidative stress.

286 metastasizing cells depend on MCT1 to manage oxidative stress.

287 age the cause of hyperglycemia and resultant oxidative stress.

288 gher levels mediates neuronal injury through oxidative stress.

289 e cycle and nutrient uptake, and lowering in oxidative stress.

290 function due to TEPP-46-induced increases in oxidative stress.

291 making them more susceptible to ROS-induced oxidative stress.

292 thelial NO pathway and increased endothelial oxidative stress.

293 1 are more resistant to both heat stress and oxidative stress.

294 released from mitochondria during sustained oxidative stress.

295 eral pathological conditions associated with oxidative stress; however, its role in periodontal disea

296 Lipofuscin is a nondegradable end-product of oxidative stress; its cerebral presence reflects the cum

297 sol and mitochondrion to cope with exogenous oxidative stresses, indicating a direct link between bot

298 ied whether SGLT2 inhibition altered cardiac oxidative substrate consumption, efficiency, and perfusi

299 osin light chain (MYL1 and MYL3) showed high oxidative susceptibility owing to peptidyl methionine an

300 Oxidative transformation of hexapyrrolylbenzenes into az