ライフサイエンスコーパス: oxygen

1 on sulfide mineral that is oxidized by trace oxygen.

2 sources such as antibiotics and supplemental oxygen.

3 cebo group were still receiving supplemental oxygen.

4 rdination between the boron and the phenolic oxygen.

5 se ratio in both the presence and absence of oxygen.

6 such as the reduction of methyl viologen or oxygen.

7 ility with reference to the mole fraction of oxygen.

8 tinguished due to the strong interference of oxygen.

9 or cemtirestat by replacement of sulfur with oxygen.

10 proliferative advantage, particularly in low oxygen.

11 Singlet oxygen ((1)O(2)), the major reactive oxygen species (ROS

12 ey, ATP was rapidly generated in presence of oxygen (100 kPa), which remained stable up to 22 h.

13 ed at baseline, 4/7 were fully recovered off oxygen, 2/7 were still hospitalized, and 1/7 had died.

14 .8-8.0), P < 0.01], and fraction of inspired oxygen [25% (IQR: 21-31) vs 42% (IQR: 30-80), P < 0.01]

15 sible proton movements during heme-catalyzed oxygen activation, as well as on ascorbate oxidation.

16 evolved-a noncooperative homodimer with high oxygen affinity that existed before the gene duplication

17 riendly and deals with greener oxidants like oxygen, affording 1,4-ketocarbonyls as a value-added end

18 This interaction is promoted during oxygen and amino acid depletion and protects LRS from de

19 ophilic aromatic substitution reactions with oxygen and carbon nucleophiles to provide access to a va

20 Impaired oxygen and cellular metabolism is a hallmark of ischaemi

21 ienced thirty-five weeks of dynamic methane, oxygen and nitrate concentrations.

22 -gas respiratory cycle-that insures adequate oxygen and nutrient delivery to meet local metabolic dem

23 Blood carries oxygen and nutrients to the trillions of cells in our bo

24 Major host insults are reactive oxygen and reactive nitrogen species as well as cellular

25 Oxygen and reactive oxygen species (ROS) have been co-op

26 a set of positive feedbacks, mediated by the oxygen and sulfur cycles, that led to permanent state ch

27 ly convert endogenous hydrogen peroxide into oxygen and then elevate the production of tumor-toxic si

28 actors included water temperature, dissolved oxygen, and baseflow.

29 l under a reaction environment with methane, oxygen, and water.

30 g noninvasive ventilation or nasal high-flow oxygen; and 6.7% receiving invasive mechanical ventilati

31 se II, 6-minute maternal hyperoxia with 100% oxygen; and phase III, 5.6-minute return to resting stat

32 lectrochemical side reactions with molecular oxygen are shown to occur during organic electrochemical

33 The method uses NHPI as the cocatalyst and oxygen as the sole oxidant.

34 methylene unit of the adamantane group by an oxygen atom increases the solubility, permeability, and

35 ch occurs via the cis form when the terminal oxygen atom of the NOO moiety reacts with the ortho posi

36 Surprisingly, 3 is a stronger oxygen atom transfer (OAT) agent than 2-trans; 3 perform

37 The developed oxygen atom transfer approach allows the generation of 1

38 metal-based redox chemistry in the enzymatic oxygen atom transfer reaction.

39 nterface favors the sequential adsorption of oxygen atoms with facile kinetics.

40 this study was to compare the ratio of renal oxygen availability (RO(2)) to glomerular filtration rat

41 Methane oxidation was strictly dependent on oxygen availability and led to enrichment of (13) C in r

42 observed interactive effects of temperature, oxygen availability, and body size predict that global w

43 P05 are more active and respire nitrate when oxygen becomes available at low concentrations suggests

44 Cm(III) mellitate that possesses only curium-oxygen bonds.

45 one of: mechanical ventilation, supplemental oxygen, bronchodilators or steroids at 28 days or discha

46 on receptors involved in chemosensitivity to oxygen, but well-established pathophysiological mechanis

47 omplex interplay between Hb (hemoglobin) and oxygen, carbon dioxide, and nitric oxide-the three-gas r

48 Accordingly, decreased oxygen concentration (hypoxia) is a major stressor that

49 1(28-36) to shift substrate utilization from oxygen-consuming fatty acid metabolism toward oxygen-spa

50 a complex and dynamic oxygen cycle in which oxygen consumption and corresponding carbon oxidation ar

51 identify parameters of a numerical model for oxygen consumption and diffusion.

52 ng circuits is directly linked to changes in oxygen consumption mediated by NNT.

53 Empagliflozin did not change myocardial oxygen consumption or MEE.

54 ar metabolic rates in vitro using changes in oxygen consumption rate as a readout.

55 cretion, partly due to reduced mitochondrial oxygen consumption rate, glucose-stimulated Ca(2+) flux,

56 irds (Aves) display high metabolic rates and oxygen consumption relative to mammals, increasing react

57 thelial cells results in decreased uncoupled oxygen consumption, increased fission, decreased membran

58 ls with LKB1 knockdown had a reduced rate of oxygen consumption, which was partially restored by PDK4

59 id not improve the 4HNE-mediated decrease in oxygen consumption.

60 f Cu in wine and their impact on the rate of oxygen consumption.

61 teraction between Pd clusters and the nearby oxygen-containing functional groups is key for the high

62 TR-FTIR) spectroscopy revealed a presence of oxygen-containing functional groups on the DLC surface.

63 surface act as electron pumps that stabilize oxygen-containing species and facilitate the continuity

64 creased with reaction time, temperature, and oxygen content in the reaction atmosphere.

65 this context, it is blood flow and not blood oxygen content that is the main driver of tissue oxygena

66 re performed in both eyes to measure retinal oxygen contents and total retinal blood flow, respective

67 ive oxygen species and a complex and dynamic oxygen cycle in which oxygen consumption and correspondi

68 Oxygen defects are essential building blocks for designi

69 Generation of the oxygen-deficient vanadium oxide, [V(6)O(6)(OC(2)H(5))(12

70 ute ventilation, cardiac output and systemic oxygen delivery did not differ between protocols (P > 0.

71 We show that oxygen delivery through PMCs is dependent on its permeab

72 In this context, increasing extracorporeal oxygen delivery, increases the respiratory quotient of t

73 ly from wastewater streams with low chemical oxygen demand (COD) to nitrogen (C/N) ratios.

74 mperatures, low oxygen supply and increasing oxygen demand, high and rising exposure to ultraviolet r

75 CoxFluor enabled the detection of oxygen-dependent changes in COX-2 activity that are inde

76 promotes FBXL5 binding to IRP2 to effect its oxygen-dependent degradation, unveiling a novel and prev

77 mors during fractionated radiotherapy, using oxygen-dependent quenching of phosphorescence, oxygen pr

78 Sinking into oxygen depleted waters explains the exceptional soft tis

79 ted an ~50-fold reduction in viability after oxygen depletion.

80 llows animals and plants to acutely adapt to oxygen deprivation, its functional and historical roots

81 fic tracheal intubation-associated events or oxygen desaturation events occurred in 40.2% of patients

82 perovskite exhibits a 12.8-fold increase in oxygen diffusivity, which matches well with the 10-fold

83 velop a non-invasive method to track spatial oxygen distributions in tumors during fractionated radio

84 quency variability in temperature, dissolved oxygen (DO), and pH increases as pCO(2) increases in the

85 O(cell) when the proportion of exchangeable oxygen during cellulose synthesis (P(ex) ) was kept cons

86 lectrochemical side-reactions with molecular oxygen during device operation.

87 dy aimed to investigate whether supplemental oxygen during hypothermic machine perfusion (HMP) could

88 Further, using a Clark-type oxygen electrode, we measured isolated rat liver mitocho

89 Herein, we show that use of the singlet oxygen ene reaction, combined with [2 + 2] cycloaddition

90 The development of oxygen evolution reaction (OER) electrocatalysts remains

91 2) for hydrogen evolution reaction (HER) and oxygen evolution reaction (OER), respectively.

92 The decrease of oxygen evolution, occurring in the presence of herbicide

93 Increased white matter oxygen extraction fraction was associated with decreased

94 Thus, solid oxygen fertilizers can potentially be used as an effecti

95 ctivated by reaction with a hematite surface oxygen first, followed by a catalytic cycle through a mo

96 have a unique ability to activate molecular oxygen for subsequent oxidative cleavage of glycosidic b

97 verns the transport rate of contaminants and oxygen from the gas phase to the liquid phase, where pol

98 ot only tolerates the presence of all of the oxygen functionalities, but also is facilitated by them.

99 t, as well as a 1.9-fold increase in singlet oxygen generation efficiency over free rose bengal.

100 2) porphyrins showed decent in vitro singlet oxygen generation, which was supported by the intracellu

101 traphenylethylene (DPPTPE) with high singlet-oxygen-generation ability both in dichloromethane and wa

102 ulation of lipopolysaccharide (LPS), KCl and oxygen/glucose deprivation (OGD) that reflect inflammati

103 Oxygen gradients inevitably arise during A. fumigatus bi

104 8 hours; 95% CI, 22 to 34); the conservative-oxygen group spent less time with an Spo(2) exceeding 96

105 The conservative-oxygen group spent more time in the ICU with an Fio(2) o

106 he ICU with an Fio(2) of 0.21 than the usual-oxygen group, with a median duration of 29 hours (interq

107 trial provides no indication that nocturnal oxygen has a positive or negative effect on survival or

108 The strained phosphorus-oxygen heterocycles open to the corresponding heterodien

109 scovered evidence for an upregulation of the oxygen homeostasis maintaining pathway involving Hypoxia

110 racellular accommodation of bacteria, nodule oxygen homeostasis, the control of bacteroid differentia

111 Successful synthesis of MXenes with oxygen, imido, sulfur, chlorine, selenium, bromine, and

112 2) or alpha-UO(3) depending on the amount of oxygen in the system.

113 f the intercalated lithium with the residual oxygen in the vacuum chamber.

114 Injury and repair caused by the oxygen-induced retinopathy model reduced visual acuity t

115 environmental half-life of DA due to singlet oxygen-induced transformations is between 5 and 63 days.

116 Iron-oxygen intermediates like iron(III)-hydroperoxo and high

117 Water electrolysis to hydrogen and oxygen is a well-established technology, whereas fundame

118 The mass-independent minor oxygen isotope compositions (Delta'(17)O) of atmospheric

119 ural observations, backed up with carbon and oxygen isotope data, which indicate an intimate associat

120 Asia, seasonally and interannually depleted oxygen isotope ratios in precipitation have been linked

121 central metabolism is poisoned by molecular oxygen itself.

122 We review oxygen K-edge X-ray absorption spectra of both molecules

123 scopy showed that water added to methane and oxygen led to surface methoxy groups and accelerated met

124 We find that low oxygen level diminishes CD8(+) T cell anti-viral respons

125 Background Despite advances, blood oxygen level-dependent (BOLD) cardiac MRI for myocardial

126 s with current noninvasive cardiac MRI blood oxygen level-dependent (BOLD) techniques.

127 ivity was assessed using resting-state blood oxygen level-dependent functional MRI to measure the fra

128 Blood-oxygen-level-dependent (BOLD) imaging showed that patter

129 Measuring molecular oxygen levels in vivo has been the cornerstone of unders

130 odone-induced decreases in nucleus accumbens oxygen levels.

131 on (*)OH versus ArS(*) transfer, whereas the oxygen-ligated complex does not.

132 However, their chemical instability towards oxygen, light and heat limits its applications in food i

133 Genomic analysis revealed adaptations to oxygen-limitation as well as pathways for mixed-acid fer

134 pment of a visible light-induced and singlet oxygen-mediated green protocol has been accomplished for

135 tinal vessels and measured the total retinal oxygen metabolic rate in rats using visible-light optica

136 igh concentrations have been observed in the oxygen minimum zones (OMZs) of the tropical Pacific but

137 ROS are highly reactive oxygen molecules that can interact with cellular macromo

138 rst oxygenation strategies included standard oxygen (n = 245, 38%), noninvasive ventilation (n = 285;

139 tion (n = 285; 44%), high-flow nasal cannula oxygen (n = 55; 8%), and noninvasive ventilation + high-

140 vasive ventilation + high-flow nasal cannula oxygen (n = 64; 10%).

141 ory distress at birth, need for supplemental oxygen, neonatal ventilator use, and neonatal resection

142 od flow (nrCBF), and tumor metabolic rate of oxygen (nTMRO(2)) was quantified.

143 r function ensure that increased delivery of oxygen, nutrients, and endocrine factors to organs durin

144 accelerator to directly reduce the dissolved oxygen (O(2) ) to reactive oxygen species (ROS).

145 eli phases through a double annealing route: oxygen (O(2)) annealing followed by annealing in 2% hydr

146 us computational analysis indicated that the oxygen (O(2)) status of the tumor and HBOC O(2) affinity

147 dial growth along with stable carbon (C) and oxygen (O) isotope composition (delta(13) C and delta(18

148 is reflected in the reactivity-reaction with oxygen occurs selectively at a position with the highest

149 when oxides, allow for face-sharing of metal-oxygen octahedra or trigonal prisms within their structu

150 Oxygen octahedral rotation forms a fundamental atomic di

151 ch water molecules take the place of lattice oxygen of alpha-MoO(3).

152 of this study was to evaluate the effect of oxygen on wine colour during ageing in barrels and bottl

153 er conditions for low-temperature NH(3)-SCR, oxygen only reacts with Cu(I) ions, which are present as

154 ing, but not limited to, pathogen intrusion, oxygen or nutrient starvation, proteotoxic and organelle

155 ICIRD spectroscopy on the light, oxygen, or voltage (LOV) domains of the blue light recep

156 The effects of a systematic modulation of oxygen over-stoichiometry of VO(2) from 1.86 to 2.44 on

157 onally, they show a more stable pH, a higher oxygen partial pressure (pO(2)), and a lower carbon diox

158 dominantly a result of the enhanced lattice oxygen participation.

159 ygen-dependent quenching of phosphorescence, oxygen probe Oxyphor PtG4 and the radiotherapy-induced C

160 ucts is complex, initiated by regioselective oxygen radical addition to the carbon-carbon double bond

161 ioned, among other factors, by the amount of oxygen received during this process, which thus impacts

162 oxyfluoride, Li(2)MnO(2)F, we show that the oxygen redox process in such materials involves the form

163 on phenomenon is attributed to the nature of oxygen redox which is very likely mostly associated with

164 Li(0.25)Mn(0.75)]O(2), present in almost all oxygen-redox compounds, is lost on charging, driven in p

165 hain reaction, initiated by electrocatalytic oxygen reduction on the cathodic membrane side and subse

166 retic propulsion mechanism and establish the oxygen reduction reaction (ORR) as the major surface rea

167 The SACs catalyze the oxygen reduction reaction (ORR) via a 2 e(-) pathway wit

168 y to improve selectivity and kinetics of the oxygen reduction reaction (ORR).

169 omposite endpoint of nonrebreather or higher oxygen requirement and death (n events = 25 of 77) inclu

170 se (odds ratio [OR], 7.4; 95% CI, 2.5-22.0), oxygen requirement at hospitalization (OR, 2.9; 95% CI,

171 to be peripherally restricted, affect brain oxygen responses induced by intravenous heroin at low, h

172 l powerfully attenuates heroin-induced brain oxygen responses.

173 s with ACS, fever (>38.5 degrees C), reduced oxygen saturation (<95) and asplenia significantly diffe

174 ts of both temperature (11-37 degrees C) and oxygen saturation (17-206% or 3.6-43.6 kPa).

175 , and cardiovascular risk factors, only mean oxygen saturation during sleep was associated with bilat

176 HBOC-201 achieved tissues oxygen saturation equivalent to blood.

177 s severe desaturation (defined as peripheral oxygen saturation reading < 80% during intubation).

178 s, comorbidities, symptom type and duration, oxygen saturation, and laboratory values.

179 Lower oxygen saturation, viral detection, and comorbidities we

180 was used to measure cerebral hemoglobin and oxygen saturation.

181 n arterial pressure, and jugular venous bulb oxygen saturation.

182 evealed he was afebrile and had normal pulse oxygen saturation.

183 cribe the understanding of the mechanisms of oxygen sensing and hypoxia signaling that resulted in th

184 Combining oxygen sensing, X-ray scattering, and Atomic Force Micro

185 Oxygen-sensing mechanisms of eukaryotic multicellular or

186 In this way, the anaerobic excellence and oxygen sensitivity of B. thetaiotaomicron are two sides

187 s of the prolyl hydroxylase domain isoform 1 oxygen sensor in mice (PHD1(KO)) reduces muscle mass.

188 The heme-based oxygen sensor protein AfGcHK is a globin-coupled histidi

189 ssing a constitutively stable version of the oxygen sensor RELATED TO APETALA2 12 (RAP2.12), indicati

190 However, the oxygen sensors based on beta-Ga(2)O(3) and other existin

191 ral ORs in triggering brain hypoxia, we used oxygen sensors in freely moving rats to examine how nalo

192 AGO1 and AGO4 RNA-silencing pathways in low-oxygen signaling in Arabidopsis.

193 ALA2 12 (RAP2.12), indicating convergence of oxygen signaling with epigenetic regulation of gene expr

194 xygen-consuming fatty acid metabolism toward oxygen-sparing glycolysis and glucose oxidation and to i

195 Contrary to common belief, reactive oxygen species (ROS) accumulation did not appear to medi

196 eprivation leads to accumulation of reactive oxygen species (ROS) and consequent oxidative stress, sp

197 Clearance and adaptation to reactive oxygen species (ROS) are crucial for cell survival.

198 lium-dependent vasodilation through reactive oxygen species (ROS) formation.

199 ion relative to mammals, increasing reactive oxygen species (ROS) formation.

200 Ang II)-induced AAA by facilitating reactive oxygen species (ROS) formation.

201 Here, we show that reactive oxygen species (ROS) function as signaling molecules tha

202 herapy (CDT), resulting in enhanced reactive oxygen species (ROS) generation.

203 Oxygen and reactive oxygen species (ROS) have been co-opted during evolution

204 re helped in the photogeneration of reactive oxygen species (ROS) in water.

205 Reactive oxygen species (ROS) increased in human donor lungs star

206 Since reactive oxygen species (ROS) is vitally involved in tissue infla

207 -mediated increase of intracellular reactive oxygen species (ROS) levels and Src protein phosphorylat

208 As results, cellular ATP and reactive oxygen species (ROS) levels are elevated, mTOR and IRF/I

209 Reactive oxygen species (ROS) oxidize nucleotide triphosphate poo

210 Singlet oxygen ((1)O(2)), the major reactive oxygen species (ROS) produced in chloroplasts, has been

211 Hydrogen peroxide (H(2)O(2)) is a reactive oxygen species (ROS) that mediates essential signaling i

212 n, calcium signaling, generation of reactive oxygen species (ROS), and additional determinants of cel

213 cetylcysteine (NAC), a scavenger of reactive oxygen species (ROS), diminished the morphological and m

214 ted to the AMP sensitizing cells to reactive oxygen species (ROS), making them more susceptible to RO

215 Reactive oxygen species (ROS), mitochondrial dysfunction, telomer

216 of NADPH oxidase 2 (NOX2)-dependent reactive oxygen species (ROS), signals we term X-ROS.

217 y specific demethylase(s) generates reactive oxygen species (ROS), which oxidize DNA and other cellul

218 lammatory responses is the burst of reactive oxygen species (ROS), with hydrogen peroxide (H(2) O(2)

219 oxidative stress while serving as a reactive oxygen species (ROS)-activated persulfide donor.

220 locally increases the abundance of reactive oxygen species (ROS).

221 uce the dissolved oxygen (O(2) ) to reactive oxygen species (ROS).

222 organelle stress, and elevation of reactive oxygen species (ROS).

223 zed that an increase in calcium and reactive oxygen species activate a large conductance channel in t

224 derscore prolific marine sources of reactive oxygen species and a complex and dynamic oxygen cycle in

225 phaSyn that form in the presence of reactive oxygen species and cytochrome c, a proapoptotic peroxida

226 the substrates, which is caused by reactive oxygen species and leads to a mixture of products, the m

227 Reactive oxygen species are centrally involved in the pathophysio

228 cally with doxorubicin by producing reactive oxygen species but also provide catechol moieties for th

229 , low adiponectin levels, increased reactive oxygen species damage, and elevated GDF15 and FGF21 leve

230 LBL favored reactive oxygen species homeostasis and metabolic activities invo

231 rited ability of TiO(2) to generate reactive oxygen species is used as a strategy to avoid adding H(2

232 osine 5'-triphosphate contents, and reactive oxygen species levels in hepatoma cells (Hepa1-6), a pro

233 bition resulted in reduced cellular reactive oxygen species production compared with negative control

234 ophil extracellular trap formation, reactive oxygen species production, and released human neutrophil

235 as well as downstream intracellular reactive oxygen species production.

236 In addition, the reactive oxygen species scavenger glutathione (GSH) was upregulat

237 important role in the resistance to reactive oxygen species through the regulation of manganese trans

238 Further, reactive oxygen species were produced in both endoperoxide- and t

239 'Reactive oxygen species' (ROS) is an umbrella term for an array o

240 damaged by spontaneous hydrolysis, reactive oxygen species, aberrant cellular metabolism or other pe

241 diseases, including neural fibrils, reactive oxygen species, and cofilin-actin rods, present numerous

242 creased survival factors, increased reactive oxygen species, and excessive neuroinflammation.

243 drial depolarization, generation of reactive oxygen species, and PARP cleavage.

244 ATP production, such as calcium and reactive oxygen species, are also key regulators of mitochondrial

245 These OH precursors include reactive oxygen species, reactive nitrogen species, and sulfur di

246 responsible for the Fls3-sustained reactive oxygen species, suggesting involvement of multiple struc

247 of surface hydroxyl groups and (sub-)surface oxygen species.

248 reby allowing toxic accumulation of reactive oxygen species.

249 n of mitochondria and production of reactive oxygen species.

250 ion trigger release of DNA damaging reactive oxygen species.

251 byproducts of which are deleterious reactive oxygen species.

252 ers the radical SAM reaction to complete the oxygen-sulfur swapping.

253 itional severity criteria as follows: use of oxygen supplementation of more than 4 L/min flow; use of

254 and hypoxemic respiratory failure requiring oxygen supplementation whose Pao(2) increased >=20% with

255 ich include low but rising temperatures, low oxygen supply and increasing oxygen demand, high and ris

256 Improper or poor oxygen supply to cells within the tissue bulk severely l

257 tivity reverses these alterations, restoring oxygen supply to the tissue and enabling recovery of mus

258 ctivity normalized these effects, increasing oxygen supply, restoring optimal diaphragm functional pr

259 admission to intensive care unit, length of oxygen support, and overall survival.

260 (HMDSO) for accurate measurements of tissue oxygen tension (pO(2)) using Proton Imaging of Siloxanes

261 the jugular venous bulb oxygen tension-brain oxygen tension gradient (16 mm Hg [sd, 6] vs 39 mm Hg SD

262 of jugular venous bulb oxygen tension-brain oxygen tension gradient to cerebral perfusion pressure (

263 n tissue oxygen tension, jugular venous bulb oxygen tension, and cerebral perfusion pressure were 29

264 Patients underwent monitoring with brain oxygen tension, intracranial pressure, cerebral perfusio

265 In the normoxia group, the mean brain tissue oxygen tension, jugular venous bulb oxygen tension, and

266 icant differences in the jugular venous bulb oxygen tension-brain oxygen tension gradient (16 mm Hg [

267 d in the relationship of jugular venous bulb oxygen tension-brain oxygen tension gradient to cerebral

268 For the oxygen-tethered substrates, the reaction can proceed at

269 olved strategies to neutralize the impact of oxygen that can be added to and integrated into the exis

270 erm for an array of derivatives of molecular oxygen that occur as a normal attribute of aerobic life.

271 fect on survival or progression to long-term oxygen therapy in patients with COPD.

272 Rationale: Aerosol generation with modes of oxygen therapy such as high-flow nasal cannula and nonin

273 9, 95% CI 1.4 to 746, P = 0.03) and need for oxygen therapy with C4d (11.7, 1.1 to 130, P = 0.045).

274 uces reintubation compared with conventional oxygen therapy, but not compared with noninvasive ventil

275 Mammalian cells respond to insufficient oxygen through transcriptional regulators called hypoxia

276 d blood cells [RBCs]) as passive carriers of oxygen to recognizing the complex interplay between Hb (

277 levate the production of tumor-toxic singlet oxygen to significantly enhance PDT.

278 Red blood cells (RBCs) transport oxygen to tissues and remove carbon dioxide.

279 creasing I above 1 m and adding diacids with oxygen-to-carbon molar ratio (O:C > 1) significantly red

280 Isotopic oxygen tracer experiments support a Mars van Krevelen re

281 and beta-subunits that mediates respiratory oxygen transport and exchange by cooperatively binding o

282 onsible for numerous functions, ranging from oxygen transportation to host defense, to injury repair.

283 Oxidation reactions include molecular oxygen under solvent control or using different dioxiran

284 eters of aerobic performance are the maximal oxygen uptake ( VO2max ), critical power (CP) and speed

285 Peak oxygen uptake, a primary determinant of prognosis, morta

286 Fe, Y) controls the concentration of surface oxygen vacancies (V(O)) in these materials whereby an in

287 behavior disappears, further confirming the oxygen vacancies as being responsible for the memristive

288 wever, the use of ionic interfaces to create oxygen vacancies has not been fully explored.

289 results verify the existence of bifunctional oxygen vacancies in the 2D black In(2) O(3-) (x) nanoshe

290 These mechanisms are (i) the formation of oxygen vacancies that couples to the electrostatic energ

291 en annealed at high temperature to eliminate oxygen vacancies, Al is doped into the ZnO nanosheet, an

292 in the crystalline structure in the form of oxygen vacancies.

293 acts as an effective dopant to modulate the oxygen vacancy (V(O) ) concentration and Ti(3+) formatio

294 onstructing nanoheterojunctions and creating oxygen vacancy (V(O)) in spinel NiCo(2)O(4).

295 because the statistical ensemble behavior of oxygen vacancy defects is deterministic even when indivi

296 underscore the importance of the interfacial oxygen vacancy migration and redistribution in controlli

297 ionship between short range order and cation-oxygen-vacancy coordination remains a subject of active

298 nsport and exchange by cooperatively binding oxygen with moderate affinity.

299 ma(2-) with bridging sulfurs and carboxamide oxygens within Mn-mu-S-CH(2) -C-O, 5-membered rings.

300 sive care unit; 46.8% receiving supplemental oxygen without positive pressure; 11.5% receiving noninv