ライフサイエンスコーパス: oxygen delivery

1 ing to provide enough material for efficient oxygen delivery.

2 on, thereby increasing tissue blood flow and oxygen delivery.

3 y after major surgery is associated with low oxygen delivery.

4 uires rapid removal of CO and restoration of oxygen delivery.

5 asis on improvements in biocompatibility and oxygen delivery.

6 required for blood circulation and systemic oxygen delivery.

7 ation regarding the quality of perfusion and oxygen delivery.

8 r cellular protection in the face of reduced oxygen delivery.

9 uction from parasite sequestration decreases oxygen delivery.

10 veolar PO2, by increasing the extracorporeal oxygen delivery.

11 only occur if oxygen consumption depends on oxygen delivery.

12 utomatically increases to preserve sustained oxygen delivery.

13 an reduction in circulating blood volume and oxygen delivery.

14 rtially related to hypoxemia and compromised oxygen delivery.

15 on is dependent on continuous and controlled oxygen delivery.

16 oxygen saturation, and systemic and cerebral oxygen delivery.

17 situations in which disease reduces cardiac oxygen delivery.

18 sition can alter cell circulation and impede oxygen delivery.

19 , and optimizing cardiovascular function and oxygen delivery.

20 on blood vessel diameter, tissue volume, and oxygen delivery.

21 systemic blood pressure, cardiac output, and oxygen delivery.

22 essure, while maintaining cardiac output and oxygen delivery.

23 imulate erythropoiesis, which improves organ oxygen delivery.

24 ls has been demonstrated to improve cerebral oxygen delivery.

25 ic hypertension, reduced cardiac output, and oxygen delivery.

26 lone, resulting in improved hemodynamics and oxygen delivery.

27 esult in molecules better suited for in vivo oxygen delivery.

28 uring detachment in addition to insufficient oxygen delivery.

29 n, it is unwise to concentrate on maximizing oxygen delivery.

30 ng interest in perioperative optimization of oxygen delivery.

31 the gut oxygen consumption beyond splanchnic oxygen delivery.

32 al heart rate, carotid blood flow or carotid oxygen delivery.

33 purpose of maintaining brain blood flow and oxygen delivery.

34 ne and a reduction in both cardiac index and oxygen delivery.

35 a measure of the efficiency of microvascular oxygen delivery.

36 nticoagulation, and optimization of systemic oxygen delivery.

37 of circulating blood and thus improve tissue oxygen delivery.

38 d enhance exercise performance by increasing oxygen delivery.

39 ues as goals or when therapy did not improve oxygen delivery.

40 1) was administered to enhance the patient's oxygen delivery.

41 local HCT(m) has also been shown to increase oxygen delivery.

42 ical partial pressure of oxygen by improving oxygen delivery.

43 hysiological adaptions that optimized tissue oxygen delivery.

44 tic adaptation in the regulation of cerebral oxygen delivery.

45 nesis, osteogenesis, coronary perfusion, and oxygen delivery.

46 ne did not significantly reduce RBF or renal oxygen delivery.

47 ble to the profound impact of AngII on renal oxygen delivery.

48 including influence on systemic and cerebral oxygen deliveries.

49 7 mL/min/kg [95% CI, 39-57], p = 0.002), and oxygen delivery (7.6 mL O2/min/kg [95% CI, 6.4-9.0] vs 5

50 or muscle blood flow and systemic and muscle oxygen delivery accompanies marked dehydration and hyper

51 xtracorporeal CO2 removal and extracorporeal oxygen delivery affect the respiratory quotient of the n

52 fabrication of a polymer-based intravascular oxygen delivery agent.

53 eneity, and the intracapillary resistance to oxygen delivery, all decrease with depth, reaching a min

54 e no differences in myocardial blood flow or oxygen delivery among groups; however, at 45 min of isch

55 eltaMAP 51 +/- 5 mmHg), and leg and systemic oxygen delivery and (.)VO2 .

56 erence in the diffusion gradient of cellular oxygen delivery and 2) the presence of diffusion limitat

57 mpaired peripheral O2 extraction, constrains oxygen delivery and aerobic capacity in ePVH.

58 Maintenance of systemic oxygen delivery and alleviation of pulmonary hypertensio

59 Global hypoxia-ischemia interrupts oxygen delivery and blood flow to the entire brain.

60 and directly measured parameters of systemic oxygen delivery and blood flow, NIRS can certainly assis

61 Oxygen delivery and blood gases were measured during bot

62 hyxia result in brain injury from inadequate oxygen delivery and constitute a major and growing world

63 iophysical models of layer-specific cerebral oxygen delivery and consumption and improve our understa

64 Better determinations of oxygen delivery and consumption are needed to guide clin

65 Impairments in oxygen delivery and consumption can lead to reduced musc

66 ent directed at achieving survivor values of oxygen delivery and consumption in critically ill patien

67 hree dimensions is crucial for understanding oxygen delivery and consumption in normal and diseased b

68 due to inadequate tools to quantify cerebral oxygen delivery and consumption non-invasively and in re

69 lid tumors arising from an imbalance between oxygen delivery and consumption.

70 good concurrent validity between convective oxygen delivery and DCS-derived blood flow index, as wel

71 al muscle vascular tone during mismatches in oxygen delivery and demand (e.g. exercise) via binding t

72 rum malaria result from an imbalance between oxygen delivery and demand.

73 There was no relationship between oxygen delivery and disease severity (P = .64) or outcom

74 l hypoperfusion (following INDO) on cerebral oxygen delivery and extraction.

75 tochrome c oxidase (CcO), thereby decreasing oxygen delivery and inhibiting oxidative phosphorylation

76 y, blood transfusion does not always improve oxygen delivery and is associated with ischemic events.

77 cellular response to hypoxia which promotes oxygen delivery and metabolic adaptation to oxygen depri

78 impaired by hypoxia despite global cerebral oxygen delivery and metabolism being maintained.

79 impaired by hypoxia despite global cerebral oxygen delivery and metabolism being maintained.

80 adjusting for various variables of cerebral oxygen delivery and metabolism, the only statistically s

81 rebral oxygen diffusion or reflects cerebral oxygen delivery and metabolism.

82 blood flow, which maintains global cerebral oxygen delivery and metabolism.

83 blood flow, which maintains global cerebral oxygen delivery and metabolism.

84 omyocyte function and metabolism rather than oxygen delivery and microvascular function.

85 Hypovolemia with reduced oxygen delivery and microvascular obstruction have diffe

86 We investigated whether oxygen delivery and muscle metabolism of the lower extre

87 hospital cardiac arrest may lead to improved oxygen delivery and organ perfusion.

88 t will reflect the unique pathophysiology of oxygen delivery and peripheral oxygen offloading are nee

89 cells invoke adaptive mechanisms to enhance oxygen delivery and promote energy conservation.

90 O from carboxyhemoglobin, improving systemic oxygen delivery and reversing the inhibitory effects of

91 g hypertrophy necessitate increased fuel and oxygen delivery and stimulate angiogenesis in the left v

92 is protective to the organ, ensures optimal oxygen delivery and supports metabolic function.

93 ntage saturation of haemoglobin and hindlimb oxygen delivery and the increase in P(a,CO2) were sustai

94 o review recent publications in the field of oxygen delivery and tissue oxygenation.

95 development has challenged understanding of oxygen delivery and use.

96 IRS-DCS system with conventional measures of oxygen delivery and utilization during handgrip exercise

97 ompared NIRS-DCS to conventional measures of oxygen delivery and utilization in an exercising limb.

98 examined the efficiency of coupling between oxygen delivery and utilization using the sd of the oxyg

99 etabolites, neural signaling, alterations in oxygen delivery and utilization, and by modifications in

100 erse processes that in turn orchestrate both oxygen delivery and utilization.

101 ation of an arteriovenous shunt may increase oxygen delivery and, hence, improve patients' functional

102 s little reserve to tolerate interruption of oxygen delivery and, thus, is at risk for hypoxemia duri

103 their relationship with cerebral blood flow, oxygen delivery, and carbon dioxide reactivity remain un

104 flow, regional cerebral blood flow, cerebral oxygen delivery, and cerebral metabolic rate of oxygen i

105 post-ischemic defects in neovascularization, oxygen delivery, and chemokine expression, and normalize

106 lation of tissue blood flow distribution and oxygen delivery, and could further reduce skeletal muscl

107 gen binding affinity of Hb, increases tissue oxygen delivery, and increases maximal exercise capacity

108 ial for many biological processes, including oxygen delivery, and its supply is tightly regulated.

109 olume, total peripheral resistance, systemic oxygen delivery, and organ blood flow were determined by

110 reased leg blood flow, vascular conductance, oxygen delivery, and oxygen consumption during exercise;

111 CHD was linearly related to reduced cerebral oxygen delivery, and that cardiac lesions associated wit

112 r perfusion can be uncoupled from convective oxygen delivery, and that tissue saturation seemingly co

113 s influenced by many interactions, including oxygen delivery (angiogenesis, permeability, and HgB) an

114 ses aiming to improve the cardiac output and oxygen delivery are commonly administered in children wi

115 thout an increase in hematocrit (eliminating oxygen delivery as an etiologic factor in myocyte surviv

116 y be associated with relatively lower tissue oxygen delivery as reflected in higher erythropoietin co

117 ver, higher SpvO2 and SaO2 enhanced systemic oxygen delivery, as demonstrated by improvement in oxyge

118 Perioperative periods of diminished cerebral oxygen delivery, as indicated by rSo(2), are associated

119 nsfusion resulted in a greater (16%) rise in oxygen delivery associated with reduction in oxygen extr

120 olids, arterial oxygen content, and systemic oxygen delivery below baseline (p <.05).

121 Hypoxia is a reduction in oxygen delivery below tissue demand, whereas ischemia is

122 20% and when therapy produced differences in oxygen delivery between the control and protocol groups.

123 ncreases in (.)Q , LBF, and systemic and leg oxygen delivery, but central venous pressure and muscle

124 hought to function primarily in nutrient and oxygen delivery, but recent evidence suggests that it ma

125 Additionally, given VP can only approximate oxygen delivery by capillaries, we show that their gener

126 Oxygen delivery by Hb is essential for vertebrate life.

127 me and cardiac output, and ensuring adequate oxygen delivery by maintaining arterial oxygen partial p

128 set physiological hypoxia and achieve normal oxygen delivery by means of higher blood flow enabled by

129 We report measurements of oxygen delivery by the retinal circulation (DO2_IR) and

130 We have explored the coupling of CBF and oxygen delivery by using two complementary methods.

131 HIFs act to promote oxygen delivery (by stimulating erythropoietin and eryth

132 HIF-1 controls oxygen delivery, by regulating angiogenesis and vascular

133 The oxygen delivery capacity of the ceria nanoparticles embe

134 severe reductions in cerebral blood flow and oxygen delivery capacity.

135 bral blood flow (CBF) by 20-30% and cerebral oxygen delivery (CDO(2) ) by 12-19% at sea level and hig

136 lood flow (CBF, duplex ultrasound), cerebral oxygen delivery (CDO(2) ), oesophageal temperature, and

137 Cerebral oxygen delivery (CDO(2)) was measured in infants with CH

138 d flow (gCBF) increases to preserve cerebral oxygen delivery (CDO2) in excess of that required by an

139 ow) and cerebral oxygen metabolism (cerebral oxygen delivery, cerebral metabolic rate of oxygen, and

140 h may provide inferior systemic and cerebral oxygen deliveries compared with either of the 2 surgical

141 Oxygen delivery, consumption, and extraction ratio were

142 d gases; electrolytes; lactate; base excess; oxygen delivery, consumption, and extraction ratio; hema

143 hat the coupling between neural activity and oxygen delivery could be imaged at the single-RBC level

144 Oxygen delivery decreased by 50% after infusion of AngII

145 significantly change cardiac index, but the oxygen delivery decreased due to a hemodilution-induced

146 n extraction, whereas both cardiac index and oxygen delivery decreased for patients in the 546C88 coh

147 the while increasing in mass to provide the oxygen delivery demands of embryonic growth.

148 demonstrate that critical force reflects an oxygen-delivery-dependent balance between motor unit act

149 5 N). These data suggest that CF reflects an oxygen-delivery-dependent balance between motor unit act

150 ute ventilation, cardiac output and systemic oxygen delivery did not differ between protocols (P > 0.

151 sm (MO(2)) due to inadequate compensation by oxygen delivery (DO(2)) and extraction fraction (OEF) af

152 about alterations of retinal blood flow (F), oxygen delivery (DO(2)), oxygen metabolism (MO(2)), oxyg

153 ic capacity could be impaired by the fall in oxygen delivery (DO2) during endotoxic shock.

154 pressin levels were determined, and systemic oxygen delivery (Do2I) and extraction ratio were calcula

155 Impaired oxygen delivery due to reduced cerebral blood flow is th

156 rebral and other organ perfusion, as well as oxygen delivery during cardiopulmonary resuscitation.

157 chanism underlying impaired vasodilation and oxygen delivery during hypoxemia with advancing age.

158 n II AT(1) receptors has a major impact upon oxygen delivery during normal and low Na(+) conditions,

159 nical implications for disease featuring low oxygen delivery (e.g. heart failure, pulmonary disease).

160 In general, there was no association of oxygen delivery (except for ventriculomegaly in the BDG

161 eased diffusion barriers may reduce cellular oxygen delivery following head injury and attenuate the

162 rest and during exercise, despite attenuated oxygen delivery following NO-PG blockade, due to an incr

163 The increase in mean oxygen delivery following transfusion was associated wit

164 .4 to 10.8 +/- 1.4 g/dL (12%; p < 0.001) and oxygen delivery from 5.0 (interquartile range, 4.4-6.6)

165 mechanism of improvement in VO2 is increased oxygen delivery from increased hemoglobin concentration.

166 coustic flowoxigraphy (FOG), which can image oxygen delivery from single flowing RBCs in vivo with mi

167 rates that RBC transfusion improves cerebral oxygen delivery globally and particularly to vulnerable

168 ia, in which cerebral perfusion pressure and oxygen delivery have gained new importance.

169 ted at reducing mortality through increasing oxygen delivery have not been successful.

170 esulted in a significant decrease of hepatic oxygen delivery (hDO2, 63% and 12% of baseline, respecti

171 Systemic oxygen delivery improved dose-dependently with Hb-200 bu

172 col-driven therapy targeting optimization of oxygen delivery improves outcomes in the management of m

173 urons are threatened by markedly constrained oxygen delivery, improving the latter by increasing arte

174 es of SNO-Hb could be manipulated to enhance oxygen delivery in a hypoxic tumor.

175 tabolic demand and the relatively inadequate oxygen delivery in affected synovium, can both be object

176 Haemodynamic therapy aimed at increasing oxygen delivery in an effort to reduce oxygen debt, tiss

177 Mechanisms involving enhanced tissue oxygen delivery in comparison to Lowlander populations h

178 We hypothesised that impaired cerebral oxygen delivery in infants with CHD is a cause of impair

179 sympatholysis and thus tissue blood flow and oxygen delivery in older adults.

180 unction (defined as the capacity to increase oxygen delivery in response to ischemia) and oxygen cons

181 To examine potential differences in cerebral oxygen delivery in Sherpa compared to lowlanders we meas

182 The observed lower cerebral oxygen delivery in Sherpa likely represents a positive a

183 oxide (NO)-derived vasoactivity to optimize oxygen delivery in the arterial periphery.

184 sion and vasodilation (required for adequate oxygen delivery in the face of chronic anemia) are media

185 The lower cerebral oxygen delivery in the Sherpa potentially represents a p

186 the spatial distribution and extent of tumor oxygen delivery in vivo.

187 Cardiac output and oxygen delivery increased significantly from 4.1 L/min a

188 Renal oxygen delivery increased while renal oxygen consumption

189 rterial pressure, cardiac index and systemic oxygen delivery, increases in heart rate and systemic va

190 In this context, increasing extracorporeal oxygen delivery, increases the respiratory quotient of t

191 rterial oxygen content, it further threatens oxygen delivery increasing the risk of cerebral infarcti

192 emic vascular resistance index), metabolism (oxygen delivery index and consumption index, oxygen extr

193 rdiac output/index, stroke volume index, and oxygen delivery index and increases in systemic vascular

194 Oxygen delivery index increased in both success and fail

195 The oxygen delivery index, oxygen consumption index, central

196 L/min/m (71.1-122.5 mL/min/m) (p < 0.01) for oxygen delivery index; 2.9% (2.2-3.5%) (p < 0.01) for mi

197 (66%) of 187 patients achieved preoperative oxygen delivery (irrespective of intervention).

198 l cerebral blood flow is abnormal, postnatal oxygen delivery is decreased, and intraoperative support

199 ications for disease states in which cardiac oxygen delivery is impaired.

200 e local oxygen saturation of hemoglobin when oxygen delivery is limiting.

201 parallel with contractile activity such that oxygen delivery is sufficient to meet metabolic demand.

202 ed blood cell (RBC) unit storage duration on oxygen delivery is uncertain.

203 experience impaired blood supply and reduced oxygen delivery, leading to altered metabolic and mechan

204 regions were defined as those with baseline oxygen delivery less than 4.5 mL/100 g/min.

205 to the same functional outcome of successful oxygen delivery, long-term persistence and high function

206 suggest that strategies to improve cerebral oxygen delivery may help reduce brain dysmaturation in n

207 g, deep breathing, and coughing.Conclusions: Oxygen delivery modalities of humidified high-flow nasal

208 spiratory tract of humans exposed to various oxygen delivery modalities.Methods: Ten healthy particip

209 ylephrine, probably because it reduced renal oxygen delivery more than did phenylephrine.

210 hagic shock, Hb-200 infusion may not improve oxygen delivery more than hetastarch, likely due to hemo

211 toregulation (hypoxic vasodilation governing oxygen delivery) observed by Guyton, to current understa

212 nary artery occlusion pressure of <18 mm Hg, oxygen delivery of >600 mL x min(-1) x m(-2), and oxygen

213 model to investigate possible limitations of oxygen delivery on size.

214 tivity of oxygen binding to Hb affect tissue oxygen delivery, only the former was thought to determin

215 O2 rather than a direct measurement of total oxygen delivery or cerebral oxygen metabolism.

216 associated with changes in renal blood flow, oxygen delivery, or histological appearance.

217 Heart rate, blood pressure, cardiac output, oxygen delivery, oxygen consumption, SMA blood flow, ile

218 cardiac output, stroke volume, and systemic oxygen delivery (p < .05 vs. controls).

219 d whether transfusion could augment cerebral oxygen delivery, particularly in vulnerable brain region

220 numerous physiological traits comprising the oxygen delivery process.

221 Any reduction in myocardial oxygen delivery relative to its demands can impair cardi

222 emodynamic stability and monitoring cerebral oxygen delivery remain important goals of perioperative

223 n (6 times basal), only minor differences in oxygen delivery resulted between the sprouting and split

224 predict whether a fluid-induced increase in oxygen delivery results in an increase in oxygen consump

225 of insufficient numbers of erythrocytes for oxygen delivery, SCD patients constantly face hypoxia.

226 of results from clinical trials, unnecessary oxygen delivery should be avoided in critically ill vent

227 of results from clinical trials, unnecessary oxygen delivery should be avoided in ventilated stroke p

228 Oxygen delivery significantly increased in these 25 pati

229 cits in RBC-mediated vasodilation to improve oxygen delivery-steps toward effective microvasculature-

230 ., the late stage), cardiac output, systemic oxygen delivery, stroke volume, total peripheral resista

231 Improved gas exchange and higher systemic oxygen delivery suggest that calpain inhibition may be a

232 Hemoglobin A (HbA), the oxygen delivery system in humans, comprises two alpha an

233 We hypothesised that individualised oxygen delivery targeted haemodynamic therapy (goal-dire

234 , but this was not affected by the use of an oxygen delivery targeted strategy.

235 a significant increase in cardiac output and oxygen delivery , the creation of an arteriovenous shunt

236 se of its essential role in gas exchange and oxygen delivery, the lung has evolved a variety of strat

237 rocytes has been investigated as a potential oxygen delivery therapeutic.

238 NOS controls blood pressure, blood flow and oxygen delivery through its effect on vascular smooth mu

239 We show that oxygen delivery through PMCs is dependent on its permeab

240 Thus, AVD ensures increased oxygen delivery to active muscle fibres by reducing upst

241 or ascending vasodilatation ensure increased oxygen delivery to active skeletal muscle.

242 st be tightly balanced to guarantee adequate oxygen delivery to all tissues in the body.

243 n and prevents neuron apoptosis by promoting oxygen delivery to brain or by direct interaction with n

244 ing, resulting in compromised blood flow and oxygen delivery to contracting skeletal muscle during ex

245 regulation of skeletal muscle blood flow and oxygen delivery to contracting skeletal muscle is comple

246 2+/-3 mL; p<or=.05), with worse matching of oxygen delivery to demand (p<.001).

247 The optimal method of oxygen delivery to donor kidneys during ex vivo machine

248 n-utero hemodynamics and increasing cerebral oxygen delivery to enhance brain development.

249 ces a systemic response designed to increase oxygen delivery to hypoxic tissues.

250 red ability of the microvasculature to match oxygen delivery to increased oxygen demand.

251 is critical to ensure proper blood flow and oxygen delivery to metabolically active skeletal muscle.

252 tory and ventilatory responses that increase oxygen delivery to muscle during exercise.

253 is not known whether VB can provide adequate oxygen delivery to restore or maintain renal function.

254 that facilitate increases in blood flow and oxygen delivery to the active tissue and the sympathetic

255 not they display differential regulation of oxygen delivery to the brain compared to lowland natives

256 ermine the quality of cerebral perfusion and oxygen delivery to the brain.

257 peripheral vasoconstriction, a reduction in oxygen delivery to the femoral circulation, worsening fe

258 o the placental vasculature that compromises oxygen delivery to the fetus.

259 l-induced focal arterial lesions that impair oxygen delivery to the heart.

260 emoglobin-based oxygen carrier (HBOC-201) in oxygen delivery to the kidney for renal protection.

261 Here we have shown that endotoxemia reduces oxygen delivery to the kidney, without changing tissue o

262 Increased oxygen delivery to the microcirculation might have contr

263 ndurance in smokers may result from impaired oxygen delivery to the mitochondria and ability of the m

264 le microvasculature and subsequent diffusive oxygen delivery to the mitochondria were diminished in p

265 r cessation of coronary blood flow such that oxygen delivery to the myocardium is insufficient to mee

266 , perfusion pressure was 5.8+/-3.3 mmHg with oxygen delivery to the organs in excess of 3.5 times the

267 During maximal KE, both convective arterial oxygen delivery to the skeletal muscle microvasculature

268 At high altitude oxygen delivery to the tissues is impaired leading to ox

269 t homeostatic regulation to maintain optimal oxygen delivery to the tissues.

270 Limited oxygen delivery to tissues (hypoxia) is common in a vari

271 rculating blood and their promise to improve oxygen delivery to tissues supports the potential for th

272 a fundamental physiologic response ensuring oxygen delivery to tissues under metabolic stress.

273 ust be tightly controlled to ensure adequate oxygen delivery to tissues without causing thrombosis or

274 ces a systemic response designed to increase oxygen delivery to tissues.

275 sidents, resulting in greater than sea level oxygen delivery to tissues.

276 ysical mechanism, losartan improves drug and oxygen delivery to tumours, thereby potentiating chemoth

277 skeletal muscle blood flow and microvascular oxygen delivery-to-utilization matching during exercise.

278 ely seem best to distribute flow to maximize oxygen delivery (total, upper body, or lower body), we f

279 ted to achieve their individual preoperative oxygen delivery value (goal-directed therapy) or standar

280 Achievement of preoperative oxygen delivery values in the postoperative phase was as

281 In these Sherpa, cerebral oxygen delivery was also lower compared to lowlanders (~

282 Cerebral oxygen delivery was calculated using phase contrast angi

283 Systemic oxygen delivery was higher after calpain inhibition comp

284 Oxygen delivery was maintained at 76 +/- 13 mL/min at ba

285 Although cerebral oxygen delivery was maintained during ascent in lowlande

286 Total systemic oxygen delivery was markedly reduced in the hybrid palli

287 We also demonstrated that single-RBC oxygen delivery was modulated by changing either the inh

288 d hybrid 475 mL . min(-1) . m(-2)). Cerebral oxygen delivery was similarly lower in the hybrid pallia

289 Volume status and oxygen delivery were assessed using transpulmonary therm

290 lesions associated with the lowest cerebral oxygen delivery were associated with the greatest impair

291 epsis maintained cardiac output and systemic oxygen delivery, whereas it increased oxygen consumption

292 serve as initial therapy to maintain tissue oxygen delivery while awaiting the maximal effect of rec

293 omes in critically ill patients by enhancing oxygen delivery while minimizing the risks of toxic effe

294 These data demonstrate that restoration of oxygen delivery with a small volume of MP4 yields signif

295 the brain susceptible to large reductions in oxygen delivery with concurrent cold stress, which might

296 oad, afterload, and contractility to balance oxygen delivery with oxygen demand.

297 By providing oxygen delivery with slow, limited infusion, new hemoglo

298 However, CO2 reactivity and oxygen delivery (with 1 exception) were not associated w

299 esions most associated with reduced cerebral oxygen delivery would demonstrate the greatest impairmen

300 ction of whether a fluid-induced increase in oxygen delivery would result in an increase in oxygen co