ライフサイエンスコーパス: oxygen saturation

1 ow supplemental oxygen to normalize arterial oxygen saturation.

2 hemoglobin concentration, blood volume, and oxygen saturation.

3 with greater alterations accompanying lower oxygen saturation.

4 percentage of sleep time with less than 90% oxygen saturation.

5 tSo2 level were poor predictors of cerebral oxygen saturation.

6 lator, in response to reduced blood arterial oxygen saturation.

7 airway hyperreactivity, and diminished blood oxygen saturation.

8 death at 36 weeks in the group with a lower oxygen saturation.

9 esults may help determine the optimal target oxygen saturation.

10 ogy but maintained normal levels of arterial oxygen saturation.

11 in tumor oxygen concentration and hemoglobin oxygen saturation.

12 not be used as surrogate for central venous oxygen saturation.

13 h cardiac index, lactate, and central venous oxygen saturation.

14 was used to measure cerebral hemoglobin and oxygen saturation.

15 ia time was not accompanied by a decrease of oxygen saturation.

16 n arterial pressure, and jugular venous bulb oxygen saturation.

17 evealed he was afebrile and had normal pulse oxygen saturation.

18 ent for thousands of RBCs from their overall oxygen saturation.

19 ial pressure cardiac index, and mixed venous oxygen saturations.

20 al pressure, cardiac index, and mixed venous oxygen saturations.

21 eaths/min [95% CI, -1.32 to 1.67]; P = .82); oxygen saturation (-0.04% [95% CI, -0.53% to 0.46%]; P =

22 ts of both temperature (11-37 degrees C) and oxygen saturation (17-206% or 3.6-43.6 kPa).

23 d venous oxygen saturation or central venous oxygen saturation; -3.7% (-4.4% to -3.0%) (p < 0.01) for

24 oreal resuscitation alone (regional cerebral oxygen saturation, 73% +/- 3% vs 52% +/- 8%; p < 0.05).

25 icant differences were found in jugular vein oxygen saturation (83.2% [79.2-87.6%] vs. 86.7% [83.2-88

26 tion and to lower or higher target ranges of oxygen saturation (85 to 89% or 91 to 95%).

27 Health Stroke Scale score, 5; mean baseline oxygen saturation, 96.6%) were enrolled.

28 It remains uncertain what values of arterial oxygen saturations achieve this balance in preterm infan

29 rebral oximetry showed fast rise in regional oxygen saturation after carbon monoxide treatment at 0.5

30 n patients treated with CPAP, mean nocturnal oxygen saturation and baseline IL-1beta were independent

31 on between obtained values of femoral venous oxygen saturation and central venous oxygen saturation (

32 is lack of agreement between femoral venous oxygen saturation and central venous oxygen saturation i

33 Results for changes of femoral venous oxygen saturation and central venous oxygen saturation w

34 Correlation and agreement of femoral venous oxygen saturation and central venous oxygen saturation w

35 luding the difference between femoral venous oxygen saturation and central venous oxygen saturation.D

36 our study was to determine if central venous oxygen saturation and femoral venous oxygen saturation c

37 n (n = 136); or level 4 (L4), which included oxygen saturation and heart rate (n = 135).

38 showed a significant decrease in local brain oxygen saturation and in brain tissue PO(2) alongside br

39 Lower oxygen saturation and intravenous iron may be modifiable

40 of BV5/TBV were directly related to resting oxygen saturation and inversely associated with both the

41 We compared the ability of central venous oxygen saturation and markers of anaerobic metabolism to

42 rived blood flow index (BFI), and (b) venous oxygen saturation and NIRS-derived tissue saturation.

43 In the presence of pain, tissue oxygen saturation and perfusion index are further reduce

44 in must be considered when evaluating tissue oxygen saturation and perfusion index as markers of hypo

45 Tissue oxygen saturation and peripheral perfusion index are pro

46 sculature and providing images of hemoglobin oxygen saturation and pulsation.

47 nic therapy can improve microcirculation and oxygen saturation and reduce vessel calibers in patients

48 atment with adenosine or ATPgammaS increased oxygen saturation and reduced histopathological signs of

49 Healthcare providers should monitor oxygen saturation and requirements during and after IS c

50 ous pressure, respiratory rate, and arterial oxygen saturation and treatment with vasopressors target

51 Daily home monitoring of oxygen saturation and weight has been reported to improv

52 ts receiving bag-mask ventilation had higher oxygen saturations and a lower incidence of severe hypox

53 lder, more often had diabetes, and had lower oxygen saturations and higher National Early Warning Sco

54 roup analysis of children by malaria status, oxygen saturation, and age.

55 with oxygenation, ventilation, mixed venous oxygen saturation, and cardiac output.

56 enting symptoms, laboratory data, peripheral oxygen saturation, and comorbid diseases, were recorded.

57 FDNIRS) to measure hemoglobin concentration, oxygen saturation, and indices of cerebral blood flow an

58 s, comorbidities, symptom type and duration, oxygen saturation, and laboratory values.

59 2), improved hemodynamics and central venous oxygen saturation, and less organ dysfunction.

60 Concentrations of algal pigments, dissolved oxygen saturation, and pH rapidly declined following ces

61 ated through histological studies, degree of oxygen saturation, and responsiveness to carbachol.

62 attering exponent, hemoglobin concentration, oxygen saturation, and sampling depth are presented alon

63 Perfusion, Intravascular Venous Oxygen saturation, and T2* (PIVOT), a recently developed

64 eters, including heart rate, blood pressure, oxygen saturation, and ventilation parameters, in inpati

65 eathing, inaccuracy of pulse oximetry at low oxygen saturations, and temperature-induced shifts in th

66 nction derived from baseline CMR and resting oxygen saturation are associated with mortality in adult

67 us oxygen saturation >70% than thenar tissue oxygen saturation (area under the curve, 0.80; 95% confi

68 tory rate >/= 25/min, PaO2 </= 50 mm Hg, and oxygen saturation (arterial [SaO2] or measured by pulse

69 Changes in tumor hemoglobin oxygen saturation as a function of time after treatment

70 ypopnea index, <30 events per hour) + (nadir oxygen saturation as measured by pulse oximetry >82.5%)

71 (Pi(O(2))) was decreased stepwise to achieve oxygen saturation as measured by pulse oximetry (Sp(O(2)

72 the two groups, the default lower limit for oxygen saturation as measured by pulse oximetry (Spo(2))

73 onservative oxygenation strategy with target oxygen saturation as measured by pulse oximetry (SpO2) o

74 ygen therapy (target Pao(2), 55 to 70 mm Hg; oxygen saturation as measured by pulse oximetry [Spo(2)]

75 dicted; FEV(1)/FVC = 31.6 +/- 7.1%; exercise oxygen saturation as measured by pulse oximetry [Spo(2)]

76 Nadir oxygen saturation as measured by pulse oximetry, apnea-h

77 Among components of the index, oxygen saturation as measured by pulse oximetry/Fi(O(2))

78 ndex (termed ROX and defined as the ratio of oxygen saturation as measured by pulse oximetry/Fi(O(2))

79 ut also essentially stabilized gas exchange (oxygen saturation) as an overall measure of lung functio

80 comes included PRAM score; respiratory rate; oxygen saturation at 60, 120, 180, and 240 minutes; bloo

81 xygen therapy was 11.6 hours, and the median oxygen saturation at the end of the treatment period was

82 Targeting an oxygen saturation below 90% with the use of current oxim

83 The primary outcome was lowest arterial oxygen saturation between induction and 2 minutes after

84 -saline group had significantly lower tissue oxygen saturation, brain tissue PO2, and venous oxygen s

85 was not predicted by baseline central venous oxygen saturation but by high baseline lactate and (P(v

86 atients underwent protocolized assessment of oxygen saturation by pulse oximetry (SpO(2) ), arterial

87 ical coherence tomography and measurement of oxygen saturation by spectrophotometry.

88 venous oxygen saturation and femoral venous oxygen saturation can be used interchangeably during sur

89 have developed a system that quantifies the oxygen saturation, cell volume, and Hb concentration for

90 ex, oxygen consumption index, central venous oxygen saturation, central venous-to-arterial carbon dio

91 HR and oxygen saturation changes were not significantly associa

92 was associated with higher regional cerebral oxygen saturation compared with manual chest compression

93 comes of time to intubation, lowest arterial oxygen saturation, complications, and in-hospital mortal

94 peritoneal BLM model as assessed by arterial oxygen saturation (control, 84.4 +/- 1.3%; C-188-9, 94.4

95 including height, weight, respiratory rate, oxygen saturation, cough, or respiratory symptom scores.

96 Delta - DeltaPCO2 and central venous oxygen saturation could predict extubation failure with

97 tation of the RUPP surgical model, placental oxygen saturation decreased 12% in comparison with NP.

98 Central venous oxygen saturation decreased in the failure group and inc

99 cyanosis confirmed by medical staff when his oxygen saturation decreased to the 60% level, and he had

100 ea was induced by stopping ventilation until oxygen saturations decreased to 80%.

101 aPCO2 (Delta - DeltaPCO2) and central venous oxygen saturation (DeltaScvO2) during spontaneous breath

102 venous oxygen saturation and central venous oxygen saturation.Despite significant correlation betwee

103 kers of anaerobic metabolism, central venous oxygen saturation did not allow the prediction of whethe

104 e)V ratios were inversely related to resting oxygen saturation, diffusing capacity of carbon monoxide

105 e primary outcome was longitudinal change in oxygen saturation divided by the FIO2 (S/F) through day

106 Dissolved oxygen saturation (%DO) decreased by -0.24 +/- 0.036% yr

107 We measured intubating conditions, oxygen saturation during and 5 mins following intubation

108 on does not seem to increase lowest arterial oxygen saturation during endotracheal intubation of crit

109 ring the temporal dynamics of blood flow and oxygen saturation during reactive hyperemia than by conv

110 -DLPFC GABA levels, but not Glx, and minimal oxygen saturation during sleep (r = 0.62, P = 0.0005).

111 Lower mean oxygen saturation during sleep was associated with atrop

112 , and cardiovascular risk factors, only mean oxygen saturation during sleep was associated with bilat

113 ation with the apnea-hypopnea index, average oxygen saturation during sleep, and average respiratory

114 easures included noninvasive cerebral tissue oxygen saturation during the first transfusion, clinical

115 nd that Delta - DeltaPCO2 and central venous oxygen saturation, during spontaneous breathing trials,

116 Clinical assessment, cerebral oxygen saturation, electrolyte abnormalities, adverse ev

117 HBOC-201 achieved tissues oxygen saturation equivalent to blood.

118 Tissue oxygen saturation (except cerebral) and perfusion index

119 ould be considered instead of central venous oxygen saturation for starting hemodynamic resuscitation

120 % (15.8 vs. 25.0%; P = 0.22), or decrease in oxygen saturation greater than 3% (53.9 vs. 55.6%; P = 0

121 supplemental oxygen in order to maintain an oxygen saturation greater than 92%.

122 of 612), versus 27.5% of those in the higher-oxygen-saturation group (171 of 622) (relative risk, 1.1

123 8), and in 30.2% of the infants in the lower-oxygen-saturation group (185 of 612), versus 27.5% of th

124 al ventilation titrated to maintain arterial oxygen saturation > 90%), "hyperoxia" (standard resuscit

125 o "control" (FIO2 0.3, adjusted for arterial oxygen saturation >/= 90%) and "hyperoxia" (FIO2 1.0 for

126 presenting with isolated fast breathing and oxygen saturation >/=90% were randomly assigned to recei

127 ation was better predictor of central venous oxygen saturation >70% than thenar tissue oxygen saturat

128 The presence of normal oxygenation (oxygen saturation >96%) decreased the likelihood of pneu

129 infants, those in the lower-target group for oxygen saturation had a reduced rate of retinopathy of p

130 R or noninvasive measurement besides resting oxygen saturation (hazard ratio, 0.90 [0.83-0.97]/%; P=0

131 ow (BF), blood volume (BV) and intravascular oxygen saturation (Hb(sat)) acquired concurrently using

132 Mean oxygen saturation, heart rate and hematocrit were not si

133 Oxygen saturation, heart rate and rhythm, and blood pres

134 Safety indicators included spirometry, oxygen saturation, heart rate, and symptoms.

135 re no significant differences in heart rate, oxygen saturation, hospitalization rate, or other outcom

136 Secondary outcomes included vital signs, oxygen saturation, hospitalization, physician clinical i

137 Pain (cold pressor test) reduces tissue oxygen saturation in all measurement sites (except cereb

138 , MSOT provided images reflecting hemoglobin oxygen saturation in blood vessels, clearly identifying

139 venous oxygen saturation and central venous oxygen saturation in both stable and unstable medical co

140 bserved higher cerebral blood flow and lower oxygen saturation in females compared to males.

141 The safest ranges of oxygen saturation in preterm infants have been the subje

142 The clinically appropriate range for oxygen saturation in preterm infants is unknown.

143 ese values are significantly higher than the oxygen saturation in seawater at the contemporary atmosp

144 on, cerebral abscess was associated with low oxygen saturation (indicating greater right-to-left shun

145 egivers to record measurements of weight and oxygen saturation into a binder and requires families to

146 on less than 90% (44.7 vs. 47.2%; P = 0.87), oxygen saturation less than 80% (15.8 vs. 25.0%; P = 0.2

147 c oxygenation and usual care in incidence of oxygen saturation less than 90% (44.7 vs. 47.2%; P = 0.8

148 opnea index (AHI) and percent nighttime with oxygen saturation less than 90% (TSat(90)) were used as

149 rapid eye movement sleep and time spent with oxygen saturation less than 90% were associated with inc

150 ), and the percentage of sleep time with the oxygen saturation less than 90%.

151 for shortness of breath or pneumonia and (2) oxygen saturation less than 92% on room air or need for

152 e oximeter reading, and of these, 10% had an oxygen saturation level below 90%.

153 ts of apnea-hypopnea index (AHI), peripheral oxygen saturation level, and number of cortical arousals

154 HSI acquisition software capable to compute oxygen saturation levels (StO2), near infrared perfusion

155 significantly higher mean regional cerebral oxygen saturation levels during cardiopulmonary resuscit

156 f deterioration) during hospitalization when oxygen saturation levels were 90% or higher.

157 en saturation, percentage of sleep time with oxygen saturation < 90%, apnea-hypopnea index, and oxyge

158 The presence of moderate hypoxemia (oxygen saturation </=96%; LR, 2.8 [95% CI, 2.1-3.6]; sen

159 Episodes of hypoxemia (pulse oximeter oxygen saturation <80%) or bradycardia (pulse rate <80/m

160 t pain, plus temperature >=38.0 degrees C or oxygen saturation <90%) presenting to a clinic underwent

161 at chest radiography, very severe pneumonia, oxygen saturation <92%, C-reactive protein >/=40 mg/L, a

162 ia and signs of severe respiratory distress, oxygen saturation <93% (when not at high altitude), mode

163 -confirmed SARS-CoV-2 infection and room air oxygen saturation <=94% whose clinicians requested remde

164 ients had confirmed SARS-CoV-2 pneumonia and oxygen saturations <90% on oxygen support with most intu

165 s with ACS, fever (>38.5 degrees C), reduced oxygen saturation (<95) and asplenia significantly diffe

166 ar systolic dysfunction had a central venous oxygen saturation<70%.

167 Oxygen saturation mapping in the intact lung yields uniq

168 h multispectral oximetry for two-dimensional oxygen saturation mapping.

169 howed some correlation between perfusion and oxygen saturation maps and the ability to sensitively mo

170 o capillaries and venules in two-dimensional oxygen saturation maps.

171 49 L/min/m; p = 0.00001), and central venous oxygen saturation (mean difference, -5.07; 95% CI, -7.78

172 ore ranging from 0 to 10, consisting of age, oxygen saturation, mean arterial pressure, blood urea ni

173 e the apparent diffusion coefficient, tissue oxygen saturation, mean transit time, and blood volume f

174 ficient (n = 11 rats per group); local brain oxygen saturation, mean transit time, and blood volume f

175 xygen saturation [Spo2] <90%) per hour, with oxygen saturation measured continuously for 48 postopera

176 he effects of hypovolemia and pain on tissue oxygen saturation (measurement sites: cerebral, deltoid,

177 casian individuals) underwent retinal vessel oxygen saturation measurements using dual-wavelength oxi

178 y has a significant impact on retinal vessel oxygen saturation measurements using dual-wavelength ret

179 High flash intensities lead to supranormal oxygen saturation measurements with a magnified effect i

180 ificant difference in mean regional cerebral oxygen saturation (median % +/- interquartile range) in

181 s in other clinical outcomes for either home oxygen saturation monitoring or home weight monitoring.

182 ed on a physiological basis (with the use of oxygen-saturation monitoring in selected infants), at 36

183 ysplasia, confirmed by means of standardized oxygen-saturation monitoring, at a postmenstrual age of

184 nds, body position, electrocardiography, and oxygen saturation (n = 136); or level 4 (L4), which incl

185 re for various home monitoring strategies of oxygen saturation (n=494) or weight (n=472), adjusting f

186 he apnea-hypopnea index (AHI), and nocturnal oxygen saturation (O2sat) parameters, and relevant comor

187 The primary outcome was the lowest oxygen saturation observed during the interval between i

188 Oxygen saturation of </=90%, tachypnea, and tachycardia

189 al dysfunction, 24-h AHI, CAI, and time with oxygen saturation of <90% were independent predictors of

190 ls, we evaluated the effects of targeting an oxygen saturation of 85 to 89%, as compared with a range

191 mmends a permissive hypoxaemic target for an oxygen saturation of 90% for children with bronchiolitis

192 with suspected myocardial infarction and an oxygen saturation of 90% or higher were randomly assigne

193 ith COVID-19 symptom onset within 7 days and oxygen saturation of 92% or greater.

194 r or need for supplemental oxygen to achieve oxygen saturation of 92% or greater.

195 breaths per min for children) and a room air oxygen saturation of 93% or more.

196 mptom onset to enrolment of 12 days or less, oxygen saturation of 94% or less on room air or a ratio

197 on during pain crisis could affect the local oxygen saturation of hemoglobin when oxygen delivery is

198 incidence of severe hypoxemia, defined as an oxygen saturation of less than 80%.

199 Patients with an oxygen saturation of less than 90% for at least 30% of t

200 tion or exposure, severe malnutrition, or an oxygen saturation of less than 90%.

201 f mannitol on brain tissue PO2 and on venous oxygen saturation of the superior sagittal sinus (n = 5

202 gen saturation, brain tissue PO2, and venous oxygen saturation of the superior sagittal sinus values

203 In extremely preterm infants, targeting oxygen saturations of 85% to 89% compared with 91% to 95

204 with mild to moderate bronchiolitis and true oxygen saturations of 88% or higher.

205 2.9% (2.2-3.5%) (p < 0.01) for mixed venous oxygen saturation or central venous oxygen saturation; -

206 We did not find any associations of home oxygen saturation or weight monitoring with mortality or

207 ry rate (OR, 1.05 [95% CI 1.03-1.07]), lower oxygen saturation (OR, 0.94 [95% CI 0.93-0.96]), and chr

208 No pulmonary emboli, alterations in oxygen saturation, or hemodynamics occurred with either

209 ygen saturation (p = .04) and deltoid tissue oxygen saturation (p = .002), and masseter tissue oxygen

210 was consistently higher than masseter tissue oxygen saturation (p = .04) and deltoid tissue oxygen sa

211 was consistently higher than deltoid tissue oxygen saturation (p = .04).

212 High brain regional oxygen saturation (P = 0.007) and low middle cerebral ar

213 ied between global brain volumes and resting oxygen saturations (P<=0.04).

214 month increase; OR 1.23, 95% CI 1.16-1.30), oxygen saturation (per 1% decrease from 100%; 1.09, 1.01

215 The other respiratory variables-lowest oxygen saturation, percentage of sleep time with oxygen

216 ed by respiratory inductive plethysmography, oxygen saturation, perfusion index, regional cerebral an

217 erial blood pressure, electrocardiogram, and oxygen saturation plethysmography activity were recorded

218 ysmography), heart rate (electrocardiogram), oxygen saturation (pulse oximetry), and brachial artery

219 a lower (85 to 89%) or a higher (91 to 95%) oxygen-saturation range.

220 s severe desaturation (defined as peripheral oxygen saturation reading < 80% during intubation).

221 wine "forced ageing" process under different oxygen saturation regimes at 60 degrees C.

222 the procedure to record oxygen requirement, oxygen saturation, respiratory rate, consciousness level

223 venous oxygen saturation and central venous oxygen saturation (rs = 0.55; p < .001), the limits of a

224 REI and oxygen saturation (SaO(2)) were primary outcomes, while

225 as a respiratory rate (RR) < 55 and room air oxygen saturation (SaO2) >/= 97%, and severe illness (n

226 Overnight nocturnal oxygen saturation (SaO2) is a clinically relevant and ea

227 t of postoperative IS on hypoxemia, arterial oxygen saturation (Sao2) level, and pulmonary complicati

228 ts and seconds with less than 90% hemoglobin oxygen saturation (Sao2) per hour of recording.

229 Distal occlusive pressure and toe oxygen saturation (Sao2) were measured for 5 minutes und

230 including end-tidal carbon dioxide (ETCO2), oxygen saturation (SaO2), intra-arterial blood pressure,

231 nd between: left thalamus mI/Cr and baseline oxygen saturation (SaO2); right putamen tCho/Cr and apne

232 ed spectroscopy (NIRS)-based cerebral tissue oxygen saturation (SctO2).

233 rstand the relationship among central venous oxygen saturation (Scv(O(2))), lactate, and base excess

234 Central venous oxygen saturation (ScvO2) in the superior vena cava is p

235 This suggests that oxygen saturation should not be the only factor in the d

236 Thus, femoral venous oxygen saturation should not be used as surrogate for ce

237 pressure, heart rate, respiratory rate, and oxygen saturation, showed no significant changes over ti

238 endered values of 3.9% to 21.2%, whereas the oxygen saturation (sO(2)) rate declined at 1.7% to 2.3%

239 Assessing oxygen saturation (sO(2)) remains challenging but is non

240 mor total hemoglobin concentration (THb) and oxygen saturation (sO(2)) were imaged in control and bev

241 investigate alterations in retinal vascular oxygen saturation (SO(2)), vessel diameter (D) and tortu

242 pectroscopy (FDNIRS-DCS) to measure cerebral oxygen saturation (SO2) and an index of cerebral blood f

243 y of PAI for analysis of placental and fetal oxygen saturation (sO2) in mice.

244 e used to compare change in tumor hemoglobin oxygen saturation (sO2) levels and BOLD signal in respon

245 oxyhemoglobin (HbO2), deoxyhemoglobin (HbR), oxygen saturation (sO2), blood flow (BF) and rate of oxy

246 rameters, the blood flow rate and hemoglobin oxygen saturation (sO2), must be measured together.

247 ding total hemoglobin concentration (C(Hb)), oxygen saturation (sO2), sO2 gradient (VsO2), flow speed

248 ges in portable oximeter-measured peripheral oxygen saturation (Spo(2) or O(2)sat) in older adults be

249 ficant differences in reductions in arterial oxygen saturation (SpO(2)) between the sexes.

250 pressure (DBP), rate-pressure product (RPP) oxygen saturation (SpO(2)), and heart rate variability (

251 The optimal oxygen saturation (SpO2) target for extremely preterm in

252 ences in mortality rates using <90% and <93% oxygen saturation (SpO2) thresholds and World Health Org

253 ailure (partial oxygen pressure <60 mm Hg or oxygen saturation [SpO2] </=90% when breathing room air

254 the total duration of hypoxemia (hemoglobin oxygen saturation [Spo2] <90%) per hour, with oxygen sat

255 eters (patients treated with oxygen if pulse oxygen saturation [SpO2] <94%) or modified oximeters (di

256 line flights (decreased peripheral capillary oxygen saturation [SpO2] and increased radiation exposur

257 r patients with PBC exhibit reduced cerebral oxygen saturation (StO(2) ) and altered patterns of micr

258 aneous reflectance spectrophotometry (venous oxygen saturation StO2 and relative tissue hemoglobin co

259 h native endobronchial tissues, donor tissue oxygen saturations (Sto2) were reduced in the upper lobe

260 he cohort of 17 patients with baseline tumor oxygen saturation (%StO2) greater than the 77% populatio

261 Perfusion, venous oxygen saturation SvO2, and T2* were each quantified in

262 nagement of infants with bronchiolitis to an oxygen saturation target of 90% or higher is as safe and

263 Use of an oxygen-saturation target range of 85 to 89% versus 91 to

264 d assess the benefits and risks of different oxygen saturation targets in acute respiratory infection

265 n and to base subsequent oxygen titration on oxygen saturation targets.

266 A drop in oxygen saturation that required supplemental oxygen was

267 es of diffusing capacity of carbon monoxide, oxygen saturation, the 6-minute-walk distance, St George

268 onocyte count, and ratio of peripheral blood oxygen saturation to fraction of inspired oxygen (SpO(2)

269 al pressure of oxygen or arterial hemoglobin oxygen saturation to individualized target values, with

270 ume (respiratory inductive plethysmography), oxygen saturation, transcutaneous carbon dioxide, and in

271 oembolism; oxygen therapy for those with low oxygen saturation; treatment of left ventricular failure

272 turation in cerebral tissue (cerebral tissue oxygen saturation [tSo2]) before, during, and after bloo

273 PAT was demonstrated by mapping the cerebral oxygen saturation via multi-wavelength irradiation in be

274 Lower oxygen saturation, viral detection, and comorbidities we

275 s 0.94 (0.89-1.05) (p = 0.027), jugular vein oxygen saturation was 79.2 (71.1-81.8) versus 83.3% (76.

276 Median lowest arterial oxygen saturation was 92% with apneic oxygenation versus

277 given via nasal tubes at 3 L/min if baseline oxygen saturation was 93% or less and at 2 L/min if oxyg

278 the 401 patients enrolled, the median lowest oxygen saturation was 96% (interquartile range, 87 to 99

279 curves analyses showed that masseter tissue oxygen saturation was better predictor of central venous

280 Retinal arteriolar and venular oxygen saturation was comparable at flash settings of 27

281 n saturation (p = .002), and masseter tissue oxygen saturation was consistently higher than deltoid t

282 the 6-hr resuscitation period, thenar tissue oxygen saturation was consistently higher than masseter

283 Regional blood oxygen saturation was determined by near-infrared spectr

284 ctal pulse oximetry, while regional cerebral oxygen saturation was estimated using near-infrared spec

285 The tissue oxygen saturation was evaluated using a recently develop

286 saturation was 93% or less and at 2 L/min if oxygen saturation was greater than 93%.

287 t was highly constrained, and red blood cell oxygen saturation was low and inappropriately variable.

288 eter in the Australian trial and those whose oxygen saturation was measured with a revised oximeter i

289 is outcome was evaluated among infants whose oxygen saturation was measured with any study oximeter i

290 The blood oxygen saturation was negatively correlated with the lev

291 ts at H0 and H6, whereas mean central venous oxygen saturation was preserved but significantly lower

292 venous oxygen saturation and central venous oxygen saturation were assessed, including the differenc

293 the vastus lateralis and intercostal muscle oxygen saturation were higher in IE than CLE (p = 0.014

294 rebral artery flow velocity and jugular vein oxygen saturation were measured at the end of each step.

295 ow velocities using Doppler and jugular vein oxygen saturation were measured in both strategies 18 ho

296 Heart rate (HR) and oxygen saturation were monitored.

297 venous oxygen saturation and central venous oxygen saturation were similar.

298 nopathy of prematurity when lower targets of oxygen saturation were used.

299 pressure, heart rate, respiratory rate, and oxygen saturation) were collected at the same time point

300 n the redox chemistry of Cu species, than at oxygen saturation, where the value was 0.6.

301 ral microvascular blood flow and dynamics of oxygen saturation with Perfusion, intravascular SvO2, an