ライフサイエンスコーパス: pain perception

1 ization (CS; alteration and amplification of pain perception).

2 en GBOs and the cortical activity subserving pain perception.

3 nwanted abnormalities in mechanosensation or pain perception.

4 entral role in cognition, affective mood and pain perception.

5 nd's adjuvant (CFA), without affecting basal pain perception.

6 ent chest pain caused by heightened coronary pain perception.

7 tigated whether vision of the body modulates pain perception.

8 E(2), and capsaicin as well as reduced cold pain perception.

9 endogenous nNOS-2 activity acted to minimize pain perception.

10 (SNP) rs563649 and individual variations in pain perception.

11 n, context, injury) can separately influence pain perception.

12 triction, reduction of edema, and diminished pain perception.

13 rve as a neural center for the modulation of pain perception.

14 nsity on afferent nociceptive processing and pain perception.

15 te to subsequently emerging abnormalities in pain perception.

16 ical responses, including blood clotting and pain perception.

17 a significant role of top-down processes in pain perception.

18 upporting the behavioural results of reduced pain perception.

19 be important targets for agents that modify pain perception.

20 n endogenous cannabinoid tone that modulates pain perception.

21 laces VRs in a much broader perspective than pain perception.

22 lgesic consumption was generally parallel to pain perception.

23 early places VR1 in a much broader role than pain perception.

24 ies in locomotor activity, visual tasks, and pain perception.

25 neuropathic pain, as well as in fundamental pain perception.

26 ermal pain thresholds are related to anginal pain perception.

27 that this suppression is causally related to pain perception.

28 ate the spinal cord dorsal horn and modulate pain perception.

29 sic and counteranalgesic pathways modulating pain perception.

30 e control of memory, cognition, movement and pain perception.

31 ntify neuroanatomical correlates of abnormal pain perception.

32 spinal projection neurons are essential for pain perception.

33 erse health outcomes that include heightened pain perception.

34 stem dynamics, each contributing uniquely to pain perception.

35 our understanding of the basic physiology of pain perception.

36 tion, and descending modulation circuits for pain perception.

37 effects of zona incerta stimulation on human pain perception.

38 d behaviors including feeding, sleeping, and pain perception.

39 ive affective qualities of acute and chronic pain perception.

40 ation in which listening to music influences pain perception.

41 hin the aMCC highlight a change in affective pain perception.

42 ease of activation in brain areas related to pain perception.

43 ies nociceptive input to the brain, altering pain perception.

44 knee-joint and articular cartilage, reduced pain perception.

45 had no mental illness or disorder affecting pain perception.

46 uch as energy balance, stress responses, and pain perception.

47 associated with within- and interindividual pain perception.

48 , whereas resting ANS activity can influence pain perception.

49 ror size has an immediate functional role in pain perception.

50 for the sensory and affective components of pain perception.

51 sal horn is a key regulator of physiological pain perception.

52 psy, musculo-skeletal anomalies, and altered pain perception.

53 chanism for their integration underlying our pain perception.

54 e tone, motor coordination, respiration, and pain perception.

55 hich may contribute to altered interoceptive pain perception.

56 siological roles, including cold sensing and pain perception.

57 bfamily, member 1, a nociceptor for heat and pain perception.

58 brainstem circuitry responsible for altering pain perception.

59 sting a molecular link between EGFR/KRAS and pain perception.

60 t afferent nociceptive signals into a stable pain perception.

61 n the brain's own mechanisms for controlling pain perception.

62 , weight of surgical specimen, and patients' pain perception.

63 we investigated which computations underlie pain perception.

64 of nociceptive inputs by SpVc, and regulate pain perception.

65 g of the functional postnatal development of pain perception.

66 channel subtype that has been implicated in pain perception.

67 al nociceptive circuit and are essential for pain perception.

68 al nociceptive network and are essential for pain perception.

69 close relationship between inflammation and pain perception.

70 ffective and cognitive factors can influence pain perception.

71 nomas and is implicated in heart failure and pain perception.

72 ems become sensitized, leading to heightened pain perception.

73 domains including working memory, mood, and pain perception.

74 phin that is implicated in the modulation of pain perception.

75 le contribution to individual differences in pain perception.

76 ntense than the aversiveness associated with pain perception.

77 alysis on neuroimaging studies of empathetic pain perception.

78 ntitatively the temporal dynamics of thermal pain perception.

79 ansient receptor potential (TRP) channels in pain perception?

80 question: does statistical learning modulate pain perception?

81 We compared pain perception (100 mm visual analogue scale), muscle s

82 bo hyperalgesia is an increase in subjective pain perception after a patient or subject underwent an

83 There are limited data on pain perception after periodontal or implant surgery or

84 regulate diverse brain functions, including pain perception, alcoholism, and substance addiction.

85 at while inequality per se did not influence pain perception, altruistic behavior had an intrinsic an

86 dulation (CPM), may play a role in enhancing pain perception among some RA patients.

87 e, painful stimulation can lead to increased pain perception and activation in pain-processing brain

88 opioid receptor (OPRM1) plays a key role in pain perception and addiction.

89 in motor control, in motor behavior, and in pain perception and also predict involvement of Go in Ca

90 ting many physiological functions, including pain perception and analgesia, responses to stress, aggr

91 Secondary outcomes of patient pain perception and analgesic intake were also evaluated

92 These results further the understanding of pain perception and are potentially relevant for the dec

93 rticularly in regions of the cord related to pain perception and autonomic tone.

94 gated the effects of attention on esophageal pain perception and brain activity.

95 of controllability and uncontrollability on pain perception and cerebral pain processing.

96 genetic polymorphisms are mediators of human pain perception and clinical pain phenotypes.

97 stand the ultimate role of this structure in pain perception and descending control of pain.

98 king to explore the role of baroreceptors in pain perception and descending modulation.

99 worms-from mate attraction and aggression to pain perception and empathy.

100 These results are evidence that pain perception and engagement of endogenous opioid syst

101 Our study indicates for the first time that pain perception and expectation elicit different hemodyn

102 te pain associated with differences in acute pain perception and fibromyalgia symptoms.

103 While the relation between pain perception and homeostatic regulation of bodily sys

104 is fundamental to our understanding of both pain perception and how opioids modulate pain.

105 le of voltage-gated sodium channel Nav1.7 in pain perception and how we can advance our understanding

106 tablished the participation of the cortex in pain perception and identified a long list of brain stru

107 h a role for neuronal polyamine transport in pain perception and identify a target for therapeutic in

108 significant role of higher-order schemas in pain perception and indicates that pain perception is bi

109 ABAergic neurotransmission, is implicated in pain perception and is a potential marker of individual

110 common features that reliably contribute to pain perception and its modulation.

111 eptor (KOR) may represent a means to control pain perception and modulate reward thresholds.

112 he subthalamic nucleus is a key structure in pain perception and modulation.

113 to, as well as coordinate, effects of SP on pain perception and mood.

114 ntially valuable and quantifiable markers of pain perception and placebo response.

115 pectations and confidence were used to weigh pain perception and prediction.

116 for modulation of neural systems subserving pain perception and processing in Parkinson's disease.

117 Pain perception and QOL were evaluated by using specific

118 eatures, we developed a system for detecting pain perception and reaction in the brain, which success

119 that low dose ketamine administration blunts pain perception and reduces blood pressure, but not musc

120 rtex (rACC), a region putatively involved in pain perception and regulation.

121 Pain perception and related cortical activation patterns

122 whereas their acute activation reduces basal pain perception and relieves inflammatory and neuropathi

123 sly reported potential gender differences in pain perception and reporting, reinforces that gender di

124 tion between the brain mechanisms underlying pain perception and representation of the body.

125 other populations, our results suggest that pain perception and severity are important when evaluati

126 ssociated with thermoregulation, action, and pain perception and showed positive functional connectiv

127 The relation between pain perception and spatial representation of the body i

128 s finding provides fresh insights into human pain perception and suggests a new avenue for the develo

129 ning the trial-by-trial relationship between pain perception and task performance revealed that pain'

130 re associated with larger trade-offs between pain perception and task performance, suggesting that th

131 te diverse physiological responses including pain perception and the control of vascular tone.

132 vations illustrate the complexity of nNOS in pain perception and the existence of opposing nNOS syste

133 numerous physiological functions, including pain perception and the motivational drive associated wi

134 Continued research into the neurobiology of pain perception and the placebo effect has also played a

135 relationship, although their involvement in pain perception and their functioning in chronic pain ha

136 MC1R variants may influence orofacial pain perception and, in turn, predispose individuals to

137 ls are believed to play an important role in pain perception, and anesthetic steroids such as alphaxa

138 y directly changes breathing rate, affective pain perception, and anxiety.

139 and impairments in cognitive function, mood, pain perception, and autonomic activity.

140 and (b) evaluate the quality of life (QOL), pain perception, and efficacy in terms of time to local

141 ntromedial medulla bidirectionally influence pain perception, and locus coeruleus activity mirrors th

142 ponse, muscle atrophy, exercise intolerance, pain perception, and mitochondrial energy metabolism.

143 reatment sessions and surgical intervention, pain perception, and procedure time.

144 HCN channel activation contributes to pain perception, and propofol, a widely used anesthetic,

145 enabled light-inducible inhibition of acute pain perception, and reversed mechanical allodynia and t

146 ver activation in rACC leads to control over pain perception, and that these effects were powerful en

147 play important roles in cognitive function, pain perception, and the reinforcing properties of nicot

148 lved in modulation of dopaminergic circuits, pain perception, and thermoregulation.

149 g the cingulate cortex's role in suppressing pain perception, any harm inflicted upon this region of

150 Changes in pain perception are a feature of C9orf72 expansion carri

151 the dynamics of neuronal activity underlying pain perception are not fully known.

152 Individual differences in pain perception are of interest in basic and clinical re

153 ments strongly suggest that these changes in pain perception are predominantly based on altered perce

154 ensory neurons transducing thermal, itch and pain perception are specified in early development is un

155 Pain perception arises from the integration of prior exp

156 Thus, our finding suggests pain perception as an aspect of the nervous system that

157 Predictive cues dynamically modulated pain perception as contingencies changed, regardless of

158 eta-band power are covarying with subsequent pain perception, as well as lowered frontolateral theta-

159 (NSAIDs) and demographic characteristics on pain perception associated with panretinal photocoagulat

160 The VAS showed a significantly lower pain perception at 6 h after the surgery for the DEX + E

161 enhanced sensitivity to morphine in tests of pain perception attributable to impaired desensitization

162 but not nitroglycerin reduced aggregate and pain perception averaged over four distention levels.

163 athways specific to chronic and acute sickle pain, perception-based targets of "top-down" mechanisms

164 ect cortical processing involved directly in pain perception, because their magnitude correlates with

165 servationally learned information can affect pain perception, both consciously and non-consciously.

166 7 is therefore not only essential for normal pain perception but also for normal C-LTMR function, coo

167 the importance of expectations in modulating pain perception, but in everyday life we don't need an e

168 e encoding in the periaqueductal gray during pain perception, but no cue or prediction error-related

169 Expectations substantially influence pain perception, but the relationship between positive a

170 egative expectations substantially influence pain perception, but their relationship remains unclear.

171 -gated sodium channel is implicated in human pain perception by genetics.

172 nnabinoids may affect memory, cognition, and pain perception by means of this cellular mechanism.

173 pose that the DLPFC exerts active control on pain perception by modulating corticosubcortical and cor

174 It is known that pain perception can be altered by mood, attention and co

175 Pain perception can be powerfully influenced by an indiv

176 creased mortality, but it is unknown whether pain perception can directly affect aging.

177 thyltransferase (COMT) is a key regulator of pain perception, cognitive function, and affective mood.

178 All interventions significantly decreased pain perception compared to a hemostatic collagen sponge

179 these effects of prestimulus connectivity on pain perception covary with pain-relevant personality tr

180 ls do not allow for consistent modulation of pain perception depending on movement.

181 from cognitive schemas continue to influence pain perception despite increasing prediction errors ari

182 ide range of behaviors, including cognition, pain perception, drug addiction, and memory consolidatio

183 reas related to the consequence of increased pain perception during CS, we found that only cortical a

184 e 1:100 000 to a physiologic level decreases pain perception during periocular, subcutaneous anesthes

185 ve activity and haemodynamics, but decreased pain perception, during a cold pressor test compared wit

186 e sensors and restoring tactile perceptions, pain perception dynamics and its decoding using effectiv

187 Pain perception elicited late theta and alpha activity (

188 It is well known that pain perception for patients and normal subjects can be

189 While the static magnitude of thermal pain perception has been shown to follow a power-law fun

190 Pain perception has evolved as a warning mechanism to al

191 citance on atrial defibrillation success and pain perception has not been studied in patients.

192 de range of functions, including tactile and pain perception, hearing, proprioception, and control of

193 ons and to an efficacious attenuation of the pain perception in an in vivo model of NP.

194 showed greatly reduced thermal inflammatory pain perception in AQP1(-/-) mice evoked by bradykinin,

195 Neural correlates of altered pain perception in C9orf72 expansion carriers were the b

196 oved efficacy of almost 10-fold in relieving pain perception in diabetic neuropathic rats as compared

197 Hypervigilance is considered important in pain perception in functional gastrointestinal disorders

198 ur understanding of the neural correlates of pain perception in humans has increased significantly si

199 connectivity patterns related to subsequent pain perception in humans, we contrasted painful with no

200 e that variation in the TRPA1 gene can alter pain perception in humans.

201 oint to a mechanism by which the body blocks pain perception in moderate states of tissue damage, all

202 n in participants with no-pain but increases pain perception in participants with chronic pain.

203 ithin the central nervous system, increasing pain perception in patients.

204 A number of potential mechanisms underlie pain perception in PCNL, and these mechanisms can be lev

205 ltered and whether it is related to abnormal pain perception in people with FM.

206 score of ankle and knee joints and enhanced pain perception in the C-Ab induced RA animals. Ashwashi

207 ecally administered 8d significantly reduced pain perception in the formalin model of rat sensory ner

208 ed significant alterations in locomotion and pain perception in the knock-out mouse.

209 During pain perception, in the absence of any treatment, an exp

210 In mice, it attenuates visceral pain perception, indicating an antinociceptive effect, p

211 We argue that pain perception involves some of the representational pr

212 tion of the stress hormone axis by conscious pain perception is a likely explanation, but the magnitu

213 after periodontal or implant surgery or how pain perception is affected by presurgical anxiety.

214 r periodontal surgery and implant treatments pain perception is affected by the level of presurgical

215 chemas in pain perception and indicates that pain perception is biased more toward predictions and le

216 Pain perception is decreased by shifting attentional foc

217 Pain perception is one of the most complicated measurabl

218 Pain perception is strongly influenced by descending pat

219 jor factor that influences the likelihood of pain perception is the threshold for activation of nocic

220 Sex dependency in pain perception is well documented and is thought to be

221 ce has severe consequences on TRPV1-mediated pain perception leading to altered capsaicin consumption

222 nmental and endogenous irritants to initiate pain perception, local inflammation, and protective beha

223 nd blood, many of which were associated with pain perception/maintenance (SRP14/BMF, GDAP1, MLLT10, B

224 Visceral hyperalgesia or heightened central pain perception may contribute to pain in chronic pancre

225 Mean pain perception measured with a visual analog scale decr

226 rectal barostat studies to evaluate visceral pain perception measured with a visual analog scale.

227 ll as potentially contributing to changes in pain perception, memory and synaptic plasticity.

228 sex differences in somatic but not visceral pain perception, motility, and central processing of vis

229 Ratings of muscle pain perception (MPP) and perceived exertion (RPE), musc

230 s disorder (PTSD) is associated with altered pain perception, namely increased pain threshold and hig

231 ng machine-learning algorithms to infer that pain perception occurred.

232 ce and hyperalgesia, without affecting basal pain perception or locomotor functions.

233 ence in early palatal wound healing, patient pain perception, or analgesic consumption between use of

234 l intensity result in minimal habituation of pain perception (over 60 s) and minimal stimulation arte

235 all test groups indicated significant lower pain perception (P < 0.0001), lower analgesic consumptio

236 urgical specimen (P = 0.54) or the patients' pain perception (P = 0.28).

237 differences between repeated VAS scores for pain perception (P = 0.91) or anxiety (P = 0.75) from tw

238 There were significant improvements in pain perception (P:=0.026) and role limitation resulting

239 ility in the GABAB pathway of inhibition, in pain perception pathways via opioid receptors, and is al

240 The data of pain perception (PP), quantity of analgesics (QA), secon

241 cortex-spinal connectivity directly changes pain perception (r = 0.55).

242 on in acute pain decreases the activation in pain perception regions while activating the pain modula

243 malities encompass emotional, autonomic, and pain perception regions, implying that they likely play

244 ut the extent to which schemas can influence pain perception relative to bottom-up sensory inputs and

245 We propose that human pain perception relies on an ancient chemical sensor con

246 portance of genes versus experience in human pain perception remains unclear, rodent populations disp

247 oid hormone signaling to insulin release and pain perception, respectively.

248 enetic variability clearly appears to affect pain perception, response to analgesics and predispositi

249 th exaggerated (orthostasis) and suppressed (pain perception) responses, compared with healthy young

250 id receptor agonists alter food consumption, pain perception, responses to stress, and drug reward.

251 anism underlying the effects of ELP on adult pain perception.SIGNIFICANCE STATEMENT Pain and stress i

252 cotransporter NKCC1 in hearing, salivation, pain perception, spermatogenesis, and the control of ext

253 the CNS and can serve as a means to modulate pain perception, stress responses, and affective reward

254 result in deficits in explicit and implicit pain perception, supporting a critical role of anterior

255 subdivisions for monetary reward and thermal pain perception tasks: pshell signaling impending pain a

256 Pain perception temporarily exaggerates abrupt thermal s

257 ing the fundamental mechanism that underlies pain perception, these new results indicate that decepti

258 s stimuli leads to a progressively increased pain perception; this temporal summation is enhanced in

259 d with angina; symptoms have been related to pain perception thresholds.

260 e hypothesis that catastrophizing influences pain perception through altering attention and anticipat

261 atastrophizing has been suggested to augment pain perception through enhanced attention to painful st

262 o be equally diverse and range from roles in pain perception to male aggression.

263 ain and helping understand how VR influences pain perception.Trial registration: NCT04245475.

264 cending pain modulatory system (DPMS) shapes pain perception via monoaminergic modulation of sensory

265 The role of H(3) receptors in regulating pain perception was further demonstrated using other str

266 In all participants, pain perception was higher in females (p < 0.001), patie

267 Pain perception was measured at the same time intervals

268 Heat pain perception was never affected.

269 when prediction errors (PEs) increased, but pain perception was only partially updated when measured

270 Pain perception was separately scored to a pain-inducing

271 rgeting the human Nav1.7 channel involved in pain perception, we present a protein-engineering strate

272 vity of the somatosensory system involved in pain perception, we show that Cav3.2 channels are expres

273 y hypnotizable subjects (HHs) who eliminated pain perception were included in the present study.

274 In contrast, no changes in coordination or pain perception were observed using the rotarod or hot-p

275 Presurgical anxiety and surgical pain perceptions were measured by visual analog scale (V

276 cephalographic (EEG) response correlate with pain perception when stimuli are presented in isolation,

277 ion to allow reemergence of consciousness or pain perception, whether resumption of cardiac activity

278 tations and prediction errors is crucial for pain perception, which suggests that aversive prediction

279 brain NMDA receptors can therefore influence pain perception, which suggests that forebrain-selective

280 strongly supports the phenomenon of reduced pain perception whilst attention is distracted away from

281 ing studies to date have confounded visceral pain perception with anticipation.

282 depression undergoing DBS during two tasks: pain perception with painful/non-painful scenarios and a