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4 c signaling pathways in fibroblasts from the palmar and nonpalmar dermis of Dupuytren's patients and
6 ling arthritic changes, marked osteoporosis, palmar and plantar subcutaneous nodules and distinctive
8 rome include multiple basal cell carcinomas, palmar and/or plantar pits, odontogenic keratocysts, ske
11 measure the pressure-derived function of the palmar arch and forearm arterial collateral circulation
12 irect invasive hemodynamic assessment of the palmar arch and forearm arterial function reveals collat
14 Before invasive CFI measurements, arterial palmar arch and forearm function was tested noninvasivel
19 romoted differentiation into specifically of palmar dermal fibroblasts from Dupuytren's patients in t
20 ith the peak systolic velocity of the second palmar digital artery (Pearson coefficient: 0.621; p < 0
21 CR) analysis and immunohistochemistry of the palmar epidermis demonstrated significantly increased ex
22 ratum granulosum of both normal and affected palmar epidermis, indicating that the altered AQP5 prote
24 ive fibroproliferative disease affecting the palmar fascia of the hands, causing fingers to irreversi
25 disabling fibroproliferative disorder of the palmar fascia, which leads to flexion contractures of th
33 mediate phenotype most readily identified by palmar hyperlinearity and in some cases fine-scale and/o
38 ong adduction of the thenar, hypothenar, and palmar interosseous muscles offer powerful rigidity to t
39 il thickening in PC-K6a and PC-K17; (3) more palmar keratoderma in PC-K16; (4) cysts primarily in PC-
41 pe of EPP which is characterized by seasonal palmar keratoderma, relatively low erythrocyte protoporp
43 he new species exhibits the oldest record of palmar (or true) opposition of the pollex, which is unpr
47 nt but required early dose reductions due to palmar plantar erythrodysesthesia, and liver decompensat
51 worse treatment-related adverse events were palmar-plantar erythrodysaesthesia (1 [<1%] of 367 patie
52 ing placebo, the most frequent of which were palmar-plantar erythrodysaesthesia (13 [10%] vs 0), hype
53 , fatigue (24 [8%]), dyspnoea (21 [7%]), and palmar-plantar erythrodysaesthesia (18 [6%]) in the sora
54 vs 39 [12%] of 320 in the sunitinib group), palmar-plantar erythrodysaesthesia (25 [8%] vs 26 [8%]),
55 ase (37 [9%] vs eight [4%] vs 18 [10%]), and palmar-plantar erythrodysaesthesia (35 [8%] vs 17 [8%] v
57 n with sorafenib than with axitinib included palmar-plantar erythrodysaesthesia (PPE; 37 [39%] of 96
58 %]), diarrhoea (109 [27%] vs 105 [54%]), and palmar-plantar erythrodysaesthesia (seven [2%] vs 100 [5
59 d to erdafitinib were stomatitis (25 [12%]), palmar-plantar erythrodysaesthesia syndrome (12 [6%]), a
60 ] vs 7 [2%]), fatigue (36 [11%] vs 24 [7%]), palmar-plantar erythrodysaesthesia syndrome (27 [8%] vs
61 5%] of 269 patients in the sorafenib group), palmar-plantar erythrodysaesthesia syndrome (33 [12%] vs
62 5%] of 269 patients in the sorafenib group), palmar-plantar erythrodysaesthesia syndrome (33 [12%] vs
63 hoea (103 [21%] of 488 patients) followed by palmar-plantar erythrodysaesthesia syndrome (87 [18%]),
64 fatigue in the axitinib arm, and diarrhoea, palmar-plantar erythrodysaesthesia, and alopecia in the
66 atigue (6% v 15%), hypertension (28% v 22%), palmar-plantar erythrodysesthesia (8% v 4%), and hematol
67 a (n = 69 [48.6%]), nausea (n = 65 [45.8%]), palmar-plantar erythrodysesthesia (n = 62 [43.7%]), cons
69 cause of adverse events related to the drug (palmar-plantar erythrodysesthesia [PPE], n = 3; asthenia
71 worse treatment-related adverse events were palmar-plantar erythrodysesthesia syndrome (18 [10%] in
73 fatigue, hypertension, febrile neutropenia, palmar-plantar erythrodysesthesia syndrome, and stomatit
74 ts in the tucatinib group included diarrhea, palmar-plantar erythrodysesthesia syndrome, nausea, fati
75 ncidences of diarrhea, nausea, vomiting, and palmar-plantar erythrodysesthesia were higher with lapat
77 mg twice per day; n = 1); grade 3 mucositis, palmar-plantar erythrodysesthesia, and hypokalemia (400
79 of whom had three dose-limiting toxicities: palmar-plantar erythrodysesthesia, cerebral ischaemia, a
80 associated adverse events included diarrhea, palmar-plantar erythrodysesthesia, decreased weight and
81 5% of patients) were diarrhea, nausea, rash, palmar-plantar erythrodysesthesia, mucositis, vomiting,
82 as 1,657 mg/m2/d with limiting toxicities of palmar-plantar erythrodysesthesia, nausea, vomiting, ver
83 ction, elevated thyroid stimulating hormone, palmar-plantar erythrodysesthesia, weight loss, and head
84 grade 3 events were skin toxicity (rash and palmar-plantar erythrodysesthesia; five [4%]) and hypert
86 on in a family with diffuse nonepidermolytic palmar-plantar keratoderma was shown to be the loss in o
87 Ewing sarcoma, three [7%] for osteosarcoma), palmar-plantar syndrome (three [7%] for Ewing sarcoma, t
90 ressed in ectoderm-derived appendages and in palmar/plantar epidermis and is robustly induced when th
96 , radioscapholunate, dorsal radiotriquetral, palmar scaphotriquetral, and dorsal scaphotriquetral lig
97 Results were compared to tape strips from palmar skin of age/race/sex-matched healthy controls (HC
98 By simulating skin deformations across the palmar surface of the hand and tiling it with receptors
99 Injection of capsaicin into the plantar or palmar surface of the paws produced a depression of brad
104 rgone AHSCT, namely finger pad inflammation, palmar violaceous papules, and digital ulcerations.
105 rgone AHSCT, namely finger pad inflammation, palmar violaceous papules, and digital ulcerations.