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1 psoriasis," "pustular," "skin," "rash," and "palmoplantar." All relevant articles in English were rev
2 he protein expression levels of DKK1 between palmoplantar and non-palmoplantar areas and the effects
3 p may help efforts to reproduce them at both palmoplantar and nonpalmoplantar body sites.
4  can cause Olmsted syndrome characterized by palmoplantar and periorificial keratoderma, itch, and ha
5                Human and mouse WNT10A mutant palmoplantar and tongue epithelia also display specific
6 pression differences between volar (ventral; palmoplantar) and nonvolar (dorsal) human skin.
7 ffering types of psoriasis (plaque, guttate, palmoplantar, and late-onset), which excluded the possib
8 xplain why melanocytes stop migrating in the palmoplantar area during human embryogenesis.
9  levels of DKK1 between palmoplantar and non-palmoplantar areas and the effects of DKK1 on melanocyte
10 apping skin disorders: multiple self-healing palmoplantar carcinoma (MSPC) and familial keratosis lic
11 verlap exists between different inflammatory palmoplantar diseases that supersedes clinical and histo
12                           In addition to the palmoplantar distribution seen in other epidermolysis bu
13 idermolytic palmoplantar keratoderma (PPK or palmoplantar ectodermal dysplasia type III) is associate
14 moplantar psoriasis (palmPP) and dyshidrotic palmoplantar eczema (DPE).
15   We demonstrate here a role for AQP5 in the palmoplantar epidermis and propose that the altered AQP5
16 hanisms underlying hyperproliferation of the palmoplantar epidermis in both physiological and disease
17                                          The palmoplantar epidermis is a specialized area of the skin
18  protein which is predominantly expressed in palmoplantar epidermis is implicated in AD may shed new
19 ferentiation, and help explain why DKK1-rich palmoplantar epidermis is paler than non-palmoplantar ep
20 ich palmoplantar epidermis is paler than non-palmoplantar epidermis via mesenchymal-epithelial intera
21 clusively in the suprabasal keratinocytes of palmoplantar epidermis, have previously been demonstrate
22  focal epidermolysis in the spinous layer of palmoplantar epidermis, in comparison with cases of bull
23 riate keratoderma particularly affecting the palmoplantar epidermis, woolly hair and a dilated left v
24  is confined to the suprabasal layers of the palmoplantar epidermis.
25 lele is sufficient for the developing normal palmoplantar epidermis.
26  and maintaining the mechanical integrity of palmoplantar epidermis.
27 anges, with conjunctival injection (n = 21), palmoplantar erythema (n = 18), lip hyperemia (n = 17),
28 order characterized by recurrent episodes of palmoplantar erythema and epidermal peeling.
29 iting, diarrhea, skin rashes, mucositis, and palmoplantar erythrodysesthesias.
30                            Understanding how palmoplantar features develop may help efforts to reprod
31                                              Palmoplantar fibroblasts significantly suppressed the gr
32                    The melanocyte density in palmoplantar human skin (i.e., skin on the palms and the
33 ual facies, dental anomalies, hypotrichosis, palmoplantar hyperkeratosis and onychodysplasia, syndact
34 f life, affected individuals later developed palmoplantar hyperkeratosis with patchy erythema and sca
35 odermal dysplasia characterized by alopecia, palmoplantar hyperkeratosis, and nail dystrophy.
36 m periods the skin may be normal, except for palmoplantar hyperkeratosis.
37 y the triad of nail dystrophy, alopecia, and palmoplantar hyperkeratosis.
38 alopecia, severe dystrophy of the nails, and palmoplantar hyperkeratosis.
39 which is characterized by diffuse, yellowish palmoplantar hyperkeratosis.
40    For example, the transcriptome of in vivo palmoplantar (i.e., volar) epidermis is globally unique,
41                         The establishment of palmoplantar identity occurs through keratinocyte-fibrob
42 t, likely contributing to a unique niche for palmoplantar inflammatory diseases.
43                                          The palmoplantar keratinization and esophageal cancer syndro
44 in a patient with autosomal dominant striate palmoplantar kerato-derma.
45                                Epidermolytic palmoplantar keratoderma (EPPK) is an autosomal dominant
46                                Epidermolytic palmoplantar keratoderma (EPPK) is one of >30 autosomal-
47                                Epidermolytic palmoplantar keratoderma (EPPK, MIM #144200) is an autos
48 a congenita (PC) and focal non-epidermolytic palmoplantar keratoderma (FNEPPK).
49 (PC, OMIM:167200) or focal non-epidermolytic palmoplantar keratoderma (FNEPPK, OMIM:613000), which ea
50 ified KRT6C mutations in patients with focal palmoplantar keratoderma (FPPK), but debate concerning o
51 ophageal cancer (TOC), and non-epidermolytic palmoplantar keratoderma (NEPPK); increased iRhom2 cleav
52                      Focal non-epidermolytic palmoplantar keratoderma (PPK or palmoplantar ectodermal
53 mutations, Cx26-H73R and Cx26-S183F, causing palmoplantar keratoderma (PPK) and deafness.
54  desmoglein (DSG) 1, cause the skin diseases palmoplantar keratoderma (PPK) and severe dermatitis, mu
55                                              Palmoplantar keratoderma (PPK) are debilitating lesions
56                  Mutations in SLURP1 cause a palmoplantar keratoderma (PPK) known as mal de Meleda.
57 delG in the V2 domain of keratin 5), striate palmoplantar keratoderma (PPK), and ichthyosis hystrix C
58 in SLURP1 lead to a rare autosomal recessive palmoplantar keratoderma (PPK), Mal de Meleda (MdM), whi
59 RPINA12 as a cause of diffuse, transgradient palmoplantar keratoderma (PPK).
60 ations in SLURP1 cause mal de Meleda, a rare palmoplantar keratoderma (PPK).
61 hic nail dystrophy, oral leukokeratosis, and palmoplantar keratoderma (PPK).
62 a dominantly inherited skin disease, striate palmoplantar keratoderma (SPPK), mapping to chromosome 1
63  MAPK/ERK signaling and is linked to striate palmoplantar keratoderma (SPPK).
64            The locus for a syndrome of focal palmoplantar keratoderma (Tylosis) associated with squam
65  characterize three Cx43 mutations linked to palmoplantar keratoderma and congenital alopecia-1, eryt
66  autosomal recessive pattern, manifests with palmoplantar keratoderma and early, destructive periodon
67 ause a dominant Mendelian disorder featuring palmoplantar keratoderma and lamellar ichthyosis, a phen
68 olidify a link between Slurp2 deficiency and palmoplantar keratoderma and to be confident that the di
69 eratitis-ichthyosis-deafness (KID) syndrome, palmoplantar keratoderma associated with hearing loss, a
70  paper, we report three patients with severe palmoplantar keratoderma associated with ichthyosis and
71 ed patients and affected family members with palmoplantar keratoderma between September 1, 2016, and
72 h mild skin fragility, woolly hair, and mild palmoplantar keratoderma but without a cardiac phenotype
73                                Patients with palmoplantar keratoderma due to DSP variants were found,
74             Genetic heterogeneity of striate palmoplantar keratoderma has been demonstrated with path
75 d, scaly skin at birth, experience worsening palmoplantar keratoderma in childhood, and develop hundr
76 msted syndrome with severe periorificial and palmoplantar keratoderma in multiple unrelated kindreds.
77 of keratin 1 (G4134A) that segregates with a palmoplantar keratoderma in three kindreds.
78                                              Palmoplantar keratoderma is a hallmark of pachyonychia c
79                          The striate form of palmoplantar keratoderma is a rare autosomal dominant di
80  Autosomal-dominant diffuse nonepidermolytic palmoplantar keratoderma is characterized by the adoptio
81 8 years or older either newly diagnosed with palmoplantar keratoderma or being followed up for the di
82                                              Palmoplantar keratoderma poses diagnostic challenges due
83 smoplakin mutation in a proband with striate palmoplantar keratoderma that also results in a null all
84 abrogated by a mutation that causes punctate palmoplantar keratoderma type 1 (PPKP1), a human skin di
85 d in patients with the skin disease punctate palmoplantar keratoderma type 1 (PPKP1).
86 ated in the inherited skin disorder punctate palmoplantar keratoderma type I.
87 ion, the patient's dermatologist treated his palmoplantar keratoderma with systemic retinoids.
88             Tylosis (focal non-epidermolytic palmoplantar keratoderma) is an autosomal dominant skin
89 osis on the volar surface of the paws (i.e., palmoplantar keratoderma), increased keratinocyte prolif
90 the K9 gene are known to cause epidermolytic palmoplantar keratoderma, a rare dominant-negative skin
91 , scaly skin with or without hyperkeratosis, palmoplantar keratoderma, and erythroderma.
92 ypes: nonsyndromic SNHL, syndromic SNHL with palmoplantar keratoderma, and KID.
93 n that presents with nail dystrophy, painful palmoplantar keratoderma, and other clinical manifestati
94              Mal de Meleda is a rare form of palmoplantar keratoderma, and recently mutations in the
95 inant skin disease, diffuse nonepidermolytic palmoplantar keratoderma, and was investigated as a poss
96 s for the triad of autosomal recessive ARVC, palmoplantar keratoderma, and woolly hair (Naxos disease
97 gene that results in skin fragility, diffuse palmoplantar keratoderma, and woolly hair with no sympto
98  the triad of ARVC, diffuse nonepidermolytic palmoplantar keratoderma, and woolly hair.
99 s leading to keratinopathies such as striate palmoplantar keratoderma, as reported in this study.
100  of the autosomal dominant disorder, striate palmoplantar keratoderma, as well as an autosomal recess
101 ermatosis characterized by focal and diffuse palmoplantar keratoderma, hyperkeratotic plaques on the
102 prised nail dystrophy or nail loss, marginal palmoplantar keratoderma, hypodontia, enamel hypoplasia,
103  bilateral hearing loss, blinding keratitis, palmoplantar keratoderma, ichthyosiform erythroderma and
104 epidermolysis bullosa simplex, epidermolytic palmoplantar keratoderma, junctional epidermolysis bullo
105 characterized by sensorineural hearing loss, palmoplantar keratoderma, knuckle pads, and leukonychia,
106                                              Palmoplantar keratoderma, nail dystrophy, and enamel def
107 ensive mucocutaneous blisters, epidermolytic palmoplantar keratoderma, nail dystrophy, enamel dysplas
108                       Focal nonepidermolytic palmoplantar keratoderma, natal teeth, and pili torti ma
109  in silico panel containing genes related to palmoplantar keratoderma, or by Sanger sequencing for sp
110 autosomal-dominant syndrome characterized by palmoplantar keratoderma, oral precursor lesions, and a
111 l for pachyonychia congenita (PC)-associated palmoplantar keratoderma, prompted us to examine the rol
112  with AAGAB variants presented with punctate palmoplantar keratoderma, showing a clear genotype-pheno
113  dominantly inherited skin disorder, striate palmoplantar keratoderma.
114 ence causes skin fragility, woolly hair, and palmoplantar keratoderma.
115 t lays the foundation for future research in palmoplantar keratoderma.
116 inical and genetic spectrum of patients with palmoplantar keratoderma.
117 calized or generalized scaling, and frequent palmoplantar keratoderma.
118 ic cardiomyopathy and the cutaneous disorder palmoplantar keratoderma.
119  for understanding the pathogenesis of human palmoplantar keratoderma.
120  predominantly by nail dystrophy and painful palmoplantar keratoderma.
121 a S233L(K1) mutation linked to epidermolytic palmoplantar keratoderma.
122 eleda is a rare, autosomal recessive form of palmoplantar keratoderma.
123 ation family of British descent with striate palmoplantar keratoderma.
124 recapitulated the clinical features of human palmoplantar keratoderma: overcornification and thickeni
125                                              Palmoplantar keratodermas (PPKs) are a group of disorder
126                                          The palmoplantar keratodermas (PPKs) are a large group of cl
127 roup of disorders, known collectively as the palmoplantar keratodermas, are unified by the phenotypic
128                               Nagashima-type palmoplantar keratosis (NPPK) is an autosomal recessive
129                           The mice exhibited palmoplantar keratosis and progressive alopecia, leading
130                      Some patients have only palmoplantar keratosis or periodontitis, and in rare ind
131 nce of cardiac dysfunction or fibroadiposis, palmoplantar keratosis, and alopecia, resembling the hum
132                                              Palmoplantar keratosis, varying from mild psoriasiform s
133 ersalis hereditaria in addition to punctuate palmoplantar keratosis.
134  mechanisms responsible for the formation of palmoplantar lesions in PC and FNEPPK patients.
135 e generated a mouse model to replicate these palmoplantar lesions.
136 involved 21 adults with PPP (11 patients) or palmoplantar plaque psoriasis with pustules (10 patients
137            Palmoplantar pustulosis (PPP) and palmoplantar plaque psoriasis with pustules remain chall
138 d be an appealing approach for treating PPP, palmoplantar plaque psoriasis with pustules, and SAPHO s
139 Eleven patients had isolated PPP, and 10 had palmoplantar plaque psoriasis with pustules.
140 r pustular psoriasis (PPPP) and non-pustular palmoplantar psoriasis (NPPP) are localized, debilitatin
141 difficult to differentiate from non-pustular palmoplantar psoriasis (palmPP) and dyshidrotic palmopla
142 was reported in 68.1% (n = 28), clearance of palmoplantar psoriasis was reported in 90.0% (n = 10), c
143                                              Palmoplantar pustular psoriasis (PPPP) and non-pustular
144 er, this was not the case when patients with palmoplantar pustular psoriasis were not included in the
145 ns including generalized pustular psoriasis, palmoplantar pustular psoriasis, acrodermatitis continua
146 mutations were identified in both localized (palmoplantar pustular psoriasis, acrodermatitis continua
147  synergistically in these diseases to induce palmoplantar pustules.
148                                              Palmoplantar pustulosis (PPP) and palmoplantar plaque ps
149                                              Palmoplantar pustulosis (PPP) is a is a chronic, orphan
150                                              Palmoplantar pustulosis (PPP) is a severe inflammatory s
151                                              Palmoplantar pustulosis (PPP) is an inflammatory disease
152                                              Palmoplantar pustulosis (PPP) is an inflammatory skin di
153 d nonlesional skin from patients with GPP or palmoplantar pustulosis (PPP) with skin from healthy vol
154 rders: generalized pustular psoriasis (GPP), palmoplantar pustulosis (PPP), and acute generalized exa
155 nical data from 125 patients with AGEP, GPP, palmoplantar pustulosis (PPP), plaque psoriasis (PSO), a
156 tular psoriasis [GPP]) or chronic localized (palmoplantar pustulosis [PPP] and acrodermatitis continu
157  30.6%; eczema, 23.5%; xerosis cutis, 10.6%; palmoplantar pustulosis, 5.3%; psoriasis, 3.8%; other, 2
158 onpustular psoriasis, 127 patients developed palmoplantar pustulosis, and 12 patients developed gener
159  atopic dermatitis, Mal de Meleda, vitiligo, palmoplantar pustulosis, and pemphigus may be mediated,
160 skin from hair bearing, nonhair bearing, and palmoplantar regions.
161 ermis elicits the hypopigmented phenotype of palmoplantar skin due to suppression of melanocyte funct
162 icroarray analysis, fibroblasts derived from palmoplantar skin expressed high levels of dickkopf 1 (D
163                                              Palmoplantar skin has several unique characteristics suc
164 l innervation and substantial alterations in palmoplantar skin immune composition in Slurp2X knock-ou
165 pression patterns of those proteins in human palmoplantar skin.
166 n patterns of those key proteins observed in palmoplantar skin.
167     Subsequently, iRhom2 was found to affect palmoplantar thickening to modulate the stress keratin r
168 h oesophageal cancer (TOC), characterized by palmoplantar thickening, upregulate K16 with robust down
169 lar) skin of amputees to pressure-responsive palmoplantar (volar) skin to enhance prosthesis use and

 
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